environment and exercise Flashcards

1
Q

heat related injuries

A

hyperthermia: body temp inc too high

heat syncope: fainting and strength loss

heat cramps: serious when multiple muscles

heat exhaustion: may need med attention, dizzy, nausea

heat stroke: med emergency, body temp too high and cause organ damage…confusion, seizure, high core temp

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2
Q

treatment of heat injuries

A

cold water immersion is best, most rapid dec in body temp

drink cold fluid, cold compress

w cramps, mult cramps = sign of heat stroke, treat as heat exhaustion

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3
Q

factors related to heat injury

A
  1. fitness: more fit = dec risk
    - tolerate inc work in heat, acclimatize faster, inc sweat
  2. acclimatization: best protection against heat stroke
    - exercise in heat 10-14 days
    - low intensity, long duration i.e. less than 50% vo2max, 60-100mins
    - mod intensity, short i.e. 75% vo2, 30-35 mins
    - adaptations: inc plasma vol, vo2max, sweat, qmax…dec body temp and HR resp, dec sodium loss
  3. hydration
  4. environ temp: convection and radiation dependent on gradient b/w skin and air temp
    - high temp = heat not lost, can even gain some
  5. clothing: exposure as uch skin as possible, materials that wick sweat i.e. cotton, polyester
  6. humidity: water vapor pressure
    - evaporation dependent on grad b/w skin and air
  7. metabolic rate: core temp is proportional to work rate
    - high work rate inc metabolic heat production
  8. wind: inc heat loss by evaporaton and convection
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4
Q

implications of heat on fitness

A
  • know signs of heat illness i.e. lightheaded
  • exercise cooler times
  • gradual inc to acclimatize
  • light clothes
  • monitor HR and alter exercise intensity…stay w/in target HR
  • water always
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5
Q

WBGT

A

wet bulb globe temperature

quantifies overall heat stress

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6
Q

dry bulb temp

block globe temp

wet bulb temp

A

dry bulb temp: Tdb, ordinary measure of air temp in SHADE

block globe temp: Tg, measure of radiant heat taken in by DIRECT SUNLIGHT

wet bulb temp: Twb, uses mercury bulb thermometer
- sensitive to relative humidity
- index of ability to evaporate sweat
- MOST IMP in determining overall heat stress

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7
Q

environ heat stress

A

risk of heat stress depends on WBGT

hypothermia when WGBT less than 10degC
hyperthermia when WGBT greater than 27.9deg

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8
Q

hypothermia

A

core temp below 35deg
- 2deg drop assoc w max shivering
- 4dec = ataxia/clumsiness and apathy
- 6deg = unconscious
- further drop = arrhythmia, dec brain BF, asystole, death

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9
Q

conduction

A

from body thru phys contact

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10
Q

convection

A

from body thru air or water

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11
Q

radiation

A

no phys contact i.e. infrared rays

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12
Q

evaporation

A

body to water on surface or skin i.e. sweat

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13
Q

what to prioritize in hypothermia

A

core temperature over limbs

peripheral vasoconstriction occurs, dec skin BF

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14
Q

environmental factors of hypothermia

A
  1. temp: gradient for convective heat loss
  2. vapour pressure: low pressure inc evap
  3. wind: loss is impacted by wind speed, windchill index
  4. water immersion: 25x heat loss than air of same temp
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15
Q

insulating factors of hypothermia

A
  1. subcutaneous fat: esp effective in cold water
  2. clothing: clo units…1 clo = insulation needed to maintain core temp at rest 21degC

inc clohtes in windy, cold, wet conditions
dry > wet
amount of insulation needed dec w exercise

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16
Q

heat production in cold

A

heat production inc upon cold exposure
- inc vo2, dec body fat
- earlier onset of shivering in lean men

inc cold water: resting vo2 and core temp maintained in fat men
- vo2 and core temp dec w thin ppl

fuel use: fat is used for shivering, similar to MICT
- can lead to muscle glycogen depletion bcs inc number of bursts

17
Q

the cold and personal characteristics

A

bio sex: at rest, women have faster dec in body temp despite more fat
- inc cold water, similar heat loss among sexes
- body composition is explanation i.e. muscle produces heat

age: 60+ less tolerant, kids have fast loss

18
Q

treating hypothermia

A

symptoms: slurred speech, impaired judgment, dec coordination

treating from mild to severe:
- remove from cold, wind, rain
- remove wet clothes
- warm drink and dry clothes
- sleeping bag w other person
- find heat source
- prep for emergency and evac

19
Q

altitutde

A

is atmospheric pressure, dec at higher altitude

dec partial pressure of gases

20
Q

altitude impact on performance

A

dec PO2 has NO EFFECT on short term anaerobic performance
- o2 transport to muscle isn’t limitation
- dec air resistance may inc performance

long term aerobic performance: dec PO2 will dec performance bcs dependent on o2 delivery to muscles

21
Q

max aerobic power at altitude

A

high alt = dec vo2max bcs dec o2 extraction

up to moderate altitudes, dec vo2 max because of dec arterial po2

at high alt, dec vo2max because decrease in Qmax
- max HR dec at altitude

22
Q

heart rate and altitude

A

at altitude, inc HR bcs dec o2 in arterial blood

altitude requires inc ventilation, bcs fewer o2 molecules in air

23
Q

acclimatization to high altitude

A

inc RBC production, inc hemoglobin concentration because of HIF-1 and erythropoietin (hormone stim RBC prod)
- more hemo, fewer o2

attempts to counter desaturation by lower po2

inc o2 saturation bcs in BF to lungs
- inc nitric oxide
- 1deg adaptation in himalayan sherpas

lifetime altitude residents: complete adaptations in arterial o2 content and vo2max

24
Q

high altitude climbling

A

successful climbers have high capacity for hyperventilation
- drives down PCO2 and H in blood
- allows more o2 bind to hemo w same low PO2

climbers contend w appetite loss
- weight loss causes dec type 1 and type 2 muscle diameter

25
everest
first successful climb 1953, w/o supplement in 1978 prev thought impossible bcs vo2max at summit is just above resting lvl actual vo2max 15ml/kg/min, miscalculated barometric pressure abt 4 METS....walking 5.5km/h is 3.6
26
goal of training at altitude
goal to inc performance by inc o2 carrying capacity of blood inc RBC via erythropoietin also venti, neural, and peripheral adaptations
27
LHTH
live high, train high: traditional method where live and train at moderate altitude couple times a yr for 2-4wks progressive phases: 1. primary training - 2-3wks 2. recovery and prep - 2-5 days, full recovery 3. return to sea lvl low intensity acclimatization
28
LHTL
live high, train low: altitude 2000-2500m, elicits inc RBC via erythropotein...also inc VO2 min 12h/day for 4 weeks intermittent hypobaric hypoxia 3100m needed for non-blood adaptations train at low alt to maintain training quality - some athletes experience hemoglobin desaturation improvement 1-1.5%
29
intermittent hypoxic exposure
actually train at altitude, live low, train high may not inc RBC bcs training intensity is compromised induces muscle adaptations unclear if inc performance
30
reflecting on acclimatization techniques
LHTH = best o2 adaptations intermittent hypoxic exposure = inc o2 utilization
31
lactate paradox
altitude exposure inc HR, VE and lactate bcs of hypoxia after acclimatization, lactate dec despite hypoxia - same hypoxic stim, but dec lactate - because of dec plasma epi or muscle adaptations - less gluconeogenesis some studies don't show it when dec o2, produces lactate bcs anaerobic metabolism...this doesn't occur in lactate paradox
32
air pollution
dec health and performance, especially CO - dec o2 transport, inc airway resistance - alters perception of effort i.e. muggy day response depends on dose - volume of air: inc w exercise - conc of air, exposure duration
33
air quality index
health concern on numerical scale - dec exposure when high stay away from blow amaounts: smoking areas, traffic, urban areas most polluted 7-10am, 4-7pm inc co will dec vo2max