ECG heart rhythm 1/18 Flashcards

1
Q

what info can a 12 lead ECG provide?

A
HR, rhythm, conduction
areas ischemia and infarction
hypertrophy
electrolyte imbalance
systemic pathologies - COPD, cerebral T-waves, etc.
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2
Q

what ECG part represents

atrial depolarization

A

p wave

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3
Q

what ECG part represents

time required for impulse to travel from atria through conduction system to Purkinje fibers

A

P-R interval

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4
Q

what ECG part represents

ventricular depolarization

A

QRS wave

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5
Q

what ECG part represents

beginning ventricular repolarization

A

ST segment

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6
Q

what ECG part represents

ventricular repolarization

A

T wave

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7
Q

what ECG part represents

time for electrical systole

A

QT interval

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8
Q

what are you calculating?

count # intervals between QRS complex in 6 second strip x 10

A

HR

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9
Q

causes of arrythmias?

A
ischemic conditions of myocarium
electrolyte imbalance
acidosis, alkalosis
hypoxemia
hypotension
emotional stress
drugs, alcohol, caffeine
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10
Q

what term?

  • premature beat from ventricle
  • ocassionally occurs in majority normal pop
A

PVC premature ventricular contractions

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11
Q

ECG:
no P wave
bizarre and wide QRS
long compensatory pause

A

PVC premature ventricular contractions

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12
Q

when to be concerned about PVCs

A

> 6 per minute of in sequential runs
multtifocal
very early PVC (R on T phenomenon)

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13
Q

next step if PVCs increase with activty

A

stop exercise, review cardiac

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14
Q
what term?
3+ PVCs sequentially
very rapid HR 150-200bpm
may have abrupt onset
seriously compromised CO
typical cause?
A

ventricular tachycardia

cause ischemic ventricle

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15
Q
ECG:
no p wave
wide bizarre QRS waves
3+  sequentially
types?
A

ventricular tachycardia
non sustained terminates spontaneously in <30sec
sustained >30sec

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16
Q

what term?

  • pulseless emergency situation, need CPR, defibrillation, medication
  • multiple foci
  • unable to determine rate
A

ventricular fibrillation

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17
Q

ECG:

  • bizarre, erratic activity
  • no QRS complexes
  • no cardiac output
A

ventricular fibrillation

18
Q

if ventricular fibrillation, cardiac death within how long?

A

4-6 min

19
Q

what term?

Rapid repetitive firing of one or more ectopic foci in atria outside of sinus node

A

atrial arrhythmia, supraventricular

20
Q

ECG:
p wave abnormal, variable in shape or not identifiable
- rhythm irregular - chronic or abrupt
- CO maintained if rate maintained

A

atrial arrhythmia, supraventricular

21
Q

ECG:
p wave abnormal, not identifiable
- rhythm irregular - chronic or abrupt

A

atrial fibrillation

22
Q

HR?

  • atrial tachycardia
  • atrial flutter
  • atrial fibrillation
A

tachy 140-250bpm
flutter 250-350bpm
fibrillation >300bpm

23
Q

ECG:

sawtooth appearance

A

atrial flutter

24
Q

what term? - abnormal delay or failure to conduct through conducting system (node>bundle of his>bundle branch > purkinje fibers)
which is life threatening?

A

atrioventricular blocks

- third degree, complete block is life threatening

25
Q

what to do if 3rd degree heart block?

A

complete heart block life threatening

  • medication atropine
  • surgical implant pacemaker
26
Q

when does ST segment depress?

A

impaired coronary perfusion- ischemia or injury

27
Q

what is abnormal ST segment change?

A
  • abnormal depress/elevate >1mm in 2 consecutive leads (except V2-V3)
  • elevate >2mm men >=40yr
  • elevate >2.5mm men <40yr
  • elevate >=1.5mm women
28
Q

what pathology?

acute ST elevations in leads over affected part of heart

A

acute MI

29
Q
hyper/hypokalemia?
ECG:  
wide PR interval
wide QRS
flatten P wave, may disappear
tall T wave
>bradycardia
A

hyperkalemia, increase K+

30
Q
hyper/hypokalemia?
ECG: 
wide PR interval
wide QT interval 
flatten or inverse T, U wave
A

hypokalmeia, decrease K+

31
Q
hyper/hypocalcemia?
ECG:  \
wide QRS
short QT interval
increase
A

hypercalcemia, increase heart action

32
Q

hyper/hypocalcemia?
ECG:
long QT interval

A

hypocalcemia, depress heart action

33
Q

hyper/hypothermia?
ECG:
elevate ST
slow rhythm, decrease PR interval

A

hypothermia, decreased body temp, decrease HR

34
Q
ECG: what medication?
depress ST segment
flatten T wave, or invert
QT shortens
shorten PR interval
A

digitalis - cardiac glycoside
- slow HR
treat congestive heart failure (CHF), heart rhythm problems (atrial arrhythmias).
-works directly on the heart muscle to strengthen and regulate the heartbeat.

35
Q

ECG: what medication?
QRS lengthens
QT lengthens
T wave flattens or inverse

A

quinidine
antiarrhythmic agent in the heart
causes increased action potential duration, as well as a prolonged QT interval.

36
Q

what medication increase HR?

A

nitrates, nitroglycerin

37
Q

what medication prolong QRS and QT intervals?

A

antiarrhythmic agent, eg. quinidine

38
Q

where do V1-V6 precordial chest leads go?

A

V1 - 4th IC, R sternum
V2- 4th IC, L sternum (Tricuspid auscultation)
V3- between V2 and V4
V4- 5th IC, midclavicular line (Bicuspid auscultation)
V5- 5th IC, anterior axillary line
V6- 5th IC, midaxillary line

39
Q

where do 5 lead ECG leads go?

A

RA, RL - white snow over green grass
LA, LL - black smoke over red fire
and Chest

40
Q

what do inverted T wave mean?

A
  • Left Ventricular Hypertrophy

* Myocardial ischemia