ch 25 Respiratory assessment Flashcards

1
Q

is gas exchange

A

respiratory system

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2
Q

transfer of oxygen (O2) and carbon dioxide (CO2) between the atmosphere and blood.

A

respiratory system

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3
Q

the upper respiratory tract and the lower respiratory tract

A

respiratory system is divided into 2 parts:

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4
Q

the nose, mouth, pharynx, epiglottis, larynx, and trachea.

A

upper respiratory tract includes

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5
Q

the nose

A

Air enters the respiratory tract through

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6
Q

bone and cartilage

A

nose is made of

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7
Q

divided into 2 nares by the nasal septum

A

nose

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8
Q

inside of the nose is shaped into 3 passages by projections called

A

turbinates

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9
Q

increase the surface area of the nasal mucosa that warms and moistens the air as it enters the nose

A

turbinates

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10
Q

with the pharynx

A

nasal cavity connects

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11
Q

the nasopharynx, oropharynx, and laryngopharynx

A

nasal cavity connects with the pharynx.
- It is a tubular passageway that is subdivided into 3 parts:

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12
Q

to protect the lower airway by warming and humidifying air and filtering small particles before air enters the lungs.

A

nose functions

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13
Q

, found within the mucosa of the upper part of the nasal cavity, is responsible for the sense of smell.

A

olfactory nerve

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14
Q

Air moves through the oropharynx to the laryngopharynx. It then travels through the epiglottis to the larynx before moving into the trachea.

A

Route of air through nose to ody

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15
Q

is a small flap behind the tongue that closes over the larynx during swallowing

A

epiglottis

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16
Q

prevents solids and liquids from entering the lungs.

A

epiglottis function

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17
Q

are in the larynx

A

vocal cords location

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18
Q

(the opening between the vocal cords) and into the trachea.

A

air passes through the glottis

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19
Q

U-Shaped cartilages keep the trachea open but allow the adjacent esophagus to expand for swallowing.

A

trachea

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20
Q

into the right and left mainstem bronchi at a point called the carina.

A

trachea bifurcates

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21
Q

is located at the angle of Louis, which is at the level of the 4th and 5th thoracic vertebrae.

A

carina

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22
Q

this area during suctioning causes vigorous coughing

A

Stimulation of carnia

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23
Q

Once air passes the carina, it is in the

A

considered lower respiratory tract

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24
Q

bronchi, bronchioles, alveolar ducts, and alveoli

A

lower respiratory tract consists

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25
Q

Except for the right and left mainstem bronchi,

A

all lower airway structures are found within the lungs.

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26
Q

-upper, middle, and lower

A

right lung is divided into 3 lobes

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27
Q

-upper and lower

A

left lung into 2 lobes

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28
Q

mainstem bronchi, pulmonary vessels, and nerves enter the lungs through a slit called the

A

hilus

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29
Q

is shorter, wider, and straighter than the left mainstem bronchus

A

right mainstem bronchus

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30
Q

is shorter, wider, and straighter than the left mainstem bronchus.

A

right mainstem bronchus vs left

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31
Q

right mainstem bronchus is shorter, wider, and straighter than the left mainstem bronchus.

A

aspiration is more likely to occur in the right lung than in the left lung.

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32
Q

several times to form the lobar, segmental, and subsegmental bronchi.Further divisions form the bronchioles.

A

mainstem bronchi subdivide

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33
Q

the respiratory bronchioles.

A

most distant bronchioles are

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34
Q

are encircled by smooth muscles that constrict and dilate in response to variousstimuli.

A

bronchioles

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35
Q

to a decrease or increase in the diameter of the airways caused by contraction or relaxation of these muscles

A

bronchoconstriction and bronchodilation refer

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36
Q

act as a pathway to conduct gases to and from the alveoli

A

trachea and bronchi

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37
Q

volume of air in the trachea and bronchi is called the
-This air does not take part in gas exchange.

A

anatomic dead space (VD)

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38
Q

is about 500mL (in a 150-lb man). Of each 500 mL inhaled, about 150mL is VD.

A

adults, normal tidal volume (VT), or volume of air exchanged with each breath,

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39
Q

are small sacs in the lungs that are the primary site of gas exchange for O2 and CO2

A

Alveoli

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40
Q

They allow movement of air from alveolus to alveolus

A

alveoli are interconnected by pores of Kohn

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41
Q

air movement through these pores and helps move mucus out of the respiratory bronchioles.

A

Deep breathing promotes

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42
Q

can also move through these pores, spreading infection to previously uninfected areas.

A

Bacteria (alveoli)

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43
Q

of about 2500 mL with a surface area for gas exchange that is about the size of a tennis court.

A

Alveoli have a total volume

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44
Q

, where the alveoli come in contact with pulmonary capillaries

A

Gases are exchanged across the alveolar-capillary membrane

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45
Q

excess fluid fills the interstitial space and alveoli, markedly reducing gas exchange.

A

pulmonary edema

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46
Q

is a lipoprotein that lowers the surface tension in the alveoli
-prevents alveoli collapse

A

Surfactant

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47
Q

reduces the amount of pressure needed to inflate the alveoli and makes them less likely to collapse

A

Surfactant

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48
Q

each person takes a slightly larger breath, termed a sigh, after every 5 or 6 breaths. This sigh stretches the alveoli and promotes surfactant secretion.

A

sigh

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49
Q

refers to collapsed, airless alveoli.

A

atelectasis

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50
Q

because of the effects of anesthesia, decreased mobility, and pain, which can alter breathing and lung expansion.

A

postoperative patient is at risk for atelectasis

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51
Q

, lack of surfactant contributes to widespread atelectasis and collapse of lung tissue

A

acute respiratory distress syndrome (ARDS)

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52
Q

pulmonary and bronchial

A

lungs have 2 different types of circulation:

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53
Q

provides the lungs with blood that takes part in gas exchange

A

Pulmonary circulation

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54
Q

receives deoxygenated blood from the right ventricle of the heart and delivers it to pulmonary capillaries that lie directly alongside the alveoli.

A

pathology of pulmonary artery

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55
Q

starts with the bronchial arteries, which arise from the thoracic aorta

A

Bronchial circulation

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56
Q

but provides O2 to the bronchi and other lung tissues. Deoxygenated blood returns from the bronchial circulation through the azygos vein into the superior vena cava.1

A

Bronchial circulation does not take part in gas exchange

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57
Q

is shaped, supported, and protected by 24 ribs (12 on each side

A

chest wall (description)

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58
Q

which consists of the ribs and sternum, protect the lungs and the heart from injury.

A

thoracic cage function

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59
Q

is the space in the middle of the thoracic cavity.

A

mediastinum

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60
Q

It contains the major organs of the chest, including the heart, aorta, and esophagus.

A

mediastinum consists

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61
Q

chest cavity is lined with a membrane called the

A

parietal pleura

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62
Q

lungs are lined with a membrane called the

A

visceral pleura

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63
Q

join to form one continuous membrane.

A

parietal and visceral pleurae join

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64
Q

does not have any sensory (pain) fibers or nerve endings.

A

visceral pleura consists

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65
Q

This is why irritation or inflammation of the parietal pleura can cause pain with each breath.

A

parietal pleura has pain fibers.

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66
Q

is the space between the pleural layers
-Normally this space contains 10 to 20 mL of fluid.

A

intrapleural space

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67
Q

(1) it provides lubrication, allowing the pleural layers to slide over each other during breathing
(2) it increases unity between the pleural layers. This promotes expansion of the pleurae and lungs during inspiration.

A

intrapleural space (fluid ) serves 2 purposes:

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68
Q

via lymphatic circulation

A

Fluid drains from the pleural space

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69
Q

Several pathologic conditions may cause the accumulation of greater amounts of fluid, termed

A

pathology of pleural effusion.

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70
Q

-Pleural fluid may accumulate because of blockage of lymphatic drainage (e.g., from cancer)
- an imbalance between intravascular and oncotic fluid pressures, as in heart failure. —–Purulent pleural fluid with bacterial infection is called empyema.

A

cause of pleural effusion.

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71
Q

Purulent pleural fluid with bacterial infection is called

A

empyema.

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72
Q

is the major muscle of respiration

A

Diaphragm

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73
Q

, moves downward, and increases intrathoracic volume.

A

During inspiration the diaphragm contracts

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74
Q

At the same time, the internal intercostals relax and the external intercostal muscles contract. This increases the lateral and anteroposterior (AP) dimension of the chest

A

During inspiration the diaphragm contracts

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75
Q

, each innervated by the right and left phrenic nerves

A

diaphragm is made up of 2 hemidiaphragms

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76
Q

the spinal cord between C3 and C5, the 3rd and 5th cervical vertebrae

A

phrenic nerves arise from

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77
Q

in hemidiaphragm paralysis on the side of the injury. Complete spinal cord injuries above the level of C3 result in total diaphragm paralysis and dependence on a mechanical ventilator.

A

Injury to the phrenic nerve results

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78
Q

refers to the process of obtaining O2 from the atmospheric air and making it available to the organs and tissues of the body

A

Oxygenation

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79
Q

by partial pressure of O2 in arterial blood (PaO2), arterial O2 saturation (SaO2)

A

lungs’ ability to oxygenate arterial blood adequately is evaluated

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80
Q

dissolved O2 and hemoglobin-bound O2

A

O2 is carried in the blood in 2 forms:

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81
Q

the amount of O2 dissolved in the plasma

A

PaO2 represents

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82
Q

is the amount of O2 bound to hemoglobin in comparison with the amount of O2 the hemoglobin can carry.

A

SaO2 (defined)

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83
Q

-Unexplained apprehension
-Unexplained restlessness or irritability
-Unexplained confusion or lethargy (can be early or late)

A

Early signs of inadequate Oxygen (CNS)

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84
Q

-Combativeness
-Coma

A

Late signs of inadequate Oxygen (CNS)

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85
Q

-Tachypnea
Dyspnea on exertion

A

Early signs of inadequate Oxygen (respiratory)

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86
Q

-Dyspnea at rest
-Use of accessory muscles
-Retraction of intercostal spaces on inspiration
-Pause for breath between sentences, words

A

Late signs of inadequate Oxygen (respiratory)

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87
Q

-Tachycardia
-Mild hypertension
-Dysrhythmias (can be early or late)

A

Early signs of inadequate Oxygen (cardiovascular)

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88
Q

-Hypotension
-Cyanosis
-Cool, clammy skin

A

Late signs of inadequate Oxygen (cardiovascular)

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89
Q

-Diaphoresis
-Decreased urine output
-Unexplained fatigue

A

can be early or late signs of inadequate Oxygen

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90
Q

involves inspiration, or inhalation (movement of air into the lungs), and expiration, or exhalation (movement of air out of the lungs)

A

Ventilation

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91
Q

from an area of higher pressure (atmospheric) to one of lower pressure (intrathoracic).

A

Gas(o2 & Co2) flows

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92
Q

occurs, neck and shoulder muscles, as well as other accessory muscles of respiration, can aid the effort

A

dyspnea (shortness of breath)

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93
Q

is passive

A

expiration

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94
Q

is the tendency for the lungs to return to their original size after being stretched or expanded.

A

Elastic recoil

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95
Q

is due to the elastin fibers found in the alveolar walls and surrounding the bronchioles and capillaries.

A

elasticity of lung tissue

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96
Q

allows the chest to passively decrease in size (volume). When intrathoracic pressure rises, air moves out of the lun

A

elastic recoil of the chest wall and lungs

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97
Q

expiration to become an active, labored process

A

Exacerbations of asthma or chronic obstructive pulmonary disease (COPD) cause

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98
Q

Abdominal, intercostal, and accessory muscles (e.g., scalene, trapezius) help expel air during labored breathing.

A

body parts help expel air during labored breathing

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99
Q

is a measure of the ease of expansion of the lungs

A

Compliance (distensibility)

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100
Q

-This is a product of the elasticity of the lungs and elastic recoil of the chest wall

A

Compliance (distensibility)

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101
Q

, it is harder for the lungs to inflate

A

compliance is decreased

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102
Q

when there is destruction of alveolar walls and loss of tissue elasticity, as in COPD.

A

Compliance increases

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103
Q

occurs with conditions that increase fluid in the lungs (e.g., pulmonary edema, ARDS, pneumonia), make lung tissue less elastic or distensible (e.g., pulmonary fibrosis, sarcoidosis), or restrict lung movement (e.g., pleural effusion)

A

cause of compliance when it’s decreased

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104
Q

refers to any obstacle to airflow during inspiration and/or expiration

A

resistance

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105
Q

is changes in the diameter (size) of the airways

A

main factor affecting airway resistance

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106
Q

patient with an acute asthma attack has narrowed airways, resulting in
- presence of secretions in the bronchi

A

example of increased resistance.

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107
Q

decreases resistance by increasing the diameter of the bronchi, promoting air entry.

A

bronchodilators

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108
Q

Changes in compliance and/or resistance can seriously

A

affect both oxygenation and ventilation.

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109
Q

responds to chemical and mechanical signals

A

Located in the brainstem, the respiratory center (the medulla)

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110
Q

sends impulses to the respiratory muscles through the spinal cord and phrenic nerves.

A

medulla (function)

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111
Q

is a receptor that responds to a change in the chemical composition (PaCO2 and pH) of the fluid around it

A

chemoreceptor

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112
Q

are found in the medulla.

A

Central chemoreceptors (located)

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113
Q

They respond to changes in the hydrogen ion (H+) concentration.
-An increase in the H+ concentration (acidosis) causes the medulla to increase the respiratory rate and VT.
-A decrease in H+ concentration (alkalosis) has the opposite effect.

A

Central chemoreceptors (function)

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114
Q

causes the medulla to increase the respiratory rate and VT.

A

An increase in the H+ concentration (acidosis) // chemoreceptors

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115
Q

has the opposite effect

A

A decrease in H+ concentration (alkalosis) // chemoreceptors

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116
Q

primarily by their effect on the pH of the cerebrospinal fluid.

A

Changes in PaCO2 regulate ventilation

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117
Q

This lowers the cerebrospinal fluid pH and stimulates an increase in respiratory rate.

A

PaCO2 level is increased, more CO2 is available to combine with H2O and form carbonic acid (H2CO3).

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118
Q

are found in the carotid bodies at the bifurcation of the common carotid arteries and in the aortic bodies above and below the aortic arch.

A

Peripheral chemoreceptors

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119
Q

and may result in chronically elevated PaCO2 levels.
- largely because of a hypoxic drive from the peripheral chemoreceptors

A

COPD change lung function

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120
Q

are found in the conducting upper airways, chest wall, diaphragm, and capillaries of the alveoli.

A

Mechanical receptors (location)

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121
Q

They are stimulated by a variety of physiologic factors, such as irritants, muscle stretching, and alveolar wall distortion.

A

Mechanical receptors (stimulated)

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122
Q

are irritant, stretch, and juxtacapillary (J) receptors

A

3 major types of mechanical receptors

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123
Q

are found in the conducting airways.

A

Irritant receptors (location)//(type of mechanical receptors)

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124
Q

sensitive to inhaled particles and aerosols and, when stimulated, initiate the cough reflex.

A

Irritant receptors (effect)//(type of mechanical receptors)

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125
Q

, in the smooth muscle of the airways, aid in the control of respiration.

A

Signals from stretch receptors//(type of mechanical receptors)

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126
Q

lungs inflate, stretch receptors activate the inspiratory center to inhibit further lung expansion. This is called the Hering-Breuer reflex, and it prevents overdistention of the lungs.

A

stretch receptors (function)// (type of mechanical receptors)

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127
Q

prevents overdistention of the lungs.

A

Hering-Breuer reflex (from stretch receptor)

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128
Q

found in the capillaries of the alveoli, occurs with increased pulmonary capillary pressure. This causes rapid, shallow respiration (tachypnea) seen in pulmonary edema

A

Stimulation of J receptors//(type of mechanical receptors)

129
Q

are efficient in protecting the lungs from inhaled particles, microorganisms, and toxic gases

A

Respiratory defense mechanisms

130
Q

air filtration, mucociliary clearance system, cough reflex, reflex bronchoconstriction, and alveolar macrophages.

A

Respiratory defense mechanisms include

131
Q

filter inspired air

A

Nasal hairs ( air filtration)

132
Q

the abrupt changes in direction of airflow that occur as air moves through the nasopharynx and larynx increase air turbulence.
-causes particles and bacteria to contact the mucosa lining these structures.
-particles (greater than 5 μm) are less dangerous

A

abrupt changes in direction of airflow ( air filtration )

133
Q

velocity of airflow slows greatly after it passes the larynx, facilitating the deposition of smaller particles (1 to 5 μm). They settle out the way that sand does in a river, a process termed sedimentation.
- smaller particles (1 to 5 μm)

A

velocity of airflow slows greatly after it passes the larynx,( air filtration ) aka sedimentation.

134
Q

e are too small to settle in this manner and are deposited in the alveoli. An example of small particles that can build up is coal dust, which can lead to pneumoconiosis

A

Particles less than 1 μm in siz (dangerous)

135
Q

Below the larynx, the mucociliary clearance system, also called the

A

mucociliary escalator

136
Q

, is responsible for the movement of mucus.

A

mucociliary escalator (function)

137
Q

continually secrete mucus at a rate of about 100 mL/day. This mucus forms a blanket that contains the impacted particles and debris from distal lung areas

A

Goblet cells and submucosal glands

138
Q

helps protect against bacteria and viruses.

A

Secretory immunoglobulin A (IgA) in the mucus

139
Q

cover the airways from the level of the trachea to the respiratory bronchioles

A

Cilia

140
Q

about 200 cilia. They beat rhythmically about 1000 times per minute in the large airways, moving mucus toward the mouth.
-We normally swallow the mucus without noticing.

A

Each ciliated cell has

141
Q

the tracheobronchial tree. As a result, we remove particles that penetrate more deeply into the airways less rapidly.

A

ciliary beat is slower further down

142
Q

Dehydration, smoking, inhalation of high O2 concentrations, infection, and drugs, such as atropine, anesthetics, alcohol, or cocaine,

A

causes of impair ciliary action

143
Q

Patients with COPD and cystic fibrosis often have repeated lower respiratory tract infections. These conditions are associated with destroyed cilia, resulting in impaired secretion clearance, a chronic productive cough, and chronic colonization by bacteria. These lead to frequent respiratory tract infections.

A

conditions are associated with destroyed cilia,

144
Q

is a protective reflex that clears the airway by a high-pressure, high-velocity flow of air. It is a backup for mucociliary clearance, especially when this clearance mechanism is overwhelmed or ineffective.

A

cough

145
Q

especially when this clearance mechanism is overwhelmed or ineffective. =COUGH

A

backup for mucociliary clearance,

146
Q

Coughing is effective in removing secretions only above the subsegmental level (large or main airways)

A

Coughing is effective in removing secretions ONLY above the subsegmental level

147
Q

Secretions below this level must be moved upward by the mucociliary mechanism before we can remove them by coughing.

A

Secretions below subsegmental level

148
Q

another defense mechanism

A

Reflex bronchoconstriction is

149
Q

response to the inhalation of large amounts of irritating substances (e.g., dusts, aerosols), the bronchi constrict to prevent entry of the irritants

A

Reflex bronchoconstriction (function)

150
Q

, such as a person with asthma, may have bronchoconstriction after inhalation of triggers, such as cold air, perfume, or other strong odors.

A

hyperreactive airways ex asthma

151
Q

Because there are no ciliated cells below the level of the respiratory bronchioles, the primary defense mechanism at the alveolar level is

A

alveolar macrophages

152
Q

rapidly phagocytize inhaled foreign particles, such as bacteria. The debris is moved to the level of the bronchioles for removal by the cilia or removed from the lungs by the lymphatic system.

A

Alveolar macrophages

153
Q

tend to remain in the lungs for indefinite periods and can stimulate inflammatory responses

A

Particles (e.g., coal dust, silica) that cannot be adequately phagocytized

154
Q

Because alveolar macrophage activity is impaired by cigarette smoke, the smoker who is employed in an occupation with heavy dust exposure (e.g., mining, foundries) is at an especially

A

high risk for lung disease

155
Q

can be divided into alterations in structure, defense mechanisms, and respiratory control

A

Age-related changes in the respiratory system

156
Q

Structural changes include calcification of the costal cartilages, which can interfere with chest expansion. The outward curvature of the spine is marked, especially with osteoporosis, and the lumbar curve flattens

A

Structural changes in elder

157
Q

chest may appear barrel shaped, and

A

chest shape elder

158
Q

the older person may need to use accessory muscles to breathe. Respiratory muscle strength progressively declines after age 50. Overall, the

A

lungs in the older adult are harder to inflate.

159
Q

Chest wall stiffening
Costal cartilage calcification
↑ Anteroposterior diameter
↓ Elastic recoil
↓ Chest wall compliance
↓ Functioning alveoli
↓ Respiratory muscle strength

A

Changes in structure of elder

160
Q

↓ Cell-mediated immunity
↓ Specific antibodies
↓ Cilia function
↓ Cough force
↓ Alveolar macrophage function
↓ Sensation in pharynx

A

Changes in defense mechanism of elder

161
Q

↓ Response to hypoxemia
↓ Response to hypercapnia

A

Changes in Respiratory Control of elder

162
Q

dyspnea can occur if their activity exceeds their normal exercise.

A

older adults have less tolerance for exertion

163
Q

because of a decline in both cell-mediated and humoral immunity (ability to produce antibodies).

A

older adults, respiratory defense mechanisms are less effective

164
Q

for aspiration.

A

older adult is at greater risk

165
Q

resulting in a more gradual response to changes in blood O2 or CO2 level.

A

aging process alters respiratory control,

166
Q

if respiratory distress is severe, only obtain pertinent information and defer a thorough assessment until the patient’s condition stabilizes.

A

physical assessment= if respiratory distress is severe

167
Q

lower respiratory problems, such as asthma, COPD, pneumonia, and tuberculosis (TB).

A

ask about lower history problems

168
Q

such as medications, pollen, smoke, mold, or pet exposure.

A

Ask the patient with allergies about possible precipitating factors or triggers,

169
Q

such as runny nose, wheezing, scratchy throat, or chest tightness.

A

Record the characteristics and severity of the allergic reaction, example

170
Q

Determine the frequency of asthma exacerbations.

A

frequency of asthma

171
Q

. Cough is a common side effect

A

effect of angiotensin-converting enzyme (ACE) inhibitors.

172
Q

, dyspnea and decreased exercise tolerance may occur very quickly.

A

effect of upper respiratory tract infection is superimposed on a chronic problem

173
Q

in the presence of mold, or with changes in temperature or air pollution.

A

asthma, symptoms may occur or worsen during exercise,

174
Q

occurs with secretions

A

loose-sounding cough

175
Q

may mean an airway irritation or obstruction.

A

dry, hacking cough

176
Q

suggests upper airway obstruction from inhibited vocal cord movement due to subglottic edema.

A

harsh, barky cough

177
Q

amount, color, consistency, and odor
-Quantify the amount of sputum per day.

A

If the patient has a productive cough, evaluate the following characteristics of sputum:

178
Q

clear or slightly whitish.

A

Sputum is normally

179
Q

is usually clear to gray with occasional specks of brown.

A

If a patient is a cigarette smoker, the sputum

180
Q

may have clear, whitish, or slightly yellow sputum, especially in the morning on rising.

A

patient with COPD sputum

181
Q

suspect pulmonary complications

A

If the patient reports any change from baseline color of sputum

182
Q

any changes in consistency of sputum to thick, thin, or frothy and pink tinged.

A

sputum may indicate dehydration, postnasal drip or sinus drainage, or possible pulmonary edema

183
Q

suggests an infectious process.

A

foul odor or exceptionally bad breath or taste in the mouth

184
Q

spitting or coughing up blood

A

hemoptysis

185
Q

(vomiting blood).

A

hematemesis

186
Q

such as pneumonia, TB, lung cancer, and severe bronchiectasis.

A

Hemoptysis occurs with a variety of conditions

187
Q

are musical sounds that can be audible

A

wheezes

188
Q

some degree of airway obstruction, such as asthma, foreign body aspiration, and emphysema.

A

Wheezing indicates

189
Q
A

Respiratory problems that have a strong genetic link

190
Q

is the most important risk factor for COPD and lung cancer.

A

Smoking

191
Q

Do this by multiplying the number of cigarettes smoked per day by the number of years smoked

A

Quantify smoking habits in pack-years

192
Q

prior residence in China, India, Africa, the former Soviet Republics, Latin America, or any developing country.

A

Risk factors for TB include

193
Q

exposure to people with high rates of TB transmission, such as the homeless, injection drug users, and people with HIV infection

A

other Risk factors for TB include

194
Q

those exposed to bird and rodent feces or polluted water, those working closely with the soil, and immunocompromised patients.

A

Risk factors for fungal lung infections include

195
Q

can be a symptom of respiratory disease

A

Weight loss (resp disease)

196
Q

are common in patients with COPD, lung cancer, TB, and chronic severe infection (bronchiectasis)

A

Anorexia, weight loss, and chronic malnutrition

197
Q

Determine if weight loss was intentional, and, if not, if food intake is changed by anorexia (from medications), fatigue (from hypoxemia, increased work of breathing), or feeling full quickly (from lung hyperinflation).

A

cause of weight loss

198
Q

can cause sputum to thicken and obstruct the airway

A

Dehydration (effect airway)

199
Q

from fluid retention may impair gas exchange.

A

Rapid weight gain ( (effect airway)

200
Q

of limited mobility, which can cause constipation.

A

Dyspnea can be the cause

201
Q

leading to atelectasis or pneumonia.

A

Immobility and sedentary habits are risk factors for hypoventilation

202
Q

with chest tightness, wheezing, or coughing. This suggests a need for adjunct therapy or other medication changes

A

patient with asthma or COPD may awaken (symptoms)

203
Q

and may cause sleep apnea

A

Excess weight interferes with normal ventilation

204
Q

while awake or asleep

A

Extremely obese persons may hypoventilate

205
Q

snoring, insomnia, abrupt awakenings, daytime drowsiness, and early morning headaches.

A

Manifestations of sleep apnea include

206
Q

of TB.

A

Night sweats may be a sign

207
Q

hypoxia can cause neurologic symptoms, ask the patient about apprehension, restlessness, irritability, and memory changes.

A

indicate inadequate cerebral oxygenation

208
Q

pleurisy, fractured ribs, and costochondritis .

A

cause chest pain

209
Q

Hobbies, such as woodworking (sawdust) or pottery (silica), and animal exposure (allergies) can

A

work environments cause respiratory problems.

210
Q

Because of hyperreactive airways, exposure to fumes, smoke, and other chemicals may

A

trigger an attack in the patient with asthma.

211
Q

perform good pulmonary hygiene (bronchodilators, coughing, deep breathing) before intimacy. They may need to use O2 therapy equipment during intercourse just like they would with any strenuous physical activity.

A

strategies improving sexual activity

212
Q

Check each nare for air patency with respiration while briefly occluding the other nare. Tilt the patient’s head backward and push the tip of the nose upward

A

how to assess pts nose

213
Q

mucous membrane should be pink and moist, with no evidence of edema (bogginess), exudate, or bleeding.
-Some septal deviation is normal in an adult.

A

Normal nose observation description

214
Q

, which are abnormal, fingerlike projections of swollen nasal mucosa. Polyps may result from long-term irritation of the mucosa (e.g., from allergies)

A

turbinates for polyps (definition)

215
Q

could occur with a foreign body.

A

Purulent and malodorous discharge (nose)

216
Q

could be related to allergies or from cerebrospinal fluid.

A

Watery discharge (nose)

217
Q

could be from trauma or dryness.

A

Bloody discharge (nose)

218
Q

could mean an infection

A

Thick mucosal discharge (nose)

219
Q

should be smooth and moist, with no evidence of exudate, ulcerations, swelling, or postnasal drip.

A

normal pharynx

220
Q

that cranial nerves IX (glossopharyngeal) and X (vagus) are intact and that the airway is protected.

A

normal response (gagging) means

221
Q

while the patient is sitting erect with the neck slightly flexed

A

how to Palpate the lymph nodes

222
Q

Tender, hard, or fixed nodes

A

abnormal feeling of node

223
Q

small, mobile, nontender nodes (shotty nodes)

A

normal feeling of lymph node

224
Q

in a well-lit, warm room with measures taken to ensure the patient’s privacy

A

Chest examination is best done (environment)

225
Q

It is best to begin on the posterior chest, particularly with female patients

A

chest examination best done on

226
Q

, ask the patient to lean forward with arms folded. This position moves the scapulae away from the spine, exposing more of the area you need to examine.

A

assessing the posterior chest

227
Q

have the patient sit upright or position the patient supine with the head of the bed elevated to 30 degrees (semi-Fowler’s position)

A

assessing the anterior chest,

228
Q

should be less than the side-to-side or transverse diameter
-AP ratio is 1:2.

A

anterior-posterior (AP) diameter normal

229
Q

(e.g., barrel chest) may be due to normal aging or result from lung hyperinflation.

A

increase in (anterior-posterior) AP diameter cause

230
Q

(e.g., pectus carinatum [a prominent protrusion of the sternum]) and pectus excavatum (an indentation of the lower sternum above the xiphoid process)

A

abnormalities in the sternum

231
Q

include kyphosis, scoliosis, and kyphoscoliosis.

A

Spinal curvatures that affect breathing

232
Q

Inspiration (I) should take half as long as expiration (E) (I/E ratio = 1:2)

A

Inspiration duration vs expiration normally

233
Q

rapid, deep breathing

A

Kussmaul

234
Q

abnormal respirations characterized by alternating periods of apnea and deep, rapid breathing

A

Cheyne-Stokes (like dog)

235
Q

irregular breathing with apnea every 4 to 5 cycles respirations.

A

Biot’s (increase Co2-sound like running fast)

236
Q

, a late sign of hypoxemia, is best seen in light-skinned patients as a bluish tinge to the mucous membranes, lips, and palms of the hands.

A

Cyanosis ( best seen in light-skinned patients )

237
Q

may be seen as a gray or white discoloration in the conjunctivae or around the mouth.

A

In dark-skinned persons, cyanosis

238
Q

include hypoxemia or decreased cardiac output.

A

Causes of cyanosis

239
Q

increase in the angle between the base of the nail and the fingernail to 180 degrees or more

A

Clubbing

240
Q

occurs away from the side of a tension pneumothorax or a neck mass, but toward the side of a pneumonectomy or lobar atelectasis.

A

Tracheal deviation

241
Q

occurs when air entry is limited by conditions involving the lung (e.g., atelectasis, pneumothorax) or the chest wall (e.g., incisional pain)

A

Unequal expansion

242
Q

occurs in conditions that produce a hyperinflated or barrel chest or in neuromuscular diseases (e.g., amyotrophic lateral sclerosis, spinal cord lesions)

A

Equal but decreased expansion in chest

243
Q

over a pleural effusion, an atelectasis, or a pneumothorax.

A

chest Movement may be absent or unequal due to

244
Q

is the vibration of the chest wall made by vocalization.

A

Fremitus

245
Q

is most intense by the sternum and between the scapulae because these areas are closest to the major bronchi

A

Tactile fremitus

246
Q

occurs when the lung becomes filled with fluid or is denser.

A

Increased fremitus

247
Q

This happens with pneumonia, lung tumors, thick bronchial secretions, and above a pleural effusion (the lung is compressed upward).

A

condition with voice that moves through a dense tissue or fluid, you can feel that the vibration is increased.

248
Q

if the hand is farther from the lung (e.g., pleural effusion) or the lung is hyperinflated (e.g., barrel chest)

A

Fremitus is decreased

249
Q

may occur with pneumothorax or atelectasis

A

Absent fremitus

250
Q

is harder to palpate for fremitus because of the large muscles and breast tissue

A

anterior of the chest

251
Q

is used to assess the density or aeration of the lungs

A

Percussion

252
Q

area over lung tissue should be resonant, except for the area of cardiac dullness

A

anterior chest resonant

253
Q

over lung tissue to the level of the diaphragm

A

posterior chest should be resonant

254
Q

should proceed from the lung apices to the bases, comparing opposite areas of the chest

A

Auscultation begin

255
Q

or you think the patient will tire easily, start at the bases

A

If the patient is in mild respiratory distress (start from base)

256
Q

are bronchial, bronchovesicular, and vesicular.

A

3 normal breath sounds

257
Q

are loud, high-pitched sounds that resemble air blowing through a hollow pipe

A

Bronchial sounds

258
Q

have an inspiratory to expiratory (I/E) ratio of 2:3, with a gap between inspiration and expiration

A

Bronchial sounds

259
Q

have a medium pitch and intensity.

A

Bronchovesicular sounds

260
Q

best heard anteriorly between the first and second intercostal space and posteriorly between the scapulae.
- have a 1:1 ratio, with inspiration equal to expiration.

A

Bronchovesicular sounds

261
Q

are relatively soft, low-pitched, gentle, rustling sounds.
-They are heard over all lung areas except the major bronchi.
- 3:1 ratio, with inspiration 3 times longer than expiration.

A

Vesicular sounds

262
Q

normal and abnormal (adventitious).

A

classify breath sounds into 2 main categories:

263
Q

crackles (fine and coarse), wheezes, stridor, and pleural friction rub

A

Adventitious breath sounds include

264
Q

is used to evaluate the status of previously identified respiratory problems and to monitor for signs of new problems.

A

focused assessment

265
Q

-Arterial blood gases
-Chest x-ray
-Hemoglobin
-Hematocrit

A

objective Diagnostic

266
Q

Interstitial edema

A

early pulmonary edema

267
Q

alveolar filling

A

pneumonia

268
Q

loss of lung volume (alveoli deflate)

A

atelectasis

269
Q

(1) pulse oximetry and (2) analysis of arterial blood gases (ABGs)

A

Two methods are used to assess the efficiency of gas transfer in the lung and tissue oxygenation:

270
Q

patient with impaired cardiac output or hemodynamic instability (e.g., altered level of consciousness, changes in heart rate and rhythm, low BP) may have inadequate tissue O2 delivery and/or abnormal O2 consumption.

A

evaluate a mixed venous blood gas.

271
Q

Values obtained by pulse oximetry are less accurate if the SpO2 is less than 70%
-may display a value that is ±4% of the actual value

A

less accurate value of pulse Ox

272
Q

hemoglobin variants (e.g., carboxyhemoglobin, methemoglobin) are present

A

Pulse oximetry is inaccurate if

273
Q

motion, low perfusion, anemia, cold extremities, bright fluorescent lights, intravascular dyes, thick acrylic nails, and dark skin color.

A

Other factors that can alter the accuracy of pulse oximetry include

274
Q

, obtain an ABG analysis to verify the values.

A

if there is doubt about the accuracy of the SpO2 reading

275
Q

are obtained to determine oxygenation status and acid-base balance.

A

ABGs (function)

276
Q

measurement of the PaO2, PaCO2 (the partial pressure of CO2 in arterial blood), acidity (pH), bicarbonate (HCO3−), and SaO2

A

ABG analysis includes

277
Q

can be obtained by arterial puncture or from an arterial catheter, which is usually inserted into the radial or femoral artery.

A

Blood for ABG analysis

278
Q

arterial catheter permits ABG sampling without repeated arterial punctures.

A

arterial catheter vs arterial puncture

279
Q

Sensor may be attached to an adaptor on endotracheal or tracheostomy tube. A nasal cannula with a sidestream capnometer can be used in patients without an artificial airway.

A

how to measure End-tidal CO2(PetCO2) (capnography)

280
Q

is 2–5 mm (dead space)
-if it increases= pulmonary embolism

A

Normal difference between PaCO2 and PetCO2

281
Q

transcutaneous CO2 (PtCO2) and end-tidal CO2 (PetCO2) capnography

A

CO2 can be monitored using

282
Q

is a noninvasive method of estimating arterial pressure of CO2 (PaCO2) using an electrode placed on the skin

A

Transcutaneous measurement of CO2

283
Q

is the noninvasive measurement of alveolar CO2 during exhalation when CO2 concentration is at its peak

A

PetCO2

284
Q

is a flow-directed catheter

A

pulmonary artery (PA) catheter

285
Q

Blood drawn from a PA catheter is called a

A

mixed venous blood gas (SvO2)

286
Q

(1) it consists of venous blood that has returned to the right side of the heart from all parts of the body and (2) it samples blood just before blood enters the lungs to be oxygenated. Normal SvO2 is 60% to 80%.

A

mixed venous blood gas (SvO2) (function)

287
Q

intermittently by obtaining a blood sample from the PA catheter or continuously via a fiberoptic sensor as part of a specific central line with SpO2 monitoring capability.

A

SvO2 can be monitored

288
Q

in cardiac output or tissue O2 delivery.

A

Changes in SvO2 provide an early warning of a change

289
Q

that less O2 is being delivered to the tissues or that more O2 is being consumed.

A

A decrease in SvO2 suggests

290
Q

expectoration, tracheal suction, or bronchoscopy.

A

obtain a sputum sample by

291
Q

if pt is unable to expectorate spontaneously, sputum may be collected by inhaling an irritating aerosol, usually hypertonic saline. This is called

A

sputum induction

292
Q

that the patient has been exposed to the antigen. It does not mean that the patient has TB

A

a positive result on a TB skin test means

293
Q

either no exposure or a depression of cell-mediated immunity, which occurs in HIV infection.

A

negative TB result means

294
Q

be sure that the injection is intradermal and not subcutaneous

A

prevent a false-negative reaction, (TB)

295
Q

is a procedure in which the bronchi are visualized through a fiberoptic tube

A

Bronchoscopy

296
Q

diagnostic purposes (obtain biopsy specimens) and for treatment (e.g., to remove mucous plugs, foreign bodies).

A

Bronchoscopy may be used for

297
Q

Laser therapy, electrocautery, cryotherapy, and stents may be placed through a bronchoscope to

A

ways to achieve patency of an airway that has been partially or nearly fully obstructed by tumors

298
Q

can be done in an outpatient procedure room, in a surgical suite, or at the bedside in the critical care unit or on a medical-surgical unit.

A

Bronchoscopy performed (environement)

299
Q

patient may be positioned supine, in low-Fowler’s, or even be seated.

A

position of patient during Bronchoscopy

300
Q

HCP inserts the bronchoscope through the nose or mouth. Depending on the approach, the nasopharynx or oropharynx is anesthetized with local anesthetic spray.

A

insertion of bronchoscopy

301
Q

is coated with water-soluble lubricant and inserted down into the airways. Small amounts (30 mL) of sterile saline may be injected through the scope and withdrawn and examined for cells, a technique termed bronchoalveolar lavage (BAL)

A

bronchoscope (how its used)/ termed bronchoalveolar lavage (BAL)

302
Q

Bronchoscopy can be done through the endotracheal tube of a mechanically ventilated patient.

A

Bronchoscopy can be done through the endotracheal tube

303
Q

(1) transbronchially, (2) percutaneously or via transthoracic needle aspiration (TTNA), (3) by video-assisted thoracic surgery (VATS), or (4) as an open lung biopsy

A

Lung biopsy may be done

304
Q

is to obtain tissue, cells, and/or fluid for evaluation.

A

purpose of a lung biopsy

305
Q

involves passing a forceps or needle through the bronchoscope for a specimen.

A

Transbronchial biopsy (lung biopsy)

306
Q

is used to distinguish infection and rejection in lung transplant recipients.

A

A combination of transbronchial lung biopsy and BAL

307
Q

involves inserting a needle through the chest wall, usually under bedside ultrasound or CT guidance. Because of the risk for a pneumothorax, a chest x-ray is done after TTNA.

A

percutaneous or TTNA biopsy

308
Q

a rigid scope with a lens is passed through a trocar placed into the pleura via 1 or 2 small incisions in the thoracic cavity. The HCP views the lesions in the pleura or peripheral lung on a monitor directly via the scope and can obtain biopsy specimens. A chest tube is kept in place until the lung expands. VATS is much less invasive than open lung biopsy. It is associated with shorter hospital stays and reduced mortality.13 It is ideal for pleural biopsy and resecting lung nodules.

A

VATS, (lung biopsy)

309
Q

is used when other procedures cannot diagnose pulmonary disease. With the patient under anesthesia, the chest is opened with a thoracotomy incision and a biopsy specimen is obtained. Postprocedure care is the same as after thoracotomy

A

open lung biopsy

310
Q

is the insertion of a large-bore needle through the chest wall into the pleural space to obtain specimens for diagnostic evaluation, remove pleural fluid, or instill medication

A

Thoracentesis

311
Q

patient is positioned sitting upright, leaning on an overbed table with feet supported. The skin is cleansed and a local anesthetic (lidocaine) is injected subcutaneously. A percutaneous catheter may be left in to allow further drainage of fluid

A

Thoracentesis procedure

312
Q

measure lung volumes and airflow.

A

Pulmonary function tests (PFTs)

313
Q

can diagnose pulmonary disease, monitor disease progression, assess response to bronchodilators, and evaluate disability.

A

results of PFTs (Pulmonary function tests)

314
Q

Airflow measurement is obtained by trained personnel using a spirometer.

A

Pulmonary function tests Airflow measurement

315
Q

are 80% to 120% of the predicted valu

A

Pulmonary function test normal value

316
Q

Volume of air inhaled and exhaled with each breath. Only a small proportion of total capacity of lungs

A

Tidal volume (Vt)

317
Q

may be used to monitor lung function in people with asthma, cystic fibrosis, or COPD, as well as before and after lung transplantation or other thoracic surgeries

A

Home spirometry

318
Q

changes can warn of early lung transplant rejection or infection.

A

Spirometry