Urology Flashcards
(13 cards)
what are the determinants of obstructive uropathy
Severity depends on cause of lesion, duration of blockage, location of blockage, degree of completeness, whether it involves one or both urinary tract systems, further classified as upper or lower urinary tract
what causes the formation of kidney stones
As urine filiters, solute concentrations get high, causing stone formation. Masses of crystals proteins mineral salts of the urinary tract. Supersaturation of one or more salts leads to precipation then crystallization or aggregation into stone.
what are clinical manifestations of kidney stones
renal pain, sharp stabbing back pain
inflammation of the bladder typically caused y genitourinary bacterial flora which overgrown or travel backwards into the host body ( retrograde movement) ( common pathogens E coli and Staph saprophyticus
cystitis ( UTI)
clinical manifestations of cystitis
can be asymptomatic, frequent urgent dysuria ( pain with urination), hematuria, cloudy foul smelling urine, flank pain, determined with urine culture
infection of upper urinary tract system ( uretuer, renal pelivis kidney) more severe than UTI, CAT scan determines, have symptoms of UTI along with fever, chills, costovertebral angle tenderness
pyelonephritis
clinical onset of pyelonephritis
acute onset of sustemic systems (fever and chills), costovertebral angle tenderness dsyrua ( pain with urination), hematuria, cloudy foul smelling urine, plank pain,
pyelonephritis is diagnosed with
CAT scan
is a sudden decline in kidney function measured in decreased GRF, increased serum creatine, increased blood urea nitrogen (BUN), reduced urine output
acute kidney injury
this kind of acute kidney injury is most common
prerenal AKI
this kind of AKI is inadequate renal perfusion leads to eventual decrease in glomerular filtration pressure (ex: hypotension of any cause, hypocolemia from acute blood loss/hemorrhage, renal vasoconstricition caused by NSAIDS)
prerenal AKI
this kind of AKI is direct injury of renal parenchyma or intersitial cells. (ex: acute tubular necrosis, exposure to nephrotoxic contrast dye, acute glomerulnephrtits, graft rejection)
intra renal
this kind of AKI is : urinary tract obstruction leading to increased intrabulular pressure and decreased GRF ( ex: tumors, kidney stones, neurogenic bladder)
Post renal