US Diets and MNT - Abali 3/15/16 Flashcards Preview

Gastrointestinal System > US Diets and MNT - Abali 3/15/16 > Flashcards

Flashcards in US Diets and MNT - Abali 3/15/16 Deck (22):
1

metabolic syndrome

 

aka

  • dysmetabolic syndrome
  • hypertriglyceridemic waist
  • insulin resistance syndrome
  • obesity syndrome
  • syndrome X

2

BMI ranges

healthy: 18.5-24.9

overweight: 25-29.9

obese: 30-39.9

extreme obesity: 40 or above

  • eligible for bariatric surgery

 

3

clinical ID of metabolic syndrome

waist circumference: M > 40; F > 35

TG: > 150

HDL chol: M < 40; F < 50

bp: > 135/85

fasting glucose: > 100

 

NEED 3 OR MORE OF THE ABOVE 

4

causes and risks of metabolic syndrome

causes:

abd obesity + insulin resistance, compounded by physical inactivity

 

risks: 2x CVD; 5x DM2 → 3x CVD

5

development of unhealthy fat stores

 

healthy vs dysfunctional adipose tissue

how your body processes positive energy balance is affected by:

  • smoking
  • unfavorable genotype
  • maladaptive response to stress

influences fat deposition!

 

healthy adipose tissuesubcutaneous obesity : no ectopic fat

dysfx adipose tissuevisceral obesity : ectopic fat

  • muscle fat (assoc with intracellular lipids)
  • epicardial fat
  • liver fat, fx

6

3 pieces of long-term weight loss puzzle

1. healthy diet

2. physical activity

3. behavioral modification

 

*diets/interventions that fail to target all three will prob yield only short term/temp effects

7

risks and benefits of Atkin's diet (low carb diet)

benefits

  • less hunger : higher protein content, more satiety
  • lower chol : decrease in LDL
  • rapid wt loss : ketosis (soluble molecule, lose some water wt) , utilization of stored fat

risks

  • lack of energy/weakness : ketosis, low stores of glycogen
  • high sat fat : 25% calories from sat fat
  • low fiber
  • ketogenesis
  • low vitamins

8

metabolic effects of low carb diets

reduction in circulating insulin, increase in circulating glucagon

  • favors gluconeogenesis

low carbohydrate intake → increased rate of FA mobilization → buildup of acetyl CoA → conversion to ketone bodies

  • favors ketogenesis, ketosis

brain will be counting on the a.a.s from your diet, not from muscle tissue you're breaking down

9

high carbohydrate diet (Ornish diet)

benefits

  • high fiber, low fat → lower chol, lower bp

risks/challenges

  • so little fat that you might run risk of lacking essential FAs
  • limits fish/nuts/olive oil which can protect against heart disease
  • difficult to maintain long term adherence!

10

fad diets

  • associated with short term effectiveness
    • after initial pd, results may fall off
  • nutrition plan isnt always safe/effective
    • may exclude/restrict healthy foods → deficiencies
  • could have long term effects on health, cause tissue wasting

11

Mediterranean diet

focus on whole foods, food patterns (versus macronutrients)

  • lifestyle : cooking (awareness of what you eat), eating together (slow consumption)
  • high fluid (incl alcohol)
  • small portions of meat/fish, high content of nuts/beans

12

maintaining weight loss

fad diets often result in weight cycling

key: maintain habits used to achieve loss after loss

new lower weight requires less calories to maintain

  • hard to change eating habits, cut down further
  • instead, 60-90 min moderate exercise can maintain

 

13

medical nutrition for diabetes

restrict carbs (< 130 g/day)

  • low glycemic index

increase fiber

  • improves glycemic reg

increase proteins (?)

  • protein ingestion lowers absorption of glucose → improves glycemic reg

  • high protein could have renal effects → bad

-alcohol in moderation (interferes with gluconeogenesis)

 

14

medical nutrition therapy for dyslipidema

avoid sat fat foods

reduce saturated FA

  • sat FAs → decreased production of LDL receptors
  • 1% increase in sat fat → 2% increase in LDL

increase PUFA (polyunsaturated FA) → lower LDL, lower CVD risk

  • omega 3 FAs (linolenic acid) > omega 6
  • fatty fish (SMASH - salmon, mackerel, albacore, sardines, herring) > supplement

increase MUFA (monounsaturated FA) → lowers LDL, TAGs without lowering HDL

  • olives, olive oil, nuts, bananas, dark choc

*degree of weight loss directly related to magnitude of decrease in TAGs

 

increase soluble fiber

  • bind/remove bile acids from body → force body to compensate by raiding its chol stores → increased LDL receptor synth → decrased circ LDL!
  • increase satiety

increase fruits/veggies/whole grains

  • each fruit, 4% decrease in CHD
  • rich in antioxidants (recommended; vitamins rich on antiox not recommended)

alcohol

  • increased HDL, decreased oxidation of LDL

 

15

hypertension

 

when to treat

contributions

over 60? > 160/90

under 60? >140/90

 

risk factors

  • obesity
  • high Na
  • low K, low Ca
  • excessive alcohol

nutrition therapy

  • weight loss, physical activity
  • lower Na
  • increase K, increase Ca
  • decrease alcohol

16

DASH plan for HTN

& other lifestyle changes to drop bp

Dietary Approaches to Stop HTN

  • lower sodium intake (tons of added salt in preservatives and processing)
  • lots of fruits and vegetables

lifestyle changes

  • weight reduction, physical activity
  • DASH diet, Na restriction
  • moderate alc consumption

17

chronic kidney disease

gradual, irreversible deterioration of kidney fx

causes

  • DM, 45%
  • HTN, 27%
  • inflamm/immuno/heretidaty disease that affect kidneys

end stage renal disease (ESRD) : requires either dialysis or kidney transplant to survive

 

consequences of kidney dysfx

  • Na retention → volume overload
  • hyperkalemia
  • metabolic acidosis
  • increase in PO4, decrease in Ca
  • anemia

18

medical nutrition therapy for chronic kidney diease (CKD)

lower protein intake → avoid uremic toxicity

need alt sources of energy 

  • complex and simple carbs + MUFA and PUFA
  • monitor for dyslipidemia!

adjust Na, K, Ca, P, vit D (bc you cant make active form without kidney fx)

supplement with folic acid and Fe (bc you cant get them through protein sources anymore)

19

CKD and...

sodium

potassium

in extremely low GFR, Na excretion drops

  • water retention
  • hypertension
  • edema

in diabetic nephropathy, K excretion drops

  • need to restrict K in diet in later stages of renal disease

20

recommendations for protein-uremic toxicity

CKD stages 1-3

drop protein intake to .75g/kg body weight

  • should pick "high bio value" proteins (complete, all essential a.a.s)
  • supplement deficiencies that pop up due to reduced protein intake [vitamin D!!!]

 

21

bariatric surgery

 

  • requirements
  • conseqs of malabs

need to be able to demonstrate ability to lose wt and maintain it

need to be morbidly obese

need to pass psych evals

 

more malabsorption → more weight loss, but also higher risk

  • less weight loss/risk : gastric banding, sleeve gastrectomy
  • medium weight loss/risk : roux en y gastric bypass
  • high weight loss/risk : biliopancreatic diversion with duodenal switch

22

post bariatric surgery diet, deficiencies

  • small portions, eat slow
  • no beverages during, up to 30min after eating
  • min 60g protein/day
  • no grazing, yes physical activity

 

  • many will experience dumping syndrome!

 

need nutrient/vitamin supplementation!

  • protein malabs : jejunum and mid ileum bypassed
  • iron deficiency → need low pH to reduce iron for absorption, have less stomach space to make this happen
  • Ca and vit D def  → leading to secondary hyperparathyroidism
  • deficiency in folic acid - low dietary intake
  • deficieny in soluble vitamins (bc we don't have good store besides folic acid and B12)
    • esp thiamine (measure thiamine or measure RBC transketolase activity)
  • vit B12 deficiency → R protein and IF swap happens in stomach → might not get good B12 abs