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OP - Yield > 3 pulpal and periapical > Flashcards

3 pulpal and periapical Flashcards

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1
Q

pulp polyp =

A

chronic hyperplastic pulpitis

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2
Q

accelerated aging and teeth

A

progeria

widespread deposition of secondary dentin

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3
Q

obstruction of pulp chamber and canal with secondary dentin

A

calcific metamorphosis
usually trauma
yellow discoloration of crown

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4
Q

calcific metamorphosis

A

obstruction of pulp chamber and canal with secondary dentin

usually trauma
yellow discoloration of crown

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5
Q

3 shapes of pulpal calcifications

A

denticles, pulp stones, diffuse linear calcifications

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6
Q

globules of gamma globulin

A

russel bodies

plasma cell product

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7
Q

russel bodies

A

globules of gamma globulin

plasma cell product

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8
Q

rushton bodies

A

linear or arch-shaped calcifications

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9
Q

linear or arch-shaped calcifications

A

rushton bodies

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10
Q

pyronine bodies

A

basophilic particles (plasma cell product)

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11
Q

pulse granuloma

A

hyaline bodies/giant-cell hyaline angiopathy
eosinophilic material surrounded by lymphocytes and giant cells
may contain inflammatory cells or calcifications
pools of inflammatory exudate that undergoes fibrowiw and dystrophic calcifications

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12
Q

celluitis what is and 2 relevanet types

A

abscess spread through facial soft tissues

ludwing’s angina and cavernous sinus thrombosis

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13
Q

three features of cavernous sinus thrombosis

A

proptosis (exophthalmos), chemosis (edematous conjunctiva), ptosis (dropping eyelid)

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14
Q

proptosis, chemosis, ptosis

A

caverbous sinus thrombosis

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15
Q

broadly, courses of osteomyelitis

A

acute suppurative and chronic suppurative

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16
Q

necrotic bone separated from adjacent vital bone

A

sequestrum

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17
Q

necrotic bone surrounded by newly-formed vital bone

A

involucrum

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18
Q

sequestrum vs involucrum

A

sequestrum: necrotic bone separated from adjacent vital bone
involucrum: necrotic bone surrounded by newly-formed vital bone

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19
Q

types/causes of **diffuse sclerosing osteomyelities

A

diffuse ssclerosing – infx present
primary chronic – no bacterial sourse
chronic tendoperiostitis

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20
Q

primary chronic osteomyelitis 2/2 masticatio muscle overuse

A

chronic tendoperiostitis

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21
Q

chronic tendoperiostitis

A

primary chronic osteomyelitis 2/2 masticatio muscle overuse

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22
Q

widespread primary chronic osteomyelitis – what is; skin lesions

A

chronic recurrent multifocal osteomylitis

no skin lesions

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23
Q

sy ndrome with primary chronic osteomylitis

A 
SAPHO
synovitis
acne
pustulosis
hyperostosis
osteomyelitis
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24
Q

alveolar osteitis aka and what;s going n

A

fibrinolytic alveolitis

lysis of fibrin releases kinins (pain mediators)

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25
role of fibrin in alevolar osteitis
lysis --> release of kinins (pain mediators)
26
when is primary dentin formed
before completion of crown
27
when is seconadry dentin formed
after primary; throughout life
28
when is tertiary dentin formed
laid down in areas of focal injury
29
demographic for more rapid secondary dentin
more rapid in M | also in Ca-related dzz eg arthritis, gout, kidney stones, gall stones, atherosclerosis, HT
30
accelerated aging and teeth
progeria | widespread deposition of secondary dentin
31
calcific metamosprhosis of teeth why and look
early obstruction of pulp chamber and canal w secondary dentin, after trauma yellow discoloration of teeth
32
interface dentin aka and what is
fibrodentin | initial layer of reparative dentin (atubular)
33
initial layer of reprative dentin
interface dentin | atubular
34
tubules from dead primary odontoblasts
dead tracts | filled w degenerated odontoblastic processes
35
pulp calcs prevalence and assoc
20%; assoc w chronic pulpitis, age, and familial
36
conditions w prominent pulp calcs
dentin dysplasia Id and II, pulpal dysplasia, tumoral calcinosis and calcinosis universalic, Ehlers Danlos, ESRD
37
forward metaplasia
proroplasia | eg when odontogenic lining of inflammatory cysts becomes resspiratory
38
pulse granuloma what is
pool of inflammatory exudate; fibrosis and dystrophic calc; surrounded by lymphocytes and giant cells
39
parulis
mass of granulation tissue at opening of sinus tract
40
conditions that favor widespread infx
diabetes, neutropenia, malignancy, immunosuppression, corticoid use, cytotoxic drug use
41
abscess draining thorugh skin
cutaneous sinus
42
acute edematous spread of acute inflammation through soft tissue
cellulitis
43
dangerous head and neck cellulitides
Ludwings angina and cavernous sinus thrombosis
44
ludwigs angina location and origin
cellulitis of submandibular region 70% from lower molar teeth infx also tonsillar/pharyngeal abscess, oral laceration, frx of md or dub-md sialadenitis more in AIDS, tranplsant, aplastic anemia, diabetes pts airway obstruction major concern
45
tongue in ludwig
woodt | elevated, enlarged, protruded when Ludwig in subligual space
46
neck in ludwig
bull | enlarged and tender (subMD space spread)
47
cavernous sinus thrombosis location and origin
cavernous sinus in dura; mx infections
48
cavernous sinus thrombosis clinical look
periorbital enlargement involving eyelid and conjunctiva | proptosis, chemosis (conjunctival edema), ptosis in 90% of cases
49
osteomyelitis locations
most in MD of males | mx cases when assoc w NUG or noma
50
three categoreis of diffuse sclerosing osteomyelitis
diffuse sclerosing osteomyelitis -- infx present primary chronic osteomylitis -- similar to classic chronic but no bacteria, suppuration, or sequestra chronic tendoperiostitis
51
chronic tendoperiostitis
primary chronic osteomylitis 2/2 overuse of masseter (MD angle/body) or digastric (anterior MD/PM region) can see parafunctional habits (bruxism, clenching, nail biting)
52
SAPHO molecular basis
autoimmune to derm bacteria, cross react with bone | poss HLA-B27?
53
CRMO and skin
chronci recurrent multifocal osteomyliis | no skin lesions but can appear up to 20 years later -- palmoplants pustulosis, psoriasis, acne, hidradenitis suppurativa
54
condensing osteitis aka, who, where
focal sclerosing osteomyelitis localized areas of bone sclerosis assoc w apices of teeth w pulpitis or necrosis children and young adults ddx: FCOD (has RL border) and idiopathic osteosclerosis (separated ffrom apex)
55
bone scar
residual area of condensing osteitis after imflammation resolves
56
residual area of condensing osteitis after imflammation resolves
bone scar
57
garre osteomyelitis better term:
periostitis ossificans or osteomylitis w proliferative periostitis
58
radio look of osteomylitis w proliferative periostitis
onion skinning | active osteoblasts on covex surface of curvy bony spicules
59
causes of periosteal bone formation
most -- osteomylitis and neoplasms (Ewing sarcoma, LCH, osteosarc, hemangioma) also: trauma, cysts, infantile, cortical hyperostosis, fluororis,s avitaminosis C, hypertrophic osteoarthropathy, and congenital syphilis)
60
histo periosteal rxn to inflammation
parallel rows of vital bone
61
locations for periosteal rxn to inflammation
most are unifocal in molar and PM 2/2 caries | also perio infx, frx, buccal bifurcation cysts, and non-odontogenic infx
62
dry socket what happened
clot is lost --> bare bony socket
63
why can clot get lost
trauma, estrogen, bacteria can stimulate fibrinolysins increased freq in impacted 3 molars, poor oral hygiene, inexperienced surgeon, traumatic exo, oral contraceptive use, and presurgical infx (pericoronitis) also inadeq irrigation, smoking, heavy spitting/sucking
64
dry socket tx:
xray to rule out tip or foreign body | irrigation, analgesics, maybe topical abx
65
normal pulp: | cold, heat, electric, percussion, pain
``` cold - 1-2 sec heat - no EPT - normal percussion - no pain - severe ```
66
reversible pulpitis: | cold, heat, electric, percussion, pain
``` cold - acute pain, sweets also heat - sometimes EPT - lower percussion - no pain - acute ```
67
irreversible pulpitis: | cold, heat, electric, percussion, pain
``` cold - early: uncomfy; lateL cold relieves pain heat - severe/sharp EPT - early: low; late: high percussion - no pain - throbbing lingering ```
68
periapical infection clinical
acute: dull throbbing pain, negative or delayed vitality, pain on percussion chronic: no pain
69
reactionary vs reparative dentin
 Reactionary: mild stimuli |  Reparative: Severe stimuli; first -- interface dentin = atubular + acellular = fibrodentin
70
detectable pulpal calcs
>200 μm to be detected by radiograph
71
ludwig and teeth:
70% related to molars submand/subling/submental spaces submand --> pharyng --> retropharyng
72
cavernous sinus thrombosis and teeth
10% related to teeth anterior pathway: canine tooth --> canine space --> valveless facial veins --> angular vain --> inf ophth vein --> sinus psoterior pathway: mx PM/M --> buccal/infratemporal space --> emissary vein --> inferior petrosal sinus --> cavernous sinus
73
SAPHO what is, cause, location, components
``` cause unknown, individuals with Autoimmune are predisposed  Axial skeleton (anterior chest)  Concurrent neutrophilic skin  Adults  -ve for bacteria Synovitis Acne Pustulosis Hyperostosis Osteitis that mirrors primary chronic osteomyelitis ```
74
CRMO what is
``` Believed to be a pediatric variant of SAPHO or wide spread variant of primary chronic osteomyelitis. Chronic Recurrent Multifocal Osteomyelitis ```

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