Biological approach to explaining OCD Flashcards

1
Q

strength of the genetic explanation

A
  1. research support
    - evidence from sources which suggest that some people are vulnerable to OCD as a result of their genetic makeup
    - Nestadt et al (2010) reviewed twin studies and found that 68% of identical twins shared OCD as opposed to 31% of non identical twins
    - research has found a person with a family member diagnosed with OCD is 4 times as likely to develop it as someone without
    - these research suggest there must be some genetic influence in the development of OCD
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2
Q

limitation of the genetic model

A
  1. environmental risk factor
    - strong evidence for the idea that genetic variation can make a person more or less vulnerable to OCD
    - OCD does not appear to be entirely genetic in origin and environmental risk factors can trigger the risk of developing OCD
    - in a study by Cromer et al (2007) found that over half the OCD clients in the sample had experienced a traumatic event in their past
    - genetic vulnerability only provides a partial explanation for OCD
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3
Q

strength of the neural model of OCD

A
  1. research support
    - antidepressants that work purley on serotonin are effective in reducing OCD symptoms
    - suggest that sertonin amy be involved in OCD
    - OCD symptoms form parts of conditions that are known to be biological in origin such as parkinson’s disease which causes muscle tremors and paralysis
    - if a biological disorder produces OCD symptoms then we may assume the biological processes underline OCD
    - suggests that biological factors may also be responsible for OCD
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4
Q

limitation of neural model

A
  1. no unique neural system
    - serotonin OCD link may not be unique to OCD
    - may people with OCD also experience CLinical depression (co morbidity)
    - depression involves disruption to the action of serotonin
    - could simply be that serotonin activity is disrupted in many people with OCD because they are depressed as well
    - serotonin may not be relevant to OCD symptoms
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5
Q

the role of serotonin in neural explanation

A
  • neurotransmitter serotonin helps to regulate mood
  • neurotransmitters are responsible for relaying information from one neuron to another
  • if a person has low levels of serotonin then normal transmission of mood relevant information does not take place and a person may experience low moods
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6
Q

Decision making systems in neural explanations

A
  • some cases of OCD seem to be associated with impaired decision making
  • this is associated with abnormal functioning the the lateral of the frontal lobes of the brain
  • frontal lobes are responsible for logical thinking and decision making
  • evident to suggest that the left parahippocampal gyrus is associated with processing unpleasant emotions which functions abnormally in OCD
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7
Q

Different types of OCD

A
  • aetiologically heterogeneous = the origins of OCD vary from person to person
  • evidence to suggest that different types of OCD may be the result of particular genetic variations
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8
Q

OCD is polygenic

A
  • OCD is not caused by one single gene by a combination of genetic variations that together significantly increase vulnerability
  • Taylor (2013) found that there are up to 230 different genes involved in OSC
  • genes are associated with the actions of dopamine and serotonin
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9
Q

candidate genes

A
  • create vulnerability for OCD
  • some of these are involved in regulating the development of the serotonin system
  • 5HT1-D beta gene is implicated in the transport of serotonin across synapses
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10
Q

genetic explainations

A
  • lewis (1936) observed that of his OCD patients 37% had parents with OCD and 21% had siblings with OCD
  • suggest that OCD runs in the family
  • genetic vulnerability is passed on from one generation to the next
  • according to the diathesis stress model certain genes leave some people more likely to develop a mental disorder
  • some environmental stress is necessary to trigger the condition
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