ECG abnormalitys Flashcards

(33 cards)

1
Q

What are the causes of bradycardia

A

The conduction blocks
Reduced autonomality

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2
Q

What is reduced autonomality

A

Low metabolic rate (hypothermia or hypothyroidism)
The endurance runners
The increased vagal tone - parasympathetic (so the vasodilation)

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3
Q

What are the forms of conduction (heart) blocks

A

1st:the elongation of the PR interval
2nd: mobitz 1 - the elongation of the PR interval each beat, this would happenuntil there would be no QRS
Mobitz 2 - no elongated PR but would just have the loss of the QRS
3rd: the beats of the ventricles and the atria would not match,they would each be conducting a different rhythm

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4
Q

How do you measure a normal heart rate

A

300/n
N being the number of boxes between the next beat

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5
Q

How do you measure a irregular heart rate

A

The number of beats in the 10s time frame (either the p or the r waves)
Then X6

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6
Q

What is the length and the height of a big box in the ECG

A

Length: 0.25 seconds
Height: 0.5 volts

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7
Q

What are the reasons for tachycardia

A

Increased autonomality
Triggered activity
Re-entered currents

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8
Q

What is the cause of the increased autonomality

A

Sympathetic overstimulation
High metabolic rate
Anxiety or stress

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9
Q

What is the re-entered currents

A

When would have the scar tissue within the atria, this would be able to conduct some of the impulse
The impulse would then be carried here and would go round in a Circle, so would have the increased stimulation

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10
Q

What is involved in the triggered activity

A

The EAD (early after depolarisation)
The DAD (delayed after depolarisation)

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11
Q

What is E.A.D

A

Current would continue from the phase 2 and 3
When the Na+ channels would not be ready
Ventricles would not contact properly
Normally caused by Drugs

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12
Q

What is D.A.D

A

Would occur at phase 4 of depolarisation
Ca+ channels would not be ready
Ventricles would not pump well
Normally caused by the ischemia

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13
Q

How many small boxes should a P wave be

A

3 small boxes

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14
Q

How many boxes would the PR interval be

A

3-5 small boxes

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15
Q

What should the QRS complex look like

A

Should be upright
Less the 3 small boxes

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16
Q

What should the T-wave look like

A

Upright and not inverted
Should be half the size of the QRS complex

17
Q

What does the QT segment look like and what does it show

A

Should be 9-11 boxes
Would show the depolarisation and repolarisation

18
Q

What should the ST segment look like

A

Should be a straight line

19
Q

What is atrial fibrillation

A

The irregularly irregular rhythms
Would have the many P waves
Would look like f-waves

20
Q

What is the main issue with the atrial fibrillation

A

The formation of the clot in the heart from the pooling of the blood
This would then lead to thrombosis and the scar tissue forming
Clots can form in the brain

21
Q

What is an eptoic heart beat

A

Would have the large ventricle beat and depolarisation
So would have the large repolarisation
Would feel as if miss a beat

22
Q

What is is monomorphic ventricular tachycardia

A

The QRS complexes would be wide and slow
Would lose the other waves

23
Q

What is polymorphic ventricle tachycardia

A

The messed up ventricle contractions
Would not be pumping enough blood
AKA tosades depointes

24
Q

What would happen in ventricular tachycardia

A

Would have the heart failure
The ventricles would not be pumping blood out the body

25
What would happen to the heart rate with the VTs
Low Still not pumping enough blood out the heart, so the HR can’t increase
26
What causes the myocardial infarction
The plaques in the coronary artery’s Would go to the heart and could lead to the damage Would stop oxygen from getting to a certain area So would have the necrosis and the infarct form
27
What is the subendocardial infarction
When would have plaque effect the ventricles tissue LEADS TO AN INVERTED T WAVE AND ST DEPRESSION
28
What is a trans mural infarction
When have the whole area effected THE ST ELEVATION OCCURS and missing T wave HAVE THE T WAVE INVERSION WHEN HAVE THE INTERVENTION AND REPERFUSION OF THE BLOOD
29
What does hypokalemia do to the ECG
The depression of the ST interval small or absent T waves Slow depolarisation and repolarisation
30
What does hyperkalemia do to the ECG
The t wave peak, flat P wave, PR prolonged Quick repolarisation The depolarisation would take a long time so would have this area prolonged (due to the inactive Na+ channels)
31
What does myocardial ischaemia normally come with
The hyperkalemia The graph for them would therefor be similar
32
What can hypokalemia deteriorate to
The polymorphic ventricle tachycardia (torsades de pointes) Ventricular tachycardia Ventricular fibrillation
33
Why would diabetics get hyperkalemia
Insulin activates the Na+/k+ pump This would then allow K+ to move into the cells If not would have the high extracellular K+ levels