Week 26 Flashcards

(41 cards)

1
Q

What are the key differences between oogenesis and spermatogenesis?

A
  • Oogenesis: One gamete formed per stem cell.
    Spermatogenesis: Four gametes per stem cell.

Oogenesis has interruptions; spermatogenesis is continuous.

Oogenesis occurs in the ovary; spermatogenesis in the testis.

Oogenesis starts before birth; spermatogenesis starts at puberty.

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2
Q

What are the stages of oogenesis?

A
  • Oogonium → Mitosis → Primary oocyte (2n).

Meiosis I begins before birth, arrested in prophase I.

Completes at puberty → Secondary oocyte (n) + polar body.

Meiosis II starts, arrested at metaphase II. If fertilised: Meiosis II completes → Ovum (n) + 2nd polar body.

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3
Q

What hormonal feedback mechanisms control oogenesis?

A
  • Hypothalamus releases GnRH → Stimulates anterior pituitary to release FSH and LH. FSH: follicle growth. LH: ovulation. Follicle releases oestrogen → negative feedback to hypothalamus. High oestrogen → positive feedback → LH surge → ovulation.
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4
Q

What structures are visible in a cross-section of the ovary?

A
  • Primary follicle (small, single layer). Developing follicle (multiple granulosa layers). Graafian follicle (fluid-filled cavity). Corpus luteum (yellow body). Corpus albicans (degenerate corpus luteum).
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5
Q

What is the process of spermatogenesis?

A
  • Occurs in seminiferous tubules. Spermatogonia (2n) → Mitosis → Primary spermatocytes (2n) → Meiosis I → Secondary spermatocytes (n) → Meiosis II → Spermatids (n) → Maturation → Spermatozoa (n).
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6
Q

Describe the early stages of spermatogenesis.

A
  • Spermatogonia undergo mitosis. Some remain stem cells; others become primary spermatocytes. Primary spermatocytes begin Meiosis I.
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7
Q

What happens after meiosis in spermatogenesis?

A
  • Secondary spermatocytes → Meiosis II → Spermatids. Spermatids → Spermiogenesis: lose cytoplasm, grow flagellum, become spermatozoa.
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8
Q

How is spermatogenesis hormonally regulated?

A
  • Hypothalamus releases GnRH → Anterior pituitary releases LH & FSH. LH → Leydig cells → Testosterone. FSH → Sertoli cells → Support spermatogenesis. Testosterone → Negative feedback to hypothalamus.
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9
Q

What structures are visible in a cross-section of the testis?

A
  • Seminiferous tubules (spermatogenesis). Sertoli cells (support sperm). Leydig cells (interstitial space, secrete testosterone).
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10
Q

What is the structure and function of the seminiferous tubules?

A
  • Lined with germinal epithelium.
    Basal layer: spermatogonia.
    Luminal layer: spermatids/spermatozoa.
    Sertoli cells support and nourish developing sperm.
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11
Q

What are the roles of Sertoli and Leydig cells?

A
  • Sertoli: Blood-testis barrier, nourish sperm, secrete inhibin (inhibits FSH).
    Leydig: Between tubules, produce testosterone in response to LH.
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12
Q

What are the key hormones involved in pregnancy?

A
  • hCG: Maintains corpus luteum.
    Progesterone: Maintains endometrial lining.
    Oestrogen: Stimulates uterine growth.
    Hormones produced by placenta after corpus luteum degenerates.
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13
Q

What are the two main types of reproduction in animals?

A
  • Asexual reproduction (offspring genetically identical to parent)
  • Sexual reproduction (genetic recombination between two individuals)
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14
Q

What are the costs of sexual reproduction?

A
  • Cost of meiosis: only 50% of genes passed on
  • Energy and time spent finding a mate
  • Increased risk of predation and disease
  • Risk of gametes not finding each other
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15
Q

What is the benefit of sexual reproduction?

A
  • Creates genetic variation, which enables natural selection
  • Improves adaptability and survival of species
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16
Q

What is August Weismann’s hypothesis about sex?

A
  • Sex increases variation in gene combinations
  • Allows selection to operate more efficiently
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17
Q

What does the Hardy-Weinberg principle state about genetic variation?

A
  • Genetic variation will be maintained in a population under certain conditions (no selection, mutation, migration, or drift)
18
Q

How does mimicry illustrate the importance of genetic variation?

A
  • Variability in phenotype helps individuals avoid predation by resembling toxic species
19
Q

How does the sickle cell anaemia example support sexual reproduction?

A
  • Heterozygotes have malaria resistance without disease
  • Maintains genetic variation in population
20
Q

What is the Red Queen hypothesis?

A
  • Constant coevolution with parasites drives the need for genetic variation from sex
21
Q

How do mud snails support the Red Queen hypothesis?

A
  • Populations with more males (sexual reproducers) are less infected by parasites
22
Q

Why is behaviour important in sexual reproduction?

A
  • Gametes must meet and timing must be correct
  • Behaviour helps attract mates and synchronise reproduction
23
Q

What behaviours help with successful sexual reproduction?

A
  • Courtship
  • Parental care
  • Nest building
  • Feeding and protecting offspring
24
Q

What are the different mating strategies in the animal kingdom?

A
  • Monogamy: 1 male, 1 female
  • Polygyny: 1 male, multiple females
  • Polyandry: 1 female, multiple males
  • Promiscuity: multiple partners
  • Polygynandry: multiple males & females
25
How do elephant seals demonstrate sexual selection?
- Males compete to control harems - Few males do most mating
26
How is mating behaviour different in bonobos?
- Use sex for bonding and conflict resolution - Promiscuous mating system
27
What is sperm competition?
- When multiple males mate with a female, their sperm compete to fertilise the eggs
28
What anatomical adaptations aid sperm competition?
- Larger testes - Penis morphology - Sperm plugs
29
How does sexual conflict drive the evolution of anatomy?
- Males evolve traits to increase fertilisation success - Females evolve resistance or choice mechanisms
30
What are the two primary methods of plant fertilisation?
- Abiotic: relies on physical forces (e.g., wind, water) - Biotic: involves animal pollinators
31
What are the sexual organs within angiosperms?
- Male: Stamen (Anther produces pollen) - Female: Carpel (Ovary contains ovules, becomes fruit after fertilisation)
32
What else can be found in angiosperm flowers besides sexual organs?
- Corolla (petals): attract pollinators - Sepals: protect flower in bud form
33
What is biotic pollination?
- Pollination involving animals (e.g., insects, birds) - Animals are attracted by color, shape, scent
34
What are variations of flowers in terms of reproductive structure?
- Hermaphrodite: both male and female organs in same flower - Monoecious: separate male and female flowers on same plant - Dioecious: male and female flowers on different plants
35
What is the risk of self-fertilisation?
- Reduces genetic variation - Increases risk of deleterious allele expression - Many species have evolved to avoid it
36
What are dichogamous flowers?
- Flowers where male and female organs mature at different times - Prevents self-pollination
37
What is self-incompatibility (SI)?
- Genetic mechanism preventing self-pollen from fertilising the ovule - Maintains genetic diversity
38
What is the SI system in Brassica species?
- Uses the S-locus with SCR (S-locus Cysteine-Rich protein) and SRK (S-locus Receptor Kinase) - SCR is produced in pollen, SRK in stigma - Matching alleles inhibit pollen germination
39
How does self-compatibility differ from SI?
- SC plants allow self-pollen to fertilise - Advantageous in absence of pollinators
40
What are the benefits of self-compatible crops?
- More consistent yields - Don’t require pollinators - Can be easily bred for uniformity
41
What are the negatives of self-compatible crops?
- Lower genetic diversity - Increased vulnerability to disease and environmental changes