Word Reading and Dyslexia Flashcards

1
Q

The word identification problem

A

A typical reader can identify 2-3 words/second.
• Even at presentations of 10 words/second,
meanings are activated.
• Identification takes in the region of 200ms./word
• Once you’ve learnt to read, you can’t stop yourself
– E.g. the Stroop effect.

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2
Q

How do we recognise words?

A
  1. Direct access hypothesis: visual pattern
    recognition.
    • We can go straight from visual processing to meaning
    without sounding out words, otherwise we would have
    trouble with:
  2. Indirect access/phonologically-mediated
    hypothesis: translation from print to sound.
    • we can also sound out letter strings, as we can
    read non-words, like tegwop, burd.
  3. Dual-route hypothesis: either of the above
    routes.
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3
Q

How is word recognition assessed?

A

Naming aloud or lexical decision. lexical decision task, subjects have to
respond “yes” rapidly when the see a word, “no”
when they see a non-word.

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4
Q

What makes a word easy/hard to read?

A
  1. Spelling-to-sound Regularity.In general, irregular words take longer to read aloud (name)
  2. Frequency (how often a word occurs)
  3. Semantic Priming (context)
    • Single Words
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5
Q

The Logogen Model

first incarnation, Morton, 1969

A

An example of a Parallel Search Model. Essential features:
• Every word we know has its own simple feature counter
called a logogen corresponding to it.
• Information sent in parallel to all the word detectors
(logogens) in the lexicon.
• The more information that is consistent with a word the
greater the activation of the logogen.
• A logogen accumulates evidence (more activated) until its
threshold level is reached, then the word is recognized.
• More frequent words have lower thresholds.
• Recently read words/associates have lower thresholds

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6
Q

Morton’s (1969) Logogen model

A

Logogen model: some issues:
• The simple logogen model could not explain
effects of ‘higher level’ information on ‘lower
level’ processing (classic example: sentence
context effects).
• The logogen model attempts to explain how we
recognise words, but has nothing to say about how
we read words or non-words aloud.
• Nonwords can sometimes be rejected very rapidly

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7
Q

How many routes to reading aloud?

A

Dual Route Model: driven by the idea that there are
two aspects to reading: rules and memory.
• One route needed for reading irregular words (e.g.
yacht, pint). The lexical route
• One route needed for reading non-words (e.g. blik).
The sub-lexical route.

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8
Q

Evidence for two routes

A

Pronunciation of nonwords cannot be done by the Lexical
route (no lexical entries for them)
• Pronunciation of exception words cannot be done by Sublexical
route (e.g. aisle)
• Two different variables: Word frequency affects the lexical
route; regularity (consistency) affects the sub-lexical route.

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9
Q

Neuropsychological Evidence for Dual

Routes: Dyslexia

A

Dyslexia: An impaired ability to read, which can’t
be attributed to more general factors such as visual
impairment, low intelligence, motivation.
• Acquired dyslexics: people who developed
normal reading ability but have lost the ability to
read properly, usually as a result of brain injury.
• Developmental dyslexics: people in whom the
ability to read has never properly developed.

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10
Q

The major acquired dyslexias

A

Surface dyslexia (e.g. Marshall & Newcombe, 1973, patient JC)
– Impaired ability to read irregular words (e.g.
have, pint, colonel)
– Regularisations (e.g. “kol-o-nell” for colonel)
• Phonological dyslexia
– Impaired nonword reading
– Relatively preserved ability to read words

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11
Q

Dual route theory and dyslexia

A

The dual-route model accounts for:
• Surface dyslexia by assuming that it is caused by
damage to the lexical route (the intact sub-lexical
route can deal with regular words and non-words).
• Phonological dyslexia by assuming that it
represents damage to the sub-lexical route (the
intact lexical route can deal with words).

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