ENDO-PERIO RELATIONSHIP Flashcards
(21 cards)
most direct route of communication to the
periodontium, but by no means is it the only location where pulpal and periodontal tissues
communicate with each other
A. Apical Foramen
B. Dentinal Tubules
C. Lateral/ Accessory Canals
D. Pulp Chamber
A
contain cytoplasmic extensions or odontoblastic
processes that extend from the odontoblasts at
the pulp dentin interface to the dentinoenamel
junction (DEJ) or the cementodentinal junction
(CDJ)
A. Apical Foramen
B. Dentinal Tubules
C. Lateral/ Accessory Canals
D. Pulp Chamber
B
mainly in the apical area and in the furcation of
molars, also connect the dental pulp with the
periodontal ligament
A. Apical Foramen
B. Dentinal Tubules
C. Lateral/ Accessory Canals
D. Pulp Chamber
C
communicate with the external root surface by
way of dentinal tubules, especially when the
cementum is denuded and dentinal tubules are
exposed
A. Apical Foramen
B. Dentinal Tubules
C. Lateral/ Accessory Canals
D. Pulp Chamber
D
T/f
The rare prevalence of endodontic periodontal lesions argues that
periradicular pathoses in general has little effect on localized
inflammatory bone resorption, given that few combined lesions are
seen in cases of apical periodontitis
T
results in necrotic debris, bacterial byproducts, and other toxic irritants that can move toward the apical foramen, lateral canals causing periodontal tissue destruction apically and potentially migrating toward the gingival margin
A. pulp degeneration or necrosis
B. periodontal tissue destruction
C. inflammatory process in PDL
D. unresolved/untreated
A
to differentiate the process from marginal periodontitis, in which
the disease proceeds physically from the gingival margin
toward the root apex
A. pulp degeneration or necrosis
B. periodontal tissue destruction
C. inflammatory process in PDL
D. unresolved/untreated
B
when pulpal disease progresses beyond the confines of the
tooth, inflammation extends and affects the adjacent
periodontal attachment apparatus
A. pulp degeneration or necrosis
B. periodontal tissue destruction
C. inflammatory process in PDL
D. unresolved/untreated
B
the endodontic infection has been regarded as a local
modifying risk factor for periodontitis progression if left
untreated
A. pulp degeneration or necrosis
B. periodontal tissue destruction
C. inflammatory process in PDL
D. unresolved/untreated
C
these may aggravate periodontal pocket formation and bone
loss and impair wound healing to accelerate further periodontal
disease development and progression
A. pulp degeneration or necrosis
B. periodontal tissue destruction
C. inflammatory process in PDL
D. unresolved/untreated
D
T/f
it is believed that an unresolved periapical infection could
sustain endodontic pathogen growth, and infectious products
would regress into the periodontium by way of the apex and lateral or accessory canals, as well as encourage osteoclastic
activity
T
T/f
it is also recognized that infection from a periodontal pocket
may spread to the pulp through accessory canals, which occur
most often in the furcation and closer to the apex of teeth
T
→ sinus tract, if present, can be traced to the apex of
the involved tooth
→ abnormal response to vitality testing
→primary endodontic lesions
→primary endodontic lesions with secondary periodontal
involvement
→primary periodontal lesions
→primary periodontal lesions with secondary endodontic
involvement
→true combined lesions
→concomitant endodontic and periodontal lesions (added by Belkand Gutman)
→primary endodontic lesions
→plaque and calculus accumulation in the sulcus leading
to pocket formation
→lowering of the epithelial attachment
→primary endodontic lesions
→primary endodontic lesions with secondary periodontal
involvement
→primary periodontal lesions
→primary periodontal lesions with secondary endodontic
involvement
→true combined lesions
→concomitant endodontic and periodontal lesions (added by Belkand Gutman)
primary endodontic lesions with secondary periodontal
involvement
→
the lesion exhibits signs of periodontal disease such as
pocket formation and horizontal/angular bone loss
→
signs of pulpal involvement including episodes of
acute pulpal pain
→primary endodontic lesions
→primary endodontic lesions with secondary periodontal
involvement
→primary periodontal lesions
→primary periodontal lesions with secondary endodontic
involvement
→true combined lesions
→concomitant endodontic and periodontal lesions (added by Belkand Gutman)
Primary Periodontal
Lesions with Secondary
Endodontic Involvement
endodontic therapy followed
by radisection/hemisection
→primary endodontic lesions
→primary endodontic lesions with secondary periodontal
involvement
→primary periodontal lesions
→primary periodontal lesions with secondary endodontic
involvement
→true combined lesions
→concomitant endodontic and periodontal lesions (added by Belkand Gutman)
True Combined Lesions
distinct etiological factors
of pulpal and
periodontal diseases
which do not influence
one another
→primary endodontic lesions
→primary endodontic lesions with secondary periodontal
involvement
→primary periodontal lesions
→primary periodontal lesions with secondary endodontic
involvement
→true combined lesions
→concomitant endodontic and periodontal lesions (added by Belkand Gutman)
Concomitant Lesions
binds to the tooth surface with a color similar to that of the stain
A. Nathoo Type I
(N1)
B. Nathoo Type II
(N2)
C. Nathoo Type III
(N3)
A
caused by chromogenic bacteria, coffee,
tea, wine and metals
A. Nathoo Type I
(N1)
B. Nathoo Type II
(N2)
C. Nathoo Type III
(N3)
A
a prechromogen or in its base state,
colorless material that binds to the tooth
and causes a stain after a chemical
reaction
A. Nathoo Type I
(N1)
B. Nathoo Type II
(N2)
C. Nathoo Type III
(N3)
C
carbohydrate-rich foods (apples,
potatoes), stannous fluoride or
chlorhexidine
A. Nathoo Type I
(N1)
B. Nathoo Type II
(N2)
C. Nathoo Type III
(N3)
C
