cardiac muslce heart pump and contraction ch 9 Flashcards Preview

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Flashcards in cardiac muslce heart pump and contraction ch 9 Deck (46)
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Name the 2 heart pumps and where they pump

right pump- pulmonary circulation,
left pump- peripheral circulation


What is Cardiac rhythmicity?

sequence of cardiac contractions


name the muslce types in the heart

atrial, ventricular, specialized excitatory and conductive fibers


whats special about the atrial and ventricular muslce fibers?

they contract longer than skeletal muslce


what are intercalated discs?

connections between heart muscle cells. they have gap junctions


What does the AV bundle do?

transmits conduction from atrium synctium to ventricular. It allows the atria to contract before the ventricles


Cardiac action potential

~105 millivolts, The intracellular potential rises from -85 to +20 mv during each beat. The membrane remains depolarized for about .2 seconds( plateau)


Whats the reason for the plateau in action potential?

allows the ventricular contraction to be as much as 15 times longer than skeletal muscle


what allows the cardiac fibers to have a prolonged AP plateau?

instead of just sodium channels opening they have calcium-sodium channels ( slow calcium channels) The slow channels maintain the depolarization period which causes the plateau


what else allows the cardiac fibers to have a prolonged AP and plateau?

after the onset of the AP the permeability of cardiac fibers for potassium ions decreases about 5 fold which doesnt happen in skeletal muscle. This decreases the outflux of potassium ions which prevents early return.


Conduction speed of Cardiac cells

.3-.5 seconds. This is slower than nerve fibers and skeletal fibers


whats a problem with Tachycardia in regards to the atria?

the fast heart beat doesnt allow the atria to fill the ventricles quick enough.


P wave

depolarization through the atria and followed by atrial contraction. This causes a slight rise in atrial pressure


QRS waves-

depolarization of Ventricles and causes the ventricular pressure to begin rising


T wave-

repolarizing of the Ventricles and ventricular muslce starts to relax


Atria as primers

~80 % of blood flows directly through ventricles even before contraction. The Atria simply act as primers that increase ventricular pumping effectiveness as much as 20%.


A wave-

Atrial contraction


C wave

ventricles begin to contract and blood backflows into atria


V wave

occurs toward the end of ventricular contraction. This is when blood is slowly filling the atria while the AV valves are closed


Period of rapid filling of ventricles

blood pushes AV valves open and fills the ventricle during the first 1/3 of diastole. Middle 1/3 of diastole involves blood straight from the veins into the ventricles. Last 1/3 of diastole is when the atria contract accounting for an additional 20% of filling


isovolumic contraction

period between when the AV valves close and the ventrilces contract and push the semilunar valves open.


End diastolic volume

normal filling of ventricles. about 110-120 ml


stroke volume output

when ventricles contract and empty to about 70ml


end systolic volume

remaining volume in each ventricle after contraction. about 40-50 ml.


ejection fraction

fraction of end diastolic volume that is ejected from the heart( jusually about 60%)


in regards to end diastolic and end systolic volume, how can stroke volume increase?

increase end diastolic and decrease end systolic volume


Semilunar valves functions

Pressure from arteries close them rapidly.


first heart sound

closure of AV valves. Low pitch and long lasting


second heart sound

aortic and pulmonary valves closing. High pitch and fast


stroke work output

amount of energy the heart converts to work during each heartbeat