Clinical arrhythmia lecture Flashcards

(45 cards)

1
Q

Some antiarrhythmics (Class 1A, 1C, Class III), erythromycin, antifungals, tricyclic antidepressants

…can cause?

A

Prolonged QT interval

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2
Q

Atrial fibrillation with controlled or slow rate in absence of meds that slow AV conduction.

A

Sick sinus syndrome variation

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3
Q

Brady-Tachy Syndrome- paroxysmal atrial tachyarrhythmias accompanied by sinus node dysfunction and symptomatic bradycardia.

A

Sick sinus syndrome variation

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4
Q

Presence of symptoms clearly related to sinus node dysfunction (pauses) proven by ECG.
40% will have concomitant AV Node dysfunction, heart block.

A

Sick sinus syndrome

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5
Q

Most common cause of sick sinus syndrome?

A

AGING

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6
Q

Sinus node dysfunction
AV node dysfunction

…treatment

A

permanent pacemaker

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7
Q

Sick sinus syndrome tx

A

step 1…eliminate offending drug (if applicable)

step 2..pacemaker

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8
Q

PAC precipitating factors

A

ETOH
caffeine
adrenergic stimulation

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9
Q

*Paroxysmal and persistent forms.**
May be seen in healthy individuals (5%) without HD
*can be precipitated by emotional stress, use of stimulants, following surgery, or with acute ETOH intoxication (“holiday heart”).
*Also occurs in absence of a precipitant

A

A fib

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10
Q

95% of A fib cases seen in the presence of…

A

underlying cardiac or pulmonary pathology

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11
Q

_____ most often seen in valvular, hypertensive and coronary heart disease
*frequently develops in adults with atrial septal defects

A

A fib

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12
Q

Irregularly irregular rhythm
atrial depol 400-600/min

*if new/untreated…ventricular rate tachycardia (often 120-180)

A

A fib

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13
Q

If rate continuously exceeds 200 bp, consider A fib with…

A

WPW

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14
Q

First priority when assessing A fib patient….

A

ASSESS HEMODYNAMIC STABILITY

most yes. if no…cardioversion

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15
Q

Must worry about thromboembolic if in A fib for longer than….

A

48-72 hours

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16
Q

Diagnostic test that all new onset A fib patients must have

A

Cardiac ultrasound (echo)

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17
Q

If hemodynamically stable, initial goal for a fib tx

A

Rate control

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18
Q

IV _____ is best for rapid rate control in ED for A fib

A

Diltiazem

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19
Q

Oral diltiazem, verapamil, or beta blockers

A

Best for chronic rate control of A fib

20
Q

If A fib present for longer than 72 hours…

A

MUST FULLY ANTICOAGULANT

no cardioversion until 3 weeks of anti coagulation

21
Q

2 chronic A fib management strategies

A
  1. rhythm control

2. rate control

22
Q

cardioversion to NSR+ drug Rx to maintain sinus rhythm→↑ CO/function, but side effects of meds, recurrences of Afib.

A

Rhythm control strategy of A fib

23
Q

leave in afib, control ventricular rate + anticoagulate (warfarin* for most patients)

A

Rate control strategy of A fib

24
Q

CHADS2 or CHADS VAS

A

used to calculate risk of stroke

25
If a pt with a fib has a CHADS score of 0...
Can leave in a fib, don't need to anticoagulate
26
Definitive treatment for A fib rhythm control...
Ablation with surgery or catheter | 65-90% success
27
This drug class appears to increase risk of developing a fib or a flutter
NSAIDs
28
Underlying heart disease always present. **Paroxysmal and persistent forms. Embolization risk less than Afib but occurs. *Rate usually 240-350; ventricular rate most often 1/2 the flutter rate; less stable hemodynamically; difficult to slow with meds.
A flutter
29
Most effective tx for A flutter
DC cardioversion | often need meds to prevent recurrence
30
If you see a 4:1 atrial flutter..is this person already being treated?
Most likely yes
31
Re-entry is responsible for vast majority of cases Seen in any age group, especially healthy young adults. ***Narrow complex, regular tachycardia, rate usually 150-220. **P waves usually not identifiable; may come after QRS
PSVT
32
2 pathways of AV node
Alpha and beta
33
Requires dual AV nodal (α & ß pathways) that can conduct in either direction. **Initiated by PAC conducted antegrade down one (β) AV pathway; if conduction slow enough to allow previously refractory other (α) pathway time to recover, impulse will be conducted retrograde back through the AVN initiating a re-entry circuit
PSVT
34
DOC for PSVT
Adenosine
35
Interventions that increase vagal tone (e.g. carotid message or valsalva) may abruptly terminate the rhythm.
PSVT
36
Selective ablation of AV node can be tx for
PSVT
37
Accessory A-V bypass tract (Kent Bundle) allows conduction to ventricles as alternative to the AV node. ECG findings: Short PR interval and Delta waves
WPW
38
Can you use Adenosine, ß-Blockers, Diltiazem, Verapamil in a WPW pt with A fib or A flutter?
NO!!!! | can use Procainamide or Amiodarone
39
Lidocaine will slow conduction through...
kent bundle! (in WPW)
40
Radio Frequency Ablation: Catheter ablation of the bypass tract is effective (90%) and curative. Treatment of choice for most ____ patients.
WPW
41
Most patients developing VF within 1st 48 hrs of an acute MI have a _____ prognosis if successfully resuscitated
good long term
42
Resuscitated VF unassociated with acute MI has a very high recurrence rate (> 30% recur within the first year following the initial event), and carries a....
very poor prognosis *tx with ICD
43
most common cause of sudden cardiac death
V fib
44
V fib sometimes occurs following administration of antiarrhythmic drugs - especially in patients with....
prolonged QT interval
45
prominent jugular pulsation and palpable parasternal lift * harsh systolic murmur best heard at second and third left intercostal space * radiates to left shoulder * early systolic sound precedes the murmur during expiration
Pulmonic stenosis