2 Flashcards

(86 cards)

1
Q

when were xrays used diagnostically

A

1896

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2
Q

when did local anesthetics become populat

A

late 1800s

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3
Q

what was heroic medicine

A

medicine before osteopathy used to preserve life force

stimulants- given to drowsy pts
hypnotics- given to agitated pts

purgatives and cathartics were rampent, as well as blood letting

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4
Q

conquer dz

A

if enough force or drugs were used, it would cast out demons

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5
Q

when did at still and fam move?

A

1830’s

MO

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6
Q

what did AT study

A

ministry and med from his dad. at the time, education was mainly apprenticeship and little class time

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7
Q

becomes the first state to legally

license DOs, then North Dakota

A

vermont

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8
Q

Missouri grants DO’s licensure

A

1897

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9
Q

goal of OMT

A

remove somatic dysfunction and restore homeostasis

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10
Q

somatic system

A

SAM

skeletal
arthoidial
myofascial

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11
Q

systems related to somatic system

A

VLAN

vascular
lymphatics
neural

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12
Q

__________ disrupts unity of body mind and spirit

A

somatic dysfx

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13
Q

___________ impairs bodys ability to self-regulate, heal and maintain

A

somatic dysfx

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14
Q

osteopathic philosophy: DOs treat what?

A

DO whole patient

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15
Q

what is homeostasis?

A

level of well-being of an individual is maintained by INTERNAL physiologic harmony that is a result of a stable state among the interdependent body functions

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16
Q

are somatic dysfxs unique?

A

yes

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17
Q

must all of tart be present to diagnose a somatic dysfx

A

no. any 1 of TART

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18
Q

bogginess

A

tissue texture abnormality characterized by spongy tissue due to increased fluid content

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19
Q

tissue texture abnormality represents combo of which signs

A
vasodilation
edema
flaccidity
hypertonicity
contracture
fibrosis
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20
Q

tissue texture abnormality is assx with the following sx

A

itching
pain
tenderness
paresthesias

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21
Q

tone

A

normal feel of relaxed muscle

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22
Q

what can tone be contrasted w

A

hypertonicity

hypotonicity

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23
Q

hypertonicity

A

at extreme

spastic paralysis

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24
Q

hypotonicity

A

no tone at all.

flaccid paralysis

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25
contraction
normal tone of muscle when it shortens or activated against resistance
26
contracture
abnormal shortening of muscle due to fibrosis, most often in the tissue itself. muscle can no longer reach normal length
27
contracture is often a result of what
chronic condition
28
spasm
abnormal, sudden and involuntary m contraction maintained beyond physiological need. results in abnormal mtn.
29
ropiness
hard, firm, rope like muscle tone
30
ropiness indicates what
chronic condiiton
31
acute vascular tissue texture abnormality
inflamed vessel wall injury; endogenous peptide released
32
chronic vascular tissue texture abnormality
sympathetic tone increases vascular constriction
33
acute ympathetic TT change
local vasoconstriction, overpowered by local chm release. net effect is vasodilation
34
chronic sympathetic TT change
vasoconstriction hypersympathetic tone may be regional
35
acute musclarature TT change
locan increase in tone, muscle contraction, spasm- mediated by spindle activity
36
chronic musclarature TT change
decreased muscle tone flaccid limited ROM due to contracture
37
asymettry
absence of symmery in POSITION or MOTION
38
how can we determine asymmetry
vision | palpation
39
AB (anatomic barrier)
limit of motion imposed by anatomical structure
40
PB physiologic barrier
limit of active motion
41
elastic barrier
range in between physiogic and anatomic barrier
42
what is the area that warms up with stretch
elastic barrier
43
restrictive barrier
functional limit that abnormally diminishes the physiologic range
44
PROM
patient relax and you must block linkage of assx structures
45
what is block the linkage?
stabilization of associated and adjacent structures so that you can focus only of the joint you are accessing.
46
what is more: PROM or AROM
PROM because the patients muscles are relaxed
47
how does somatic dys alter ROM
adds a restrictive barrier, decreasing the physiologic range
48
AROM
motion by patient, reaching physiological barrier
49
PROM
motion by DO, reaching anatomical barrier
50
end feel
palpatory experience when a joint is moved to its limit
51
ex of restricted ROM and abnormal end feel
1. early muscle spasms 2. Late muscle spasms 3. Hard capsular 4. Soft capsular
52
early muscle spasms
protective spasms after injury empty, guarding
53
late muscle spasms
chronic spasms; chronic tissue changes
54
hard capsular
frozen shoulder
55
soft capsular
synovitis- swelling of knee after injury
56
how do you know when you are at end feel
experience
57
to look at skin vs muscle, we should consider
tissue movement
58
to look at muscle vs bone, we should consider
deep palpation
59
what is tenderness
pain or discomfort elicited by DO through palpation
60
pain
unpleasant senation caused by noxious stimuli and received by specialized nerve endings.
61
acute tenderness pain visceral fx visceral dysfx
pain is sharp, severe, Min somatoviseral effects visceral dysfunction may/may not be present
62
Chronic tenderness pain visceral fx visceral dysfx
pain is dull, ache, paresthesia 9tingling, burning, gnawing, itching somatovisceral effects are common visceral dysfx is involved in somatic dysfx
63
acute TART TTA Asymmetry Restriction Tenderness
1. red, swollen, boggy, increased tone 2. asymmetry present 3. restriction is present and painful w motion 4. sharp pain
64
chronic TART TTA Asymmetry Restriction Tenderness
1. dry, cool, ropy, pale, decreased tone 2. asymmetry is present but compensation occurs 3. Restriction is present but maybe not. may be garded or empty 4. pain is dull and achy
65
Tenderpoints
small discrete hypersensitive areas within myofascial structures that result in localized pain
66
Trigger points
small discrete hypersensitive areas within myofascial structures which palpation causes reffered pain away from site
67
what is the goal of OMT
remove SD and restore homeostasis
68
how are somatic dysfunctions named
position of ease where they live
69
somatic dysfunction results in
local changed in skeletal, arthroidal, myofascial, neural, lympathic and vascular structure.
70
omt indications
somatic dysfunction and or visceral dysfunction
71
adverse reactions
soreness similar to workout, acute illness exacerbation of current complaints
72
precautions
cancer | frailty due to severity, dz, youth and or elderly
73
recommendations
rest 1-4 days, hydration 1-2 liters per day
74
when are direct techniques contraindicated
ligamentous laxity states
75
role of omt in biochemical model
optimize myofascial and joint fx
76
role of omt in neurologival
remove neurologic imbalance
77
role of omt in respiratory/circulatory
maximize fx
78
role of omt in metabolic
structure and fx are recipricolly related
79
role of omt in behavior
more of a cause than effect. how we spend out ime affects the above 4. the exerces teaches patients to tx themselves
80
direct techniques
action engaging the restrictive barrier directly
81
indirect techniques
action involving postitioning away from the restrictive barrier
82
direct omt techniques
``` MFR INR ST MET HVLA Visceral ```
83
combo OMT techniques
MFR | Still
84
Indirect OMT techniques
MFR INR BLT/LAS FPR
85
does somatic dysfx impaire the 4 tenants
yes
86
acute somatic dysfunction is characterized by
``` vasoDILATION edema -tenderness -pain -tissue contraction ```