Chapter 6: perception Flashcards

1
Q

Rods

A

Rods: work very well in a lot of light and are important for color perception.

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2
Q

Cones

A

Work very well in the dark and are sensitive to light.

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3
Q

Vertral pathway

A

The signal transfers from the primary visual cortex (V1) to the secondary areas V2 and V3, to V4 and then to the inferotemporal cortex. Important for the ‘what’.

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4
Q

Dorsal pathday

A

The signal transfers form V1 to V2 and V3, to V5 (motion perception area) and then to the parietal cortex. Important for the ‘where’ and ‘how’.

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5
Q

Gestalt principles

A
  1. Proximity
  2. Similarity
  3. Common motion
  4. Good continuation
  5. Closure
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6
Q

Visual field deficits

A

Results from damage between the retina and V1, which can cause a loss of part of the visual field. Brain damage in the left hemisphere leads to loss of vision in the right visual field and damage in the right hemisphere leads to loss of vision in the left visual field.

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7
Q

Homonymous hemianopia

A

When visual field loss occurs in the same visual field in both eyes.

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8
Q

Quadrantanopia

A

Blindness for a specific quadrant of the visual field.

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9
Q

Blindsight (due to cortical blindness)

A

Damage that completely destroyed the V1 and resulted in blindness for the full visual field. Someone with cortical blindness can experience blindsight: someone does perceive what is there but is nog conscious about this. The patients show eye movement to a stimulus and perform above chance level on discrimination tasks, but to them they are just guessing.

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10
Q

Visual anosognosia

A

People are cortically blind but not aware of their conditions, they are fully convinced that they can see.

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11
Q

Cerebral achromatopsia

A

Loss of color vision due to damage in the V4 in the ventral pathway. People percieve the world in shades of gray.

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12
Q

Cerebral dyschromatopsia

A

Damage to the V4 that leads to a limited ability to distinguish between colors.

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13
Q

Akinetopsia

A

Inability to perceive motion. Caused by damage to the V5.

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14
Q

Visual agnosia

A

Inability to recognise objects based on visual perception. These patients do have an intact visual field, but the information is not sufficiently integrated.

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15
Q

Optic ataxi

A

Difficulty with reaching to and grasping of objects despite intact basis visual functions and object recognition. It’s caused by damage to the inferior parietal lobe and superior occipital cortex.

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16
Q

Balint’s syndrome

A

A combination of optic ataxia, ocular apraxia and simultanagnosia. Ocular apraxia is the loss of control over intentional eye movement. Caused by bilateral lesions in the posterior parietal lobe.

17
Q

Visual neglect

A

Caused by a lesion in the parietal lobe that leads to a deficit in attention to half of the visual field. They can see, but don’t consciously perceive the information.

18
Q

Auditory perception

A

Results form the translation of vibrations into electrical signals. These vibrations move the eardrum back and forth, which is amplified in the middle ear, which sets fluids in motion behind the oval window. By moving the hair cells inside the cochlea, the vibrations are converted into electrical signals. The hair cells at the beginning of the cochlea respond to high requencies and the hair cells at the end respond to low frequencies. This electrical signal is sent to the brainstem, after which it is processed in the midbrain and the auditory cortex.

19
Q

Cortical deafness

A

Being unable to percieve sound even though there is no damage to the cochlea or middle ear that can explain this.

20
Q

Auditory agnosia

A

Impairments in receiving / recognizing auditory information. This can be for various types of auditory information of specifically for verbal information, environemental sounds or music.

21
Q

Amorphagnosia

A

A deficit in object discrimitation through tactile perception that effects the perception of macrogeometrical features, like shapes and sizes.

22
Q

Tactile agnosia

A

Patients can recognize an boject only with the ipsilateral hand. This is not a disorder in sensory perception, but a disconnection in areas for tactile perception and multimodel integration / semantic memory.

23
Q

Tactile apraxia

A

A disorder in the execution of different exploratory movements of the contralateral hand. This means for example feeling in your bag and making the movements with you finger to determine your are holding the keys.

24
Q

Ahylognosia

A

A deficit in object discrimination through tactile perception that affect the perception of microgeometrical features, like texture, material, hardness, weight and temperature.

25
Q

The gustarory system

A

Consists of tastebuds that transmit electrical signals to the cranial nerve, which projects to the solitary nucleus in the medulla and from there, via the pons and thalamus, to the insula.

26
Q

Olfactory receptors

A

Are embedded in the olfatory epithelium and send signals via the olfactory nerve to the olfactory bulb and then to the primary olfactory cortex. This is connected to the thalamus, hippocampus and other limbic areas so that the smell can be associated with emotions and memories.

27
Q

Ageusia

A

Loss of taste

28
Q

Hypogeusia

A

Reduced taste

29
Q

Dysgeusia

A

Change of taste

30
Q

Gustatory agnosia

A

Inability to recognize or idefntify taste

31
Q

Anosmia

A

Loss of smell

32
Q

Hyposmia

A

Reduced smell

33
Q

Phantosmia

A

Aolfactory hallucinations (smell)

34
Q

Olfactory agnosia

A

Inability to recognize or identify smell

35
Q

Spatial allignment

A

When 2 things originate from the same spatial location.

36
Q

Temporal allignment

A

When 2 things are percieved approximately at the same time.