Clinical Psychology Flashcards

(55 cards)

1
Q

Mental disorder/psychopathology

A

Persistent disturbance or dysfunction in behavior, thoughts, or emotions that causes significant DISTRESS and IMPAIRMENT

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2
Q

Medical model of mental disorders

A

Like physical illnesses, mental illnesses have biological and environmental causes, defined symptoms, and cures

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3
Q

Disorder vs disease vs diagnosis

A

Disorder: common set of signs (OBJECTIVELY observed indicators) and symptoms (SUBJECTIVELY reported thoughts, behaviors, emotions)

Disease: known pathological process affecting the body

Diagnosis: determination of whether a disorder or disease is present

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4
Q

The DSM

A
  • DSM = Diagnostic & Statistical Manual of Mental Disorders
  • Standardization of diagnoses; classification system for each known mental disorder, symptoms/diagnostic criteria, etc
  • Most recent release is DSM-5 + DSM-5-TR (test revision)
  • Used to use roman numerals to number them but now use arabic numbers so we can do updates to the same version (5.1, 5.2, etc) –> publish updates more frequently instead of every 20 yrs

Drawbacks:
- May feel like “labeling”
- Can be problematic if just “below” the cutoff
- Doesn’t acc for subjective experience (ppl can experience things in different ways)

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5
Q

The ICD

A

USA uses DSM but many other countries use WHO’s International Classification of Diseases (ICD)

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6
Q

The RDoC

A
  • Research Domain Criteria project
  • Initiative aiming to guide classification of mental disorders by focusing on underlying processes
  • Addresses growing concern that research findings on biopsychosocial factors that appear to cause psychopathology don’t neatly map onto DSM/ICD diagnoses
  • Can help explain comorbidity
  • Not meant to replace DSM/ICD in any way, but serve as a guide+ inform future revisions
  • CONSTRUCT: biopsychosocial processes that, at extremes, can give rise to mental disorders (e.g. fear, anxiety)
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7
Q

Biopsychosocial model of causation

A

States that mental disorders are the result of interactoins btw biological, psychological, and social factors

Biological: genetics, epigenetics, chem imbalances, brain structure

Psychological: maladaptive learning + coping, biases, dysfunctional attitudes

Social: poor socialization, stress life experience, cultural and social inequities

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8
Q

Diathesis-stress model

A
  • Specific theory within biopsychosocial model; DESCRIBES how the biological, psychological, and social factors interact w each other
  • States that a person may be predisposed to a psychological disorder that remains unexpressed unless triggered by stress
  • Diathesis –> stress –> psychological disorder

Diathesis: the predisposition (e.g. brain structure, hormones, genes)

Stress: the catalyst (e.g. abuse, loss, onset of physical illness)

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9
Q

Comorbidity

A

Co-occurrence of 2+ diseases in a person

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10
Q

Anxiety disorder

A
  • Anxiety is the predominant feature
  • Significant comorbidity w depression
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11
Q

4 main types of anxiety disorder

A

Phobic disorders, panic disorders, social anxiety disorder, Generalized Anxiety Disorder (GAD)

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12
Q

Phobic disorders + 2 main types

A
  • Persistent, excessive fear and avoidance of spceific objects, activities, or situations
  • Ppl w phobic disorders recognize the fear is irrational but can’t prevent it from interfering w everyday function
  • 2 main types: specific and social phobia
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13
Q

Specific vs social phobia

A

Specific phobia: irrational fear of particular object or situation that markedly INTERFERES w everyday func

Social phobia: irrational fear of public humiliation or embarrassment
- Could be specific situations like public speaking or eating in public, but could also include general social situations involving interacting w unfamiliar ppl
- Social phobia dependent on subjective experience, not physiological response

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14
Q

Preparedness theory

A
  • Subtype of diathesis-stress model focusing on fear and phobias specifically
  • Ppl evolutionarily predisposed to fear objects we’re supposed to avoid – supported by heritability
  • Temperament (e.g. shyness) + neurological factors may also play role
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15
Q

Panic disorder & agoraphobia

A

Panic disorder: sudden occurrence of multiple physiological and psychological symptoms that contribute to stark feeling of terror (i.e. panic attacks)
- Acute symptoms can last a few mins and include shortness of breath, heart palpitations, sweating, dizziness

  • Occasional panic attack not sufficient for diagnosis; must cause significant dread and anxiety + IMPAIRMENT in person’s life
  • Intense anxiety and avoidance related to attack for at least 1 mo

Agoraphobia: specific phobia involving public places
- Often comorbid w panic disorder
- Not frightened of public places themselves but afraid that smthn terrible will happen and they won’t be able to escape or get help

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16
Q

Social anxiety disorder

A

Fear of social situations –> worry and diminished day-to-day func

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17
Q

Generalized Anxiety Disorder (GAD)

A
  • Chronic excessive worry abt everyday things that is out of proportion to the specific cause of worry
  • Generalized bc worries aren’t focused on a particular threat
  • At least 6 mo of excessive anxiety + symptoms like fatigue, restlessness, irritability, conc problems
  • Mild-modest heritability
  • Unpredictable experiences in childhood increase risk of developing GAD
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18
Q

PTSD

A
  • Post-traumatic stress disorder
  • Caused by exposure to traumatic event
  • Chronic psychological arousal, recurrent unwanted thoughts or images of the trauma, avoidance of things that call the traumatic event to mind for 1+ mo
  • Most evident in soldiers returning from war; not everyone develops PTSD –> supports preparedness theory
  • Cortical regions: heightened amygdala activity, smaller hippocampus
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19
Q

OCD

A
  • Obsessive-Compulsive Disorder
  • Obsessive thoughts and/or compulsions that seem irrational or nonsensical
  • Obsession: intrusive, obsessive thoughts that produce anxiety
  • Compulsion: repetitive, often ritualistic behavior to remedy intrusive thoughts; may cause relief, but only temporarily
  • Classified separately from anxiety disorders bc researchers believe it has a distinct cause maintained by different neural circuitry than anxiety disorders
  • Obsession suppression can backfire
  • Take up significant amt of time (1+ hrs/day)
  • Cause significant distress or impairment in func
  • Moderate-strong heritability
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20
Q

Mood disorder

A
  • Mood disturbance as predominant feature
  • 2 main forms: depression/depressive disorders and bipolar disorder
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21
Q

MDD

A
  • Major depressive disorder/unipolar depression
  • Severely depressed mood and/or inability to experience pleasure
  • Must have either depressed mood, anhedonia (reduced pleasure in things that used to cause joy), or both + other symptoms
  • Symptoms must last 2+ wks + cause significant distress or impairment in function
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22
Q

PDD

A
  • Persistent depressive disorder
  • Same cog and physiological symptoms as depression; less severe but lasts longer (2+ yrs)
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23
Q

Double depression

A
  • MDD and PDD co-occur
  • Moderately depressed mood that persists for 2+ yrs; punctuated by periods of MDD
24
Q

SAD

A
  • Seasonal affective disorder
  • Recurrent depressive episodes in a seasonal pattern
  • Episodes tend to begin in fall or winter and remit in spring due to lower light lvls in colder seasons
25
Depression biopsychosocial contributing factors
Biological: genetics, neurotransmitters (esp serotonin and NE) Psychological: negative thoughts contribute to depression Social: stressful life events, interpersonal factors
26
Caspi et al study on serotonin transporter gene
- Serotonin (5-HT) transporter gene that faciliates serotonin reuptake - One allele is longer (l), one is shorter (s) - Gene itself is not related to depression, but it seems to make indivs more sensitive to life stressors (gene x env interaction) - Study examined s/s, l/l/, and s/l combo of alleles - As # of stressful life events increased, odds of MD episode increased BUT s/s had much higher prob than l/l --> shorter allele correlated w depression
27
Beck's cognitive model of depression
States that biases in how info is processed and remembered leads to + maintains depression
28
Helplessness theory
Ppl prone to depression automatically attribute neg experiences to causes that are: - INTERNAL (e.g. my bad math grade = I'm stupid) - STABLE (e.g. I'll always be stupid) - GLOBAL (e.g. I'll fail in all future life endeavors)
29
Negative schema
- Deeply ingrained neg beliefs abt oneself, the world, etc - Caused by combo of genetic vulnerability + neg early life events in ppl w depression - Seeing the world thru "gray glasses"
30
Bipolar disorder
- Cycles of mania and depression - Manic phase must last 1+ wks to fit DSM reqs - Depressive phase often indistinguishable from MDD - Depressive episodes tend to last longer than manic episodes (5mo vs 3mo) - Has one of the highest rates of heritability - Can be hard to ID -- risk factors and symptoms also associated w MDD, autism, schizophrenia, and ADHD - Biological: genetics (twin studies) - Psychosocial: life stressors + positive life events can lead to more manic episodes
31
Bipolar I vs II disorder
Bipolar I: person experiences at least 1 depressive episode and at least 1 manic episode Bipolar II: person experiences at least 1 depressive episode followed by HYPOMANIC episode -- elevated mood but lower intensity than mania
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Rapid cycling bipolar disorder
- At least 4 mood episodes (either manic/hypomanic or depressive) - Esp difficult to treat
33
Schizophrenia
Psychotic disorder; characterized by profound disruption of basic psychological processes, distorted perception of reality, blunted emotion, and more - Diagnosed when 2+ symptoms emerge during continuous period of 1+ mo and signs persist for 6+ mo - Often has onset from 20-29 - Ppl w schizophrenia do not know their perception is disordered - 3 types of symptoms: positive, negative, cognitive - Hereditary + prenatal (e.g. toxins in mom's blood) and perinatal env also affect prognosis - Social factors include family env and severely disturbed fams - Dopamine hypothesis: schizophrenia involves excess of DA activity -- largely proven to be inaccurate - Enlarged ventricles (cavity in brain) -- suggests loss of brain-tissue mass
34
Schizophrenia positive symptoms
- Things ADDED when disease is present - Hallucinations, delusions, disorganized speech, disorganized behavior HALLUCINATIONS: false perceptions - Can involve any of the senses - ~65% report hearing voices repeatedly -- voices are negative, scolding, etc; can sound like they're internal or external - Auditory hallucinations accompanied by activation of Broca's area DELUSIONS: false beliefs that are often bizarre and grandiose but maintained despite irrationality - Delusions of identity (e.g. person thinks they're Ariana Grande) - Delusions of persecution (e.g. person believes the CIA is conspiring to harm them) DISORGANIZED SPEECH: in verbal communication, ideas shift rapidly and incoherently - Different than Wernicke's aphasia bc they understand the question DISORGANIZED BEHAVIOR: behavior inappropriate for situation or ineffective in attaining goals - E.g. childlike silliness, disheveled appearance - Can also be motor disturbances like strange movements and bizarre grimacing
35
Schizophrenia negative symptoms
- LOSS of normal function - Anhedonia, amotivation, alogia, flat effect, catatonic behavior ANHEDONIA: things that used to make you happy don't anymore AMOTIVATION: no motivation to do the things you like ALOGIA: decrease in speech + delayed speech response FLAT EFFECT: flattening of emotion; apathy CATATONIC BEHAVIOR: marked decrease in all movement; or increase in muscle rigidity - Ppl in CATATONIA may actively resist movement or become unresponsive/unaware of their surroundings - Those doing drug therapy might also exhibit moto symptoms as a side effect
36
Schizophrenia cognitive symptoms
- Can be present BEFORE onset of distinct schizophrenia symptoms - Difficulty w attention, executive func, problem-solving, working memory - Least noticeable of the symptoms but often plays large role in keep ppl w schizophrenia from achieving high lvl of func (making friends, keeping a job)
37
Psychotherapy
Ineraction btw sanctioned clinician and someone suffering from a psychological problem; VERY broad term
38
Eclectic psychotherapy
Involves drawing techniques from different forms of therapy; very adaptable depending on the client and the problem
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Psychoanalytic therapy
- Patient does most of the insight - Main goal of bringing repressed material to the surface - Focuses on how unconscious thoughts and early childhood experiences influence behavior - Rooted in Freud
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Psychodynamic therapy
- Therapist actively works w patient to increase self-awareness - Emphasis on understanding emotional patterns in real-time (unlike psychoanalytic therapy, which focuses on the unconscious and past) - Rooted in Freud
41
Existential-humanistic therapy
- Assumes human nature is generally positive - Emphasizes natural tendency of each indiv to strive for personal improvement - Assumes that psychological problems stem from feelings of alienation and loneliness Ex: Person-centered therapy, Gestalt therapy
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Person-centered therapy
- Type of existential-humanistic therapy - Therapist tends to be more PASSIVE and just paraphrase the client's words back at them to show support; goal is NOT to uncover repressed conflicts
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Gestalt therapy
- Type of existential-humanistic therapy - Therapist ACTIVELY helps client become aware of their own thoughts, behaviors, etc and to take responsibility for them - Emphasis on stuff occurring at that moment in the therapy session
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Cognitive behavior therapy (CBT)
- Teach ppl new/more constructive ways of thinking and acting (e.g. was the event really negative, or was it neutral and your interpretation was negative?) - Often involves homework (e.g. exposure exercises, diary, etc) - Can be better for long-term than
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Psychopharmacology
Study of effect of drugs and mind on behavior
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Antipsychotic medicine
Ex: chlorpromazine/Thorazine for schizophrenia - Reduces psychotic symptoms (e.g. hallucinations) - Blocks DA receptor sites + some also affect 5-HT ssystem - Treats some positive symptoms but not the negative ones
47
Conventional vs atypical antipsychotics + which is more commonly used
Conventional: blocks DA receptors Atypical: blocks DA AND serotonin receptors ATYPICAL antipsychotics more commonly used as frontline today; conventional antipsychotics usually involve having to take another medication to address side effects
48
Antianxiety medications
- Drugs that help reduce a person's experience of fear or anxiety - Commonly benzodiazepines -- class of drugs that increases GABA (primary inhibitory neurotransmitter) - Doctors are cautious when prescribing bc of potential for abuse
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Antidepressant medications (4 main types)
MAOIs, TCAs, SSRIs, SNRIs - Antidepressants not recommended for ppl w BPD bc the antidepressant effect may inadvertently trigger manic episode 1) MAOIs - Monoamine oxidase inhibitors - Inhibits enzyme from breaking down neurotransmitters like DA, NE, and serotonin in synapse 2) TCAs - Tricyclic antidepressants - Blocks reuptake of NE and serotonin 3) SSRIs - Selective serotonin reuptake inhibitors - Inhibits reuptake of serotonin in synaptic cleft - Can also help treat anxiety and eating disorders 4) SNRIs - Serotonin & norepinephrine reuptake inhibitors - Blocks reuptake of NE and serotonin but has less severe side effects than TCAs
50
True or false: antidepressant meds work right away
FALSE Unlike antianxiety meds, antidepressants can take up to a month to have an effect
51
Mood stabilizers
- Can help suppress swings btw mania and depression in those w BPD - Can also help w depression
52
Is medication better than psychotherapy or vice versa?
- Not necessarily -- depends on condition being treated - For schizophrenia, medication is more effective, but for mood and anxiety disorders, meds and psychotherapy can be equally helpful - One issue is that the 2 treatments are often provided by 2 different ppl --> lots of coordination required btw psychologists and psychiatrists
53
Electroconvulsive therapy (ECT)
- Shocks delivered to brain via electrodes on scalp - Primarily used to treat severe depression that hasn't responded to medication; has also been shown to be effective in treating BPD
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Transcranial magnetic stimulation (TMS)
- Powerful pulsed magnet placed over person's scalp to alter neuronal activity - Can treat depression - New but exciting -- non-invasive + fewer side effects than ECT, but just as effective
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Deep brain stimulation (DBS)
- Small battery-powered device implanted in brain to deliver elec stim to specific areas of the brain - Has worked for severe depression, OCD, and tremor for Parkinson's