Test 8 Notes Flashcards

1
Q

What happens when you place a patient on a ventilator

A

we reverse the normal pressure changes in the chest

  • Normal insp causes negative pressure in chest, this expands blood vessels and heart aiding in blood delivery to the heart
  • PPV will put pressure on the blood vessels and heart impeding blood delivery to the heart and lung
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2
Q

Minimize effects Positive Pressure ventilation

A

Primary way to diminish effects of PPV on pulmonary and cardiovascular system is to keep mean airway pressure at its lowest

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3
Q

Cardiovascular - Most likely to affect cardiac patients and patient without lung disease

A

Decreased compliance (stiff lungs) and airway disease pressure not transferred to heart

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4
Q

PPV in chest of pt with CHF may improve

A

cardiac function

  • pushes blood out of heart and vessels so makes it easier for the muscle to pump the blood
  • PEEP is commonly used to treat left heart failure
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5
Q

Cardiac monitorings

A

ECG, a common cause of dysrhythmias is hypoxia

  • Arterial blood pressure monitoring
  • Continuous monitoring via arterialcatheter (Art-line)
  • Oxygen delivery is dependenton a good BP and CO
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6
Q

Hemodynamics Direct measurements

A

Pulmonary Artery Catheter (Swan-Ganz)
Central Venous Pressure (CVP)0-8 mmHg
Right Atrial Pressure (RAP) 0-8 mmHg
Pulmonary Artery Pressure (PAP) 15-30/4-12 mmHg
Mean Pulmonary Artery Pressure (PAP) 9-18 mmHg
Pulmonary Capillary Wedge Pressure (PCWP) 4-12 mmHg
Cardiac Output (CO) 4-8 L/mi

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7
Q

Barotrauma vs. Volutrauma

A

Caused by overdistention
-Barotrauma caused from too much pressure in lungs.
Volutrauma caused from too much volume in lungs, regardless of pressure
controversy over pressure vs volume, usually both pres

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8
Q

Overdistention of lungs can cause

A
Alveolar capillary level damage
From:
Stiff lungs
ETT in rt mainstem bronchi
Too much volume in lungs
Vt too high
Auto PEEP
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9
Q

pressure ventilation can cause

A

lung damage to tissue leading to ALI and ARDS causes of overdistention

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10
Q

Avoid VILI monitor

A

Peak Pressures and set alarms appropriately
Plateau pressures
Auto-PEEP
CX

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11
Q

Prevention of VILI

A
small Vt
decrease Vt when raising PEEP
keep plateau below 30 mcH2O
keep peak pressure below 35 cmH2O
avoid mainstem intubation
don’t use pause pressure with volume ventilation
monitor and treat autoPEEP
use PEEP for optimal lung recruitment 
permissive hypercapnia
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12
Q

Other causes of auto PEEP

A

high minute ventilation>10
High RR
I;E

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13
Q

how to find auto PEEP

A
EASIEST WAY: FLOW TIME CURVE
-exp flow not returning to baseline
Increased resonance on percussion
Decreased BS
Expiratory Hold
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14
Q

How to get rid of auto PEEP

A
increase E time
-faster flows, smaller Vt
Lower rate
Large ETT
or allow permisive hypercapnia
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15
Q

Inverse I:E ratios cause

A

autoPEEP and improves O2

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16
Q

O2 toxicity leads to

A

ALI/ARDS

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17
Q

In pneumothorax mediastinum moves

A

towards affected side, tension moves away

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18
Q

Pneumothorax

A

air enters pleural space

  • increased pressure in pleural space crushes the heart and great vessels causing cardiovascular collapse and death
  • treat all with chest tube unless very small and no problem
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19
Q

Signs of pneumothorax

A

subcutaneous emphysema

20
Q

Subcutaneous emphysema

A

air leaks out of lungs and into soft tissue

which is a sign of pneumo, always get CXR

21
Q

signs of tension pneumo

A

-Increased Peak Pressures
-Increased WOB
-Absent BS on affected side
-Mediastinal shift away
-Increase in HR and decrease Spo2
-Loss of BP/CO
CHEST XRAY IS BEST DX

22
Q

Emergent tx for tension pneumo

A

14 guage needle

  • anterior 2nd and 3rd on affected side, midclavicular space
  • pt head up position
23
Q

papillary response

A

pinpoint, drug overdose
dilated and fixed, atropine
mid-position and fixed, severe cerebral damage

24
Q

gag reflex used to

A

used to test cranial nerve function but also to assure airway protection post extubation.
-Place a tongue depressor in back of throat and there should be a response of those muscle

25
what controls breathing
brainstem, there would be no breathing if this is affected
26
Cheyenne stokes breathing
increase in ICP, CHF, Hyoxia
27
ICP
amount of pressure in brain - increased pressures cause decreased perfusion in brain - goal to keep lowest pressures possible in chest keeps blood from entering the chest causing a back up of blood in the skull leading to increased ICP
28
What will decrease ICP
hyperventilation- CO2 is a cerebral vasodilator | -Keep CO2 28-32 on closed head injury 24-36 hours
29
ICP Monitoring
normal mean ICP is 10-15 in a supine position 15-20 compress the capillary bed and compromise circulation 30-35 venous drainage is impeded and edema develop 40-50 perfusion cannot be maintained
30
Cerebral perfusion pressure (CPP)
blood floww through brain, maintain above 70
31
CPP=
MAP-ICP | wanna maintain above 70
32
Glasgo Coma Scale
Scale 3-15 9-13 need ICU 8 or less need an ICP
33
Endocrine effects
increase in anti-diuretic hormone(ADH) causes lower urine output several other hormone changes that may lead to lower urine output
34
ABG with RENAL FUNCTIONS
(Kidney, Endocrine, ABG) | lower PaO2 and higher PaCO2 lead to lower urine out
35
Normal urine output
is 1ml/kg/hr or about 30-70 ml/hr | -poor urine output leads to pulmonary edema from too much fluid in the body and acid base problems, usually acidosis
36
PPV and liver functions
PPV can lead to venous distention with ischemia to the liver | increased bilirubim
37
Gastric effects with PPV
PPV can lead to GI bleed and ulcers acid reflux can lead to pneumonia pt needs to be on antacids and histamine H2 blockers (Pepcid)
38
Nutrition and PPV
Nutrition not enough calories to breath too many carbs lead to increase CO2, makes weaning difficult for COPD Oral feedings best
39
Lung protective Strategies
keep peak pressures <35 Keep Plateau pressures <30 Vt 6 ml/kg may need to increase RR to compensate Permissive hypercapnia
40
Lung recruitment maneuver
``` Sustained high-pressure inflation Intermittent sigh Extended sigh Intermittent PEEP increase Pressure control + PEEP ```
41
VAP causes within
48-72 hours, usually bacterial
42
Causes of VAP
``` Aspiration of oral secretion Aspiration of gastric contents Inadvertent instillation of an infectious agent down the artificial airway Inhalation of infectious aerosol ```
43
Risks of VAP
``` nasal intubation Reintubation Low endotracheal tube cuff pressures Supine position Enteral feeding Hyperglycemia Blood transfusion Inadequate staff ```
44
CVP Catheters
Filling pressure or preload of rt atrium | -most useful in pts with volume-depleted state
45
PA catheter
Estimate SVR and PVR | -Assess pulm hypertension
46
adding a plt will
Increase the MAP add to the I time Improve O2
47
Does rate affect Pressures and volumes
NO