Test 2 Flashcards

1
Q

What is the retina and why is it important?

A

It’s the back surface of the eye that is lined with visual receptors and where incoming light hits. It detects photons of light and then fires impulses along optic nerve to the brain

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2
Q

In what way does image hit the retina and how do we see it right-side-up?

A

Light from the left side of the world hits the right side of the retina and vice versa. Light from below strikes the top half. Refraction through a convex lens flips images.

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3
Q

Cells in the retina - photoreceptors

A

Located in the back of the eye. Message bipolar cells.

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4
Q

Cells in the retina - bipolar cells

A

Located close to the center of the eye. Message ganglion cells.

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5
Q

Cells in the retina - amacrine cells

A

Helps ganglion cells respond to certain shapes, directions of movement or other visual features.

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6
Q

Cells in the retina - horizontal cells

A

If light hit one receptor but not the other ones, the horizontal cells inhibits the surrounding receptors for edge detection.

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7
Q

Cells in the retina - ganglion cells

A

Located even closer to the center of the eye.

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8
Q

Optic nerve

A

The bundled axons from ganglion cells that exit the retina.

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9
Q

Blind spot

A

Blind spot is where the optic exit and where blood vessels enter and leave. There are no receptors in this spot. We don’t notice it because 1) your brain fills in the gap, 2) anything in the blind spot in one eye is visible to the other eye.

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10
Q

Fovea

A

A tiny area specialized for acute, detailed vision.

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11
Q

Difference between foveal and peripheral vision

A

Foveal vision: useful in bright lights, but poor in dim lightning. Because there is one ganglion dedicated to each receptor, there is more acuity (better vision for details). Because many cones it’s best for color vision.
Peripheral vision: more rods than cones. One ganglion cell receives input from multiple receptors = lower acuity. Given higher number of rods = it’s good for dim lightning, not for bright light or color vision.

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12
Q

Difference between rods and cones

A

Rods: abundant in the periphery of retina. Responds to faint light but not useful in daylight because bright light bleaches them.
Cones: abundant in and near the fovea. Essential for color vision. Less active in dim light and more useful in bright light.

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13
Q

Trichromatic Theory (Young-Helmholtz Theory)

A

Hypothesized there to be 3 types of photoreceptors in the eye. Each type sensitive to a particular range of visual light. Found that people could match any color by mixing the appropriate amounts of just 3 wavelenghts. The 3 types of cone photoreceptors classified by what wavelenght they responded to. Short-preferring (blue), middle-preferring (green), and long-preferring (red). The relative strenghts of the signals detected by each type was interpreted by the brain as a visible color.

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14
Q

Color constancy

A

Our ability to recognize colors despite changes in lightning. Ex: if you wear green-tinted glasses, a banana will still be yellow for you.

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15
Q

Retinex Theory

A

Says both the eye and the brain are involved in color processing. The brain compares info from various parts of the retina to determine the brightness and color for each area. Our brain uses context to perceive color. A certain wavelenght of light can appear as different colors depending on the background or context. When we see something, we make an inference. Visual perception requires reasoning and inference, not just retinal stimulation.

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16
Q

What occurs in red-green color deficiency (protanopia)?

A

Long and medium wavelenght cones have the same photo-pigment instead of different ones. People with this can’t tell the difference between red and green. This gene is in the X chromosome so it affects more men than women.

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17
Q

Photopigments

A

Chemicals that release energy when struck by light.

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18
Q

What is 11-cis-retinal, and what does it convert to? What happens as a result of the conversion of this molecule?

A

It’s a derivative of vitamin A. Converts to all-trans-retinal. This conversion creates the electrical current that goes to our brain.

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19
Q

Opsins

A

Proteins that bind to 11-cis-retinal and modify its sensitivity to different wavelenghts of light.

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20
Q

When light strikes one photoreceptor, what happens to the bipolar cell to which it’s connected? What happens to surrounding bipolar cells? Mention horizontal cells.

A

When light hits one receptor, it will result in a exitation of its bipolar cell. It also excites a horizontal cell, which inhibits surrounding bipolar cells.

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21
Q

Receptive field

A

A point in visual space from which light strikes the cell and excites or inhibits it.

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22
Q

Describe the pathway from the retina to the brain. Discuss optic chiasm.

A

Ganglion cells’ axons form the optic nerves. The optic nerves from each eye meet at the optic chiasm. Half of the axons from each eye cross to the opposite side of the brain. From the optic chiasm, most of the ganglion cell axons go to the lateral geniculate nucleus.

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23
Q

Lateral geniculate nucleus (LGN)

A

Part of the thalamus. Receives major sensory input from retina and relays it to V1 (primary visual cortex).

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24
Q

Two layers of the LGN

A

Parvocellular: mostly in fovea. Small cell bodies and small receptive fields. Detect visual details and colors. “What” am I seeing?
Magnocellular: throughout retina. Larger cell bodies and receptive fields. Detect movement of large, overall patterns. “Where” am I seeing it?

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25
Q

Visual info from the right visual field is processed in the … occipital lobe, and visual info from the left visual field is processed in the … occipital lobe.

A

left.

right.

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26
Q

Blindsight

A

An ability to respond in limited ways to visual info without perceiving it consciously. Can occur among those with damage to V1. 2 possible explanations:
1) some healthy V1 tissue may remain but not enough fto provide conscious perception.
2) the thalamus sends visual input to other brain areas outside of the V1 to respond without conscious awareness of it.
Main point: V1 is crusial for conscious visual perception.

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27
Q

Cells in visual cortex - simple cells

A

Respond to exact location of a stimulus. Have receptive fields with fixed excitatory and inhibitory zones. The more light shines on excitatory zone, the more cell activity. The more light shines on inhibitory zone, the less activity.

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28
Q

Cells in visual cortex - complex cells

A

Larger receptive fields. Don’t respond to exact location of a stimulus. Will respond to a stimulus equally throughout a large area. Responds to patterns of light in a particular orientation anywhere within its large receptive fields. Responds most strongly to a moving stimulus.

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29
Q

Cells in visual cortex - hypercomplex (end-stopped) cells

A

Resemble complex cells, but have a strong inhibitory area at one end of its receptive field. Responds to a pattern of light anywhere in a broad field; provided the stimulus doesn’t extend beyond a certain point.

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30
Q

Ventral pathway

A

The “what” pathway. It’s going through the temporal cortex. Specialized for identifying and recognizing objects.

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31
Q

Visual agnosia

A

The inability to recognize objects despite otherwise satisfactory vision. Someone with this can point to the object and describe features, but can’t recognize what it is.

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32
Q

What type of object does the parahippocampal cortex most strongly respond to?

A

Places/landscapes

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33
Q

What type of object does the fusiform gyrus most strongly respond to?

A

Faces

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34
Q

What does the occipital face area respond most strongly to?

A

Facial features

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35
Q

Prosopagnosia

A

Impairment in ability to recognize faces. From damage or innate structural/functional differences. When people with this look at a face, they can describe a few features but can’t identify the person.

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36
Q

Dorsal pathway

A

The “how” pathway. It goes through the parietal cortex. Important for visually guided movements.

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37
Q

Damage to dorsal pathway

A

They can see objects but can’t integrate their vision with movements. Can read/recognize faces, describe objects in detail but can’t accurately reach out and grasp the object. May bump into objects. Can tell you what their furniture at home looks like, but can’t tell you how it’s spatially arranged.

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38
Q

Role of middle temporal cortex (MT) and medial superior temporal cortex (MST) play in motion perception

A

MT: detect acceleration and deceleration and speed in three dimentions. Responds to photographs that imply movement. Electrical stimulation of this area results in seeing movements that doesn’t exist.
MST: responds if something moves relative to background.

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39
Q

Sound Wave

A

Periodic compressions of air, water, or other media.

40
Q

The Components of Sound Waves

A

Amplitude: loudness. The intensity of a sound wave. In general, sounds of greater amplitude sound louder.
Frequency: the number of compressions/waves/cycles per second. 1 Hz = 1 cycle per second.

41
Q

Speech prosody

A

People communicate emotions by alternations in pitch, loudness, timbre.
Graditude, sarcasm etc. depends on the sound quality produced. Conveying emotional info by tone of voice is prosody.

42
Q

Timbre

A

the character of a sound. Tone quality or tone complexity.

43
Q

what is the function of the pinna?

A

Alter the reflections of sound waves to help locate source of sound.

44
Q

how are sound waves transmitted by the tympanic membrane and then carried through the middle ear to the inner ear

A

Sound waves pass through the auditory canal and strike the tympanic membrane (eardrum). Tympanic membrane vibrates at same frequency as sound waves. Tympanic membrane connects to 3 bones; hammer, anvil, and stirrup. The 3 bones convert sound waves into waves of greater pressure on the oval window. This transformation is important because vibrations in the air can’t produce significant vibrations in the viscous fluid behind the oval window.

45
Q

how vibrations at the oval window are transformed into auditory messages to the brain in the cochlea. Mention hair cells

A

the stapes pushes against the oval window which transmits waves of sound, and the vibrations are sent to the cochlea which is filled with fluid. The cochea consists of tiny hair cells. Hair cells line the cochlea and transmit vibrations into electrical impulses that are carried to the brain by sensory nerves.
Hair cells are located in the basilar membrane.

46
Q

What is the method by which our auditory system encodes low-frequency sounds?

A

Frequency theory: Basilar membrane vibrates in synchrony with a sound, causing auditory nerve axons to produce action potentials at that same frequency.

47
Q

What is the method by which our auditory system encodes sounds of higher frequency?

A

Volley Theory.
Volley principle: groups of neurons of respond by firing action potentials out of phase with one another. When combined, greater frequency of sound is encoded.

48
Q

primary auditory cortex; where is it located

A

Its primary function is to process sound along with its volume and pitch. The location of the sound is also processed via primary auditory cortex. This auditory cortex is essential to comprehend the spoken language and is concerned with tasks such as finding out and separating the auditory objects.
Located in the superior temporal cortex.

49
Q

tonotopic map

A

The way in which the primary auditory cortex is organized.

50
Q

Describe the following three different ways in which we localize sound - Interaural time difference

A

when concerning humans or animals, is the difference in arrival time of a sound between two ears. It is important in the localization of sounds, as it provides a cue to the direction or angle of the sound source from the head.

51
Q

Describe the following three different ways in which we localize sound - interaural intensity difference

A

arises from the fact that, due to the shadowing of the sound wave by the head (sound shadow), a sound coming from a source located to one side of the head will have a higher intensity, or be louder, at the ear nearest the sound source.

52
Q

Describe the following three different ways in which we localize sound - Interaural phase difference

A

refers to the difference in the phase of a wave that reaches each ear. A sound wave from the side strikes the two ears out of phase.

53
Q

Difference between amusia and perfect pitch

A

Amusia: The inability to recognize musical tones or to reproduce them. Amusia can be congenital (present at birth) or be acquired sometime later in life (as from brain damage). Tone deafness.
Perfect pitch: the ability to recognize the pitch of a note or to produce any given note

54
Q

conductive deafness

A

Middle ear deafness. From infection or tumorous bone growth. Corrected by surgery or hearing aids. They may accuse others of talking too softly or mumbling.

55
Q

nerve deafness

A

Inner ear deafness. From damage to cochlea, hair cells, or auditory nerve. Genetic or environmental or can result from loud noices.
Often produces tinnitus.

56
Q

McGurk Effect

A

Our tendency to integrate visual speech into what we “hear”.

57
Q

vestibular organ

A

Sensations from vestibular organ detect the direction of tilt and the amount of acceleration of the head. You use that info automatically for guiding eye movements and maintaining balance.

58
Q

Describe following types of somatosensory receptors - mechanoreceptors

A

provide info to the brain about touch, pressure, vibration, and skin tension.

59
Q

Describe following types of somatosensory receptors - thermoreceptors

A

provide info about temperature

60
Q

Describe following types of somatosensory receptors - nociceptors

A

provide info about potentially damaging stimuli (pain). Ex: sunburn.

61
Q

We have … cervical spinal nerves, … thoracic nerves, … lumbar nerves,… sacral nerves, and … coccygeal nerves for a total of … spinal nerves that enter the spinal cord and deliver info to the brain.

A
8
12
5
5
1
31
62
Q

dermatome

A

Kind of like a map.

Each spinal nerve connects to a limited area of the body called a dermatome.

63
Q

primary somatosensory cortex

A

it’s located in the parietal lobe.

64
Q

How does the pain and somatosensory information travel in the spinal cord to the brain and how do these pathways differ?

A

Paths for pain and touch are parallel, but different. Pain crosses immediately in the spinal cord. Touch info travels up the ipsilateral side to the medulla, then crosses. Pain and touch reach neighboring sites in the cerebral cortex.

65
Q

how are physical and emotional pain similar?

A

they activate the same region of the brain

66
Q

Gate Theory of Pain

A

A lot of things influence pain. What we expect of pain can influence how much pain we feel.
Spinal cord neurons that receive messages from pain receptors also receive input from touch receptors and from axons descending from the brain.

67
Q

Placebo

A

fake drugs that are taken during double blind studies. some people show favorable responses to placebos, because their brains release endorphins to kill the pain. They believe that they have taken a real drug, so they perceive that the pain has gone away.

A drug or other procedure with no pharmacological effects.

68
Q

What naturally occurring neurotransmitter reduces pain? What drugs act on this particular neurotransmitter system?

A

Endorphins; they interact with opioid receptors in the brain to reduce the perception of pain

69
Q

Olfactory cells

A

are on the roof of the nasal cavity that contains millions of olfactory sensory neurons, when particles reach the neurons and sends the signals to the brain;

70
Q

Vomeronasal organ (VNO)

A

Set of receptors located near, but separate from, the olfactory receptors.

71
Q

Pheromones

A

A chemical substance produced and released into the environment affecting the behavior of others.
Important for most mammals, but less so for humans.

72
Q

Evidence of pheromones in humans

A

Smell of women’s perspiration increases a man’s testosterone levels.
Effect is stronger in heterosexual men vs. homosexual men.

The smell of men’s perspiration increases release of cortisol (stress hormone) but not sexual arousal.

73
Q

Synesthesia

A

The experience some people have in which stimulation of one sense evokes a perception of another type of sense as well.
Most common: perceiving letters or numbers in certain colors.
Can involve any of the senses, ex:
- sounds in response to smell
- smell in response to touch
- feeling something in response to sight

74
Q

Three categories of vertebrate muscle fibers

A

1) smooth muscles: control the digestive system and other organs
2) skeletal or striated muscles: control movement of the body in relation to the environment
3) cardiac muscles: control the heart

75
Q

neuromuscular junction

A

the synapse between a motor neuron and a muscle fiber

76
Q

antagonistic muscles

A

antagonist - flexor muscle.

Muscles only move in one direction. Moving an arm or a leg requires antagonistic muscles

77
Q

Difference between fast-twitch and slow-twitch muscle fibers

A

Fast twitch fiber: fire rapidly, fatigue rapidly. Best for generating short bursts of strenght or speed, useful for sprinters. Anaerobic = don’t use oxygen.

short twitch fibers: fire more slowly, less vigorous contractions and no fatigue. Best for marathon runners and endurance sports. Aerobic = uses oxygen.

78
Q

proprioceptor

A

A receptor that detect position or movement of a body part or a muscle.

79
Q

Describe the two following proprioceptors - muscle spindle

A

sensory receptors within a muscle that detect when that muscle is stretched

80
Q

Describe the two following proprioceptors - golgi tendon organ

A

sensory receptor organ that senses changes in muscle tension.

81
Q

ballistic movements

A

a movement that is executed as a whole. Once initiated, it cannot be altered. Ex: reflex

82
Q

Central pattern generators

A

Neural mechanisms in the spinal cord that generate rhythmic patterns of motor output.

83
Q

Motor program

A

Instinctive, non-learned, fixed sequence of movements that is “hard-wired”

84
Q

two areas in brain associated with planning a movement

A

1) prefrontal cortex: active during a delay before a movement. Considers probable outcomes of possible movements. People with damage to this area exhibit disorganized/non-rational movements/behaviors. Inactive during dreams.
2) posterior parietal cortex

85
Q

Primary motor cortex (M1) - location + role in executing movement

A

Located in frontal cortex. M1 axons synapse with interneurons in the spinal cord, but some synapse directly with motor neurons that control muscles.

86
Q

Role of posterior parietal cortex in movement

A

Monitors position of body relative to world.

87
Q

Two areas involved in inhibiting a movement

A

Prefrontal cortex and basal ganglia

88
Q

Antisaccade task

A

Involves inhibiting a response and looking in opposite direction.
Requires sustained activity in prefrontal cortex and basal ganglia

89
Q

Describe two corticospinal tracts - lateral corticospinal tract

A

goes from primary motor cortex, surrounding areas and red nucleus. Axons extend from motor cortex to neurons in spinal cord. In the protrusions of the medulla, axons cross. Controls movement in peripheral areas -> ex: hands and feet.

90
Q

Describe two corticospinal tracts - medial corticospinal tract

A

Axons go to both sides of the spinal cord. Axons go to trunk, more proximal muscles.

91
Q

How does cerebellum help control movement?

A

Contains more neurons than the rest of the brain combined. It coordinates timing and force of different muscle groups for fluid limb or body movements.

92
Q

Finger-to-nose test

A

It’s used to assess neurological abnormalities with cerebellum.
If damage to cerebellum: trouble with initial rapid movement ex: finger misses nose, stops too soon, or goes too far.
If damage to cerebellum nuclei: difficulty with the hold segment -> finger reaches front of nose and then wavers.

93
Q

Role of basal ganglia in movement

A

It’s important for spontaneous, self-initiated behaviors

94
Q

Substantia nigra

A

Gradual loss of neurons in the substantia nigra (a loss of dopamine-releasing axons to the striatum).
The striatum ↓ inhibition of the globus pallidus, which then ↑ inhibitory input to the thalamus.
Result: slow, weak (less vigorous) voluntary movements.

95
Q

Parkinson’s disease

A

Rigidity, muscle tremors, slow movements and difficulty initiating physical and mental activity.
Causes: genetics and environment. More common in farmers/rural dwellers than in urban dwellers. Most likely combination of both genetics and environment.
Gradual loss of neurons in the substantia nigra.

96
Q

How does brain stimulation treat Parkinson’s?

A

It may normalize neuronal (brain cell) activity.

97
Q

Huntington’s disease

A

Deterioration of basal ganglia and cerebral cortex, leading to eventual death.
Symptoms begin with arm jerks and facial twitches.
Tremors spread to other body parts, develop into writhing. Tremors interfere with walking, speech, and other voluntary movements.
Causes: genetics. History of drug/alcohol use can increase risk of early onset.