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Veterinary Pathology > VP7+8 > Flashcards

Flashcards in VP7+8 Deck (18)
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1

Hyperaemia

The active process of increasing the blood flow to a tissue during eg. inflammation.

2

Congestion

A passive process due to decreased outflow from a tissue, eg. secondary to a local obstruction (something tight around a limb)

3

Haemorrhage

Escape of blood from vessels

4

Petechial haemorrhage

Small haemorrage, 1-2mm diameter either due to a capillary rupture or clotting problem

5

Ecchymoses haemorrhage

A heamorrhar 1-3cm diameter, your regular bruise.

6

Suffusive haemorrhage

A heamorrhage that is covering a large area

7

Rhectic haemorrhage

A rupture causing a haemorrhage, eg a fracture-related haemorrhage

8

Haemorrhagic diathesis

Increased tendency for haemorrhages after minor injuries, eg due to clotting problems.

9

Haemostasis

Blood clotting, arrests bleeding from ruptured or damaged blood vessels. Either primary, secondary or tertiary.

10

Primary haemostasis

The platelet response (turning dendritic) to exposed collagen fibers and von Willerbands factor.

11

Secondary haemostasis

Activation of the coagulation cascade by activated platelets (releasing factor 12 to activate factor 11 = thrombin).

12

Tertiary haemostasis

Fibrinolysis, the breakdown of fibrin into plasmin to balance the reaction.

13

Thrombus

A formation of thrombocytes, fibrin and other blood elements in blood vessels. Will have a firm attachment to the blood vessel wall.

14

PM clot

Coagulation of the blood as a normal post mortem change, esp found in the ventricles of the heart. Smooth surface and easily removed.

15

Embolism

The breaking off of a tissue/aggregation that will travel in the blood stream to a distant part.

16

Ischaemia

Lack of blood flow

17

Infarction

Segmental or localised area of ischaemia (failure/reduction in blood flow).

18

Disseminated intravascular coagulation (DIC)

Widespread intravascular activation of the coagulation cascade by either (1) tissue factor release (2) thrombin activation or (3) extensive endothelial damage, leading to thrombosis, ischaemia and micro-infarctions.