Chapter 7 Flashcards

1
Q

What are the two methods of testing function?

A

Nerve conduction study (NCS)

Somatosensory evoked potentials (SEP)

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2
Q

What does NCS measure?

A

peripheral nerve function

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3
Q

What does SEP measure?

A

peripheral nerves and central nervous system pathways

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4
Q

What is compared to determine if NCS is normal?

A

distal latency
amplitude of the evoked potential
conduction velocity

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5
Q

What are SEPs used to determine?

A

verify subtle signs and locate lesions of the dorsal roots, posterior columns, and brainstem.

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6
Q

What is ataxia?

A

incoordination that is not due to weakness

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7
Q

What are three types of ataxia?

A

sensory
vestibular
cerebellar

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8
Q

What is the Romberg test used for?

A

to distinguish between cerebellar ataxia and sensory ataxia

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9
Q

What is neuropathy?

A

general term for dysfunction or the pathologic condition of one or more peripheral nerves

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10
Q

What is the sensory loss in peripheral nerve lesions?

A
  1. conscious proprioception and discriminative touch
  2. Cold
  3. Fast pain
  4. Heat
  5. Slow pain
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11
Q

What are the common causes of dysfunction of the spinal region?

A

trauma
disease
virus

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12
Q

What happens with complete transection of cord?

A

All sensation is prevented at one or two levels below the lesion
Voluntary motor control below the lesion is also lost

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13
Q

What happens with hemisection of the spinal cord?

A
  • complete loss of sensation in contralateral side 2-3 levels below lesion
  • discriminative touch and conscious proprioception lost ipsilateral to lesion
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14
Q

Lesion in posterior column:

A

conscious proprioception, 2 point discrimination and vibration lost below level of lesion
-ataxic movements

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15
Q

Brainstem region lesion:

A

mix of ipsilateral and contralateral signs

-sensory loss can be contralateral in upper midbrain after discriminative sensation have crossed midline

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16
Q

Somatosensory Cortex Lesion:

A

Conscious proprioception
Two-point discrimination
Stereognosis
Localization of touch and pinprick (nociceptive) stimuli
-contralateral and decrease of above sensations

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17
Q

Fast and slow pain can occur with musculoskeletal injuried via what fibers?

A

A-delta and C fibers

18
Q

What parts of the brain are included in processing and regulating pain information?

A

brainstem, amygdala, hypothalamus, thalamus, and areas of the cerebral cortex

19
Q

What does understanding of pain matrix require?

A

understanding of:

  1. discriminative
  2. motivational affective
  3. congitive-evaluative component
20
Q

Discriminative aspect of pain matrix refers to what?

A

the ability to localize the site, timing, and intensity of tissue damage or potential tissue damage

21
Q

Motivational affective aspect of pain matrix refers to what?

A

the effects of the pain experience on emotions and behavior, including increased arousal and avoidance behavior.

22
Q

Cognitive-evaluative aspect of pain matrix refers to what?

A

meaning that the person interprets /ascribes to the pain (punishment, burden

23
Q

How is pain controlled?

A
  1. gate theory
  2. counterirritant theory
  3. dorsal horn processing of nociceptive information
  4. antinociceptive systems
24
Q

What is the gate theory of pain?

A

Typical response to hitting one’s thumb with a hammer is to withdraw the thumb, yell, and apply pressure
- TENS

25
Q

What is the counterirritant theory?

A

explains the inhibition of nociceptive signals by stimulation of non-nociceptive receptors occurs in the dorsal horn of the spinal cord

26
Q

What is the dorsal horn processing of nociceptive information?

A

Processing of somatosensory information in the dorsal horn can be altered by abnormal neural activity or by tissue injury

27
Q

What are the states of dorsal horn processing?

A

normal
suppressed
sensitized
reorganized

28
Q

Normal state of dorsal horn processing:

A

normal physiologic activity

29
Q

Suppressed state of dorsal horn processing:

A

segmental and descending inhibition

30
Q

Sensitized state of dorsal horn processing:

A

changes in neurotransmitters and receptors

31
Q

Reorganized state of dorsal horn processing:

A

cell death, degeneration of C fiber and sprouting that synapses with neurons in the nociceptive pathways

32
Q

Neuropathic pain is produced by what?

A

neuroplasticity not by stimulation of nociceptors

33
Q

Antinociception:

A

the suppression of pain in response to stimulation that would normally be painful.

34
Q

What activates antinociceptive mechanisms?

A

endorphins and endogenous substances

35
Q

Level I of antiociceptive site:

A

peipheral

36
Q

Level II of antiociceptive site:

A

dorsal horn

37
Q

Level III of antiociceptive site:

A

fast acting neuronal pathway from brainstem

38
Q

Level IV of antiociceptive site:

A

hormonal

39
Q

Level V of antiociceptive site;

A

descending cortical inhibition

40
Q

What is primary hyperalgesia?

A

refers to excessive sensitivity to stimuli in injured tissue