ECG finishing touches lecture Flashcards Preview

Clin Med-Cardio pt 2 > ECG finishing touches lecture > Flashcards

Flashcards in ECG finishing touches lecture Deck (36):
1

Bundle of Kent that directly connects atrium to ventricles

Wolff-Parkinsons White

2

PR interval shortened to under 0.12 seconds
QRS widened to more than 0.10
***delta wave present

WPW

3

hallmark*** slurring of upstroke due to Kent bundle conduction (delta wave), wide QRS at base (between 2-2.5 boxes wide), narrow QRS at top

WPW

4

Arrhythmias associated with WPW

PSVT
A fib (even faster than usual bc Kent Bundle conducts faster than AV node)

**can have vent rates up to 300 with a fib in WPW

5

Cannot read what in a WPW EKG?

Ischemia
Infarct
LVH

6

**Cannot interpret Q waves (if present) for MI in the presence of WPW abnormality on ECG.
**Cannot interpret LVH by voltage criteria (if present) in the presence of WPW abnormality.
**Cannot interpret ischemia via ST segment depression or T wave inversion in the presence of WPW abnormality.

WPW

7

99% of the time, irregularly irregular EKG means...

A fib

8

Bypass from atrium into Bundle of His

Lown-Ganong-Levine Syndrome

9

Very short PR interval
Associated w PSVT

Lown Ganong Levine Syndrome

10

This drug has a narrow therapeutic to toxic ration and is a potent stimulator of arrhythmias

Digoxin

11

ST segment scooping

Digoxin

12

Multiformed PVCs are most common EKG presentation of

Digoxin toxicity

13

Digoxin causes SA nodal suppression and...

AV block

14

*Accelerated junctional rhythm
*Atrial tachycardia with AV block

seen with Digoxin

15

mild ST segment depression
Flattening of the T wave
Appearance of a U wave

HYPOkalemia

16

T waves across the entire 12 lead EKG begin to peak

HYPERkalemia

17

PR interval become prolonged, and the P wave gradually flattens and then disappears

HYPERkalemia

18

Ultimately, QRS complex widens until it merges with the T wave, forming a SINE WAVE** pattern. V fib may eventually develop

Hyperkalemia

19

QT interval should not exceed ______ of the R-R interval

one half

20

tricyclic antidepressants
erythromycin (and other macrolides)
antifungals
myocardial ischemia
myocardial infarction

can cause...

QT prolongation

21

Pt comes in with chest pain. EKG shows HUGE!!! T waves n V3-V5..whats going on?

Early MI!!! Anterolateral*

22

Prolonged QT interval puts at risk for...

V fib

23

OD of tricyclic antidepressants can cause...

ST depression

24

Pt comes in with diffuse ST elevation..seen in all leads except aVL and aVR. What is it? And DOC?

Acute pericarditis

tx with NSAIDS

25

EKG with LOW VOLTAGE...what could be going on?

Pericardial effusion

26

EKG with narrow QRS and rate of 150. Can be 3 things....

1. atrial flutter with 2:1 AV block
2. PSVT
3. sinus tachycardia

27

Young healthy individual
Sinus bradycardia
Slight ST elevation

Early repolarization

28

deep T wave inversions until proven otherwise mean...

ischemia or NSTEMI

29

wide, bizarre QRS complexes
**small vertical line seen before every QRS

electronic ventricle pacemaker!

(ventricles are conducting on a cell to cell basis, which is why there are wide QRS)

30

vertical line seen before every P wave

atrial pacemaker

31

Causes SA nodal suppression and AV block
*can basically cause any arrhythmia

Digoxin

32

Most common arrhythmia caused by Digoxin

Multifocal PVCs

33

2 most specific arrhythmias caused by Digoxin

Accelerated junctional rhythm
Atrial tachycardia with AV block

34

Diffuse peaked T waves
no P waves
QRS widens, merges with T waves
Sine wave forms

*V fib may eventually develop

HYPERkalemia

35

Pt comes in with NVD
ECG shows BIG** T waves in precordial leads

HYPERkalemia

36

class 1a, 1c, 3 anti-arrhythmic agents
erythromycin, non sedating anti histamines
macrolides, antifunals

can all cause...

QT prolongation