Skin infections Flashcards

1
Q

prevalence of herpes simplex virus in childrfen

A

18-35% of children by 5yo

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

when should herpes simplex virus be treated promptly and with what

A

periobrital involvement
-eyes checkd for corneal involement which can cause scarring

treatment - acyclovir
-prophylaxi may be requied if reactivation becomes frequent

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

what condition can herpes simplex virus accompany in children and how does it present

A

children with atopic eczema (eczema herpeticum)

infeciton can become widespead and acute detreriation in the severiry of their eczema causing monomorphic punched out erosinos which can be painful

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

what causes impetigo
-who gets it

A

highly contagious skin infeciton caused by staph A
(occassionaly strep)

commonly seen in infants and young children

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

characterisitic features of impetigo 3

A

annular erythematous lesions w a honey coloured crust

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

when can impetigo become bullous

A

may become bullous as a result of cleaving of the epidermis by exfoliative staph endotoxins

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

management priciples of impetigo

A

swabbed and paretns advised hwo to reduce spread
-avoid sharing towels and bathing with other children

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

treatment of impetigo 3

A

topical antiseptic (cream) in bath
-also apply directly to affected areas

if only a few lesions consider topical ABx

if multiple lesions
oral ABx

Fluclox - first choice

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

how is molluscum contagiosum characterised

A

small pearl umbilicated papules on the skin of children

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

who gets molluscum contagiosum 2

A

agd 4-9

often more sever in children with eczeema and worse in flexural sites where lesiosn can become infected

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

cause of molluscum contagiosum

A

DNA Pox virus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

management of molluscum contagiosum 2

A

become inflamed and then resolve oer 18mnths or more

no treatment gnerally required
-topical antiseptics may prevent them becoming infected

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

how to get rid of molluscum contagiosum quicker 4

A

physically irritated
-rubbing
-piercing
-cryotherapy

chemically
-salicylic acid

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

how does scabies present in infants compared to adults

A

extremely itchy rash simlar to adults

difference is with burrows that are commonly present in adults -these typical burrows are not present on the skin but appear:
-on the soles of the feet. (not usually seen in adults)

can also manifest as nodules in warm moist areas such as axilla, umbililicsu groin or penis

WHOLE FAMILY AND CARERS SHOULD BE TREATED

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

treatment for scabies 2

A

permethrin

malathion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

why is tinea capatis increasing in incidence s

A

result of increaed immigration from african and carribean contunres where the incidence is higher

17
Q

main causal organism of tinea capatis 2

A

tricopyton tonsurans

-less commonly trichophyton violaceum

18
Q

how can tinea capatis present

A

spectrum can be:
-diffuse scales

-patchy alopecia w black dots (broken hairs)

-widespread pusutules associated with lymphadenopathy

19
Q

define a kerion in relation to tinea capatis
-what is this associated with in this condition

A

kerion- inflammatory pus filled abscess that sometimes oozes

can cause lymphadenopathy and can breakdown to leave a huge ulcer that scars

20
Q

how is tinea capatis diagnosed

A

micropscopy and cultures of scapring or brushgin from the scalp

21
Q

management of tinea capatis 3

A

ketoconazole shampoo help prevent spread to toerh children

but not a recongised treatment

oral griseofulvin
-licesned but cuasuses nausae

terbinafine (unlicensed)
-is most effective

review after four weeks and rescpra/contiuunue treatment if still clinically affected

22
Q

presentation of uritcaria 2

A

itchy papules (hives)

and pplaques which demonstrate a wheal and flare ( where the papule is white with surrounding eythema)

23
Q

what causes these lesions in uritcaria

-how does this cause swelling

A

histamine release from mast cells in the skin which have degranulated after stimulation by a trigger

-the histamine causes vasodilation of the blood vessels (hence erythema) and leakage of fluid from the capillaries causing swelling in the skin

24
Q

timeframe for acute uritcaria

A

less than 6-8 weeks

25
Q

how can causes of uritcaria be classified 5

A

5 ‘i’s

infection
infestation
injection
ingestion
inhalation

26
Q

how can causes of uritcaria be classified 5

A

5 ‘i’s

infection
infestation
injection
ingestion
inhalation

27
Q

regarding causes of urticaria describe each subset
-infection 3

A

bacterial

viral

fungal

28
Q

regarding causes of urticaria describe each subset
infestation 1

A

worms (diarrhoeal/perianal itch assocated)

29
Q

regarding causes of urticaria describe each subset
injection 3

A

drugs

immunisation

blood products

30
Q

regarding causes of urticaria describe each subset
ingestion 2

A

drugs- while exposed

foods -lasts hours

31
Q

regarding causes of urticaria describe each subset
inhalation 2

A

pollen

mould - lasts hours/while exposed

32
Q

what is the basic pathophsy of chronic urticaria
-what is it associated with 3

A

usually autoimmune trigger
-assoc w other autoimmune conditions such as thyroid disease

rarely associated with underlying chronic infection like gut paraisetes or tuberculosis

extremely rarely can occur as part of antiinflammatory syndrome when it presents in infancy assoc w pyrexia, malaise and joint or abdo pain

33
Q

management of uritcria if under 6 months 1

A

chlorpheniramine

34
Q

managment of uritcaria if over 6 months 2

A

chlorpheniramine
+
long acting antihistamine (astemizole)

35
Q

other parts of uritcaria managemnt 1

A

add sedative antihistamine at light if sleep is distrubed

36
Q

when should a child with uritcaria be referred

A

if they are unresponsive to 3 differnt antihistamines each for 4-6wks or they have additional symptoms or brusing