Week 4 Flashcards
(42 cards)
Sensation & transduction
Sensation: Stim of sense organ
Transduction: receptors convert physical from environment to signals for brain to interpret
Perception
Interpretation of a sensation –> mental representation
Sensory adaptation
Sensitivity to prolonged stim tends to decline over time as one adapts to current conditions
Psychophysics
Study of how physical stim affect the senses
Absolute threshold
Minimal intensity needed to barely detect a stim 50% of the time
Sensitivity
How responsive one is to faint stim
Acuity
Differentiating btw two stim (e.g. two similar tones)
Just noticeable difference (JND)
Minimal change in a stim that can barely be detected
Weber’s law
Amount of change needed for change to be noticed is constant ratio of the original stim
Signal detection theory
Response to a stim depends on person’s sensitivity to stim and a person’s decision critereon
How is light interpreted in the eye?
(keywords: cornea, pupil, iris, lens, accommodation, retina, photoreceptor cells, optic nerve)
1) Light passes through CORNEA (clear protective layer around eye)
2) Light passes through PUPIL (hole in the IRIS, the colored part of the eye that controls how much light is let in)
3) Light passes through LENS, which is shaped by eye muscles so the light is focused on the RETINA in a process called ACCOMMODATION (lens flatter for objects further away, lens more curved for objects nearby)
4) Retina lined with PHOTORECEPTOR CELLS called RODS and CONES
5) Signals from rods and cones transducted to brain via the OPTIC NERVE (OPTIC DISK is the beginning of the nerve in the eye)
Myopia vs hyperopia
Myopia/nearsighted - eyeball too long –> image focused in FRONT of retina
Hyperopia - eyeball too short –> image focused BEHIND retina
Rods
- Peripheral and night vision, brightness
- More rods than cones (~120M rods)
- Distributed evenly in retina except for area in the macula called the FOVEA
- No rods in fovea –> reduced clarity in low light but increased sensitivity to light in periphery
Cones
- C words - “Clarity and Color”
- ~6M cones; concentrated in the FOVEA but sparse in macula/retina –> we see things much clearer if we look right at them
- 3 different cones for 3 different wavelengths: L-cones (long - red), M-cones (medium - green), S-cones (short - blue)
- Missing cones –> color blindness
Trichromatic color theory
We perceive color by combining RGB wavelengths
Color-opponent system
Pairs of cone types work against each other
What causes negative afterimages?
Opponent process theory: cones are together in opposing pairs: red/green, yellow/blue, black/white
- Cells stimulated by green inhibit the cells stimulated by red
- When changed, the previously inhibited red cells fire, while the green PREVIOUSLY-stimulated cells are tired and don’t fire –> we see red
Neurological pathway from photoreceptor cells to brain
rods/cones –> bipolar cells –> retinal ganglion cells –> lateral geniculate nucleus (part of thalamus) –> area v1 –> other parts of the brain
NOTE: bunded RGCs form the optic nerve
Dorsal vs ventral vision pathways + purpose
Dorsal: “where” – where an object is and its movement; upper pathway to parietal lobe
Ventral: “what”/”how” – shape and identity of an object; lower pathway to temporal lobe
Purpose: Neurons in area v1 only perceive small details like edge –> signals to regions farther from V1 respond to more complex features
Visual agnosia vs prosopagnosia
Visual agnosia: inability to recognize objects
Prosopagnosia: inability to recognize faces
Patient DF
Injured ventral stream –> couldn’t recognize objects
Bottom-up processing
Sensory receptors pick up signals and send them to the brain
Top-down processing
Perceiving things based on your prior knowledge of the world
Monocular/pictorial depth cues
- Can be seen w one eye
- Linear perspective/vanishing point, light and shadow, interposition, relative height, relative size, texture gradient