Brainscape's Knowledge GenomeTM


Top Flashcards (Certified)
All Flashcards

Cardiology > CAD > Flashcards

CAD Flashcards

(85 cards)

Start Studying
1
Q

Chronic coronary syndrome can be caused by

A

Endothelial dysfn, epicardial artery atherosclerosis,coronary spasm, microvascular pathology

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Rupture of atherosclerotic plaque doesn’t cause

A

Chronic coronary syndrome

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Typical anginal pain in CCS

A

Pressure/ tightness in chest irradiating to neck ,mandible ,left arm for 2 to 5 min

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Typical angina provoking factors

A

Exercise,cold air, eating

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Typical angina relieving factor

A

Rest and nitroglycerin

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Major CAD risk factor

A

AH, smoking, sedentary lifestyle, hypercholesterolemia,( alcohol abuse is not a major one)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Stable angina class determining criteria

A

Physical exercise tolerance

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Angina fn class , symptoms appear on climbing 3 rd floor or walking 600-700m

A

2

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What is not an first line examination for stable coronary artery disease

A

CT angiography

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Drugs that relieve pain in chronic coronary syndrome

A

Beta blockers,short acting nitrates,ranolasine

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Drugs used for prevention of cardiovascular recurrent events prevention in CCS

A

Statins and aspirin

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Target LDL for pat with cardiovascular disease

A

Less than 1.4

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

In case of suspicion of coronary artery disease,stress ECG must be stopped if

A

Cardiac pain or shortness of breath,ECG,st segment deviation more than 2 mm,SBP increase to 190, Frequent extrasystole and appearance of polymorphic extrasystole

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Should we stop stress ECG if HR is 115

A

No

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Which CAD form more often cause chronic cardiac failure

A

Old MI

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

What type of CCS is divided into 4 class

A

Stable angina

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

What angina can be treated ambulatory

A

Stable angina

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Coronary artery spasm is the main mechanism of development of

A

Prinzmetal s Angina

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

What characteristic of pain is typical for MI

A

Burning type, irradiating on both hands,more intensive when walking,provocated by emotion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Is short stabbing pain typical for MI

A

No

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Stable angina most often last for

A

2 to 3 min

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

Most common pain localization in angina pectoris attack

A

Retrosternal

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

Typical characteristics for angina

A

Pain occurs due physical exercise, retrosternal pain,pain is relieved by nitroglycerin,attacks mostly occur in cold and windy weather

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

The main methods to diagnose painless MI

A

Holter monitoring,stress ECG test

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Main effect of nitroglycerin for anginal pain is
Peripheral venous system dilation
26
Most significant in ACS pathogenesis is
Plaque instability and rupture
27
Diagnostic marker for MI necrosis is
Troponin test
28
Mex time from the first medical contact and analysing ECG for pat with suspected ACS
10 min
29
The most effective treatment method for pat with ACS with ST segment elevation is
Percutaneous coronary intervention ( PCI)
30
Absolute CI to fibrinolysis are
GIT bleeding within past month, Ischemic stroke in the preceding 6 month
31
Door to balloon time means
Time from pat admission to hospital until catheter guidewire insertion in coronary blood vessels
32
What is expected delay from STEMI diagnosis to primary PCI to choose PCI strategy over fibrinolysis
Less than 120 min
33
Fibrinolytic therapy done when
Timely primary PCI can not be performed and fibrin specific agents are recommended
34
Most typical criteria for MI
Increased Troponin level
35
High risk criteria of recurrent ishemia and cardiovascular death for ACS without STs elevation are
Grace score more than 140,ECG: dynamic ST depression, Established N STEMI diagnosis
36
First choice treatment for patients with high risk ACS without ST segment elevation
PCI
37
P2 Y 12 inhibitor preferred for ACS with ST segment elevation undergoing primary PCI
Prasugrel
38
Max recommend time from successful fibrinolysis to coronary angiography with secondary PCI for stable patients are
Within 24 hrs
39
Anti thrombotic agents which are not recommended for treatment of unstable angina
Warfarin and alteplase
40
Duration of use of dual antithrobotic therapy after Acute MI for pat with sinus rhythm
12 month
41
Drugs recommend to use after acute anterior STEMI for secondary prevention of recurrent cardiovascular events
Aspirin, Beta blocker,statins,ACE I,
42
When is recommended to repeat troponin test if first one is negative for pat with suspected ACS without ST s elevation
After 1 hour
43
Score used to assess intra hospital mortality risk of pat with ACS without ST segment elevation
GRACE
44
Very high risk criteria of recurrent ischemia in ACS without ST segment elevation are
Cardiogenic shock or hemodynamic instability, recurrent or ongoing chest pain refractory to medical treatment
45
ACS without ST segment elevation with stable hemodynamic,ST segment depression in ECG and positive troponin test,how fast should we perform coronary angiography
Within 24 hr
46
Which artery branches into circumflex artery and left anterior descending artery
Left main coronary artery
47
Description of pat with non STEMI
Acute onset rest angina with normal ECG and elevated cardiac biomarkers
48
ECG patterns in unstable angina / NSTEMI
Wellens phenomena ( deeply inverted/ biphasic T wave in anterior precordial leads), Isolated T wave flattening or inversion, horizontal ST segment depression,Normal ST segment and T waves
49
Most common pathophysiological mechanism present during STEMI
Coronary plaque rupture
50
Diagnosis of NSTEMI should be based on
A combination of clinical history, symptoms,ECG and high sensitive cardiac troponin
51
ESC algorithm to measure high sensitive cardiac troponin
0h/1h or as an alternative 0h /2h
52
Score which should be considered for prognosis estimation
GRACE
53
It is not recommended to administer routine 02Y 12 I in patients with NSTEMi ,
Whom coronary anatomy is unknown and early invasive management is planned
54
In vet high risk NSTE- ACS pat irrespective of ECG or biomarker findings ----+ is recommended
Immediate invasive strategy ( less than 2 HR)
55
Early invasive strategy (<24 hr)in NSTE-ACS is indicated in
Pat with dynamic ST change and or GRACE score more than 140
56
Low risk NSTE- ACS patients should be
Selectively scheduled for coronary angiography after a positive non invasive test
57
Very high risk NSTE-ACS criteria which need immediate invasive strategy
Heamodynamic instability or cardiogenic shock,life threatening arrhythmias, mechanical complications of MI,presence of ST depression more than 1 mm in>or equal to 6 leads additional to ST segment elevation in aVR and or V1
58
High risk criterias needed to early invasive strategy (<24 hr) are
Diagnostic of NSTEMI suggested by diagnostic algorithm,new contiguous ST/ T changes suggesting ongoing ischemia,transient ST elevation
59
Is positive stress test included in universal definition of 1 st type MI
No
60
Medical treatment of CAD include
Oral medication administration
61
We can't determine the location of MI using
Cardiac enzymes
62
Tests used to diagnose CAD
ECG, treadmill stress test,cardiac catheterization
63
Post MI complications,in Cardiac intensive care unit resting time indicate
Free wall rupture
64
A pat with Killip class 3 has
Pulmonary edema
65
Pat with heart failure killip class 2 has
Mild to moderate HF with S3 gallop,less than half way up lung field or elevated jugular venous pressure
66
Killip class 4 has
Cardiogenic shock defined as SBP less than 90 and signs of hypo perfusion such as oliguria,cyanosis, sweating
67
Killip class 1
No evidence of HF
68
Rescue percutaneous therapy done when
Fibrinolytic therapy fails
69
Beta blockers are not used in STEMI if there is
Pulmonary edema
70
Most common complications after acute MI is
Arrhythmias
71
Typical anginal pain is
Substernal chest discomfort of characteristic quality ( pressure like,dull), provoked by physical exertion and relieved by nitrates and restwithin minutes
72
Signs of unstable angina
Rest angina for prolonged period ( >20 min),new onset angina class 2 or 3,crescendo angina
73
Non invasive techniques to assess coronary artery are
CT angiography and CT with coronary calcium scoring
74
ECG bof posterior MI
Isolated ST depression more than.5 mm in lead V1 to V3
75
Conditions other than MI with Troponin level elevation are
HF,Myocarditis, aortic dissection,critical illness alike shock,sepsis,Tachyarrythmias, acute neurological event
76
P2 Y 12 inhibitors used in ACS
Clopidogrel,Ticagrelor, Prasugrel
77
CI for fibrinolysis are
Aortic dissection, Known bleeding disorders,CNS neoplasm,Time from chest pain onset>12 hr
78
Dual antiplatelet therapy after Acute MI is used for
12 months
79
Mechanical complications after acute MI are
Rupture of papillary muscle,free wall rupture interventricular rupture
80
LVEF<35% 90 days after MI ,QRS more than 130 + LBBB, treatment strategy
CRT-D
81
Most common MI complications
Rhythm disorder
82
If one month after MI sT elevation persist,then we must think about
Development of heart aneurysm
83
The sudden death at the 3 rd to 4 th day of acute MI is related to
Heart free wall rupture
84
Which treatment is less important fo emergency treatment of MI induced pulmonary edema ( furosemide,morphine,Digoxin,nitrogl)
Digoxin
85
Indication for intra aortic counterpulsation in cardiology intensive care practice
The method used in case of MI mechanical complications

Cardiology (8 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2025 Bold Learning Solutions. Terms and Conditions