ABGs Flashcards

(44 cards)

1
Q

Normal pH

A

7.35-7.45

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2
Q

normal Pao2

A

75-100

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3
Q

normal paCo2

A

35-45

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4
Q

normal hco3

A

22-28

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5
Q

normal sao2

A

94-100

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6
Q

normal arterial vol

A

15-22%

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7
Q

normal venous vol

A

11-16%

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8
Q

critical pH values

A

<7.25, >7.6

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9
Q

Critical PaCo2

A

<20, >60

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10
Q

critical HC03

A

<10, >40

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11
Q

Critical PaO2

A

<40

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12
Q

O2 sat critical

A

<75%

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13
Q

What should you perform prior to ABG?

A

allen’s test

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14
Q

PaCO2 is elevated in

A

respiratory acidosis
metabolic alkalosis

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15
Q

PaCO2 is decreased in

A

respiratory alkalosis
metabolic acidosis

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16
Q

Majority of the CO2 in the blood is

A

HCO3 - regulated by the kidneys
elevated in metabolic alkalosis, decreased in metabolic acidosis

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17
Q

indirect measure of O2 in arterial blood

A

PaO2

decreased in: oxygen diffusion issues, premature mixing of arterial/venous blood (CHD), overperfusion (Pickwickian syndrome)

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18
Q

% of hemoglobin saturated w/ oxygen

A

O2 saturation
if carbon monoxide poisoning –> put O2 on

19
Q

amount of oxygen in the blood

A

O2 sat * Hgb * 1.34 * PO2 * .003

20
Q

base excess/deficit calculated by

A

pH, PaCO2, Hct, measuring amount of buffering anions
negative = metabolic acidosis
positive = metabolic alkalosis or compenstaed resp acidosis

21
Q

When to draw an ABG?

A

resp failure, acid-base disorders, monitoring critically ill patients, evaluation of organ function, guiding therapy decisions

22
Q

interpret ABG

A

1) check pH
2) analyze paCO2
3) analyze HC03
4_ compensatory mechanisms

23
Q

high PaCO2, low pH

A

respiratory acidosis from COPD, asthma, overdose

24
Q

low PaCO2, high pH

A

respiratory alkalosis from hyperventilation, anxiety, pain

25
low HCO3, low pH
metabolic acidosis from diabetic ketoacidosis, renal failure
26
high HCO3, high pH
metabolic alkalosis, vomiting, diuretic use
27
CIs to ABG
absence of pulse, infection, - allen test, AV fistula proximal to site of acess, severe coaguloopathy can cause arterial occlusion, nerve injury
28
metabolic alkalosis can be from
hypokalemia hypochloremia chronic + high vol gastric suction chronic vomiting aldosteronism
29
respiratory alkalosis can be from
CHF, cystic fibrosis, CO poisoning, anxiety, pain, preganncy
30
metabolic acidosis can be from
keto acidosis, lactic acidosis, severe diarrhea, renal failure
31
respiratory acidosis can be from
resp failure
32
Tx: respiratory acidosis
improve ventilation
33
Tx: respiratory alkalosis
treat underlying cause
34
Tx: metabolic acidosis
treat underlying cause
35
Tx: metabolic alkalosis
correct electrolyte imbalances, treat vomiting
36
COPD oversedation head trauma over-oxygenation in patients w/ COPD causes
increased PaCO2
37
hypoxemia PE anxiety pain pregnancy
decreased PaCO2
38
chronic vomiting aldosteronism use of mercurial diuretics COPD
increased HC03
39
chronic or severe diarrhea chronic use of loop diuretics starvation DKA AKI
decreased HCO3
40
polycythemia increased oxygen hyperventilation
increased PaO2 + O2 content
41
anemias mucus plug bronchospasm pneumothorax pulmonary edema ARDS
decreased PaO2 + O2 content
42
anion gap
10-16
43
high anion gap
lactate, ketones, renal, toxins
44
normal anion gap
chloride, acetazolamide, addisons GI causes - diarrhea ,vomtiing, fistulas