THE CHILD WITH CONGENITAL HEART PROBLEM Flashcards

1
Q

Weaker pulses or lower blood pressure in the lower extremities may indicate

A

coarctation of the aorta (COA)

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2
Q

Bounding pulses can indicate

A

patent ductus arteriosus (PDA) or aortic insufficiency.

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3
Q

to define silhouette of the heart.

Heart size, shape, pulmonary markings, and cardiomegaly.

A

Chest x-ray

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4
Q

n invasive test to diagnose or treat cardiac defects.
Visualizes heart and vessels.
Measures oxygen saturation of chambers.
Measures intra-cardiac pressures.

A

Cardiac Catheterization

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5
Q

Watch for signs of toxicity to the dye used during the procedure

A
Increased temperature
Urticaria
Wheezing
Edema
Dyspnea
Headache
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6
Q

Pre-cardiac Catheterization

A
Assess vital signs with blood pressure
Hemoglobin and hematocrit
Pedal pulses
Observe NPO
Hold digoxin
IV if child is polycythemic
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7
Q

ressures on the left side of the heart are normally higher than the pressures in the right side of the heart.

A

Left to Right Shunt

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8
Q

Clinical Manifestations of

Left to Right Shunt

A

infant is not cyanotic.

Tachycardia due to pushing increased blood volume.

Cardiomegaly due to increased workload of the heart.

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9
Q

Deoxygenated blood from the right side shunts to the left side

A

Right to Left Shunts

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10
Q

Clinical Manifestations Right to Left Shunts

A

Hypoxemia = the result of decreased tissue oxygenation.
Polycythemia = increased red blood cell production due to the body’s attempt to compensate for the hypoxemia.
Increase viscosity of the blood = heart has to pump harder.

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11
Q

Treatment of Heart Failure

A

Digoxin Therapy

Digoxin increases the force of the myocardial contraction.

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12
Q
Bradycardia
Arrhythmia
Nausea, vomiting, anorexia
Dizziness, headache
Weakness and fatigue
signs for?
A

Signs of Digoxin Toxicity

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13
Q

Interventions for Heart Failure

A

Fluid restriction
Diuretics – Lasix (potassium wasting) or Aldactone (potassium sparing)
Bed rest
Oxygen

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14
Q

One of the most common benign defects
Ductus normally closes within hours of birth
Connection between the pulmonary artery (low pressure) and aorta (high pressure)

A

Patent Ductus Arteriosus

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15
Q

Diagnosis of Patent Ductus Arteriosus

A

Chest x-ray – enlarged heart and dilated pulmonary artery

Echo-cardiogram

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16
Q

Treatment of Patent Ductus Arteriosus

A
  1. Indomethocin given po – constricts the muscle in the wall of the PDA and promotes closure
  2. Cardiac Catheterization – coil is placed in the open duct and acts like a plug
  3. Closed heart surgery – small incision made between ribs on left hand side and PDA is ligated o
17
Q

Blood in left atrium flows into right atrium
Pulmonary hypertension
Reduced blood volume in systemic circulation

A

Atrial Septal Defect

18
Q

Treatment For Atrial Septal Defect

A

Surgical closure of the atrial septal defect

19
Q

Opening in the ventricular septum
Left-to-right shunt
Right ventricular hypertrophy
Deficient systemic blood flow

A

Ventricular Septal Defect

20
Q

Diagnosis of Ventricular Septal Defect

A

heart murmur
Electrocardiogram – to see if there is a strain on the heart
Chest x-ray – size of heart
Echocardiogram – shows size of the hole and size of heart chambers

21
Q

Treatment of VSD

A

CHF:
Digoxin if additional force needed to squeeze the heart
FTT (Failure to thrive) o

22
Q

Congenital narrowing of the descending aorta
80% have aortic-valve anomalies
Difference in BP in arms and legs (severe obstruction)

A

Coarctation of Aorta

23
Q

Treatment of Coarctation of Aorta

A

Prostaglandin

24
Q

when a baby’s heart does not form correctly as the baby grows and develops in the mother’s womb during pregnancy.

A

Tetralogy of Fallot (TOF)

25
Tetralogy of Fallot (TOF) | first described by
Étienne-Louis Fallot in 1888
26
Tetralogy of Fallot (TOF) Four Components
VSD Pulmonary stenosis Overriding of the aorta Hypertrophy of right ventricle
27
has two rather than three leaflets. Leaflets are thickened or fused. Obstruction of blood flow from left ventricle Mild symptoms: dizziness, syncope, angina, fatigue
Aortic Stenosis
28
Diagnosis of Aortic Stenosis
Heart murmur Electrocardiogram Echocardiogram Exercise stress test
29
Treatment of Aortic Stenosis
Cardiac catheterization | Surgical valvotomy
30
Life threatening shock develops when the ductus arteriosis closes Low oxygen saturations – will not increase with oxygen administration Pulses will be weak in all extremities
HYPOSTATIC LEFT HEART SYNDROME(HLH)
31
TREATMENT FOR HYPOSTATIC LEFT HEART SYNDROME(HLH)
Norwood – right ventricle becomes the systemic ventricle pumping blood to the body Glenn – done at 3-6 months Fontan – done at 2 -3 years of age
32
Infection of endocardial surface of the heart | History of CHD, Kawasaki Disease, Rheumatic Fever, or prosthetic valves are more susceptible to infection
Bacterial Endocarditis
33
Acute-self limiting disease Generalized vasculitis Peak incidence 6 months to 2 years More common in males and Japanese
Kawasaki Disease