Anatomy & Physiology Muscle Study Flashcards

(41 cards)

1
Q

Three Types of Muscles

A
  • Smooth
  • Cardiac
  • Skeletal
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2
Q

Smooth Muscle

A

Located- digestive tract, blood vessels, bladder, airways, uterus.
Non-striated
Involuntary (contracts naturally)

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3
Q

Cardiac Muscle

A

Located- Only in the heart
Involuntary (contracts naturally)

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4
Q

Skeletal Muscle

A

Attaches to bone & causes movement in body.
Appears stripped under a microscope.
Voluntary ( contrast at will)

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5
Q

Muscle Fibers

A
  • Long, thread-like appearence.
  • Muscle cells in these fibers contain MULTIPLE NUCLEI pressed against side of plasma membranes.
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6
Q

Muscle Fibers contain ______ Structures:

A

Sarcolemma - cell membrane

Myofibrils - fundamental role in muscle contraction

Sarcoplasmic Reticulum ( endoplasmic reticulum) - activates muscle contraction mechanism when a muscle fiber is stimulated.

Myofilaments - proteins that for light & dark bands that makes muscle appear striated.

Transverse tubules - opens to the outside of muscle fiber & contain extracellular fluid, activates the muscle contraction mechanism when muscle fiber is stimulated.

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7
Q

Muscle Attachment

A

Skeletal muscle may appear in 1 of 2 ways

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8
Q

Direct Attachment

A

Muscle Fibers merge w/ the periosteum of bone to form a strong attachment.

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9
Q

Indirect Attachment

A

Epimyosium extends past the muscle as a tendon, which merges w/ the periosteum.

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10
Q

Muscle Movement

A
  • Prime movement
  • Synergists
  • Antagonists
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11
Q

Prime mover

A

Main muscle triggering the movement

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12
Q

Synergists

A

Muscles that assist the Prime Mover.

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13
Q

Antagonists

A

Muscles that oppose that action of the Prime Mover. Typically works to moderate speed/ range of movement to prevent injury.

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14
Q

How Muscles and Movement Work Together

A
  • When the Prime Mover contracts, the Antagonist must relax to give the Prime Mover control.

-Prime Mover for one movement = Antagonist for opposite movement.

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15
Q

Hypertrophy

A

Enlarging muscles ( ex. weight lifting).

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16
Q

Atrophy

A

Shrinking muscles (from lack of use).

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17
Q

Aerobic

A

Endurance (endurance exercise) stimulates the growth of blood vessels in the muscle. ( increases supply of oxygen & glucose for ATP production)

18
Q

Slow Twitch (Type 1)

A

a slower reaction to stimuli.
- contains a lot of mitochondria & rich blood supply for efficiency at using oxygen to generate ATP of energy
- responds slower but fires for a longer time before becoming fatigued.

19
Q

Fast Twitch (Type 2)

A

Better @ generating short bursts of speed/strength.
- Smaller amts of mitochondria & a poorer supply of blood, but can absorb/release calcium quickly.
- fatigues quicker than twitch fibers.

20
Q

Muscle Fatigue

A

Muscle loses ability to contract during strenuous exercise = fatigue.
- arises usually from electrolyte imbalances & decreased ATP levels
- muscle cramps are due to lack of ATP required to return calcium ions back to the Sarcoplasmic reticulum so muscle fibers can relax.

21
Q

Muscles are named according to:

A
  • Size
  • Shape
  • Location
  • Number of origins
    -Direction of muscle fibers
    -Action/Function
22
Q

Size of Muscles:

A

MAXIMUS- largest

MINIMUS- smallest

MAJOR- large

MINOR- small

LONGUS- longest

BREVIS- shortest

23
Q

Shape of Muscles

A

DELTOID- triangular

RHOMBOID- diamond shaped

SERRATUS- sawtoothed

TRAPEZIUS- trapezoidal

24
Q

Pectoralis

25
Brachio
Upper arm
26
Gluteus
Buttock
27
Fermoris
Femur
28
Sterno
Sternum
29
Cleido
clavicle
30
Mastoid
mastoid process
31
Digiti
finger or toe
32
Pollicis
thumb
33
Number of Origins
Biceps- 2 origins Triceps- 3 origins Quadriceps- 4 origins
34
Direction of Muscles
Rectus- straight Transverse- across Oblique- diagonal
35
Action of Muscles
Adductor- adducts Abductor- abducts Flexor- flexes Extensor- extends Leviator- elevates
36
Muscle Contraction
1) impulse reaches end of motor neuron, causing small vesticles to fuse w/ cell membrane & release a neurotransmitter: ACETYLCHOLINE (ACh) into the synaptic cleft. 2) ACh quickly diffuses across synaptic cleft, stimulating receptors in the sarcolemma, sending an electrical impulse over the sarcolemma & inward along the T-Tubules. - causes the sacs in the sarcoplasmic reticulum to release calcium. 3) Calcuim binds with the tropin on the actin filament to expose attachment points. - in response, the myosin heads of thick filaments grab onto thin filaments and muscle contraction occurs.
37
Muscle Relaxation
1) when nerve impulses stop arriving @ neuromuscular junction, ACh is no longer released. 2) An enzyme breaks down any remaining ACh while calcium ions are pumped back into the sarcoplasmic reticulum. 2) w/ calcium removed, troponin and tropomyosin again prevent the myosin heads from hooking onto thin filaments & muscle fiber relaxes.
38
Calcium & ATP
*calcium in required to prevent tropmyosin & tropin ( 2 protein molecules entwined in actin filament) from blocking the sites where the myosin head attaches. *ATP provides energy which allow myosin heads to release their grip on actin filaments. The ATP splits, giving the myosin fuel to form new cross bridges.
39
Strength of contraction
The nervous system controls strength of a contraction by: Altering the FREQUENCY of the stimulus & altering the INTENSITY of the stimulus.
40
Stimulus Frequency
- When a muscle contracts several times in a row, the last contraction will be stronger than the first. (the sarcoplasmic reticulum doesn't have time to completely reabsorb the calcium) - When the impulse releases the muscle fibers even faster. the fibers don't have a chance to completely relax before the next impulse arrives. ( as a result, each subsequent contraction builds on the previous one.)
41
Stimulus Intensity
A strong stimulus may stimulate all fibers in a motor nerve, causing nerve fibers to call on their accompanying muscle fibers to contract. In contrast, a weak stimulus stimulates only a few nerve fibers, which creates a weaker response.