Cardio 1 Flashcards

(48 cards)

1
Q

Rhythm originates from Sinoatrial Node;

Atrial & Ventricular rates are

A

60-100 b/m

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2
Q

Multiple rapid Impulses form many Foci Depolarize in the Atria in a Totally Disorganized Manner at a Rate of 350-600 times/min

A

Atrial Fibrillation

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3
Q

Atria Quiver, Can lead to formation of Thrombi

A

Atrial Fibrillation

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4
Q

No definitive P -Wave, only fibrillatory waves before each QRS

A

Atrial Fibrillation

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5
Q

Oxygen, Anticoagulants (risk of emboli), cardiac meds to control ventricular rhythm & assist in maintenance of CO, Cardioversion, meds to control dysrhythmia.

A

Atrial Fibrillation

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6
Q

Early Ventricular Complexes result from Increased Irritability of the Ventricles

A

PVCs

Premature Ventricular Contractions

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7
Q

Can occur in repetitive patterns: bigeminy (q other Hbeat), trigeminy (q 3rd Hbeat), quadrigeminy (q 4rth Hbeat); Or Unifocal (Upward/Downward deflection), Multifocal (diff shapes, impulses in different sites)

A

PVCs

Premature Ventricular Contractions

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8
Q

Evaluate SpO2 to assess for Hypoxemia

A

Can cause PVCs

administer O2

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9
Q

Hypokalemia

A

Can cause PVCs

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10
Q

Repetitive Firing of an Irritable Ventricular Ectopic focus at a rate of 140-250 bpm

A

VT

Ventricular Tachycardia

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11
Q

May present as a Paroxysm of 3 self-limiting beats or more or may be a sustained rhythm

A

VT

Ventricular Tachycardia

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12
Q

Can lead to Cardiac Arrest

A

VT

Ventricular Tachycardia

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13
Q
  1. oxygen

2. antidysrhythmics

A

Stable / sustained VT (Pulse, no S/S of < CO)

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14
Q
  1. oxygen
  2. antidysrhythmics
  3. Synchronized Cardioversion (unstable)
  4. Cough Cardiopulmonary Resuscitation (CPR), cough hard q 1-3 sec
A

Unstable/ VT (Pulse, S/S of <CO)

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15
Q

Defibrillation & CPR

A

Pulseless w/ VT

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16
Q

Impulses from many irritable foci in Ventricles fire in disorganized manner; chaotic rapid rhythm in which ventricles quiver & there is no Cardiac Output

A

VF

Ventricular Fibrillation

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17
Q

Fatal if not successfully terminated within 3-5 min; absent Pulse, BP, RR, H-Sounds

A

VF

Ventricular Fibrillation

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18
Q
  1. CPR
  2. O2
  3. AntiDysrhythmic therapy
A

VF

Ventricular Fibrillation

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19
Q

Induce vagal stimulation of Cardiac Conduction System & are used to Terminate Supraventricular Tachydysrhythmias

A

Vagal Maneuvers

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20
Q

bear down or induce gag reflex to stimulate vagal response; Monitor HR, Rhythm, BP, Aspiration Precautions

A

valsalva maneuver

21
Q

Synchronized Countershock to Convert an Undesirable rhythm to a stable rhythm

A

Cardioversion

22
Q

for stable tachydysrhythmias resistant to medical therapies or an Emergent procedure for hemodynamically Unstable ventricular or supraventricular tachydysrhythmias

A

Cardioversion

23
Q

Asynchronous countershock used to terminate Pulseless Ventricular Tachycardia (VT) or VF

A

Defibrillation

resume CPR, 5 Cycles or 2 min, shock as necessary if VF or pulseless VT continues

24
Q

Temporary or Permanent Device that Provides Electrical Stimulation & Maintains the HR when the Client’s Intrinsic Pacemaker fails to Provide a Perfusing Rhythm

25
Internal Pulse generator, Surgically Implanted in subQ pocket below Clavicle
Permanent Pacemakers
26
Leads passed transvenously via cephalic or Subclavian vein to Endocardium on R side of Heart; Post-op limitation of arm movement on operative side required to prevent lead wire dislodgement
Permanent Pacemakers
27
Single-Chambered : lead wire placed in chamber to be paced. | Dual-Chambered: lead wires placed in both R Atrium & Ventricle
Permanent Pacemakers
28
Biventricular pacing of ventricles allows for synchronized depolarization & is used for moderate to severe HF to improve CO
Permanent Pacemakers
29
causes decreased perfusion of myocardial tissue & inadequate myocardial oxygen supply; leads to: HypErtension, Angina, Dysrhythmias, MI, HF, & Death
CAD
30
Narrowing/ Obstruction of one or more coronary arteries from atherosclerosis (accumulation of lipid-containing plaque in arteries)
CAD
31
More than one artery supplying a muscle w/ blood, normally present in coronary arteries & older adults
Collateral Circulatio
32
symptoms occur when coronary artery is occluded to the point that inadequate blood supply to muscle occurs, causing ischemia
CAD
33
goal of tx is to alter atherosclerotic progression
CAD
34
1. Chest Pain 2. Palpitations 3. Dyspnea 4. Syncope 5. Cough/ Hemoptysis (spitting up blood) 6. Excessive Fatigue
CAD
35
ECG (Electrocardiogram) shows: ST-segment Depression &/or T-wave Inversion
Ischemia from reduced blood flow in CAD
36
ECG (Electrocardiogram) shows: ST-segment Elevation, followed by T-Wave Inversion & Abnormal Q-Wave
Infarction
37
in CAD/ ischemia/ infarction, this provides definitive source for diagnosis; shows presence of atherosclerotic lesions
Cardia Catheterization
38
elevated Blood Lipid Levels
CAD
39
Low calorie, sodium, cholest., fat diet ++ Fiber
CAD
40
to compress plaque against wall of artery & dilate vessel (CAD + Angina)
PTCA (percutaneous transluminal coronary angioplasty
41
to vaporize plaque (CAD + Angina)
laser angioplasty
42
to Remove plaque from artery (in CAD + Angina)
Atherectomy
43
to prevent artery from closing & prevent restenosis (in CAD + Angina)
Vascular stent (bare metal or drug-eluting)
44
to Improve blood flow to Myocardial Tissue at risk for ischemia or infarction b/of occluded artery (in CAD + Angina)
Coronary Artery Bypass Grafting
45
to Dilate coronary arteries & Decrease Preload & Afterload
Nitrates | CAD + Angina
46
to Dilate coronary arteries & Reduce Vasospasm
Calcium Channel Blockers | CAD + Angina
47
to reduce development of Atherosclerotic plaques
Cholesterol-Lowering medications | CAD + Angina
48
to reduce BP in Hypertensive individuals
B-Blockers | CAD + Angina