Wound Care Flashcards
(55 cards)
contraindications to e stim over a wound
cancerous lesion
osteomyelitis
location of wound over contraindicated area
sensation
cognition
current of injury
injured cells have an endogenous electrical current
this current circles around a wound bed’s edge as the wound causes electrical “leaking”
greater potential at greater distances from the wound center
exogenous current’s effect on wounds:
neural tissue: pain relief and healing
wound epidermis
migratory mesenchyme cells: vimentin protein marker on fibroblasts - these cells can create epithelial tissue by organizing into polarized sheets, which can organize to close the wound
undifferentiated cells come to the wounfd and turn into collagen to scar down
layers of the epidermis - where will cells migrate in electrical field?
upper layers - cells migrate to anode +
lower layers - cells migrate to cathode -, including monocytes, fibroblasts, macrophages
current shown to affect cell migration
50-150 mV/mm
desoluabolization
liquefied tissue that is necrotic can be easily removed with estim using negative current on the cathode
stim currents used for improved blood flow and reduced inflammation
what specific effects does it have?
HVPC
increases blood flow at contraction levels
retard inflammation
antibacterial effects of extim
cathode delivery -
gram negative and positive
low intensity
HVPC current
80 pps, 2 hrs, 4x week
which pole is for what healing effect?
cathode for bacteria retarding
anode for healing and epithelial growth
protocol with HVPC for wound healing
start w negative from cathode for 4 weeks
negative 3-7 then change to positive
acute inflammation phase
hemorrhage, necrosis, erthemia, edema, exudate, red granulation forming
how to resolve pt in chronic inflammation phase
move back into acute inflammation phase so pt can progress into proliferation phase
acute proliferation phase
inflammation, wound starts reducing in size, red granulation tissue present, serous/serosanguineous exudate, may be odor, wound edges start to adhere and progress to epithelialization
signs of chronic proliferation phase
hyper granulation, tissue growing out of wound bed as this is not effective at closing wound bed
pink granulation tissue
causes of chronic proliferation phase
infection changing granulation tissue
acute epithelialization phase
expected outcome is to resurface wound
chronic epithelialization phase
rolled wound edges, fibrotic
could be caused by drying out, poor dressing choices
one cause?
pt can be vulnerable to pressure ulcers on the sacrum if:
poor hygiene, B&B dysfx
poor pressure relief adherence
atrophy or scarred muscle not providing normal cushioning and blood supply
seated all day
remodeling phase
should result in immature scar formation if optimal healing
helped with stimulation of migrating epidermal cells
happens after wound is closed
lasts 6 mo-2 years
granulation tissue
red beefy tissue
+ sign of proliferation phase
phases of healing
Acute inflammatory
acute proliferation
acute epithelialization (part of proliferation)
remodeling
any of these phases can turn chronic if healing is interfered with
exudate
liquid drainage from wound bed, not clear
transudate
liquid drainage from wound, clear and normal
serosanguinous fluid
bloody/clear/pink fluid exudate