Common Pathologies lower limb Flashcards
Aim of physio intervention
differential diagnosis, determine appropriateness of physio/ other assessments (X-rays, scans, blood tests)/ interventions (injections/ surgery), to rehabilitate LL dysfunction conservatively
diagnosis and management
understand who gets it (typical patient), understand disease aetiology, understand typical presentation, understand why aetiology causes presentation, understand management
different groups of LL pathologies
tendinopathies (gluteal, achilleas, plantar fascia, rupture), OA (hip>knee»ankle), joint disorders (FAI, meniscal tears), sprains (ACL/PCL, MCL/LCL, ankle- LLS=ATFL, CFL), adolescents (OGS. SLJ)
LL MSK disorders- vascular, bone, muscle/tendon
Vascular= Venous (DVT) or arterial, bone= stress fracture or fracture, muscle/tendon= tendinopathies or muscle strains
LL MSK disorders- neural and joint
neural= entrapment, peripheral, spinal joint= degenerative, inflammatory, traumatic
what causes insertional tendinopathy
due to calcaneal compression
typical GTPS patients and subjective
female/ postmenopausal/ increased BMI/ comorbidities
subjective= diffuse pain, sleep disturbance, standing on one leg, walking, hills/stairs
gluteal tendinopathy treatment
reduce compression, increase strength, increase functional strength and control. increase resilience to compression
physio main treatment for GTPS
education, load management, self- management strategies, strengthening exercises targeted to hip abductors
activity modification
reduce external loading- volume, intensity, frequency, duration, heel raises
MTU- functions as a shock absorber and spring- good
good muscle coordination, increased energy efficiency, greater elastic recoil- less hear production- -0tentional to improve or protect from tendinopathy
MTU- functions as a shock absorber and spring- bad
poor muscle coordination (ineffective function)- reduced energy efficiency and greater amplitude of tendon strain or greater cumulative load- greater energy absorption (More heat)- potential to trigger cellular response and tendinopathy
neuromuscular demanding exercise programme- achilleas (step up)
work away from patient preferred speed/ tempo, ensure full DF, stop movement at different positions, make patient aware of tremor- aim to smooth this, add external load early, different knee flexion angles, movement straight up (not leaning forward), change limb alignment
what happens with OA
breakdown of articular cartilage, fibrillation, fissures, gross ulcerations, disappearance of articular surface, osteophyte formation, thickening of subchondral bone, synovial membrane changes
clinical features of LL OA
high levels of activity (early OA), hip pain on movement/ walking, pain around joint, loss of ROM (on PROM R>P), antalgic or trendelenburg gait
treatment for OA
depends on severity of condition:
mobilisations for pain and stiffness, strengthening and ROM exercises, lifestyle/weight loss, surgery where pain and stiffness are severe= TJR or compartment replacement
what muscle groups are commonly injured
hamstrings>calf>groin>quads
groin strain- muscles and causes
muscles- adductors, iliopsoas, rectus femoris
cause- inflammation from overuse muscle/tendon direct trauma/biomechanics, inflammation/ pain on movement or contraction= loss of function/ weakness, visible/ palpable defect
hamstring strain rehab
POLICE, early load, length of muscle (ROM/ stretch), strengthen, pain relief if needed
collateral ligament injury- mechanisms of injury
usually varus/valgus contract force (i.e.direct blow to the knee can also occur as a result of a varus or valgus blow to the foot, MCl most commonly injured structure in knee> common than LCL, characteristic instability caused by MCL/ LCL injury is opening of medial/lateral joint space
signs and symptoms of lig injury
any age group but often early adulthood, focal joint pain, +/- h/o clicking/ popping, giving way, locking, swelling developing (24+ hours for extra articular lig, <24 for intra articular), positive test
ACL- MOI
injury on external rotation, usually sudden deceleration and change of direction with fixed foot or hyperextension injury, often combined with valgus strain of MCL and medial meniscus
ACL- signs and symptoms
knee buckles, unable to stand, immediate swelling- haemarthrosis (within 2 hours), inability to resume sport, locking/ loss of extension, positive lachmans test
PCL- MOI and S+S
MOI- posterior force to proximal tibia, if combined with rotational force injury to P-L complex
S+S= mild to mod swelling, positive posterior draw test, often asymptomatic, pain on kneeling