Equine lameness and orthopaedics Flashcards

(40 cards)

1
Q

history questions

A
  • signalment
  • use
  • duration of ownership
  • recent management (work, feed, shoeing, housing
  • previous medical problems
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2
Q

specific history

A
  • limb affected
  • timing and nature of onset of signs
  • associated events or incidents
  • details of any swelling, heat, pain
  • progression of signs
  • treatments employed
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3
Q

aims of a lameness workup

A
  • identify limb affected
  • score severity
  • identify source/cause
  • implement training plan
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4
Q

steps of a lameness work up

A
  • physical exam
  • gait evaluation
  • flexion tests
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5
Q

gait evaluation

A
  • different surfaces (soft/hard)
  • start with walk
  • trot in line
  • lunge
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6
Q

forelimb lameness

A

head goes up as lame limb hits ground

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7
Q

hindlimb lameness

A

hindquarters raised by sound limb and sink during stance phase of lame limb

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8
Q

provocative (flexion) test

A
  • limb flexed for 1 min
  • horse trotted away as soon as limb released
  • only flex the joint being assessed
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9
Q

limitation of flexion test

A
  • lack of specificity to site
  • inconsistency
  • false positives and negatives
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10
Q

lunging

A

usually exacerbates lameness on inside leg

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11
Q

nerve block

A
  • perineural, intrasynovial or local infiltration of local anaesthetic
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12
Q

nerve block steps

A
  • start distally and work up
  • clean area with chlorhex/spirit
  • leave for 10 mins then trot up
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13
Q

sites of nerve block injection

A
  • palmar/plantar digital (medial and lateral)
  • abaxial sesamoid (site of digital pulse)
  • low 4 point (medial and lateral)
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14
Q

joint blocks

A
  • intrasynovial admin
  • sterility a must
  • evaluate at 10mins and then later
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15
Q

diagnostic imaging of lameness

A
  • performed once a narrow area source of lameness identified
  • allows interpretation of significance of findings
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16
Q

ultrasound for lameness

A
  • distinguishes tendon/ligament injury from peritendinous swelling
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17
Q

signs signalling injury in ultrasound for lameness

A
  • increase in tendon/ligament size
  • change in internal architecture
  • indistinct margination
18
Q

diagnostic arthroscopy

A
  • direct visualisation of joint cavities
  • articular cartilage, synovial membrane, intra-articular ligaments, menisci
19
Q

limitations of diagnostic arthroscopy

A
  • need GA
  • inability to examine most joints in their entirety
20
Q

CT and MRI for lameness

A
  • allows bone and soft tissue visualisation
21
Q

nuclear scintigraphy (bone scan)

A
  • injected IV
  • taken up into bone mineral lattice
  • emits gamma radiation
22
Q

lesions detected by nuclear scintigraphy

A
  • stress fractures
  • arthropathies
  • enthesiopathies
23
Q

nuclear scintigraphy steps

A
  • lunge to increase uptake if poss
  • IV catheter placed
  • horse radioactive after (isolation)
  • urine collected during image acquisition
24
Q

synovial sepsis

A
  • bacterial contamination of synovial structure
  • wounds in adults
  • causes septic arthritis and chronic lameness if not treated
25
synovial sepsis investigation
- synoviocentesis and analysis - may inject sterile saline into joint to check for egress - contrast radiography
26
arthrocentesis
- sterility a must - sedation
27
roles of nurse in arthrocentesis
- prep site - non-sterile assistant - equipment and spares ready
28
arthrocentesis samples
analysis: - cytology - protein conc - lactate
29
laminitis definition
- inflammation of laminae/lamellae in hoof - dermal/epidermal separation - can lead to rotation or sinking of p3
30
phases of laminitis
1. developmental= before onset of clin signs 2. acute= onset of clin signs - can become chronic or subacute 3. subacute= 2-3 months repair 4. chronic= structural failure
31
clinical signs of laminitis
- stilted, pottery gait - bounding digital pulses - leaning back on heels - recumbency - worse on hard ground (increased pressure) - struggle to turn - reluctance to pick up feet
32
causes of laminitis
- endocrinopathies (PPID= cushings, EMS= eq metabolic syndrome) - excessive carbs - excessive weightbearing - endotoxaemia/SIRS - corticosteroids? - potentially multiple mechanisms
33
endocrinopathic laminitis
- most cases - majority of pasture associated cases - pituitary pars intermedia dysfunction - equine metabolic syndrome
34
risk factors of laminitis
- history of laminitis - obesity - endocrinopathies (insulin resistance, PPID, EMS) - season (spring + frost)
35
management of developmental phase
- Cold therapy - NSAIDs to reduce swelling - frog supports, deep shavings bed - treat underlying disease
36
management of acute phase
- strict box rest, deep shavings - frog supports - NSAIDs - treat underlying cause
37
management of subacute laminitis
- gradually withdraw treatment - strict box rest subacute and chronic: - radiograph and farriery - shorten toe over time, remedial shoeing
38
radiography for laminitis
- measure rotations and sinking - metal marker on dorsal hoof wall - coronary band -> dorsal hoof wall
39
effects of box rest
- limited exercise - diet change - behaviour (boredom) - reduction in eating time (gastric ulcers)
40
nursing considerations for box rest
- monitor faecal output, colic signs. appetite, stress - company