carb oral topics Flashcards

(28 cards)

1
Q

lactate in meat production

A

endproduct of anaerobe glycolysis

  • short starvation: lots of glycogen and lactate in muscle
  • stress: few glycogen and lactate in muscle (incr. pH, dark meat)
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2
Q

cori cycle

A

cycle of secretion and uptake of glucose in mammals

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3
Q

complex 1 of resp chain

A

NADH dehydrogenase
generate 4 protons to intermembrane space
transport 2 to complex Q

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4
Q

complex 2 of resp chain

A
succinate dehydrogenase (TCA)
no protons are pumped out
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5
Q

complex 3 of resp chain

A

cytochrome c oxidoreductase

  • subunits: cytochrome C1, bL, bH, Fe-s, Co-Q
  • has co-enzyme Q binding spot, binds for 2H⁺ transfer via Fe-s
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6
Q

complex 4 of resp chain

A

cytochrome c oxidase

  • subunits: cytochrome a, a3, and +2Cu
  • generate water molecule
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7
Q

complex 5 of resp chain

A

aka ATP synthetase, uses proton motive force of translocated protons to form ATP in the matrix

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8
Q

H⁺ affect the complexes how

A

reduces them

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9
Q

adrenalin: effect, production and affected pathways

A

hyperglycemia
andrenal medulla
muscle: incr. glycogen degredation from several starting prods (incr. GGL, decr. GG)

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10
Q

glucagon: effect, production and affected pathways

A

hyperglycemia
Pancreas A cells
liver: incr. glucose prod and decr. its degradation (incr. GGL, GNG, decr. GL, GG)

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11
Q

insulin: effect, production and affected pathways

A

hypoglycemia
Pancreas B cells
incr. glucose uptake, decr. fat+protein degradation (incr.GG, GL, decr. GGL, GNG)

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12
Q

glucocorticoids: effect, production and affected pathways

A

hyperglycemia
adrenal cortex
increased glucose production (no glucagon degr.) (incr. GNG, decr. GL)

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13
Q

ACTH: effect, production and affected pathways

A

hyperglycemia
adenohypophysis
incr. glucocorticoids

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14
Q

STH: effect, production and affected pathways

somatotropin aka GH

A

hyperglycemia
adenohypophysis
lipolysis for glucose prod, decr. GNG from aa

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15
Q

blood sugar levels, regulation

A

= transport form of carbs,
using glucagon (working in liver) to incr. BS
using insulin (working in fat tissue) to decr. BS
–> restore normal BS levels

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16
Q

BS levels of birds, ru and non-ru

A

birds: 8-9mmol/L
non-ru: 4-5mmol/L
ru: 2-3mmol/L

17
Q

diabetes

A

type 1: low insulin production, born with it

type 2: receptors are “worn out”, caused by diet (insulin independant)

18
Q

GLUT transporters

A

GLUT-1: RBC, brain
GLUT-2: Liver, kidney, intestines
GLUT-3: brain
GLUT-4: muscle and adipose tissue (insulin dependant)

19
Q

lactolysis

A

small intestines, lactase

endprod: galactose and glucose

20
Q

galactose degr. endprod used in

A

UDP-glucose:

  • lactogenesis
  • chondroitin synthas
  • glycogenesis
21
Q

composition of milk

A

13% dry matter

  1. 8% milk sugar/lactose
  2. 5% milk fat
  3. 3% milk protein
22
Q

where is milk fat from

A

(feed, microbial FA, de novo synth in adipose tissue and mammadry gland)

23
Q

milk protein

A
mammary gland origin
 -  2.6% - casein
 -  0.1% - alpha-lactalbumin
 -  0.3% - beta-lactalbumin
 systemic synth in liver -> blood plasma
 -  0.1% - milk serum albumin and immunoglobulins (colostrum!!)
24
Q

colostrum

A

First form of milk immediately following delivery
Most species will generate colostrum just prior to giving birth. Colostrum contains antibodies to protect the newborn against disease.

25
how does the diet of a cow impact the lactose and fat % of the milk
Starch/grain rich: - GNG: increases milk sugar and decreases milk fat Fiber/cellulose rich diet: - more acetate, milk fat% increases
26
fiber/cellulose rich diet "pathway"
intensive salvia prod(buffer pH 8.2-8.4) -> increased pH to ish6.8. increased pH stim the microbes to make acetate, which produces FA -> milk Fat decr. and incr. milk sugar (lactose)
27
grain/stach rich diet "pathway"
less intensive salvia prod(buffer pH 8.2-8.4) -> decr pH to ish5.8 decr pH stim the microbes to make propionate, which is used in GNG for glucose prod -> milk fat increases
28
casein
phophoprotein -> casein micelle - carry large amounts of highly insoluble CaP to mammalian young in liquid form and to form a clot in the stomach for more efficient nutrition.