ECG finishing touches lecture

Question 1

Bundle of Kent that directly connects atrium to ventricles

Answer 1

Wolff-Parkinsons White

Question 2

PR interval shortened to under 0.12 seconds
QRS widened to more than 0.10
***delta wave present

Answer 2

WPW

Question 3

hallmark*** slurring of upstroke due to Kent bundle conduction (delta wave), wide QRS at base (between 2-2.5 boxes wide), narrow QRS at top

Answer 3

WPW

Question 4

Arrhythmias associated with WPW

Answer 4

PSVT
A fib (even faster than usual bc Kent Bundle conducts faster than AV node) 

**can have vent rates up to 300 with a fib in WPW

Question 5

Cannot read what in a WPW EKG?

Answer 5

Ischemia
Infarct
LVH

Question 6

• *Cannot interpret Q waves (if present) for MI in the presence of WPW abnormality on ECG.
• *Cannot interpret LVH by voltage criteria (if present) in the presence of WPW abnormality.
• *Cannot interpret ischemia via ST segment depression or T wave inversion in the presence of WPW abnormality.

Answer 6

WPW

Question 7

99% of the time, irregularly irregular EKG means…

Answer 7

A fib

Question 8

Bypass from atrium into Bundle of His

Answer 8

Lown-Ganong-Levine Syndrome

Question 9

Very short PR interval

Associated w PSVT

Answer 9

Lown Ganong Levine Syndrome

Question 10

This drug has a narrow therapeutic to toxic ration and is a potent stimulator of arrhythmias

Answer 10

Digoxin

Question 11

ST segment scooping

Answer 11

Digoxin

Question 12

Multiformed PVCs are most common EKG presentation of

Answer 12

Digoxin toxicity

Question 13

Digoxin causes SA nodal suppression and…

Answer 13

AV block

Question 14

• Accelerated junctional rhythm

* Atrial tachycardia with AV block

Answer 14

seen with Digoxin

Question 15

mild ST segment depression
Flattening of the T wave
Appearance of a U wave

Answer 15

HYPOkalemia

Question 16

T waves across the entire 12 lead EKG begin to peak

Answer 16

HYPERkalemia

Question 17

PR interval become prolonged, and the P wave gradually flattens and then disappears

Answer 17

HYPERkalemia

Question 18

Ultimately, QRS complex widens until it merges with the T wave, forming a SINE WAVE** pattern. V fib may eventually develop

Answer 18

Hyperkalemia

Question 19

QT interval should not exceed ______ of the R-R interval

Answer 19

one half

Question 20

tricyclic antidepressants
erythromycin (and other macrolides)
antifungals
myocardial ischemia
myocardial infarction

can cause…

Answer 20

QT prolongation

Question 21

Pt comes in with chest pain. EKG shows HUGE!!! T waves n V3-V5..whats going on?

Answer 21

Early MI!!! Anterolateral*

Question 22

Prolonged QT interval puts at risk for…

Answer 22

V fib

Question 23

OD of tricyclic antidepressants can cause…

Answer 23

ST depression

Question 24

Pt comes in with diffuse ST elevation..seen in all leads except aVL and aVR. What is it? And DOC?

Answer 24

Acute pericarditis

tx with NSAIDS

Question 25

EKG with LOW VOLTAGE...what could be going on?

Answer 25

Pericardial effusion

Question 26

EKG with narrow QRS and rate of 150. Can be 3 things....

Answer 26

1. atrial flutter with 2:1 AV block 2. PSVT 3. sinus tachycardia

Question 27

Young healthy individual Sinus bradycardia Slight ST elevation

Answer 27

Early repolarization

Question 28

deep T wave inversions until proven otherwise mean...

Answer 28

ischemia or NSTEMI

Question 29

wide, bizarre QRS complexes | **small vertical line seen before every QRS

Answer 29

electronic ventricle pacemaker! | ventricles are conducting on a cell to cell basis, which is why there are wide QRS

Question 30

vertical line seen before every P wave

Answer 30

atrial pacemaker

Question 31

Causes SA nodal suppression and AV block | *can basically cause any arrhythmia

Answer 31

Digoxin

Question 32

Most common arrhythmia caused by Digoxin

Answer 32

Multifocal PVCs

Question 33

2 most specific arrhythmias caused by Digoxin

Answer 33

Accelerated junctional rhythm | Atrial tachycardia with AV block

Question 34

Diffuse peaked T waves no P waves QRS widens, merges with T waves Sine wave forms *V fib may eventually develop

Answer 34

HYPERkalemia

Question 35

Pt comes in with NVD | ECG shows BIG** T waves in precordial leads

Answer 35

HYPERkalemia

Question 36

class 1a, 1c, 3 anti-arrhythmic agents erythromycin, non sedating anti histamines macrolides, antifunals can all cause...

Answer 36

QT prolongation