Trauma and Emergency Flashcards

(231 cards)

1
Q

causes of intrinsic hreat failure?

A

1. MI
2. cardiac failure
3. cardiac contusion
4. cardiac laceratin

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2
Q

causes of extrinsic cardiogenic shock?

A

1.tension pneumothorax
2.Hemothorax
3.cardiac tamponade

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3
Q

intrinsic cardiogenic shock means…..

A

pump failure

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4
Q

Extrinsic cardiogenic shock means…..

A

unrelated to the heart pump itself

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5
Q

parameters of cardiogenic shock regarding CO/VFP/SVR

Think of hemodynamic compansation after pump failure.

A

CO
VFP
SVR

📝The decreased cardiac output causes an increase in ventricular filling pressure and the body compansates with increasing SVR which in turn sets the stage for the cardiac decompansation , volume overload and decreased coronary perfusion.

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6
Q

Sepsis causes which type of shock?

A

septic shock ( a type of distributive shock)

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7
Q

Which type of shock caused by head trauma?

A

Neurogenic shock ( a type of distributive shock )

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8
Q

what is the main effect of distributive shock?

A

loss of vasomotor tone & ↓ SVR

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9
Q

When to start antiplatlets or anticoagulants in BCVI with intimal involvement ?

*BCVI=Blunt cerebrovascular injuries

A

Regardless of the degree of intimal involvment start as soon as possible.

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10
Q

Complications of a seat belt injury from least to most severe?

A

1. Rupture of intima with or without thrombosis
2.Damage of the entire vascular wall with pseudoaneurysm

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11
Q

Cause of death in seat belt injury?

A

1. secondary stroke
2.blood clot

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12
Q

How to manage a case of seat belt injury ?

A

Suspected Carotid Injury

CTA Neck Performed

Intimal Tear Detected?

Yes

Observe Start antiplatelet or anticoagulant

Repeat CTA in 7–10 Days

┌────────────┴────────────┐
[Complete Healing] No
↓ ↓
Stop treatment Continue same Rx for 3 months

📝1. Intial cervical CTA to detect any intimal tear of the carotid artery.
2.After detection of intimal tear»> start antiplatlets or anticoagulants as soon as possible regardless the degree of intimal injury.
3.Repeat CTA after 7-10 days if there is complete recovery stop treatment.
4. If there is still an injury continue treatment for 3 months.

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13
Q

treatment of Retroperitoneal bladder rupture?

A

decompression with Foley catheter, then cystography to confirm healing.

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14
Q

Treatment of Peritoneal bladder rupture?

A

suturing of the bladder with 2 layers of absorbable thread and leaving a Foley catheter or a suprapubic catheter.

📝 nonabsorbable thread would result in a nidus for stone formation and infection.

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15
Q

Blood loss in each stage of hemorrhagic shock?

A
  • stage1 :0-15%
  • stage 2 : 15-30%
  • stage 3 :30-40%
  • stage 4 :>40%
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16
Q

CNS assessment in the 4 classes of hemorrhagic shock?

A

class 1 🡆 slightly anxious
class 2 🡆 mildly anxious
class 3 🡆 anxious or confused
class 4 🡆 confused or lethargic

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17
Q

Pulse assessment in 4 stages of hemorrhagic shock?

A

class1:<100
class2:>100
class3:>120
class4:>140

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18
Q

Blood pressure asssessment in 4 classes of hemorrhagic shock?

A
  • class 1➡️normal
  • class 2 ➡️ normal
  • class 3 ➡️ ⬇︎
  • class 4 ➡️ ⬇︎
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19
Q

Pluse pressure assessment in 4 classes of hemorrhagic shock?

A
  • class 1 ➡️ normal
  • class 2 ➡️ ⬇︎
  • class 3 ➡️ ⬇︎
  • class 4 ➡️ ⬇︎
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20
Q

Respiratory rate assessment in 4 classes of hemorrhagic shock?

A
  • class 1 ➡️ 14-20 /min
  • class 2 ➡️20-30/min
  • class 3 ➡️ 5-15/min
  • class4 ➡️ negligible
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21
Q

urine output in 4 classes of hemorragic shock?

A

class 1➡️ >30 ml/hr
class 2➡️ 20-30 ml/hr
class 3➡️ 5-15 ml/hr
class 4 ➡️ negligible

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22
Q

Fluid treatment in 4 classes of hemorrhagic shock?

A
  • class 1 ➡️ crystalloid
  • class 2 ➡️crystalloid
  • class 3 ➡️ crystalloid &Blood
  • class 4 ➡️ crystalloid &Blood
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23
Q

Base dificit in 4 classes of hemorrhagic shock?

A
  • class1 ➡️0 to -2
  • class2 ➡️-2 to -6
  • class 3➡️ -6 to -10
  • class 4➡️ -10 to <
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24
Q

Parameters of class 1 hemorrhagic shock regarding Pulse, BP, Pulse pressure, RR& urine output?

A
  • Pulse➡️<100 bpm
  • BP➡️ normal
  • Pulse pressure ➡️ normal
  • RR➡️ 14-20 /min
  • urine output➡️>30 ml/hr
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25
Parameters of class 2 hemorrhagic shock regarding mental status, pulse, BP,pulse pressure,RR, urine output ?
**mental status**➡️ mildly anxious **Pulse**➡️ normal **BP**➡️ normal **Pulse pressure**➡️ ↓ **RR**➡️ 20-30/min **urine output**➡️ 20-30 ml/hr
26
Parameters of class 3 hemorrhagic shock regarding mental status, pulse, BP,pulse pressure,RR, urine output ?
**Mental** **status**➡️ anxious or confused **Pulse**➡️ >120 bpm **BP**➡️↓ **Pulse pressure**➡️ ↓ **RR**➡️ 30-40/min **urine output**➡️ 5-15 ml/hr
27
Parameters of class 4 hemorrhagic shock regarding mental status, pulse, BP,pulse pressure,RR, urine output ?
**Mental** **status**➡️ confused or lethargic **Pulse**➡️ >140 bpm **BP**➡️ ↓ **Pulse pressure**➡️ ↓ **RR**➡️ >35/min **urine output**➡️ negligible
28
High level of lactate indicates.....
tissue distress
29
What is the clinical significance of a base deficit ≤ -6 and a lactate level > 4 mmol/L in trauma or shock patients?
correlates with increased mortality risk.
30
How to differentiate between septic shock & neurogenic shock on physical examination?
* **Septic shock**: cold extremities due to vasoconstriction. * **Neurogenic shock**: no cold extremities because there is no peripheral vasoconstricion. | 📝Both septic &neurogenic shock are types of distributive shock.
31
The first compansation mechanism in hypovolmic shock?
increased sympathetic activity **in** **response** **to** decrease in vascular volume
32
How baroreceptors sense & respond to decrease in BP?
**1**. increase in BP is mediated by pressure receptors or baroreceptors in the aortic arch, atria, and carotid bodies **2**. Hypotension ➡︎⬇️ arterial pressure ➡︎ ⬇️stretch ➡︎⬇️ afferent baroreceptor firing ➡︎⬆️ efferent sympathetic firing and ➡︎⬇️ efferent parasympathetic stimulation➡︎ ⬆️ vasoconstriction, ⬆️ HR, ⬆️ contractility, ⬆️ BP.
33
decreased urine output as acompansatory mechanism in hypovolemic shock starts from stage .....
**stage2**
34
increased RR as a compansatory mechanism in hypovolemic shock starts from stage....
**stage****2**
35
decreased level of consciousness in hypovolemic shock is evident from which stage?
**stage 3**
36
Indications of immediate exploratory laparotomy in anterior stab wound?
**1**. hemodynamic instability **2**. peritonitis **3.** evisceration ## Footnote 📝In the absence of these signs, the remainder of patients should undergo local wound exploration to determine whether the anterior or posterior abdominal fascia is violated.
37
How to manage a patient with thoracoabdominal stab wound?
**1**. a diaphragm evaluation by chest x-ray **2**. pericardial FAST **3**. diagnostic laparoscopy
38
Management of anterior abdominal stab wound?
**1**. abdominopelvic CT **2**. local wound exploration **3**. serial abdominal exams for 24 hours **In case of** fascial violation or peritonitis ▶️ diagnostic laparoscopy or exploratory laparotomy is performed. **if not** ▶️ discharge
39
Management of flank or back abdominal wound?
**1**. abdominal CT with or without rectal contrast **2**. If positive ➡️exploratory laparotomy **3**. If not ➡️ discharge or serial exams may be considered
40
warning signs after management of an abdominal stab wound ?
**1**. peritonitis **2**. hemodynamic instability **3**. significant decreases in hemoglobin level (of more than 3 g\dL) **4**. leukocytosis ## Footnote 📝either surgery or diagnostic modalities such as CT or diagnostic peritoneal lavage are done. Otherwise, in patients without clinical or laboratory changes after 24 hours, diet is initiated and they are discharged home.
41
What does **FAST** stand for regarding trauma assessment?
Focused Assessment with Sonography for Trauma (**FAST**) is a rapid, bedside ultrasound exam used to detect free fluid (e.g., blood) in trauma patients.
42
What is the source of hemorrhage in pelvic instability in most cases?
presacral venous plexus
43
Management of a case of pelvic instability?
**Hemodynamically stable**➡️ plain radiograph **Hemodynamically unstable**➡️ Pelvic binder to compress the fracture **then** FAST if positive▶️ laparotomy
44
Indication of angiography & embolization in trauma patient ?
Arterial bleeding not responding to fluid resuscitation
45
What does ABCDE stand for in ATLS trauma assessment?
**A** - Airway & C spine protection **B** - Breathing and ventilation **C** - Circulation and hemorrhage control **D** - Disability (neurologic status) **E** - Exposure
46
How to manage a case of pneumothorax?
A tube thoracostomy
47
IS it allowed to push back the eviscerated organ in the field or in the emergency room (ER)?
**NEVER**, under any circumstances, the eviscerated organs be pushed back into the abdominal cavity outside the operating room.
48
Why ETCO2 is an important measurement in ventilation monitoring?
**1**. It serves as an indicator of systemic perfusion (with the gradient between ETCO2 and PaCO2 reflecting dead-space ventilation) **2**. identify when the esophagus was accidentally intubated by measuring no ETCO2 at all
49
ETCO2 =0 indicates.......
improper intubation ## Footnote 📝absent ETCO2 indicates the need to check whether the tube was misplaced and to correct its position for effective ventilation.
50
Which one is more common penetrating or blunt trauma of the diaphragm?
Penetrating trauma. ## Footnote 📝penetrating trauma of the diaphragm is seen more than blunt trauma (67% vs. 33% respectively)
51
Which one has a higher mortality rate blunt or penetrating trauma of the diaphragm?
Blunt injuries
52
What is the most common cause of death in diaphragmatic injuries?
damage to organs adjacent to the diaphragm. ## Footnote 📝since damage to the diaphragm itself is usually of limited threat to life.
53
What is the mechanism behind blunt diaphragmatic injury in trauma patients?
rapid increase in intra-abdominal pressure during an anterior impact ➡️ a rupture (Blow-Out) of the diaphragm.
54
Which side of diaphragm is injured the most?
the left side of the diaphragm (75%) ## Footnote 📝incidence of right side of diaphragm is low because of the coverage of the right side with the liver.
55
What are the potential complications (sequelae) of an undiagnosed diaphragmatic hernia?
expansion of the tear (hernia)➡️ herniation of the abdominal organs into the chest.
56
In case of a diaphragmatic hernia X-ray shows.......
presence of abdominal organs, usually the stomach, within the chest.
57
CT scan finding in diaphragmatic hernia?
**1**. presence of abdominal organs within the chest **OR** **2**. irregularity of the diaphragm itself (e.g. thickening, elevation or defect).
58
How can a nasogastric (NG) tube aid in diagnosing a diaphragmatic hernia?
if the tube is identified in the lower left hemithorax.
59
Next step if diaphragmatic rupture was suspected in a hemodynamically stable patient without peritonitis ?
laparoscopy ## Footnote 📝laparoscopy is recommended over CT scanning alone to reduce the incidence of missed traumatic diaphragmatic injuries.
60
During surgery how is the repair of a diaphragmatic hernia is done?
**1**. debriding the wound & suturing the defect in one layer with non-absorbable suture **2**. if the defect is too large ➡️ repair it with a synthetic material (mesh) if the surgical field is not contaminated.
61
Source of bleeding in diaphragmatic rupture in most cases?
Phernic artery
62
Next step after a confirmed case of diaphragmatic rupture with herniation of abdominal organs itto the chest?
Laparotomy
63
Next step after a confirmed penetrating injury to the stomach?
Laparotomy
64
Why laparotomy is preferred over gastroscopy in penetrtating injury to the stomach?
**1**. Gastroscopy in the setting of penetrating trauma may worsen the injury. **2**. a penetrating injury to the stomach➡️ perforation and signs of peritonitis or bloody vomiting ➡️ requiring a laparotomy.
65
A seat belt mark on the neck after a car accident indicates.....
High risk injury to the cervical blood vessels.
66
Next step after finding a neck seat belt mark in a car crash patient?
CT on the neck.
67
How is a vertebral artery pseudoaneurysm managed intraoperatively after trauma?
Stent replacement **or** embolization
68
What is Damage Control Resuscitation (**DCR**)?
**DCR**:is atrauma resuscitation strategy focused on * minimizing crystalloid use * rapidly controlling bleeding * correcting coagulopathy * preventing the lethal triad of (hypothermia, acidosis, and coagulopathy) 🧠**Mnemonic**: "**HARM-C**" Think of DCR as protecting the patient from HARM with Control. **H** – Hypothermia prevention **A** – Acidosis avoidance **R**– Rapid bleeding control **M** – Minimize crystalloids **C** – Correct coagulopathy
69
Highest priority of **DCR**? | **DCR**:Damage Control Resuscitation
Rapid control of bleeding
70
Why reducing crystalloid solution use is so important according to **DCR**? | **DCR**:Damage Control Resuscitation
As they can cause: **1**. Inflammation after hemorrhagic shock **2**. Abdominal compartment syndrome
71
According to **DCR** how to reduce the use of crystalloid solution? | **DCR**:Damage Control Resuscitation
**1**. Initial use of 5% hypertonic saline **2**. early use of blood products (PRBCs, FFP, platelets, cryoprecipitates) | **PRBCs**:Packed Red Blood Cells ## Footnote 📝**FFP**:Fresh Frozen Plasma – Plasma frozen within 8 hours of donation; contains clotting factors.
72
According to **DCR**, what is the preferred type of blood product to use first? | **DCR**:Damage Control Resuscitation
Whole blood
73
According to **DCR** ,if whole blood is not available as a first choice what’s the next best option? | **DCR**:Damage Control Resuscitation
component therapy with PRBCs, thawed plasma, and platelets in a** 1:1:1** ratio
74
How pringle maneuver is performed?
the hepatoduodenal ligament is surrounded by a device or a special rubber band **SO**, the blood flow to the liver from the hepatic artery and the portal vein is blocked.
75
Indication of pringle maneuver during abdominal surgery?
slow down or stop the blood supply to the liver to identify the source of the bleeding.
76
Bleeding persists after a pringle maneuver ,what is the likely source?
IVC **OR** hepatic vein | IVC: inferior vena cava
77
Compression of hepatoduodenal ligament in pringle maneuver leads to compression of.....
**1**. proper haptic artery **2**. portal vein **3**. common bile duct
78
What is the first step when massive bleeding occurs during surgery?
Immediate abdominal packing to control bleeding and gain exposure.
79
What is the next step if abdominal packing failed to control bleeding during abdominal surgery?
Perform a Pringle maneuver to assess bleeding from the portal triad.
80
The most reliable screening method for injuries of the genitourinary tract is?
screening for hematuria
81
Next step after detecting hematuria in a trauma patient?
Abdomenopelvic CT with contrast
82
Why CT with contrast is preferred over U/S after detecting hematuria in a trauma patient?
**Because** US may reveal fluid and the presence of an injury **but** cannot determine the degree of injury for example.
83
You found blood in the catheter of a trauma patient what is the next step?
Abdomenopelvic CT with contrast
84
Causes of peptic ulcer?
* **↑** **in offending factors**: **1**. high acidic content **2**. increase in pepsin **3**. ethanol **4**. smoking **5**. bile reflux from the duodenum **6**. ischemia **7**. NSAIDs **8**. hypoxia **9.** H. Pylori **OR** * ↓ **in Protective factors** **1**. mucosal bicarbonate secretions **2.** mucus production **3**. good vascular supply **4**.growth factors **5**.cell renewal **6**.prostaglandins.
85
sudden, sharp and acute epigastric abdominal pain in a peptic ulcer patient what do you expect?
Ulcer perforation
86
In most cases, the first symptom of peptic ulcer disease is ....
Ulcer perforation
87
Chest X-RAY finding in peptic ulcer perforation?
free air (air under diaphragm)
88
Most common site of peptic ulcer perforation?
the first part of the duodenum
89
X-ray shows perforated peptic ulcer what is the next step?
surgery
90
Through which incision can you access a perforated first part of the duodenum?
Via an upper midline laparotomy incision.
91
Managment of perforated ulcer during surgery?
🔹 **Small Perforations** (<1 cm)**: – Primary closure – Reinforced with a well-vascularized omental patch (simple omentopexy) 🔹 **Large Perforations or Fibrotic Edges** (not suitable for tension-free closure): – Graham Patch Repair using healthy omentum. – Acts as a biologic plug over the perforation
92
How does pelvic trauma most commonly occur?
Impact injuries such as car accidents.
93
Is a CT scan necessary after detecting free air on chest X-ray?
**NO**, because Obtaining a CT scan in cases of free air demonstrated on chest XRAY will not change the course of management,
94
Most dangerous complications of peptic ulcer?
**1**. Bleeding **2**. perforation **3**. peritonitis
95
Why are pelvic fractures life-threatening?
**Because** they can cause massive bleeding ➡️ (intraperitoneal and retroperitoneal) ➡️ hemorrhagic shock and death.
96
Pelvic fractures present with....
**1**. pelvic pain **2**. reduced range of motion **3**. hematomas
97
First step in management of pelvic bleeding?
Applying pelvic binder.
98
How is angioembolization performed in pelvic bleeding?
**1**. An intra-arterial catheter is inserted into a major artery (typically via the **femoral artery**). **2**. The catheter is navigated to the branches of **the internal iliac arteries** supplying the bleeding area. **3**. Thrombotic agents (such as coils, particles, or gel foam) are delivered through the catheter to embolize (block) the bleeding vessels. **4**. This stops the hemorrhage by preventing further blood flow to the injured site.
99
What is angioembolization?
an endovascular technique used to control bleeding by occluding (blocking) blood vessels.
100
Angioembolization used mainly for(arterial **or** venous ) bleeding?
arterial
101
What is the difference between angioembolization & Extraperittoneal pelvic packing regarding source of bleeding?
**Extraperitoneal Pelvic Packing**➡️Venous, bony, and some arterial bleeding **Angioembolization** ➡️ Mainly arterial bleeding
102
What is the difference between angioembolization & Extraperittoneal pelvic packing regarding Method?
**Extraperitoneal Pelvic Packing** ➡️Surgical tamponade using laparotomy sponges **Angioembolization** ➡️ Catheter-based endovascular procedure
103
Management of bleeding in pelvic fracture?
**Initial Assessment** | **Check hemodynamic stability** + **Identify pelvic fracture**] | **Immediate Action: Apply Pelvic Binder** | **Assess Source of Bleeding** | **Venous/Bony Bleeding** | **Continue Pelvic Binder** + **Consider** Extraperitoneal Pelvic Packing | **Arterial** **Bleeding** Suspected | **Angioembolization** if available | NOT available | **Perform** Extraperitoneal Pelvic Packing
104
Management of Pelvic fracture after controling the source of bleeding?
**Fracture Stabilization** | **External Fixation** (temporary) | **External Fixation** (definitive)
105
The first priority in neck injuries is....
Airway protection.
106
Next step after inspecting expanding hematoma in the neck of a trauma patient?
Immediate intubation.
107
First step in bleeding in a trauma patient?
Direct pressure on the wound until exploration in the OR.
108
List "hard signs".
**1**. Airway compromise **2**. Massive subcutaneous emphysema **3**. Air bubbles necessitating though wound **4**. Expanding or pulsatile hematoma **5**. Active bleeding **6**. Neurologic deficit **7**. Hematemesis
109
What are "hard signs" of vascular or aerodigestive injury?
They are clinical signs strongly suggesting serious vascular or airway damage that typically require immediate surgical intervention.
110
What gastrointestinal finding is considered a "hard sign"?
Hematemesis.
111
How are penetrating neck injuries categorized?
By anatomical location into Zones I, II, and III.
112
Extent & content of Zone II in penetrating neck injuries?
**Extent**: from the cricoid cartilage to the mandibular angle **Contents**: * carotid and arterial artery * the jugular vein * structures of the respiratory system. * structures ofdigestive system.
113
Extent & content of Zone I in penetrating neck injuries?
**Extent**: from the thorax to the cricoid cartilage **Contents**: * large vascular structures * the trachea * the esophagus
114
Extent & content of Zone III in penetrating neck injuries?
**Extent**: between the angle of the mandible and the base of the skull **Contents**: * contains blood vessels that are surgically difficult to reveal.
115
Which zone of the three zones of neck is the easiest location for surgical tratment?
Zone II
116
Penetrating neck injuries with evidence of hard signs or hemodynamic instability what is the next step?
surgical intervention **and** exploration in the operating room.
117
What GCS score indicates the need for intubation?
A GCS score of 8 or below.
118
**GCS** lowest score... | **GCS**: Galsgow coma scale
3
119
In trauma patients, what is the first priority in management?
Airway management according to ATLS.
120
**GCS** measures... | **GCS**: Galsgow coma scale
**1**. Eye opening **2**. Verbal response **3**. Motor response
121
Why **GCS** is so important in a trauma patient?
Because it reflects the patient’s mental status➡️ guide decision-making in the context of traumatic brain injuries.
122
**GCS** highest score... | **GCS**: Galsgow coma scale
15
123
What is the likely diagnosis in a trauma patient with hypotension, tachycardia, dyspnea, and unilateral decreased breath sounds after a stab wound?
Tension pneumothorax
124
What is the appropriate next step in management for tension pneumothorax after chest trauma in a hospital setting?
**Immediate Action**: Right chest tube insertion (Tube thoracostomy
125
Why is needle thoracostomy **NOT** the preferred first step for traumatic tension pneumothorax in the emergency department?
**1**. Needle thoracostomy is mainly for In the hospital setting **2**. chest tube thoracostomy is faster, more definitive, and immediately available.
126
When is emergency thoracotomy (resuscitative thoracotomy) indicated in chest trauma?
**Indicated only** if the patient with penetrating thoracic injury experiences cardiac arrest.
127
In trauma management following the ATLS protocol, what is assessed during Stage A and Stage B?
* **Stage** **A** Airway * **Stage** **B** Breathing
128
In a trauma patient, what is prioritized first brain injury **or** hemorrhagic shock?
hemorrhagic shock ## Footnote 📝 Always prioritize life-threatening hemorrhagic shock first before addressing brain injury.
129
In a blunt trauma patient who is hemodynamically unstable with a positive FAST exam, what is the next immediate step?
**Emergency laparotomy** to control intraabdominal bleeding.
130
Can a significant intracranial hemorrhage alone cause hemodynamic instability (shock)?
**No** ## Footnote 📝if a patient with intracranial hemorrhage became hemodynamically unstable ➡️search for another source of bleeding because intracranial hemorrhage rarely causes hemodynamic instability.
131
In trauma, what are the five locations where massive blood loss can occur?
**1**. Chest **2**. Abdomen **3**. Retroperitoneum (pelvic fractures) **4**. Long bone fractures **5**. External bleeding.
132
When is craniotomy indicated in trauma patients?
**1**. After CT imaging confirms an operable intracranial injury **&** **2**. once the patient is hemodynamically stabilized.
133
Priorities in dealing with a trauma patient with suspected brain injury?
**1**. Treat the most immediate life-threatening condition first. **2**. Control hemorrhage before addressing brain injury or proceeding to CT imaging.
134
How is Eye Opening scored in the Glasgow Coma Scale?
**Spontaneously**=4 **To voice**=3 **To pain**=2 **None**=1
135
How is Verbal Response scored in the Glasgow Coma Scale?
* **Oriented** =5 * **Confused** =4 * **Inappropriate words**=3 * **Incomprehensible sounds**=2 * **None**=1
136
How is Motor Response scored in the Glasgow Coma Scale?
* **Obeys commands** =6 * **Localizes to pain** =5 * **Withdraws from pain** =4 * **Abnormal flexion (decorticate posturing)** =3 * **Abnormal extension** **(decerebrate posturing)** =2 * **None** =1
137
What does a GCS score of ≤8 usually indicate?
Severe brain injury➡️ Often requires intubation and intensive monitoring.
138
What clinical signs suggest a tension pneumothorax after blunt chest trauma?
**1**. Dyspnea and restlessness **2**. Subcutaneous emphysema **3**. Hypotension (unmeasurable BP) **4**. Decreased or absent breath sounds on one side **5**. Tracheal deviation (in advanced cases)
139
In a suspected tension pneumothorax, what is the immediate next step in management?
**Insert a chest tube** (tube thoracostomy) or large-bore needle immediately **do not wait for imaging!**
140
Why should you not wait for a chest X-ray before treating a tension pneumothorax?
Waiting may lead to **cardiovascular collapse and death**.
141
What is the pathophysiology behind hypotension in tension pneumothorax?
Increased intrathoracic pressure ➡️ compresses large veins ➡️ decreased venous return ➡️ decreased preload ➡️ severe hypotension. ## Footnote 📝>10 mmHg drop in systolic BP during inspiration is called pulsus Paradoxus.
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What is pulsus paradoxus and what is its pathophysiology (e.g. in tension pneumothorax)?
**Definition:** An exaggerated drop (>10 mmHg) in systolic BP during inspiration. **Pathophysiology:** Inspiration ↑ venous return to RV. In tension pneumothorax: ↑ intrathoracic pressure limits RV expansion. Interventricular septum shifts left ➡️ ↓ LV filling. ↓ Stroke volume ➡️ ↓ systolic BP during inspiration.
143
Why is positive pressure ventilation risky before decompressing a tension pneumothorax?
**It can worsen the pneumothorax by:** **1**.further increasing intrathoracic pressure **2**.collapsing the cardiovascular system.
144
When should a **FAST** ultrasound be performed during trauma evaluation?
After the patient is stabilized **(secondary survey)**. ## Footnote 📝 Immediate life-threatening conditions like tension pneumothorax must be treated first.
145
Which injuries after blunt trauma can also require chest tube placement beside pneumothorax?
**1**. Massive hemothorax **2**. Tension pneumothorax ## Footnote 📝Both require tube thoracostomy for lung re-expansion and blood evacuation (in hemothorax).
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In a trauma patient with dyspnea, subcutaneous emphysema, and sudden hypotension, what diagnosis should be suspected?
**Tension pneumothorax**
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What is the **hallmark** clinical sign that suggests a tension pneumothorax after trauma?
A **sudden** drop in blood pressure (**hypotension**) in the setting of chest **trauma** and **dyspnea**.
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What is the emergency treatment for a tension pneumothorax?
Immediate large-bore needle decompression **or** Tube thoracostomy on the affected side.
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Why must treatment of a tension pneumothorax be performed immediately without imaging?
**To prevent cardiovascular collapse** ## Footnote 📝it is a clinical diagnosis requiring urgent decompression.
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Where should the needle or chest tube be inserted in tension pneumothorax?
**Needle:** 2nd intercostal space, midclavicular line **Chest tube:** 5th intercostal space, midaxillary line
151
What is the role of **FAST** ultrasound in penetrating abdominal trauma?
**Limited role.** FAST is mainly useful in blunt trauma to detect free fluid.
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What is the final step after 24 hours of stable observation without clinical or lab deterioration in abdominal stab wound patients?
Start diet **and** discharge home.
153
What are the classic clinical features of adrenal crisis?
**1**.Shock (hypotension) **2**.Abdominal pain **3**.Nausea and vomiting **4**.Fever **5**.Electrolyte disturbances **6**.Hypoglycemia
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**pathophysiology** behind hypotension in adrenal crisis?
**1**. Mineralocorticoid deficiency ➡️ Loss of sodium and intravascular volume **2**. Reduced cardiovascular responsiveness to catecholamines
155
What life-threatening condition can occur in chronic steroid users undergoing surgery without perioperative steroid coverage?
**Adrenal crisis** (acute adrenal insufficiency).
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Management of adrenal crisis?
**1**. **Large-volume IV isotonic saline resuscitation** **2**. **IV glucocorticoid administration:** (100 mg hydrocortisone bolus, then 75 mg every 8 hours **OR** 4 mg dexamethasone every 24 hours)
157
How should a Crohn’s patient on chronic steroids use be managed perioperatively?
**Give stress-dose steroids:** * IV hydrocortisone 100 mg bolus, then 50–100 mg IV every 8 hours for major surgery * Continue usual steroid dose if surgery is minor. * Taper after 24–48 hrs if stable. ## Footnote 📝**Acute adrenal crisis** (adrenal insufficiency) can be triggered in chronic steroid users by surgery or stress without appropriate perioperative glucocorticoid supplementation.
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Why is IV insulin not indicated in adrenal crisis management?
Because adrenal crisis causes **hypoglycemia** not hyperglycemia
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What are the main causes of hypoglycemia?
* 🧠**Insulin-related**: * Exogenous insulin overdose - Insulinoma * 🧠.**Critical illness** : - Sepsis - Liver failure - Renal failure * 🧠.**Endocrine deficiency**: - Adrenal insufficiency - Hypopituitarism * 🧠**Alcohol-induced** * 🧠**Reactive/postprandial hypoglycemia** * 🧠**Fasting (starvation, malnutrition)**
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Why is noradrenaline (norepinephrine) not useful initially in adrenal crisis?
**Because of** reduced vascular responsiveness to catecholamines due to mineralocorticoid deficiency ➡️ **fluids and steroids must be given first.**
161
How do you differentiate primary vs secondary adrenal insufficiency regarding **ACTH**,**Cortisol**,**aldosterone**, **hyperpigmentation**,**K levels**,**Na levels**,**cosyntropin test**?
🧠**Primary (Addison’s)** **ACTH** ↑ High **Cortisol** ↓ **Aldosterone** ↓ **Hyperpigmentation** ✅ Yes **K⁺ levels** ↑ Hyperkalemia **Na⁺ levels** ↓ Hyponatremia **Hypotension** More severe **Cosyntropin test**No cortisol response 🧠**Secondary:** **ACTH** ↓ Low or inappropriately normal **Cortisol** ↓ **Aldosterone** Normal ✅ **Hyperpigmentation** ❌ No **K⁺ levels** Normal **Na⁺ levels** Normal **Hypotension** Less severe **Cosyntropin test**Delayed but may respond
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In patients with secondary adrenal insufficiency, what perioperative steroid strategy is recommended?
* 🧠 **Continue** usual glucocorticoid dosage * 🧠**Supplement** based on surgical stress level (minor, moderate, major) using specific guidelines.
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What is the approximate risk of hypotensive crisis in secondary adrenal insufficiency patients without steroid coverage?
1-2% risk without perioperative glucocorticoid coverage.
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How are surgical procedures classified for perioperative steroid management?
**1. Minor** **2. moderate** **3. major stress.** Each category has its own glucocorticoid supplementation protocol.
165
What are the two essential components of adrenal crisis management?
**1. Large-volume isotonic IV fluids** **2. IV glucocorticoids (hydrocortisone or dexamethasone)**
166
What is the first step in the primary survey of trauma patients?
**Airway assessment** (part of ABCDE approach).
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What finding suggests malposition of an endotracheal tube?
**1**. Decreased breath sounds on one side **2.**Low oxygen saturation (Oxygen saturation is deteriorating fast)
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The appropriate position of the ET can be confirmed by:
**1.** Chest and abdomen auscultation (to rule out esophageal intubation). **2.** End-tidal carbon dioxide measurement **3.** A chest radiograph.
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* **ETCO2** is known as... * **ETCO2** normal values range between.....
* 🧠the **fractional pressure** of carbon dioxide at the **end of expiration**. * 🧠 range between **35** and **45** mmHg
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Why **ETCO2** is so important in a trauma patient?
**AS It reflects** * the cardiac output * perfusion * gas exchange occurring in the lungs.
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**ETCO2** is measured by ....
Capnometer
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Which bronchus is more likely to be unintentionally intubated during ET tube placement?
**Right main bronchus** — it is wider and more vertical than the left.
173
What is the fastest method to confirm proper ET tube placement in an emergency?
End-tidal carbon dioxide (**ETCO₂**) measurement.
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What is the appropriate next step in a trauma patient with decreased left-sided breath sounds post-intubation?
🧠Confirm endotracheal tube position immediately (preferably using **ETCO₂**).
175
What clinical features suggest **airway injury** in a trauma patient?
**1**. Inability to speak (↓ consciousness or obstruction) **2**. Noisy breathing (stridor, gurgling) **3**. Severe facial trauma (especially with bleeding) **4**. Blood or foreign bodies in oropharynx **5**. Agitation or altered mental status
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What does a drop in oxygen saturation to 85% after facial trauma and vomiting blood indicate?
Progressive **respiratory failure** due to compromised airway, likely from blood obstructing the oropharynx.
177
Next step in management after failed endotracheal intubation for multiple times?
**cricothyroidotomy**
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What is the first-line method for establishing a definitive airway in trauma?
**Endotracheal intubation**, preferably with video-assisted laryngoscopy if available.
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What is the difference in indication between cricothyroidotomy and tracheostomy in trauma?
🧠**Cricothyroidotomy:** Emergency surgical airway after failed intubation or airway obstruction. 🧠**Tracheostomy:** Performed when there is suspected laryngeal trauma or need for long-term airway management.
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🔵 What are the definitive and adjunct airway management options in trauma patients?
* 🚩 **Definitive airway:** _ Orotracheal intubation (can be video-assisted) * 🚩 **Adjuncts (temporary airways):** _ Laryngeal mask airway (LMA) _ Combitube
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⚠️ When is cricothyroidotomy indicated in trauma airway management?
🚨 **When intubation fails** and visualization is hindered (e.g., by blood), proceed directly to cricothyroidotomy. 👉🏼 Especially **after 3 failed intubation** attempts and obscured anatomy due to blood.
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⛔ Why is LMA not suitable as a definitive airway in severely hypoxic trauma patients?
**Because** LMAs do not ensure acceptable oxygenation and are only **temporary** adjuncts. 👉🏼 Not suitable when the patient cannot maintain oxygen levels.
183
In what trauma scenario is tracheostomy preferred over cricothyroidotomy?
➡️ When **laryngeal structural damage** is suspected, tracheostomy is performed. ⚠️ Usually **not** the **first-line** surgical airway in acute trauma setting.
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Why is continued bag-valve-mask (BVM) ventilation discouraged in unresponsive trauma patients?
⛔ It increases **aspiration risk** and delays the establishment of a definitive airway.
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What is the pathophysiology behind fever in diverticulitis?
🔻 **Microperforation** allows fecal flora to enter the sterile peritoneal cavity ➡️ localized infection and inflammation ➡️ fever.
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⚠️ What indicates progression from localized to diffuse peritonitis in diverticulitis?
🚨 Presence of **rebound tenderness** and **guarding** ➡️ Suggests free perforation and widespread contamination.
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What are the **typical** clinical features of diverticulitis?
🚩 **LLQ pain & tenderness** (sigmoid colon) 🚩 **Change in bowel habits** 🚩 **Anorexia, nausea** 🚩 **Fever** 🚩 **Urinary urgency** (if bladder involved)
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...... is the most common site of pain in diverticulitis due to involvement of the **sigmoid colon**.
**left lower quadrant.**
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.......causes **fever** due to infection alone.
**Diverticulitis.** ## Footnote 🔻 Here's how it happens step by step: **Microperforation**: A small tear or rupture forms in the wall of a diverticulum. **Leakage of colonic contents**: This allows bacteria, fecal material, and gas from the colon (which is full of gut flora) to escape into the surrounding sterile tissues. **Local infection & inflammation**: These bacteria trigger an inflammatory response in the colon wall and peritoneum—especially if the perforation is walled off (localized). **Immune response**: The body reacts by releasing pyrogens (fever-inducing cytokines like IL-1 and TNF-alpha) into the bloodstream → leading to fever. ✅ This is true infection, not just sterile inflammation, because it involves actual bacterial invasion and immune response to pathogens.
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Why **don’t** pancreatitis, abdominal hematoma, or lung infarction cause fever by themselves?
⛔ Because their primary mechanisms are **non-infectious**. ➡️ Fever may occur only if they become secondarily infected.
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What are the typical symptoms of uncomplicated (simple) cystitis in immunocompetent women?
🚩 **Dysuria** 🚩 **Increased frequency and urgency to void** 🚩 **Malodorous urine** ⛔ Fever is **not** typical in uncomplicated cases.
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Why is simple cystitis **not** usually associated with fever?
➡️ Because the infection is **localized** to the bladder and does **not** cause **systemic** involvement like pyelonephritis.
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👉🏼 Pancreatitis, abdominal hematoma, and lung infarction may cause fever only if ....... occurs
**Secondary infection**
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What is the underlying cause of diverticulitis?
➡️ **Microperforation** of a colonic diverticulum ➡️ **leakage** of colonic contents into the sterile peritoneal cavity.
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How does microperforation in diverticulitis lead to inflammation?
🔻 **Extravasation** of colonic flora and feces into the peritoneal cavity → triggers **localized peritoneal inflammation**.
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🔵 Where do diverticula most commonly form and why?
➡️ In **high-pressure regions** where **vasa recta** traverse the muscular layer, especially the **sigmoid** and **descending colon**.
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What type of diverticula are seen in diverticulosis?
➡️ **"False" diverticula** — they contain only mucosa and muscularis mucosa, not the full wall.
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What is the difference between diverticulosis and diverticulitis?
**Diverticulosis** ⚙️ **Definition**: Outpouchings of bowel wall Inflammation 😷 **Symptoms**: Usually asymptomatic 👁 **Pathophysiology**: Structural change (herniation) 🧠 **Type of lesion**: False diverticula (mucosa only) ❗**Complications**: Bleeding (diverticular hemorrhage) **Diverticulitis** ⚙️ **Definition**Inflammation/infection of diverticula 😷 **Symptoms**Symptomatic: fever, LLQ pain, leukocytosis 👁**Pathophysiology**Microperforation → infection/inflammation 🧠 **Type of lesion**Infected/inflamed diverticula ❗**Complications**Abscess, perforation, peritonitis, fistula
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What is the difference between true and false diverticula?
🧠**True Diverticula**:Involve all layers of the bowel wall (mucosa, submucosa, muscularis, serosa) 🧠**False Diverticula**: Involve only mucosa and submucosa (no muscularis)
200
........involve all layers of the bowel wall
**True diverticula**
201
......involve only the mucosa and submucosa.
**false diverticula**
202
What is the **diagnostic imaging** of choice for diverticulitis?
➡️ **IV-contrast CT scan** Findings include: 🔍 Presence of diverticula 🔍 Colonic wall thickening 🔍 Pericolic fat stranding ± abscess formation
203
When should **colonoscopy** be performed in diverticulitis and why?
⚠️ **Not** during acute phase (risk of perforation) ✅ Should be done **4–8 weeks later** to rule out malignancy or IBD
204
What does the Modified **Hinchey** Classification describe?
➡️ It describes the **severity** of diverticulitis: 🧠**Stage0**:Mild clinical diverticulitis 🧠**Stage1**:**A**. Phlegmon or **B**. Confined pericolic abscess 🧠**Stage2**:Abscess in the pelvis 🧠**Stage3**:Diffuse peritonitis with pus in the abdomen. 🧠**Stage4**:Diffuse peritonitis with feces in the abdomen.
205
🚩 What are risk factors that increase the likelihood of developing diverticulosis?
🔻 **Older age** 🔻 **Western diet** high in red meat, fat, and refined grains ## Footnote 📝**Obesity** is also a risk factor.
206
What factors decrease the risk of diverticulosis?
🔻 **Diet rich in fruits, vegetables, and whole grains** 🔻**High fiber intake**
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How is **uncomplicated** diverticulitis (**Stage 0**) managed?
➡️ **Conservative treatment**: 🔹 Antibiotics 🔹 Fluids 🔹 Short-term diet modifications
208
How are **Stage I & II** diverticulitis (with abscess) managed?
🚩 **Smaller abscesses**: ➡️ Antibiotics 🚩 **Larger abscesses**: ➡️ Antibiotics + Drainage ➡️ Surgery if no response 📌 Elective surgery offered after recovery
209
How is perforated diverticulitis (**Stage III & IV**) treated?
🔵 **Stable patients**: ➡️ Sigmoidectomy + Primary anastomosis + Diverting ileostomy 🔴 **Unstable / Immunocompromised patients**: ➡️ Hartmann procedure = Sigmoidectomy + Stoma without primary anastomosis
210
What is the most common type of diverticulitis-associated **fistula**?
➡️ **Colovesical fistula** 📌 Presents with: 🔹 Recurrent UTIs 🔹 Pneumaturia 🔹 Fecaluria
211
How is a colovesical fistula **treated**?
🔹**Step 1:** Antibiotics 🔹**Step 2:** Colonoscopy + Cystoscopy 🔹**Step 3:** Surgical resection of colon and fistula tract with primary anastomosis
212
👉🏼 Larger abscesses in diverticulitis require......
**antibiotics + drainage** ➡️possibly surgery if unresolved.
213
What is the most likely cause of respiratory failure, hypotension, and rhabdomyolysis in a malnourished patient started on parenteral nutrition?
✅ **Hypophosphatemia** & **hypokalemia** 📌 This is characteristic of **refeeding syndrome**
214
⚠️ What is **refeeding syndrome** and what causes it?
➡️ A **life-threatening shift** in fluids and electrolytes (especially phosphate, potassium, and magnesium) in **malnourished patients** after **starting nutrition** (especially parenteral or high-carb intake).
215
Why is phosphate critical in refeeding syndrome?
🔻 Phosphate is essential for **ATP production** ## Footnote 📝 Hypophosphatemia impairs muscle function (including respiratory muscles) → respiratory failure
216
What are the clinical features of **refeeding syndrome**?
🚨 **Respiratory failure** 🚨 **Hypotension** 🚨 **Rhabdomyolysis** 🚨 **Arrhythmias, seizures, weakness**
217
When is IV (**parenteral**) nutrition indicated?
➡️ When a patient cannot obtain adequate nutrients via **enteral feeding** (e.g., due to vomiting, fistula, or obstruction).
218
When should parenteral nutrition be initiated in **hospitalized** patients?
➡️ Usually **delayed** for **7 days** after presentation, 🔹 to assess if the patient can resume **enteral feeding**.
219
👉🏼 Parenteral nutrition is typically started only when the patient.....
**cannot meet nutritional needs enterally.**
220
👉🏼In hospitalized patients Parenteral nutrition is usually.....
**delayed for 7 days** * to allow time for possible enteral nutrition.
221
What are the infectious complications of parenteral nutrition?
➡️ **Central line–associated bloodstream infection (CLABSI)** 📌 Risk is higher than in central lines used for non-nutritional purposes.
222
What vascular complication is associated with parenteral nutrition?
➡️ **Vascular thrombosis**
223
What are the **metabolic and biochemical** complications of parenteral nutrition?
🔹 **Hypertriglyceridemia** 🔹 **Hyperglycemia** 🔹 **Electrolyte imbalances** (e.g., in refeeding syndrome or overfeeding)
224
What **hepatic** complications may occur with long-term parenteral nutrition?
* ➡️ **Hepatic steatosis** * ➡️ **Cholestasis** * ➡️ **Cirrhosis**
225
⚠️ What is **refeeding syndrome** and how can it be prevented?
➡️ Occurs with **full caloric** administration after **starvation** ⚠️ Leads to: * Hypokalemia * Hypophosphatemia * Hypomagnesemia ✅ **Prevention**: Gradual increase in caloric intake
226
What is a complication of **overfeeding** in parenteral nutrition?
* **Electrolyte imbalances** * **hyperglycemia** * **hypertriglyceridemia** * **complicates weaning from mechanical ventilation.** ## Footnote 📝 **complicates weaning from mechanical ventilation** means ➡️ Patients may fail attempts to breathe without ventilator support, delaying recovery and prolonging mechanical ventilation.
227
👉🏼 To prevent refeeding syndrome, calories should be.....
**introduced gradually** after starvation
228
👉🏼.......is more common in patients receiving parenteral nutrition through a central line.
🔹**CLABSI**= central line–associated bloodstream infection
229
👉🏼 Refeeding syndrome may result in......
**Hypo** 📌 **hypo**kalemia 📌**hypo**phosphatemia 📌**hypo**magnesemia
230
👉🏼 Overfeeding in parenteral nutrition can result in.....
**Hyper** 📌**hyper**glycemia 📌**hyper**triglyceridemia
231
What symptoms can result from **hypokalemia** and **hypophosphatemia** in refeeding syndrome?
🚨 **Respiratory failure** – due to diaphragm weakness (↓ ATP from hypophosphatemia) 🚨 **Hypotension** – from impaired cardiac and vascular muscle function 🚨 **Rhabdomyolysis** – due to muscle cell breakdown from electrolyte depletion