Acute Painless loss of Vision Flashcards

(29 cards)

1
Q

What needs to be included in an ocular history of APVL

A

Previous ocular history
Cardiovascular disease
Family history of eye disease
drugs and eye drops
Symptoms: mono-ocular or binocular
Time of event, method of becoming aware of symptoms
Change in symptoms and associated symptoms - central only, central and peripheral loss, altitudinal, haemianopia

pupil reactions, anterior segment, red reflex, fundoscopy

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2
Q

What are the monocular causes of APLV?

A
Ischaemic/vascular - thrombosis, embolism, temporal arteritis 
Occlusion of retinal artery/vein 
ACUTE CORNEAL DISEASE
anterior chamber haemorrhage 
uveitis haemorrhage 
ACUTE CATARACT 
VITREOUS HAEMORRHAGE
RETINAL DETACHMENT 
OPTIC NEURITIS OR ISCHAEMIC OPTIC NEUROPATHY
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3
Q

What is the presentation of acute corneal disease?

A

(normally painful)

rarely painless - cloudy cornea

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4
Q

What is the presentation of anterior chamber haemorrhage?

A

hyphaema - blood in the anterior chamber

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5
Q

how do acute cataracts cause APLV?

A

struck by lightning

acute cataract over weals if the lens becomes porous and takes in fluid

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6
Q

What are the common causes of vitreous haemorrhage?

A

proliferative diabetic retinopathy
retinal tears
posterior vitreous detachment

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7
Q

What is the classic presentation of optic neuritis?

A
acute visual loss
age over 60
headache 
colour vision (epspecially red) 
Pain on moving eyes
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8
Q

What % of haemorrhages affect the retina? optic nerve?

A

Retina - 80%

Optic nerve - 20%

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9
Q

Which is more common, occlusion of the vein or occlusion of the artery?

A

VEIN - veins present with haemorrhage whereas arteries do not

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10
Q

What are the clinical features of branch retinal vein occlusion?

A

Variable degree of central vision on waking in morning

Retinal signs on examination - variable degree of haemorrhage with cotton wool spots, that are limited to one sector of the retina

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11
Q

What are the risk factors for retinal vein occlusion?

A
Hypertension
High cholesterol
Diabetes
Smoking
Glaucoma
Systemic inflammatory conditions such as systemic lupus erythematosus
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12
Q

What is the prognosis for branch retinal vein occlusion

A

not good if extensive and there is the risk of developing new blood vessels in the future (i.e. diabetic retinopathy)

If mild - good prognosis, resolution and development of collaterals

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13
Q

What are the clinical features of central retinal vein occlusion?

A

Acute painless loss of vision

Acuity varies from 6/6 to CF and may have RAPD if severe.
Opposite eye should be examined for the optic disc to check for raised IOP

Fundoscopy finings: 
Flame and blot haemorrhages
Optic disc oedema
Macula oedema
(looks like a cheese and tomato pizza)
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14
Q

What investigations should be done for central retinal vein occlusion?

A

BP, bloods, IOP

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15
Q

How should patients with retinal vein occlusion be managed?

A

Referral to ophthalmology
Laser photocoagulation
Intravitreal steroids (e.g. a dexamethasone intravitreal implant)
Anti-VEGF therapies (e.g. ranibizumab, aflibercept or bevacizumab)

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16
Q

How does central retinal artery occlusion occur?

A

something blocks the flow of blood through the central retinal artery

The most common cause of occlusion of the retinal artery is atherosclerosis. It can also be caused by giant cell arteritis, where vasculitis affecting the ophthalmic or central retinal artery causes reduced blood flow

17
Q

What are the risk factors for central retinal artery occlusion?

A

Risk factors for retinal artery occlusion by atherosclerosis are the same as for other cardiovascular diseases:

Older age
Family history
Smoking
Alcohol consumption
Hypertension
Diabetes
Poor diet
Inactivity
Obesity

GCA or PMR = higher risk

18
Q

What is the presentation of central retinal artery occlusion?

A

Sudden painless loss of vision
RAPD
Fundoscopy shows pale retina with cherry red spot

19
Q

What is the management of central retinal artery occlusion?

A

Immediate referral
?Dislodge thrombus

Ocular massage
Removing fluid from the anterior chamber to reduce intraocular pressure.
Inhaling carbogen (a mixture of 5% carbon dioxide and 95% oxygen) to dilate the artery
Sublingual isosorbide dinitrate to dilate the artery

Secondary prevention of CVD

20
Q

What is the treatment for GCA?

A

Temporal artery biopsy and high dose steroids

21
Q

What is retinal detachment?

A

where the retina separates from the choroid underneath. This is usually due to a retinal tear that allows vitreous fluid to get under the retina and fill the space between the retina and the choroid

22
Q

What are the risk factors of retinal detachment?

A
Posterior vitreous detachment
Diabetic retinopathy
Trauma to the eye
Retinal malignancy
Older age
Family history
23
Q

What is the presentation of retinal detachment?

A

Dense shadow that starts peripherally progresses towards the central vision
A veil or curtain over the field of vision
Straight lines appear curved
Central visual loss

24
Q

What is the management of retinal detachment?

A

Immediate referral
For tears:
Laser therapy
Cryotherapy

For detachment: vitrectomy, scleral bulking

25
What is PVD?
Posterior vitreous detachment is a condition is where the vitreous gel comes away from the retina. It is very common, particularly in older patients.
26
What is the presentation of PVD?
Painless Spots of vision loss Floaters Flashing lights
27
What is the management of PVD?
No treatment neccessary Over time symptoms improve as brain adjusts Essential to exclude retinal detachment / tear
28
What are the binocular causes of APLV?
Optic chiasm pathology Optic nerve pathology migraine
29
What is the appearance of papilloedema on fundoscopy?
Venous engorgement, loss of venous pulsation, blurring of optic disc margin, elevation of optic disc Loss of optic cup Paton’s lines from optic disc