ECG Flashcards

1
Q

U have no Pot no T just a long PR & QT

mnemonic for?

A

Hypokalemia (no pot)
U waves
absent T waves
Prolonged PR & QT

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2
Q

Jesus Quist it’s Bloody Freezing

mnemonic for?

A
Hypothermia
J waves
prolonged QT
Bradycardia
First degree heart block
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3
Q

ECG: digoxin

long QT interval

A

false

short

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4
Q

ECG: digoxin

raised T waves

A

false

flattened or inverted

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5
Q

ECG: digoxin ST wave features?

A

down-sloping ST depression (‘reverse tick’, ‘scooped out’)

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6
Q

ECG: digoxin arrhythmias?

A

AV block, bradycardia

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7
Q

ECG: hyperkalaemia Peaked or ‘tall-tented’ T waves occurs first

A

true

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8
Q

ECG: hyperkalaemia which waves absent?

A

P waves

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9
Q

ECG: hyperkalaemia narrow/broad QRS

A

broad

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10
Q

ECG: hyperkalaemia can lead to VF

A

true

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11
Q

ECG: hyperkalaemia characteristic wave pattern

A

sinusoidal

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12
Q

WiLLiaM MaRRoW looks at changes in which leads

A

V1 & V6

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13
Q

in LBBB there is a ‘?’ in V1 and a ‘?’ in V6

A

in LBBB there is a ‘W’ in V1 and a ‘M’ in V6

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14
Q

in RBBB there is a ‘?’ in V1 and a ‘?’ in V6

A

in RBBB there is a ‘M’ in V1 and a ‘W’ in V6

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15
Q

Bifascicular block features RBBB/LBBB

A

RBBB

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16
Q

Bifascicular block features RBBB alongside right/left hemiblock

A

left anterior or posterior hemiblock

e.g. RBBB with left axis deviation

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17
Q

Trifascicular block includes

features of bifascicular and

A

1st-degree heart block

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18
Q

Posterior STEMI features which ECG changes

A

Tall R waves V1-2

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19
Q

STEMI areas and coronary artery affected

A

Anteroseptal V1-V4 Left anterior descending

Inferior II, III, aVF Right coronary

Anterolateral V4-6, I, aVL Left anterior descending or left circumflex

Lateral I, aVL +/- V5-6 Left circumflex

Posterior Tall R waves V1-2 Usually left circumflex, also right coronary

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20
Q

LBBB/RBBB may point towards a diagnosis of acute coronary syndrome.

21
Q

Acute myocardial infarction (MI) T wave changes in first few minutes are T waves typically become inverted

A

false

hyperacute T waves are often the first sign of MI but often only persists for a few minutes

22
Q

Acute myocardial infarction (MI) T wave changes in first 24 hours

A

T waves typically become inverted within the first 24 hours

23
Q

Acute myocardial infarction (MI) inversion of the T waves can last for 48 hours

A

false

days to months

24
Q

Acute myocardial infarction (MI) pathological Q waves develop after several hours to days

25
Acute myocardial infarction (MI) pathological Q waves persists infinitely
true
26
clinical symptoms consistent with ACS (generally of ≥ 20 minutes duration) with persistent (> 20 minutes) ECG features in ≥ 2 contiguous leads
true
27
A posterior MI causes ST depression not elevation on a 12-lead ECG.
true
28
ECG features STEMI | in men under 40 years
2.5 mm (i.e ≥ 2.5 small squares) ST elevation in leads V2-3
29
ECG features STEMI | in men over 40 years
≥ 2.0 mm (i.e ≥ 2 small squares) ST elevation in leads V2-3
30
ECG features STEMI in women
1.5 mm ST elevation
31
new LBBB/RBBB is ECG feature of STEMI
new LBBB
32
ECG features STEMI 1 mm ST elevation in other leads (not V2/V3)
true
33
Causes of RBBB
``` right ventricular hypertrophy chronically increased right ventricular pressure - e.g. cor pulmonale pulmonary embolism myocardial infarction atrial septal defect (ostium secundum) cardiomyopathy or myocarditis ```
34
RBBB is a normal variant - more common with increasing age
True
35
Causes peaked T waves includes hyperkalaemia & myocardial ischaemia
True
36
Inverted T waves causes
``` myocardial ischaemia digoxin toxicity subarachnoid haemorrhage arrhythmogenic right ventricular cardiomyopathy pulmonary embolism ('S1Q3T3') Brugada syndrome ```
37
Increased P wave amplitude is a feature of
cor pulmonale
38
Broad, notched (bifid) P waves a sign of left atrial enlargement, classically due to
mitral stenosis | often most pronounced in lead II
39
In atrial fibrillation, there is an absence of P waves.
true
40
Causes of ST depression
``` secondary to abnormal QRS (LVH, LBBB, RBBB) ischaemia digoxin hypokalaemia syndrome X ```
41
A prolonged PR interval may also be seen in athletes
true
42
Causes of a prolonged PR interval
``` idiopathic ischaemic heart disease digoxin toxicity hypokalaemia* rheumatic fever aortic root pathology e.g. abscess secondary to endocarditis Lyme disease sarcoidosis myotonic dystrophy ```
43
A prolonged PR interval may also be seen in WPW
false | short PR
44
Causes of left axis deviation (LAD)
left anterior hemiblock left bundle branch block inferior myocardial infarction Wolff-Parkinson-White syndrome* - right-sided accessory pathway hyperkalaemia congenital: ostium primum ASD, tricuspid atresia minor LAD in obese people
45
Causes of right axis deviation (RAD)
``` right ventricular hypertrophy left posterior hemiblock lateral myocardial infarction chronic lung disease → cor pulmonale pulmonary embolism ostium secundum ASD Wolff-Parkinson-White syndrome* - left-sided accessory pathway normal in infant < 1 years old minor RAD in tall people ```
46
The following ECG changes are considered normal variants in an athlete
sinus bradycardia junctional rhythm first degree heart block Wenckebach phenomenon
47
Causes of ST elevation include
``` myocardial infarction pericarditis/myocarditis normal variant - 'high take-off' left ventricular aneurysm Prinzmetal's angina (coronary artery spasm) Takotsubo cardiomyopathy ```
48
ECG changes WPW
short PR interval wide QRS complexes with a slurred upstroke - 'delta wave' left axis deviation if right-sided accessory pathway* right axis deviation if left-sided accessory pathway*