Theory Flashcards
(47 cards)
Describe the Gunn Neuropathic Model
the role of compression on muscle trigger points and how a sensitization can manifest (dystrophic nerve)
What is the cycle of dysfunction proposed by Gunn
spondylosis, compression of peripheral nerves, denervation supersensitivity, muscle banding / trigger points, increased nerve irritation, continued denervation supersensitivity
Describe energy crisis theory and motor end plate hypothesis as it pertains to trigger points
consider the chemical causes of prolonged contraction. The energy crisis theory considers the role of calcium, and the motor end plate hypothesis considers the role of acetyolcholine
2 general recepotros in the body
Exteroreceptors collect information from outside the body, and enteroreceptors collect information from within our body.
factors influencing nervous system
pain, injury, stress, sleep, nutrition, emotion, exercise load, enviormental stressors
Are there reflex loops in periphealr and visceral nervouse systems
Yes the peripheral nervous symptoms involve muscles for reflex loops, and visceral internal organs.
Differentiate between thes omatic afferen tinformation and visceral afferent information
The somatic nervous systems carries afferent sensory information from skin, muscles and joints. The Visceral nervous system includes afferent information from organ and blood vessels.
Describe the modulation of pain an on/off mechanism?Explain
The body can inhibit and facilitate pain. The modulation system is not an on and off system because it considers the input. It may prioritize and evaluate information - it may consider danger, and receive input from other systems of the body (i.e limbic) to modulate pain.
Nerve layers deepest to superficial
Epineurium, perinuerium, endoneurium
What are some distinguishing characteristics of each layer
Endo - more susceptible to pressure. Peri - risk of punctures. Epi - dense connective tissue
What determines if a nerve fires or transmits an impulse?
If excitatory potential exceed inhibitory potential
List the nerves in order of fasted to slowest speed of impulse conduction
Mylenated (A Alpha, B Beta, A Delta ) Unmyelinated (C fibers)
What is the difference in type of pain and modality sensed between A delta and C- Fibers?
A delta are intense signal conducting for fast nociceptive pain, damaging temperature and light mechanical deformation.
C fibers are slower adaptive receptors, sensations include warm, secondary burning to pain and muscle burn.
What are the three types of pain and their broad descriptions according to Wall?
Type 1 (Nociceptive, immediate acute pain), Type II (Inflammatory, biochemical pain), Type III (Chronic, neuropathic)
What are the time responses for the different types of pain?
Type 1 - immediate. Type II within 48 hours, Type III > 3 months persists.
What is the description of the quality of pain for each type?
Type I (sharp, stabbing, throbbing), Type II (Acute, throb), Type III (Variable)
What behaviours are associated with each type of pain?
Type I (fight or flight), Type II (concern, anxiety, care seeking), Type III (depression, introversion, resistant to treatment)
How long does the maturation phase of wound healing take?
21 days to 3+ years
After the maturation phase of wound healing is the tissue as strong as it was before?
No, it can be 20% weaker than original tissue
Describe the gate control theory of pain
The gate control theory describes how different types signals can interfere with others. For example, a A beta fibers can inbibit the effects of fast pain and slow pain.
What tissues in the body can develop denervation supersensitivity ?
Muscles, glands, vessels, organs, CNS
According to Cannon and Rosenbluth, what occurs when a nerve is injured?
It can result in super sensitivity
Describe the types of supersensitivity that can develop
Normal (normal response),
super duration (time course increases),
hyper-excitability (threshold of stimulating is lower),
increased susceptibility (threshold of stimulus is lower than previous, non-noxious or noxious),
super reactivity (capacity of tissue to respond is augmented)
What are the four main causes of neuropathy?
Mechanical, chemical, chemical (exogenous source), disease process