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Intro to Perio 511 > Week 3 > Flashcards

Week 3 Flashcards

1
Q

Steps in colonization of the oral cavity starting with birth?

A

Day 1- facultative and aerobic bacteria from birth

Day 2- anaerobic bacteria found

Day 14- mature microbiota established in gut

2 years- whole microbiota formed 10^14 total

Tooth eruption = more complex flora

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2
Q

Is the oral microbiota harmful?

A

No most are commensal and or beneficial

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3
Q

How much bacteria do we have in and on our bodies?

A

~ 2 kg

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4
Q

How many different types of bacteria are found in the mouth?

A

Over 600

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5
Q

When infection with a microbe happens what are the choices and how much damage happens over time?

A

Commensalism- no damage over time unless it gets out of control and leads to disease- in which case it damage amount goes up over time.

Colonization- damage goes up over time and can lead to clearing the infection, disease, or persistence.

Both colonization and disease can lead to persistence where damage level is constant and high over time

Disease has two outcomes: death or eradication by immune or tx

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6
Q

Name the 6 major ecosystems in the mouth?

A
  1. Keratinized intraoral supragingival tissue and hard surfaces
  2. Perio pocket (including around implants)
  3. Non-K tissues- buccal, palate and floor of mouth
  4. Dorsum of tongue
  5. Tonsils
  6. Saliva

3.

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7
Q

Describe plaque and the locations it is found?

A

Structured, layered, yellow grey,

Found on hard surfaces of teeth and restorations both above and below gums

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8
Q

What is materia alba?

A

White cheesecake like substance that is soft and easily sprayed off with water made from salivary proteins, bacteria, dead epithelial cells and some occasional food debris.

No organizational structure

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9
Q

What is plaque made of?

A

Bacteria in a matrix of salivary glycoproteins and extra cellular polysaccharides

It is a biofilm

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10
Q

Can plaque be removed by sprays or rinsing?

A

Nope impossible needs physical scraping

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11
Q

What is dental pellicle?

A

A substance that coats every surface of the mouth derived from saliva made of glycoproteins, proline rich proteins, phosphoproteins, histidine rich proteins and enzymes

Aka proteins that are a mix of glyco, phospho, histidine and proline combined with enzymes

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12
Q

How does dental pellicle adhere to teeth?

A

Electrostatic, Vanderwalls and hydrophobic forces

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13
Q

How fast does pellicle form after cleaning teeth?

A

Within nanoseconds

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14
Q

3 major phases of plaque formation?

A
  1. Pellicle forms
  2. Initial adhesion and attachment of bacteria
  3. Colonization and plaque maturation
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15
Q

Three phases of bacterial attachment?

A
  1. Random contact of bacteria with hard surface aka transport of bacteria to the tooth surface
  2. Initial reversible adhesion
  3. Attachment with a firm anchorage by reactions between bacterial adhesins and pellicle receptors

After Step 3 it needs to be scraped off aka physically removed

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16
Q

Which phases of bacterial attachment are non specific?

A

Phase 1 and 2 are random and phase 3 depends on specific interactions between bacterial surface proteins (adhesins) and pellicle receptors

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17
Q

Why are teeth so good at accumulating plaque?

A

Hard non-shedding surface to attach to

A part of our ectoderm that doesn’t shed providing a home and “port of entry” for perio pathogens

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18
Q

When full mouth extractions are done does the type of bacteria in the mouth stay the same?

A

No key perio pathogens disappear

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19
Q

Describe the specific location of plaque growth on teeth?

A

Starts at gingival margin and interdental spaces and grows toward the corona

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20
Q

What is Supragingival plaque?

A

Plaque located outside of the perio pocket that starts at the gingival margin (touch’s the margin)

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21
Q

What type of bacteria makes up the first layer (touching the tooth) of supragingival plaque?

A

Gram positive cocci and short rods

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22
Q

What type of bacteria makes up the outer layer (surface) of supragingival plaque?

A

Gram negative rods and filaments, spirochetes

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23
Q

What is a major factor is causing plaque to stick to the surface of teeth?

A

The roughness of the surface.

Fine polishing paste or glycine powder for air polishing

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24
Q

What does thicker plaque contain and is it more or less pathogenic?

A

More pathogenic

Contains more motile bacteria like spirochetes and is packed denser so more organisms

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25
Q

What areas of the mouth does plaque form the fastest?

A

Lower, interproximal, buccal areas of molars…

IP is faster than straight buccals and lower molars traps food easier

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26
Q

Does plaque form at the same rate on people?

A

Nope varies a lot

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27
Q

What can explain 90% of the variations in the rate of plaque formation?

A

Salivary induced aggregation and relative flow

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28
Q

Does more or less saliva cause plaque to form faster?

A

More saliva= faster

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29
Q

Other than amount of saliva what are some other factors in plaque formation?

A 
Diet,
Chewing fibrous foods,
Smoking,
Brushing tongue and palate,
Antimicrobial factors in saliva,
Inflamed gingiva
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30
Q

Does age influence plaque formation?

A

No

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31
Q

Does plaque in older or younger people leave to more gingivitis?

A

Older

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32
Q

Does eating remove plaque?

A

Nope

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33
Q

What makes sub gingival plaque different?

A

Different environment,

Availability of blood products and an anaerobic environment

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34
Q

Do strict anaerobes contribute to initiation of periodontal disease?

A

Most likely very little but it is under debate

Not sure yet what exactly causes the consequences of the disease

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35
Q

Can sub gingival plaque be removed completely?

A

No it is really hard to do so

What is left allows for recolonization

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36
Q

Can some sub gingival plaque pathogens penetrate soft tissue and dentin?

A

Yes they can

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37
Q

How fast does sub gingival plaque recolonize after a cleaning?

A

7 days it is back to pretreatment levels

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38
Q

How does tooth associated sub gingival plaque versus tissue associated sub gingival plaque vary throughout the pocket?

A

Tooth associated subgingival plaque is more like Supragingival plaque than tissue associated.

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39
Q

What bacteria does TISSUE associated sub gingival plaque contain?

A

Mostly- gram negative rods and cocci

Others- filaments, flagellated rods and spirochetes

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40
Q

Do plaque colonies share resources?

A

Yes there are fluid channels that nutrients run though

Plus they communicate with each other via quorum sensing

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41
Q

What binds lower layers of plaque together?

A

Polysaccharide matrix with both organic and inorganic materials

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42
Q

Can perio and cariogenic pathogenic bacteria be transmissible to other people?

A

Yes both are transmissible- most common is vertical transmission in families.

Familial horizontal is less common

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43
Q

Can pathogenic bacteria be translocated from one place in the mouth to another?

A

Yes is can be spread with things like proxa brushes

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44
Q

List non bacterial organisms that live in the mouth too?

A

Viruses,
Fungi/yeast,
Protozoa,
Archaea

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45
Q

3 plaque hypotheses that have been discarded by now?

A
  1. Non-specific plaque hypo- control plaque and you control the disease
  2. Specific plaque hypo- only certain plaque is bad and if you have it then you get the disease
    They still didn’t know if it was causal or correlation
  3. Ecological plaque hypo- little deeper both plaque levels and specific bacteria along with the disease along with some host factors
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46
Q

What is the current accepted hypothesis on perio and plaque?

A

Keystone Pathogen Hypothesis

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47
Q

What does the Keystone Pathogen Hypothesis say?

A

A specific pathogen P. gingivalis is present in low numbers but it can disrupt the perio microbiota leading to dysbiosis.

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48
Q

What type of bacteria is associated with health?

A

Mostly Facultative- Gram positive rods and cocci

With a few anaerobic both pos and neg rods along with positive cocci

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49
Q

What type of bacteria is associated with gingivitis?

A

Main point- The levels of anaerobic bacteria gram negative rods and facultative gram negative rods increases
There are gram positive rods and cocci around along with negative cocci

Extra details-
Levels of facultative gram + rods and cocci decrease

Small amounts of anaerobic + cocci turn into - cocci

Anaerobic + rods decrease

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50
Q

What type of bacteria is associated with periodontitis?

A

Anaerobic gram negative rods 75%
Anaerobic gram positive rods ~12%
Spirochetes as well now

The rest are facultative gram positive rods and cocci

Take away- during gingivitis anaerobic gram neg rods increase in numbers along with facultative gram neg rods then the anaerobic neg rod take over in Periodontitis

90% of the bacteria present in Periodontitis is anaerobic

Gram positive rods and cocci = good

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51
Q

What does the rate of plaque growth on unbrushed teeth look like?

A

Exponential at first
First 24 hrs slow,
Next 3 days rapid

After 4 days it slows and shifts to anaerobic and gram negative

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52
Q

How much of the bacteria present is periodontitis is anaerobic

A

Most approximately 90%

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53
Q

List common bacteria associated with gingivitis names?

Don’t have to know this yet but made this card to start to get familiar with them.

A

Gram +: streptoccus, actinomyces, eubacterium and parvimonas

Gram - : capnocytophaga, fusobacterium, prevotella, campylobacter gracilis, C. consisus,B. parbula, E. corrodens

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54
Q

List common bacteria associated with periodontitis names?

Don’t have to know this yet but made this card to start to get familiar with them.

A 
P. gingivalis, 
T. forsythia, 
P. intermedia, 
C. rectus, 
E. corrodens, 
F. nucleatum, 
A. actinomycetemcomitans, 
T. denticola
55
Q

Which bacteria is associated with early (age) localized aggressive bone loss?

A

A. actinomycetemcomitans

A.a. May make up 90% of bacteria found in pockets

56
Q

Generalized aggressive periodontitis is dominated by what kinds of bacteria?

A

GIFT AA or A GIFT

P. gingivalis,
P. intermedia,
T. forsythia,
treponema spp.,
And less A. actinomycetemcomitans
57
Q

Necrotizing perio disease is associated with what systemic disease?

A

HIV

58
Q

In necrotizing periodontitis what types of bacteria are associated?

A

P. intermedia and spirochetes

59
Q

What is the tx for necrotizing perio disease?

A

Debridement,
OHI,
Mouth rinse and pain meds,

If no response to the above then-
Antibiotics

60
Q

When do perio abscesses occur?

A

Untreated perio or sometimes right after srp usually

Foreign matter gets stuck under gum tissue- popcorn kernels

61
Q

Symptoms of a perio abscess?

A

Pain, swelling, suppurations(puss), BOP and mobility

62
Q

What is the tx of a perio abscess?

A

Clean it out

63
Q

Bacteria associated with a perio abscess?

A

F. nucleatum, P. intermedia, P. gingivalis, P. micros, T. forsythia

64
Q

When perio is a manifestation of a systemic disease is it specific bacteria or due to immune response issues?

A

Immune response defects

65
Q

When perio is a manifestation of a systemic disease what are some potential immune response issues?

A

Neutrophils defects,

Leukocytes adhesion defects

Severe destruction may be associated w/ mutation of the Cathepsin C receptor

66
Q

5 benefits of good bacteria?

A
  1. Passively occupy a niche
  2. Limit bad or pathogenic bacteria’s ability to adhere to tissue surfaces
  3. Adversely affect growth or vitality of pathogen
  4. Affect the ability of pathogen to produce virulence factors
  5. They degrade pathogenic virulence factor
67
Q

Drawbacks to microbial testing?

A

Presence of Pathogens doesn’t = disease

Pathogens detected in healthy people

Quality of host response isn’t looked at

68
Q

Are biofilms resistant to antibiotics?

Why or why not? How much more o less?

A

Yes 1000-1500 times more resistant

Antibiotics can’t reach inner film easily meanwhile outlayer is passing along mutations for for resistance, differences in growth rates, nutrition and pH can all affect the effectiveness of antibiotics

69
Q

What is calculus and where does it form?

A

Mineralized plaque on hard surfaces of natural teeth both sub- and supra-gingival and prosthetics

70
Q

How common is calculus?

A
  1. 9 % has supragingival
  2. 1 % has subgingival

Probably more

71
Q

What % of calculus is inorganic and what is the composition of the inorganic part?

A

70-90%
76% calcium phosphate, 3% calcium carbonate, 4% magnesium phosphate and other metals.

Another way to break it down is 2/3rds of the inorganic are crystalline: 
Hydroxyapatite,
Magnesium whitlockite,
Octocalcium phosphate
Brushite
72
Q

Does calculus have more or less inorganic mass than bone, enamel and dentin?

A 
More than bone and less than enamel….
In order most to least-
1. Enamel 96%
2. Calculus 70-90%
3. Bone 60-70%
4. Dentin 45%
73
Q

How does calculus form?

A

By precipitation of mineral salts on the the inner layer of plaque in layers and subsequent calcification

74
Q

When does calculus formation and calcification happen?

A

Precipitation for mineralization starts between 1 and 14 days of plaque formation and calcification/initial hardening can start in as little as 4-8 hours

75
Q

Why does calculus form?

A

Local rise in saturation of calcium and phosphate ions

Rise in pH causes precipitation,

when pH rises colloidal proteins settle out of saliva leading to precipitation

76
Q

Different out comes in a mouth based on high or low pH?

A

Acid is caries and basic is calculus

77
Q

What are the 4 methods of calculus attachment to teeth?

A
  1. Attaches to pellicle-no need bacteria
  2. Mechanical- locks in to surface irregularities
  3. Close adaptation of calculus under surface depressions of cementum
  4. Calculus bacteria penetrate into cementum
78
Q

Basics of supragingival calculus location, make up, color, timing?

A

Gingival margins near salivary ducts
Minerals from saliva
Whitish yellow
Forms in less than 24 hrs

Moderately hard but removable
Dry to see

79
Q

Mineral source for sub gingival calculus ?

A

Gingival crevicular fluid and inflammatory filtrate

80
Q

Location of sub gingival calculus?

A

All over but most common in interproximal areas

81
Q

Color of sub gingival calculus?

A

Brown or black

82
Q

How hard is subgingival calculus?

A

Dense, hard and tenacious

83
Q

How fast does subgingival calculus form?

A

Slower that supra

84
Q

Can we distinguish between the effects of plaque and calculus?

A

It is difficult- calculus is always covered in plaque

85
Q

Is calculus correlated with gingivitis?

A

Yes

86
Q

Cornerstone of periodontal therapy?

A

Removal of plaque and calculus

87
Q

4 perio risk categories?

A
  1. Risk Factors
  2. Risk Determinants/background characteristics
  3. Risk Indicators
  4. Risk Markers or Predictors
88
Q

3 Risk factors?

A
  1. Tobacco smoking,
  2. Diabetes,
  3. Pathogenic bacteria and microbial tooth deposits
89
Q

5 Risk Determinants/ Background Characteristics?

A
  1. Genetic factors,
  2. Age,
  3. Gender,
  4. Socioeconomic Status,
  5. Stress
90
Q

3 Risk Indicators?

A
  1. HIV/ AIDS
  2. Osteoporosis
  3. Infrequent Dental Visits
91
Q

2 Risk Markers or Predictors?

A
  1. Previous history of perio disease

2. Bleeding on probing

92
Q

What is risk assessment?

A

Probability that a person will develop a disease in a given time period

93
Q

Basics of what makes a risk factor?

A

Environmental, behavioral or biological

Identified by longitudinal studies

Exposure at single point, multiple or continuous prior to onset of disease

Intervention helps

94
Q

What percent of perio cases are smokers?

A

42% current and 11% past smokers

Current smokers are 3x more likely to have perio disease than non

95
Q

Does smoke more increase the risk?

A

Yes heavy smokers have higher rates of severe perio

96
Q

Does risk decrease with years since a smoker quit?

A

Yes

97
Q

Are the negative effects of smoking on gingivitis reversible?

A

Yes it can be cleared up perio on the other hand can be treated and stopped but not actually reversed

98
Q

Do former smokers respond well to perio therapy?

A

Yes they respond similar to non-smokers

99
Q

Effects of smoking on the microbiology of the mouth in regards to periodontitis?

A

Increases complexity and negatively effect periodontitis associated pathogen colonization both sub and supragingival

100
Q

What does smoking do to oral immune and inflammatory response? 4 things

A
  1. Alters neutrophil chemotaxis, phagocytosis and oxidative burst
  2. Increases tumor necrosis factor alpha and prostaglandin E2 in GCF (gingival crevicular fluid)
  3. Increases collagenase and elastase in GCF
  4. Increases prostaglandin E2 by monocytes in response to lipopolysaccharide
101
Q

What does smoking do to a persons physiology ? 4 things

A

Masks some key indicators of gingivitis and periodontal disease

  1. Decreases gingival blood vessels but increases inflammation
  2. Decreases GCF flow and bleeding but increases inflammation
  3. Decreases subgingival temperature
  4. Increases time need to recover from anesthesia
102
Q

What are the 5 A’s to use with patients who smoke?

A
  1. Ask if they smoke
  2. Advise of the association between oral disease and smoking
  3. Assess their interest in quitting
  4. Assist them in quitting
  5. Arrange a referral or follow up care
103
Q

What is diabetes?

A

A metabolic disease characterized by chronic hyperglycemia with a direct relationship to periodontal disease

104
Q

Is there a difference in perio risk between type I and type II?

A

Nope

105
Q

Where does perio disease rank in regards to common complications of diabetes?

A

6th most common complication

106
Q

4 things that happen in poorly controlled diabetes?

A
  1. Altered immune function in PMNs
  2. Qualitative changes in bacteria
  3. Altered collagen structure and function
  4. All this leads to severe gingival inflammation, deep pockets, rapid bone loss and perio abscesses
107
Q

3 perio rate differences between diabetics and non-diabetics?

A

Type 1 teenagers = 5x

Poorly control adults = 2.9x

Smoker and poorly controlled = 4.6x

108
Q

Do uncontrolled diabetics respond well to periodontal therapy?

A

No they have a poor response compared to both controlled and non diabetics

109
Q

Does clinical attachment loss increase with age in diabetics?

A

Yes

110
Q

What is important composition or quantity of plaque regarding it as a risk factor for perio disease?

A

Composition

111
Q

Name 3 bacteria commonly associated with perio disease?

A

A. actinomycetemcomitans
P. gingivalis
T. forsythia

112
Q

7 anatomical factors that harbor plaque?

A
  1. Furcations
  2. Root concavities
  3. Grooves
  4. Cervical enamel projections
  5. Enamel pearls
  6. Overhanging margins or restorations
  7. Calculus
113
Q

What are Risk Determinants/Background Characteristics ie general definition?

A

Risk factors that is non modifiable aka can’t change

114
Q

Name 5 Risk Determinants?

A
  1. Genetic factors,
  2. Age,
  3. Gender,
  4. Socioeconomic Status,
  5. Stress
115
Q

Have studies shown a link to perio disease in genetics?

A

Yes twin studies and Sri Lanken Tea worker

116
Q

What genetic factors have shown to be linked to severe perio?

A

Alterations in IL-1 genes

Alterations in neutrophils

and monocytic hyper responsiveness associated with severe perio

117
Q

How does age affect perio?

A

Increase prevalence with age most likely due to an increase in exposure to other risks and cumulative destruction

118
Q

Who has higher rates of perio disease- men or women and why?

A

Men most likely not as good preventative practices

119
Q

How is socioeconomic status linked to perio?

A

Lower has higher rates of perio

Maybe less education, lack of dental visits, lack of hygiene since other things like staying alive matter more

120
Q

How is stress linked to perio?

A

Higher stress decrease immune function

Periodontal disease has been shown to occur at higher rates during periods of high stress

121
Q

General definition of Risk Indicators?

A

Probable risk factors identified in cross-sectional not longitudinal studies

122
Q

Name 3 Risk Indicators?

A
  1. HIV/AIDS
  2. Osteoporosis
  3. Infrequent dental visits
123
Q

What is a prominent diagnostic feature of HIV associated perio disease?

A

Oral lesions

124
Q

7 oral manifestations of HIV?

A
  1. Oral Candidiasis
  2. Linear gingival erythema
  3. Oral hairy leukoplakia
  4. Kaposi sarcoma and other malignancies
  5. Acute necrotizing ulcerative gingivitis
  6. Necrotizing ulcerative gingivitis and periodontitis
  7. Chronic periodontitis
125
Q

How is osteoporosis linked to periodontal disease as a Risk Indicator?

A

Doesn’t initiate it
Studies are conflicting
But bone density loss may aggravate perio progression

126
Q

Do studies agree on Infrequent Dental Visits as a Risk Indicator?

A

No some say yes and some no

127
Q

What is the general definition of what defines a Risk Marker or Predictor?

A

They are things associated with perio disease but do not cause it

128
Q

Name 2 Risk Markers?

A

Previous perio disease predicts future unless well controlled

Bleeding or absence of bleeding on probing

But careful of probing force and bleeding- hard probing can cause bleeding in healthy people

129
Q

Is bleeding on probing a good predictor for perio disease or absence of it a good predictor for perio health?

A

Continuous absence of BOP is a reliable 98% predictor for maintenance of perio health

Positive prediction of disease was only 6%

ie people bleed at times for many other reasons than perio and that doesn’t mean perio disease but not bleeding does mean you are healthy

130
Q

Pathway of clinical assessment?

A
  1. Medical History, dental history, physical perio exam
  2. Identify Risks from the 4 categories
  3. Make tx decisions
  4. Treat and assess response
  5. Then either maintain or reassess risks and start again
131
Q

What should always be a part of any risk assessment and intervention/treatment strategies?

A

Patient Education!

132
Q

Does plaque form faster next to inflamed or healthy gingiva?

A

Inflamed

133
Q

On a general level how do plaque bacteria vary in the perio pocket by depth?

A

Deeper part of pocket is less filamentous,

The apical portion is dominated by smaller bacteria in no particular orientation

Intro to Perio 511 (5 decks)

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