Brainstem Disorders Flashcards

(40 cards)

1
Q

what is involved in a MEDIAL BS lesion?

A

motor tracts

MLF

medial lemniscus

motor nuclei (divide into 12)
- CN 3, 4, 6, 12

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2
Q

what is involved in a LATERAL (side) BS lesion?

A

sympathetic (hypothalamospinal) tract -Horner’s syndrome

spinothalamic tract

spinocerebellar tract

sensory nuclei (don’t divide into 12)
- CN 5, 7, 8, 9, 10

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3
Q

if there is a lesion to the lateral corticospinal tract or DCML above the caudal medulla, where is the loss? why?

A

contralateral signs bc the damage is after the decussation

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4
Q

if there is a lesion to the lateral cortiocospinal tract or DCML below the caudal medulla, where is the loss? why?

A

ipsilateral signs bc the damage is b4 the decussation

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5
Q

if there is any BS damage to the spinothalamic tract, where is the loss? why?

A

contralateral signs bc of the immediate decussation so damage is after decussation

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6
Q

if there is a lesion of the L rostral anteromedial medulla, what is the loss?

A

paresis of the R hand and foot

loss of light touch and proprioception on the R side of the body

paresis on the L side of the tongue

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7
Q

how do you differentiate the involvement of the motor tract or the motor cranial neurons?

A

ask the pt to close their eyes

hypertonicity vs hypotonicity

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8
Q

what supplies blood to the midbrain?

A

posterior cerebral artery

basilar artery

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9
Q

what supplies blood to the pons?

A

anterior inferior cerebellar artery (AICA)

basilar artery

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10
Q

what supplies blood to the medulla?

A

posterior inferior cerebellar artery (PICA)

vertebral artery

anterior spinal artery

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11
Q

what are the cardinal signs of BS dysfxn? (KNOW THIS)

A

the 4 Ds:
1) dysphagia
2) dysarthria
3) diploplia
4) dysmetria

disorders of vital fxns

disorders of consciousness

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12
Q

why does BS dysfxn cause dysphagia and dysarthria?

A

the CNs are in the pons and medulla of the BS

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13
Q

why does BS dysfxn lead to diploplia?

A

CN 3, 4, 6 control eye movement and they are in the BS

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14
Q

damage to what structure in the BS can cause disorders of consciousness?

A

reticular formation

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15
Q

what is vertebrobasilar artery insufficiency?

A

causes transient symptoms of BS ischemia reproduced with cervical extension and rotation

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16
Q

vertebrobasilar artery insufficiency is characterized by poor blood flow to what part of the brain?

A

the posterior brain

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17
Q

what are the symptoms associated with BS ischemia?

A

abrupt onset of neurologic symptoms (dizziness, weakness, incoordination, and somatosensory dysfxn)

sustained ischemia=Wallenberg syndrome, loss of consciousness

18
Q

what is the most common BS stroke?

A

Wallenburg syndrome

lateral medullary lesion

19
Q

what is Wallenburg syndrome?

A

a LATERAL medullary lesion

PICA affected

very parasympathetic

spinal trigeminal tract: loss of nociception and temp from the ipsilateral face

spinothalamic tract: loss of nociception and temp from contralateral body

spinocerebellar tract: ipsilateral ataxia

descending sympathetic tract: ipsilateral Horner’s syndrome

20
Q

does Wallenburg have contra or ipsi s/s?

21
Q

what CNs are involved in Wallenburg syndrome?

A

5, 7, 8, 9, 10, 12

22
Q

what are the CN s/s associated with Wallenburg syndrome?

A

vertigo, nystagmus, problems swallowing and speaking, hoarse voice, digestion issues, inability to slow the HR

23
Q

what CNs are involved in AICA syndrome?

25
what is AICA syndrome
lateral caudal pons lesion AICA affected spinal trigeminal tract: loss of nociception and temp from the ipsilateral face spinothalamic tract: loss of nociception and temp from contralateral body spinocerebellar tract: ipsilateral ataxia descending sympathetic tract: ipsilateral Horner's syndrome
26
what is the 2nd most common BS stroke?
lateral caudal pons lesion
27
are the s/s of AICA syndrome ipsi or contra?
both
28
what are the CN s/s associated with AICA syndrome?
loss of efferent limb of corneal reflex and stapedius reflex (sounds are louder), lack of tears and salivation, unilateral deafness, vertigo, nystagmus, nausea, vomiting
29
what is an anteromedial midbrain stroke?
the most common midbrain stroke involved the basilar artery contra signs (except CN 3) med lemniscus: contra loss of sensation red nucleus: motor dysfxn obscured by ataxia; pathologic laughing/crying motor tracts (corticopontine): contra cerebellar ataxia; (corticospinal and corticobrainstem): contra paresis of the body and lower face MLF: internuclear opthalmoplegia obscured by CN 3 lesion
30
what is internuclear opthalmoplegia?
damaged connection bw CN 6 and 3
31
what CNs are involved in anteromedial midbrain stroke?
3
32
what are the CN signs associated with an anteromedial midbrain stroke?
pupil dilation from lack of pupillary sphincter innervation inability to focus on near objects unable to move eye up, down, or in double vision
33
what structures may be involved in disorders of consciousness?
cerebrum (thalamic or hypothalamic) and BS (reticular formation)
34
what is a coma?
unarousable state no response to strong stim lost vital fxns (vent is necessary)
35
what is a vegetative state?
cycle of sleeping and waking (can yawn and vocalize) unresponsive wakeful state complete loss of consciousness w/o alteration of vital fxns (don't need a vent)
36
what is a minimally conscious state?
respond to very painful stim and follow very easy instructions some signs of consciousness in and out of consciousness
37
tumors in the cerebellum or BS cause an increase in what?
intracranial pressure
38
what are the symptoms associated with increased ICP?
headache, nausea, vomiting, CN dysfxn, and/or hydrocephalus possible ataxia
39
what is the most common BS tumor?
acoustic neuroma
40
what is an acoustic neuroma?
a benign tumor of the Schwann cells surrounding CN 8 unilateral vestibular lesion