Week 3.10 Ataxia Flashcards

(32 cards)

1
Q

what does an ataxic gait look like

A

like you are drunk!

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2
Q

damage to the ____ causes ataxia

A

cerebellum

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3
Q

the cerebellum is responsible for

A

coordinating movement

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4
Q

what does the cerebellum compare

A

the intended output and actual movement

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5
Q

what does the cerebellum do in preparation of movement

A

it makes predictive and anticipatory modifications

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6
Q

the cerebellum has a huge role in motor…

A

learning

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7
Q

TF: the cerebellum learns, memorizes and stores motor programs

A

true

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8
Q

what is so important about the role of adaptation in the cerebellum

A

the cerebellum must adapt to the changing environment, and the speed of the changing environment.

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9
Q

TF the cerebellum controls balance and equilibrium

A

true

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10
Q

TF: the cerebellum does not control motor tone

A

false, it does

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11
Q

the cerebellum makes sure movement has what 4 things

A

direction
extent
force and timing

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12
Q

how does the cerebellum affect speech and eye movements

A

responsible for all of those !

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13
Q

what are some signs and symptoms of cerebellum infarct, and what side of the lesion

A

nausea, vomit, ataxia, nystagmus, headache all ipsilateral to the lesion

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14
Q

what happens in a lateral lesion and a vermin/flocculonodule lesion

A

lateral: limb ataxia

vermis and flocculonodule: more midline, so the drunk gait.

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15
Q

name some things that result from cerebellar dysfunction

A
disorganized movements 
impaired balance and postural control  hypotonicity 
dysmetria 
decomposition 
ataxia 
dysdiadochokinesia 
tremor 
asthenia 
dysarthria 
occular dysmetria
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16
Q

what are some acquired mechanisms of cerebellar damage

A

stroke, tumor, structural changes, toxicity (alcohol drugs), immune mediated (MS, Gluten), trauma, infection (cerebellitis) or endocrine (hypothyroidism)

17
Q

what are some degenerative non-hereditary causes of cerebellar damage

A

MSA

idiopathic late onset cerebellar ataxia.

18
Q

what are some hereditary causes of cerebellar damage

A
autosomal dominant disorders (like episodic of spinocerebellar ataxia's)
autosomal recessive (Friedrich, early onset cerebellar)
X-linked disorders (mitochondrial, fragile X associated tremors)
19
Q

define/describe ataxia

A

without order, incoordination, slurred speech, stumbling, falling, incoordination, trouble eating and swallowing, eye movement abnormalities, tremors, cardiac issues

20
Q

what are 3 traditional function and activity outcome measures

A

FIM
Balance/postural control measures
gait speed

21
Q

what are 2 ataxia specific outcome measures

A

International cooperative ataxia rating scale (ICARS)

Scale for the assessment and rating of ataxia (SARA)

22
Q

TF: there is a lot of evidence for treatment of ataxia

23
Q

what do we treat in patients with ataxia

24
Q

what kinds of things can we treat in patients

A

postural stability
gait
balance
accuracy of limb movements

25
do we do an intensive long or short term motor training
long term
26
what should the HEP focus on
static and dynamic balance activities like sitting standing and walking
27
TF: there is a lot of evidence for BW supported treadmill training
false, not much evidence
28
can we use biofeedback in patients with ataxia
yes, like a mirror, EMG, or do it in a small space so they can bump into things
29
do we want to increase or decreases DOF, and how
decrease, by adding an AD, resistance, kneeling, 1/2 kneeling
30
activity should focus on what 3 things
stability co-contraction midrange control
31
can we use resisted movements
yes
32
what are some compensatory strategies
``` slow it down , reduce the number of moving segments widen BOS minimal distractions weighting axial and limb AD orthotics (decrease DOF) ```