Blonder Flashcards

1
Q

ASD

A

opening in intra-atrial septum

most common - secundum

closure - catheter device

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2
Q

patent foramen ovale

A

foramen covered by septum primum not sealed shut in 20% of normal subjects

bubble study on echo

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3
Q

over time ASD

A

resistance - stiff and non-compliant

then pressure increases

pressure follows resistance

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4
Q

myxomatous mitral valve

A

mitral valve prolapse syndrome

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5
Q

most common septal defect

A

bicuspid AV

ASD is second**

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6
Q

ASD

A

usually asymptomatic until adult - surgery by age 40

atrial arrhythmia, paradoxical emboli, cerebral abscess, right heart failure, pulmonary HTN > eisenmanger syndrome

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7
Q

ASD at bottom of septum

A

primum

-worse

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8
Q

sinus venosus ASD

A

least common
-cannot be closed percutaneously

two types
-superior (SVC) and inferior (IVC)

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9
Q

scimitar syndrome

A

partial anomalous venous return

hypoplasia of lobe of right lung

thoracic aorta > pulmonary artery collaterals

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10
Q

lesions <8mm without symptoms

A

larger lesions enlarge with age

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11
Q

pulmonary HTN and eisenmenger syndrome

A

require >2.5:1 shunt

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12
Q

wide fixed split S2

A

ASD - splitting stays equal

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13
Q

increased P2

A

pulmonary HTN

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14
Q

S1 split

A

tricuspid component

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15
Q

upper left sternal border murmur

A

systolic ejection murmur

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16
Q

most common congenital heart disease at birth

A

VSD

spontaneous closure

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17
Q

infundibular VSD

A

below aortic and pulmonic valves

leading to progressive aortic regurgitation, the hallmark

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18
Q

membranous VSD

A

conoventricular

deficiency of membranous septum

19
Q

inlet defect VSD

A

AV canal

down’s

20
Q

muscular VSD

A

in trabecular system

21
Q

small/restrictive VSD

A

orifice diameter < or = 25% aortic annulus diameter

no LV volume overload
no pulmonary HTN

22
Q

moderate size VSD

A

orifice 25-75% diameter

mild-moderate volume overload

23
Q

large VSD

A

orifice 75% diameter

moderate L>R shunts with LV volume overload

pulmonary HTN
> eisenmenger

24
Q

tetralogy of fallot

A

VSD
aorta overrides
concentric RV hypertrophy
RVOT obstruction

25
causes of AV block
-vagal tone increase -fibrosis of conduction system (50%) -IHD -cardiomyopathy and myocarditis -congenital heart disease familial AV block (auto dom)
26
latrogenic
Dr. caused AV block -digitalis, non-DHP CCBs, beta blockers, amiodarone, adenosine cardiac surgery catheter alcohol septal ablation
27
first degree AV block
PR interval >0.2 decreased CO
28
progressive PR interval prolongation
mobitz type I second degree AV block
29
PR interval unchanged, but get P wave that doesn't conduct
mobitz type II second degree AV block
30
P and QRS dissociation
third degree AV block usually need a pacemaker
31
rheumatic heart disease
mitral valve stenosis
32
indication for pacing
wide QRS
33
most common arrhythmia
atrial fib more in men, increases with age reduced CO - decreased diastolic filling time
34
risk factors for A fib
hypertensive heart disease CHD RF
35
AF that terminates spotaneously or within 7 days, may recur
paroxysmal AF
36
AF that fails to terminate within 7 days
persistent require pharm or cardioversion
37
AF longer than 12 months
long standing persistant
38
patients with persistant AF where joint decision is made to no longer control rhythm
permanent AF
39
palpitations, syncope, dyspnea, fatigue
AF
40
rate control for AF
patient anti-coag | -rate controlled by AV blockers
41
rhythm control for AF
anticoag and restore NSR by meds -or electrical cardioversion TEE used to rule out LA thrombus prior to cardioversion
42
CHA2DS2 - Vasc
``` CHF HTN Age 65-74 1 point >75 2 points DIabetes Stroke 2 points ``` Vasc disease Hx +1 Female +1 all 9 15% per year for A-fib stroke risk
43
PVCs
with syncope = serious** very common complaint - not really that big of a deal - unless with acute coronary syndromes or MI reassurance is mainstay of treatment beat after pause - very forceful