Cardio: Antidysrhythmic Drugs Flashcards
(48 cards)
How does Atropine increase HR?
What receptors does it act on?

What are the Class III drugs?

Give the Clinical Application of Verapamil
Note: More useful than digoxin for rate control in atrial fibrillation due to the Ca+2 channel blockade in AV node persisting during sympathetic stimulation.

Give the PKs and ADRs for Propranolol

What is the general mechanism of Class Ib drugs?
What happens to phase 3, APD, and QT?
What conditions are they useful in treating?

What are the Class IV Agents?

What is the Class II Agent?
Propranolol
What are the Class Ia Agents?

Describe the PKs of Procainamide

What is Ventricular Re-entry, and what kind of Disorders of Impulse is it?
Disorder of Impulse Conduction

What are the ADRs Quinidine?

Label each action potential as either
- Atrium
- Ventricle
- SA node
- AV Node
- Purkinje Fibers


What are the Pharmacokinetics for Digoxin?

Give each of the following the title as
- ST segment
- P Wave
- QT Interval
- QRS Complex
- T Wave
- PR Interval


What are the General mechanisms for Class II drugs?

Concerning AV Nodal Blocks, describe the difference in 1st, 2nd and third degree. What kind of Disorders of Impulse are they?

- What is the mechanism for Lidocaine?
- What is Lidocaine used to treat?
- What does Lidocaine do to the ERP and APD?

Concerning Class Ic drugs
- What happens to QRS, APD, QT, and PR?
- What happens to phase 0?
- What is special about the mechanism kinetics?

What are the ADRs for Amiodarone?

What is the general mechanism for all Class I Antidysrhythmic drugs?
Block voltage-sensitive Na+ channels
Concerning Quinidine
- What Clinical Applications does it have?
- What does it do to the QRS and QT?

What are the main Ventricular (alliterations at the ventricle muscle mass) Dysrhythmias?

What are the ADRs for Verapamil?

What does the Cardiac Action Potential look like for the Atria, Purkinje fibers, and Ventricles?
What about the SA and AV nodes?























