ECG abnormalitys Flashcards

1
Q

What are the causes of bradycardia

A

The conduction blocks
Reduced autonomality

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What is reduced autonomality

A

Low metabolic rate (hypothermia or hypothyroidism)
The endurance runners
The increased vagal tone - parasympathetic (so the vasodilation)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What are the forms of conduction (heart) blocks

A

1st:the elongation of the PR interval
2nd: mobitz 1 - the elongation of the PR interval each beat, this would happenuntil there would be no QRS
Mobitz 2 - no elongated PR but would just have the loss of the QRS
3rd: the beats of the ventricles and the atria would not match,they would each be conducting a different rhythm

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

How do you measure a normal heart rate

A

300/n
N being the number of boxes between the next beat

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

How do you measure a irregular heart rate

A

The number of beats in the 10s time frame (either the p or the r waves)
Then X6

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What is the length and the height of a big box in the ECG

A

Length: 0.25 seconds
Height: 0.5 volts

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

What are the reasons for tachycardia

A

Increased autonomality
Triggered activity
Re-entered currents

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

What is the cause of the increased autonomality

A

Sympathetic overstimulation
High metabolic rate
Anxiety or stress

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What is the re-entered currents

A

When would have the scar tissue within the atria, this would be able to conduct some of the impulse
The impulse would then be carried here and would go round in a Circle, so would have the increased stimulation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

What is involved in the triggered activity

A

The EAD (early after depolarisation)
The DAD (delayed after depolarisation)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What is E.A.D

A

Current would continue from the phase 2 and 3
When the Na+ channels would not be ready
Ventricles would not contact properly
Normally caused by Drugs

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What is D.A.D

A

Would occur at phase 4 of depolarisation
Ca+ channels would not be ready
Ventricles would not pump well
Normally caused by the ischemia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

How many small boxes should a P wave be

A

3 small boxes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

How many boxes would the PR interval be

A

3-5 small boxes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What should the QRS complex look like

A

Should be upright
Less the 3 small boxes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

What should the T-wave look like

A

Upright and not inverted
Should be half the size of the QRS complex

17
Q

What does the QT segment look like and what does it show

A

Should be 9-11 boxes
Would show the depolarisation and repolarisation

18
Q

What should the ST segment look like

A

Should be a straight line

19
Q

What is atrial fibrillation

A

The irregularly irregular rhythms
Would have the many P waves
Would look like f-waves

20
Q

What is the main issue with the atrial fibrillation

A

The formation of the clot in the heart from the pooling of the blood
This would then lead to thrombosis and the scar tissue forming
Clots can form in the brain

21
Q

What is an eptoic heart beat

A

Would have the large ventricle beat and depolarisation
So would have the large repolarisation
Would feel as if miss a beat

22
Q

What is is monomorphic ventricular tachycardia

A

The QRS complexes would be wide and slow
Would lose the other waves

23
Q

What is polymorphic ventricle tachycardia

A

The messed up ventricle contractions
Would not be pumping enough blood
AKA tosades depointes

24
Q

What would happen in ventricular tachycardia

A

Would have the heart failure
The ventricles would not be pumping blood out the body

25
Q

What would happen to the heart rate with the VTs

A

Low
Still not pumping enough blood out the heart, so the HR can’t increase

26
Q

What causes the myocardial infarction

A

The plaques in the coronary artery’s
Would go to the heart and could lead to the damage
Would stop oxygen from getting to a certain area
So would have the necrosis and the infarct form

27
Q

What is the subendocardial infarction

A

When would have plaque effect the ventricles tissue
LEADS TO AN INVERTED T WAVE AND ST DEPRESSION

28
Q

What is a trans mural infarction

A

When have the whole area effected
THE ST ELEVATION OCCURS and missing T wave
HAVE THE T WAVE INVERSION WHEN HAVE THE INTERVENTION AND REPERFUSION OF THE BLOOD

29
Q

What does hypokalemia do to the ECG

A

The depression of the ST interval
small or absent T waves
Slow depolarisation and repolarisation

30
Q

What does hyperkalemia do to the ECG

A

The t wave peak, flat P wave, PR prolonged
Quick repolarisation
The depolarisation would take a long time so would have this area prolonged (due to the inactive Na+ channels)

31
Q

What does myocardial ischaemia normally come with

A

The hyperkalemia
The graph for them would therefor be similar

32
Q

What can hypokalemia deteriorate to

A

The polymorphic ventricle tachycardia (torsades de pointes)
Ventricular tachycardia
Ventricular fibrillation

33
Q

Why would diabetics get hyperkalemia

A

Insulin activates the Na+/k+ pump
This would then allow K+ to move into the cells
If not would have the high extracellular K+ levels