Wound Care Flashcards

(55 cards)

1
Q

contraindications to e stim over a wound

A

cancerous lesion
osteomyelitis
location of wound over contraindicated area
sensation
cognition

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2
Q

current of injury

A

injured cells have an endogenous electrical current
this current circles around a wound bed’s edge as the wound causes electrical “leaking”
greater potential at greater distances from the wound center

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3
Q

exogenous current’s effect on wounds:

A

neural tissue: pain relief and healing
wound epidermis
migratory mesenchyme cells: vimentin protein marker on fibroblasts - these cells can create epithelial tissue by organizing into polarized sheets, which can organize to close the wound
undifferentiated cells come to the wounfd and turn into collagen to scar down

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4
Q

layers of the epidermis - where will cells migrate in electrical field?

A

upper layers - cells migrate to anode +
lower layers - cells migrate to cathode -, including monocytes, fibroblasts, macrophages

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5
Q

current shown to affect cell migration

A

50-150 mV/mm

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6
Q

desoluabolization

A

liquefied tissue that is necrotic can be easily removed with estim using negative current on the cathode

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7
Q

stim currents used for improved blood flow and reduced inflammation

what specific effects does it have?

A

HVPC
increases blood flow at contraction levels
retard inflammation

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8
Q

antibacterial effects of extim

A

cathode delivery -
gram negative and positive
low intensity
HVPC current
80 pps, 2 hrs, 4x week

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9
Q

which pole is for what healing effect?

A

cathode for bacteria retarding
anode for healing and epithelial growth

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10
Q

protocol with HVPC for wound healing

A

start w negative from cathode for 4 weeks
negative 3-7 then change to positive

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11
Q

acute inflammation phase

A

hemorrhage, necrosis, erthemia, edema, exudate, red granulation forming

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12
Q

how to resolve pt in chronic inflammation phase

A

move back into acute inflammation phase so pt can progress into proliferation phase

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13
Q

acute proliferation phase

A

inflammation, wound starts reducing in size, red granulation tissue present, serous/serosanguineous exudate, may be odor, wound edges start to adhere and progress to epithelialization

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14
Q

signs of chronic proliferation phase

A

hyper granulation, tissue growing out of wound bed as this is not effective at closing wound bed
pink granulation tissue

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15
Q

causes of chronic proliferation phase

A

infection changing granulation tissue

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16
Q

acute epithelialization phase

A

expected outcome is to resurface wound

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17
Q

chronic epithelialization phase

A

rolled wound edges, fibrotic
could be caused by drying out, poor dressing choices

one cause?

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18
Q

pt can be vulnerable to pressure ulcers on the sacrum if:

A

poor hygiene, B&B dysfx
poor pressure relief adherence
atrophy or scarred muscle not providing normal cushioning and blood supply
seated all day

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19
Q

remodeling phase

A

should result in immature scar formation if optimal healing
helped with stimulation of migrating epidermal cells
happens after wound is closed
lasts 6 mo-2 years

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20
Q

granulation tissue

A

red beefy tissue
+ sign of proliferation phase

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21
Q

phases of healing

A

Acute inflammatory
acute proliferation
acute epithelialization (part of proliferation)
remodeling
any of these phases can turn chronic if healing is interfered with

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22
Q

exudate

A

liquid drainage from wound bed, not clear

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23
Q

transudate

A

liquid drainage from wound, clear and normal

24
Q

serosanguinous fluid

A

bloody/clear/pink fluid exudate

25
maceration
surrounding tissue of wound is water logged risk for pressure ulcer/wound complications
26
rolling edges
build up of epithelial tissue if it is impeded from moving forward on the wound bed tissue rolls under itself as a nodule at the wound edge
27
tunneling
bottom of the wound bed breaks and wound progresses deeper into tissue important to visualize as it can become infected and reopen a closed wound
28
undermining
space under superficial wound edge you can stick a probe under and lift up bc edge is not adhered down
29
eschar
dark heavy scabbing comes off in chunks/pieces mainly dead tissue
30
slough
heavy, stringy fibrotic tissue can be debrided
31
ways to measure wounds
tracing tape measure depth
32
measure wound: tracing
circle with radius trace wound shape and compare size to circle
33
measure wound: tape measure
sterile, disposable, measure greatest vertical and horizontal lines to get area
34
measure wounds: depth
place qtip in wound bed and mark level even with edges measure tunneling by going clockwise around wound bed to check
35
pressure ulcer: stage 1
redness non blanchable
36
pressure ulcer: stage 2
skin breakdown through epidermis only
37
pressure ulcer: stage 3
dermal layer into fascial layer affected
38
pressure ulcer: stage 4
bone and muscle impaired
39
venous ulcer cause
PVD failure of veins to return blood to heart due to valvular dysfunction causing distal fluid accumulation
40
characteristics of venous ulcers
large >10 cm irregular edge saucer shaped swollen leg dry/itchy skin painless unless elevated red/brown coloring can be infected
41
arterial insufficiency wounds: cause
hardening/narrowing of arteries compromising blood supply to legs triggered by smoking, high BP, RA, DM, CV disease
42
arterial insufficiency wounds characteristics
cold LE white/blue/shiny appearance hard distinct edges/punched out
43
alginates
ABSORB lots of exudate by forming gel and maintaining optimal moisture levels used for infected or healthy wounds
44
antimicrobial dressing
dressing with topical antiseptics silver as antiseptic
45
collagen dressing
encourage healing, maintain moist environment, promote granulation tissue
46
contact layer dressing
thin non adherent layer placed over a wound to protect fragile tissue from other dressings or topicals
47
compression dressings
long stretchable cloth wrapped over a wound for compression
48
composite dressing
multi layer primary or secondary dressings inner layer is non adherent middle layer absorbs moisture to prevent maceration with alginate, hydrocolloid, etc outer layer is antibacterial film for protection, semi permeable
49
enzymatic debrider dressing
medicated dressing with enzyme to soften hard tissue for debridement
50
foam dressing
sheets with small open cells to absorb fluid can be layered with other materials absorptive capacity depends on size and thickness of foam non adhesive area covers wound with adhesive border or overlying film for protection
51
hydrofiber dressing
absorb exudate and promote healing transforms into gel when in contact with fluid
52
hydrogel dressing
sheet, gel, or impregnated gauze 80-99% water and glycerin absorb minimal fluid but can moisturize a dry wound less effective at protecting from bacteria
53
hydrocolloid dressing
contains hydrophilic colloidal substances with a strong film backing absorb fluid slowly turning into a gel mass protection against water, air, and bacteria
54
absorptive wound dressings
alginates foams hydrocolloid hydrofiber minimal: composite hydrogel
55
negative pressure wound therapy
sealed and fitted to skin so vacuum can draw pressure upwards pulls the wound edges towards spongy surface of flim