10/25 - Hip Intra Articular Pathology Flashcards

(121 cards)

1
Q

what are the 3 main intra-articular conditions seen

A

nonarthritic
- femoroacetabular impingement syndrome (FAIS)
- microinstability

osteoarthritis

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2
Q

what are the 3 roles of the labrum

A
  1. ext of acetabulum (enhanced joint stability)
  2. suction seal
  3. shock absorption
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3
Q

what is FAIS

A

misshapen joint leads to breakdown of intra-articular structures

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4
Q

what is a FAI-CAM lesion and what is another word for that

A

nonspherical femoral head (ie change in bone shape) rotating inside acetabulum

aka slipped capital femoral epiphysis

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5
Q

what population is FAI-CAM common in

A

peds

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6
Q

what is a slipped capital femoral epiphysis

A

aka FAI-CAM

extra bone growth at head/neck junction of femur
- can then impinge labrum there and then damage the labrum

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7
Q

what motion causes pain in CAM lesion and why

A

pain w deeper flexion or when leg crosses over body
- that’s when bony pathology engages w labrum

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8
Q

what is the alpha angle for a CAM deformity

A

> 60deg

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9
Q

what is FAI - PINCER

A

prominent anterolateral rim of acetabulum
- overgrowth of ant edge
- retroversion of acetabulum

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10
Q

describe the lateral center edge angle in normal hip vs dysplasia vs FAI Pincer

A

normal: 25-39deg
dysplasia <25deg
- under coverage
FAI pincer >40deg
- over coverage

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11
Q

what does a smaller lateral center edge angle indicate

A

smaller the angle, more shallow the acetabulum and greater risk of sublux/dislocation

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12
Q

what sign occurs when there is retroversion of the acetabulum

A

crossover sign

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13
Q

what is a crossover sign

A

there is extra coverage w ant portion of acetabulum crosses over posterior

normal: ant should be deeper, post have more coverage

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14
Q

what are 3 categories of FAIS pathomechanics which lead to symptomatic bony impingement

A
  1. abnormal bony morphology
    - CAM, Pincer, mixed
  2. susceptible populations and activities
    - prior dysplasia, SCFE, LCP
  3. abnormal hip/pelvic kinematics
    - activity w inc hip flexion (squats, drop jumps)
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15
Q

what is a “C” sign indicative of

A

deep hip pain w intra-articular path

could also be microinstability (extra-articular)

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16
Q

what are 4 characteristics of pain in FAIS

A
  1. nonspecific groin pain can radiate to medial thigh
  2. “C” sign - deep inferior hip pain
  3. pain w twisting, pivoting (aka when hip in loaded position)
  4. pain w end-range flexion
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17
Q

what is a consideration of sx of FAIS

A

cumulative effect of abnormal wear

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18
Q

what are mechanical sx of FAIS

A

intermittent sharp pain
clicking, catching, locking

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19
Q

what is a common exam finding of FAIS

A

limited hip IR
- bilateral morphology
- motion may be symmetrical

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20
Q

if someone has a bony abnormality w FAIS, what will this likely look like upon examination

A

bilateral
- will usually have limitations w ROM (IR and possibly flex)

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21
Q

what ms weakness is common in FAIS

A

glut med and max weakness

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22
Q

what ms length may be limited in FAIS

A

hip flexor length

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23
Q

why would someone w FAIS have sx when hip flex is resisted

A

hip flex close to ant portion of capsule and labrum
- if damage/irritation to that portion of ant hip capsule, will have irritation w resisted hip flex

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24
Q

what are 4 intra-articular tests that are more provocative for FAIS

A

FABER***
Scour
FADIR
log roll

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25
what are 3 intra-articular tests that test more micro-stability for FAIS
ABD-hyperext-ER (AB-HEER) prone instability HEER
26
what is the most specific special test for hip injuries? what is its sensitivity?
log roll NOT sensitive
27
what does a log roll test assess
ant laxity of hip by amt of ER
28
what are 4 other reasons intra-articular tests may be positive other than FAIS
arthritis (ie OA) synovitis labral path loose body
29
what is a good use of FADIR special test in terms of FAIS
screening tool - sensitive but not specific
30
what are 3 possible locations of pain from a FABER test and what are their MOIs
1. groin pain - ms strain vs intra-articular (no sensitive to what intra path) 2. SIJ (PSIS region) pain - sacroiliac disorder 3. post hip pain - post hip impingement
31
what are 4 things that could cause the reproduced clicking in a scour test
psoas labrum arthritis loose body
32
what does a resisted SLR test help differentiate between
hip flexor strength discomfort vs pain inside the joint (intra-articular)
33
why is the FADIR an appropriate test to use in FAIS
symptomatic position
34
why are intra-articular injections used
common dx use (intra vs extra) - does it make sx better? if helps - inflammation was in intra-articular joint, help to tolerate ADLs and PT if does nothing - think extra-articular path
35
what are the 3 components of non-operative treatment of FAIS
1. modify irritating positions and activity 2. maximize mobility of joint 3. hip/core/lumbopelvic strength
36
what are irritating positions/activities to modify in FAIS
limit ant pelvic tilt - if tight hip flex and/or weak gluts adjust seat hight to avoid inc hip flex - deeper flex = more engagement limit squat depth limit incline or stair running - inc hip flex
37
what directions should joint mobility be maximized in FAIS
into capsular restrictions post and inf (if post capsular restriction, femur will shift away to ant)
38
how can PT prognosis change depending on how they present with FAIS
good PT prognosis: - more capsular restriction, weakness, hip joint mobility limitations + bony abnormality - modifiable factors w PT poor PT prognosis: - good strength, mobility + bony abnormality - more likely to need surgery
39
what are 3 examples of interventions targetting hip / core / lumbopelvic strength in FAIS
hip ABD & rotator ms plank variations paloff press
40
hypermobility vs instability
hypermobility - greater than normal physiologic motion instability - sx such as pain and/or apprehension are present
41
what is microinstability
capsuloligamentous laxity & clinical sx such as pain with/without apprehension
42
why is microinstability under-diagnosed
subtle not as obvious as hip dysplasia or sublux sx for microinstability are similar to hypomobility
43
what is location of most microinstability in the hip and why
usually anterior - post portion of acetabulum has more bony congruence -> more susceptible ant dislocations
44
where and with what are hip microinstability sx present
sx w WBing ER and/or forceful ext groin or deep joint pain "C-sign"
45
what are strength deficits seen in microinstability in hip and is this relevant
ABD and rotators pretty much weak in everyone, isn't helpful
46
what does ROM and ms lengths look like in hip microinstability
inc ER ROM dec length of iliopsoas complex - guarding to stabilize ant capsuloligamentous complex
47
what are 5 (+) special tests w hip microinstability
1. log roll 2. FABER 3. AB-HEER 4. prone instability 5. HEER
48
if sx w hip flexion, what is it likely d/t and what is it likely not ? why?
hip flexors can be used to create dynamic stability - likely d/t instability/microinstability usually not d/t tendinitis
49
describe the use of AB-HEER in testing hip microinstability and why
provocative test - basically putting head of femur as ant as possible to see if get sx
50
what are provocative tests looking to recreate sx of hip microinstability
AB-HEER prone instability HEER
51
describe hip microinstability cluster
95% chance of microinstability if 3 (+) tests: - AB-HEER - prone instability - HEER
52
what are 4 interventions of microinstability in the hip
1. education/activity modification 2. exercises w mid-range initially 3. exercise progressions 4. caution w hip flexor stretching
53
what are activity modifications that should be made in microinstability
avoid end range ext & ER avoid pivoting movements modify running stride to dec ext
54
what are 2 interventions of initial exercises in mid-range for microinstability
prone manual resist IR/ER - alternating isometrics quadruped external perturbation
55
what is the goal of initial exercises in mid-range for microinstability
to create dynamic stability
56
how should mid-range exercises be progressed in microinstability
hip ABD and rotators lumbopelvic stabilizers
57
why should there be caution w hip flexor stretching
spasm d/t overuse avoid tension on ant capsuloligamentous structures
58
what may be a more preferred way to dec hip flexor irritation in microinstability other than stretching
create better strength w other ms
59
what is OA
disorder of synovial joint - deterioration of articular cartilage & new bone formation
60
what are radiograph findings of OA
joint space narrowing osteophyte formation
61
what are sx of hip OA
stiffness pain
62
where will the pain be and what will cause the pain d/t hip OA
groin, ant, lat hip inc w amb, stairs, squatting
63
is primary or secondary hip OA more common
secondary (80%)
64
primary vs secondary hip OA
primary - no predisposing mechanical factor secondary - end result of dz process
65
what are 6 examples of secondary OA causes
osteonecrosis legg-calve-perthes dz dysplasia slipped capital femoral epiphysis congenital coxa vara / valga hip fx
66
what is the general takehome for OA diagnostic cluster
stiffness in morning stiffness gets better w movement the more WB activity, sx get worse capsular pattern of pain
67
what are the 2 main clinical presentations of hip OA
gluteal weakness balance deficits
68
what are 2 functional tests for hip OA
6MWT TUG
69
what are two components that interventions for hip OA are working to maximize and why
strength - offload joint - stabilizing structures can absorb force mobility - larger SA distribution
70
what are 6 interventions in management of hip OA
pt education manual therapy strengthening flexibility balance aerobic exercise
71
what is included in the pt education for hip OA
avoid end-range positions, sitting cross-legged
72
what are 2 indications for manual therapy
low grade pain high grade mobility restriction
73
what ms should be targeted for strengthening in hip OA and how
glut med single leg tasks - be cautious early on w WBing
74
what should be considered of aerobic exercises in hip OA
consider impact of forces & irritability - dec pain depending on severity of sx, think ab amt of WBing you want them to do (running vs biking)
75
what are balance progressions
DL -> SL EO -> EC static -> dynamic
76
what are 4 goals of PT management of hip OA
1. maintain function 2. relieve sx 3. prevent deformity 4. education (ie hip joint protection)
77
what are 6 treatment principles of hip OA
1. inflammation (responsible for pain) 2. joint alignment 3. ROM 4. ms length 5. ms strength 6. joint protection
78
what are 3 techniques to protect the hip joint from OA
1. body wt reduction 2. load carry modification 3. AD use
79
what is an ideal way to modify the load carry in hip OA
posterior (backpack) better than a shoulder bag - also balance sides out
80
THA cemented vs cementless
cemented - tolerate load right away - potential for loosening d/t cement cementless - WB restriction early on while bone heals
81
what is a precaution of trochanteric osteotomy if done during THA
no activve/resisted ABD
82
what are the 3 types of surgical approach to THA
ant post direct lateral
83
how are precautions related to the surgical approach of THA
avoiding motion that will load the area of the capsule that was cut
84
what are precautions of a post THA and why
avoid flex, ADD, IR -> ERs divided
85
what is a complication from during a THA
femoral stem fx
86
what is the composition of a hybrid THA
acetabular component - cementless femoral - cement
87
outcomes in cemented vs uncemented
no significant difference
88
what are precautions w an ant surgical approach and why
ext, ER - minimal incision
89
what precautions are associated w a direct lateral surgical approach
none low dislocation rate ABD mechanism impacted
90
what are 3 early complications of THA
thromboembolic event (ie DVT) infection dislocation
91
what THA surgical approach is dislocation the most common in? when does this inc risk decline?
inc risk w post approach pseudocapsule formed at 6mo
92
what is a late complication of THA
implant loosening - or if person falls and damages the construct
93
what is involved in joint resurfacing and how is this different from THA
acetabular component replaced cap over fem head fem neck/shaft spared
94
when is joint resurfacing indicated over THA
joint resurfacing if arthritis only on one surface replacing both if arthritis on both surfaces
94
when is joint resurfacing indicated over THA
joint resurfacing if arthritis only on one surface replacing both if arthritis on both surfaces
95
what are determining factors of whether hip fx will be treated as ORIFs or closed
- location - displacement inc displacement -> inc likelihood to have ORIF
96
why is there an inc mortality risk with hip fx
d/t sequelae of care afterwards - pressure sores - wounds - infections - poor mobility post
97
what surgery is indicated fro a displaced femoral neck fx
hemiarthroplasty
98
what are 5 things examined in an acute care setting s/p hip fx
1. DVT risk 2. bed mobility 3. WB status ** 4. transfer ability 5. gait safety
99
what are 4 acute care interventions s/p hip fx
1. ankle pumps, quad and glut sets 2. plan room for transfers and gait 3. appropriate assistance w gait 4. AD choice
100
what makes basic LE isometrics important as an acute care intervention s/p hip fx
prevent DVTs
101
goals in acute care (3) vs sub acute (5) vs chronic (4) s/p hip fx
ACUTE 1. OOB 2. safe gait 3. d/c planning SUB ACUTE 1. gait 2. transfer ability 3. endurance 4. ADL adaptations 5. plan to return home (if possible) CHRONIC 1. strength 2. gait 3. balance 4. ADL issue
102
why is hip ABD strengthening a post-acute phase goal in s/p hip fx repair
depending on what was cut w surgery
103
what are 2 ms groups targeted w strength interventions s/p hip fx
knee ext hip ABD (post-acute phase)
104
what is a test used to monitor fall risk in s/p hip fx
TUG
105
what are 3 physical performance tests used in s/p hip fx
1. gait speed 2. 30'' STS test (post-acute) 3. 6MWT (post-acute)
106
what are 2 factors that inc mortality and poorer outcomes w hip fx
inc age (>75yo) co-morbidities (DM, chronic illness)
107
what does inc age (>75yo) inc the risk of s/p hip fx (3)
inc mortality & poorer outcome inc time to dc inc risk of subsequent fall
108
what is legg-calve-perthes disease
blood supply to head of femur disrupted - subsequent fx w poor healing
109
what population is LCPD most common in
4-10yo boys (not exclusively)
110
what is the cause of LCPD
unknown
111
what does recovery look like in LCPD
recovery may take 2 yrs - bone remodeling
112
how does LCPD present (2)
gradual onset of pain short leg
113
why is it important to get imaging if suspicious of either LCPD or SCFE
sequelae of either of these are bad - catching it is the biggest piece - kids will heal easily and well
114
what is the population that slipped capital femoral epiphysis (SCFE) is most common in
adolescent males (8-15yo)
115
what inc the risk of SCFE
obesity
116
how does SCFE present (3)
knee or groin pain short limb may be bilateral
117
what is the main thing to look at to determine how likely hip dysplasia is
how shallow the depth of the acetabulum is
118
what are 3 factors that have an inc likelihood of developmental hip dysplasia
female > male (+) family history womb position (breech = inc risk)
119
what is the cause of developmental hip dysplasia
unknown
120
what is the typical age of dx for developmental hip dysplasia
variable age of dx