Exam 3 Flashcards

(223 cards)

1
Q

body composition

A

Examining Height and Weight are not enough predict healthy weight

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2
Q

Body Mass Index

A

BMI=weight(lb)*703/height^2 (in^2)
or
BMI=weight(kg)/height(m^2)

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3
Q

BMI is an ok measure in the right circumstances

A

Valid- children & sedentary

- Invalid- many athletes

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4
Q

assessing body composition

A

body s chemical and molecular composition

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5
Q

body s chemical and molecular composition

A

Fat around internal organs, nerves, in muscle

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6
Q

component of fat mass: Sex specific

A

~9% above essen+al

- Needed for proper hormone release

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7
Q

component of fat mass: Storage fat

A

Subcutaneous and visceral

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8
Q

Subcutaneous and visceral

A
  • Muscle
  • Bone
  • Blood
  • Viscera
  • etc
    Large portion is water
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9
Q

body composition Provides more information

A
  • Height and weight not enough to know fitness status

- increase Percent body fat, decrease performance

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10
Q

Body composition measured several ways

A
  • Densitometry/hydrostatic weighing
  • DEXA
  • Air plethysmography
  • Skinfold
  • Bioelectric impedance
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11
Q

Densitometry

A
measures body density
Hydrosta.c (underwater) weighing
• Gold Standard
- Muscle heavier than water, fat lighter than water
- Most commonly used method
- Accurate to within 1-2%
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12
Q

hydrostatic weight

A

Use body density to calculate %BF

  • Mass in air
  • Mass in water
  • Volume (calculated)
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13
Q

Limitations of hydrostatic weighing

A
Lung air and intestinal volume confounding variable
oConversion of body 
density to percent fat
oFat-free density varies among people
Different %BF equations exist
Siri Equation (General Population
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14
Q

• Air plethysmography (Bod Pod)

A
  • Another densitometry technique
  • Air displacement (instead of water)
  • Easy for subject, difficult for operator, expensive
  • Similar limita+ons as underwater weight
  • Similar accuracy as well
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15
Q

Dual-energy X-ray absorptometry(DEXA)

A
  • Quantifies bone and soft-tissue composition

- Precise and reliable

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16
Q

DEXA Limitations

A

expensive to run
-$700-$1500 depending on location
size of the individual
-Table and imaging cameras only cover a certain area

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17
Q

skinfold

A
  • Most widely used field technique
  • Measures thickness at a minimum of three sites
  • Number of sites vary and sites may vary by location with gender
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18
Q

skinfold: Reasonably accurate

A
  • ~3% accurate when tester is trained
    • Pinch pressure is important
  • Visceral fat is not measured
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19
Q

Bioelectric impedance

A
  • Electrodes on ankle, foot, wrist, hand
  • Current passes from proximal to distal sites
  • Fat-free mass good conductor, fat poor conductor
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20
Q

Bioelectrical Impedence: Tanita Unit

A
  • Fast and easy means of

measuring %BF

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21
Q

Bioelectrical Impedence: Limitations

A
  • Reasonably accurate

- May be affected by hydration status

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22
Q

adipose cells are like muscles

A

as adults we are considered to have a set number of cells

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23
Q

large genetic component to obesity

A

Obese individuals will develop a greater number of adipose cells which will store fat

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24
Q

weight loss with obesity

A

no such thing as spot reduction

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25
exercise & diet
manageable exercise volume and intensity to start - aerobic and resistance training - workout with a buddy for good adherence reduce caloric intake but not drastically -200-300 / day drop
26
physiological factors:obesity
- heredity/genetic - hormonal imbalances - altered basic homeostatic mechanisms
27
lifestyle factors:obesity
- cultural habits - inadequate physical activity - improper diets
28
adipose connective tissue
- stores energy | - endocrine function
29
risks of fat/obesity
- inflammation - high blood pressure - narrowing of arteries - clots in arteries - type 2 diabetes
30
type 2 diabetes
NON-insulin dependent - used to be called "adult on set diabetes" but has changed due to prevalence of children developing the disease - 90% of all diabetes cases
31
diabetes prevalence
- 2% of world population has diabetes - 9.4% of U.S. population has diabetes - 22 million
32
diabetes: fasting glucose range
126 or more mg/dL
33
prediabetes: fasting glucose range
100-125 mg/dL
34
normal: fasting glucose range
99 or less mg/dL
35
prediabetes prevalence
86 million in U.S. >1/3 of adults -90% of them undiagnosed
36
a generally reversible condition characterized by higher than normal blood sugar
-will turn to type 2 diabetes in less than 10 years
37
true test for prediabetes and diabetes :
oral glucose tolerance test (OGTT)1
38
type 2: progressive disease related to lifestyle with a strong genetic link
as many as 220 genes have been implicated in T2DM
39
type 2: characterized by insulin resistance
cells not responding to insulin
40
type 2:
during initial years, beta cells are normal and may even increase in number
41
thoughts on causes of diabetes
- huge genetic link - chronic inflammation and oxidative stress - obesity
42
obesity : thoughts on causes
- Healthy adipose tissue releases cytokines that help maintain normal healthy blood vessels - Increased adipose tissue releases: • Inflammatory cytokines • Resistin that interferes with insulin response • Lower Adiponectin levels
43
type 2 initial signs:
polyuria- increased urination --with glucose weight loss???
44
type 2 symptoms:
- polydipsia - polyphagia - fatigue
45
complications: atherosclerosis-plaques
- coronary and systemic - can lead to gangrene - possibly stroke or heart attack
46
complications: neuropathy
loss of feeling and motor control
47
complications: glomeruloclerosis
thickening in kidney that limits function
48
complications: retinal deterioration
remember retinal deterioration
49
type 2: tx initially lifestyle intervention
diet -low glycemic load diets exercise -- depletes sugar stores in muscle and helps to naturally increase glucose uptake (without need for insulin) » Overtime will increase insulin sensi2vity - Lowers blood pressure, lower heart rate and more efficient control of blood vessels - Fat loss for improved adipokine balance - Decreases stress
50
progressive tx: oral hypoglycemics
- increase insulin in blood during all blood glucose levels | - so ingested sugar will have less effect
51
progressive tx: insulin
in late stage type 2, beta cells may die off or sop functioning, no longer producing insulin
52
endothermic
produce our own heat internally | -70kcal/hr or 81 watts
53
homeothermic
same core body temp in all environments | -core body temp: 36.5-37.5° C
54
body fluid
~60% of body weight
55
Types of Mechanisms of Heat Transfer
Radiation Conduction Convection Evaporation
56
Evaporation is
of water from body surfaces or breathing passages cool the body
57
radiation is
objects exchange radiation with each other andw ith the sky. warmer objects lose heat to cooler objects
58
convection is
heat is loss by convection when a stream of air (wind) is coolere than body surface temperature
59
conduction is
the direct transfer of heat when objects of different temperatures come into contact
60
heat sources for body
* Metabolic heat * Environment - Radiation - Conduction - Convection
61
heat loss from body
``` - Radiation • Primary during rest - Conduction - Convection - Evaporation • Primary during exercise ```
62
heat balance equation
M ± R ± C ± K - E = 0 heat balance
63
heat loss equation
If M ± R ± C ± K - E < 0
64
heat gain equation
If M ± R ± C ± K - E > 0
65
core
temperaturearound organs needed to survive
66
shell:
Changesthickness to allow for heat loss or insula+on
67
2 gradients to consider:
1. core to shell | 2. shell to environment
68
41 C is
fatal in prolonged exposure
69
42 C is the
highest recorded for short period
70
40 C normally
reached in heavy exercise
71
39.5 C
stop exercise in most labs
72
negative feedback mechanism for heat
receptor integration center effectors
73
receptors
- skin | - hypothalamic blood temp
74
integration centerq
-anterior hypothalamus
75
effectors
- skin blood vessels | - eccerine sweat glands
76
increased body temp
- Muscle metabolism - Environment • Convection - If warmer than skin temp • Radiation
77
measurements of body temp
skin and core
78
cardiovascular changes
- Vasodilate blood vessels in the skin to increase radia+on and convective heat loss - increase Heart rate and contractility in the heart • More blood circulating to the skin and less filling time lead to a decrease in SV •
79
responses to exercise in the heat
cardiovascular changes sweating sweat electrolyte content
80
sweating
- Hot environmental temperatures > skin, core temperatures - C, K, R: heat gain, E only avenue of heat loss - Eccrine sweat glands controlled by POAH
81
sweat electrolyte content < plasma
- Light sweating: very dilute sweat • Duct reabsorbs some Na+, Cl- - Heavy sweating: less dilute (more Na+, Cl- loss)
82
sweat is also occurring
- Typical sweat rate 1.5 - 2.0 L/hr • Sweat is ultimately produced from blood plasma • 2-3% of body weight per hour - increase sweating will lead to decrease blood volume • Relative increase in hematocrit; which increase blood viscosity - Greater resistance to blood flow • Further decrease stroke volume
83
hormonal response
exercise and body water loss stimulate adrenal cortex and posterior pituitary gland
84
hormonal control of fluid balance
loss of water, electrolytes triggers release of aldosterone and ADH Aldosterone: reating Na+ at kidneys ADH (vasopressin) retains water at kidneys
85
hot dry environment
- very warm air - high solar radiation - high reflective radiation - low humidity
86
hot wet environment
- warm air - high humidity - evaporation is less effective - increase sweating and faster dehydration
87
acclimatization
- Adaptation which occurs as an individual undergoes prolonged repeated exposure to a natural stressful environment
88
acclimation
- Adaptations that occur in artificial environment | • Lab setting
89
Repeated exercise in heat: rapid changes for better performance in hot conditions
- Fast short term response (~10 days) | - Need ~1.5 hours of heat exposure per day
90
plasma volume(increase)
- Allows for adequate muscle and skin blood flow - increase Heart rate, increase cardiac output - Supports increase skin blood flow - Greater heat loss, decrease core temperature
91
widespread sweating earlier; more dilute
prevents dangerous Na+ loss | optimized E heat loss
92
earlier release of fluid preserving hormones:
- aldosterone - antidiuretic hormone - renin-angiotensin mechanism
93
elephant in the room:
- humans are smart - drink water (2% in body water before thirsty) - during prolonged exercise take in 8 oz of fluid every 15 mins - choose time of day to exercise
94
how can you monitor hydration levels/fluid loss?
changes on body weight | clothing
95
changes in body weight:
-weight self prior to and after exercise
96
Changes in body weight: calculation
%Bodyweightchange=[(pre-exercisebodyweight-postexercisebody | weight)/pre-exercise body weight] × 100.
97
changes in body weight: loss of weight
* Fluidlossof1%-2%is likely an is of no major concern,but losses above this should be avoided * Fluidlossof3%-5%of body weight results in cardiovascular strain and impaired ability to dissipate heat * Fluidloss>7%is very dangerous
98
clothing
• "Heat Gear" isn't as good as no clothes - Societal norms are a consideration • Dissipate heat better without interference - Radiation and Evaporation more efficient - But, wear sun screen
99
health risks during exercise in the heat:
• Measuring external heat stress | - Heat index does not reflect physiological stress
100
health risks during exercise in the heat: Wet-bulb globe temperature equation (gauge of thermal stress)
- Dry-bulb T: actual air temperature (i.e., C) - Wet-bulb T: reflects evaporative potential (i.e., E) - Globe T: measures radiant heat load (i.e., R) - WBGT=0.1Tdb +0.7Twb +0.2Tg
101
cold is
relative
102
cold is huge range on earth:
10 to -56 degrees C | 50 to -70 degrees C
103
white adipose
- Energy storage to be released into circulation - Insulation - Hormonally controlled
104
brown adipose
- Energy storage to be used for metabolic heat | - Innervated by SNS
105
response to cold: vasoconstriction in skin
- Particularly the extremities - Limits heat loss via - Limits radiation - Thicker insulation layer
106
response to cold: cold induced vasodilation
Period of vasodilation to try to save tissue from death
107
response to cold: non shivering thermogenesis
BAT metabolizing fatty acids to increase heat production
108
response to cold: skeletal muscle shivering(shivering thermogenesis)
- Involuntary muscle twitches to increase metabolism for heat production - Asynchronous MU activation - Controlled by posterior hypothalamus
109
response to cold: frontal cortex
Again, humans are smart and capable - We can make clothes and put them on - Maybe go inside - Drink warm liquids
110
exercise in the cold: muscle function
Critical Muscle temp ~27° C Nerves and muscles slow Altered fiber recruitment --> decreases contractile force Shortening velocity and power decreases
111
exercise in cold: as fatigue increase , metabolic heat production decrease:
Energy reserve depletion with endurance exercise potential for hypothermia
112
decrease in fat metabolism
Normally, catecholamines increase-->FFA oxidation increase Exercise in Cold--> increase catecholamine secretion but no FFA increase VC in subcutaneous fat --> decrease FFA mobilization
113
glucose metabolic responses
- Blood glucose maintained well during cold exposure - Muscle glycogen utilization increase - Hypoglycemia suppresses shivering
114
We do not truly Acclimate or Acclimatize to cold
Up for debate Cold is huge range on earth 10° C to -56° C
115
The things that do change are really just nervous system control of the system
and you are still uncomfortable
116
cold habituation
Occurs after repeated cold exposures without significant heat loss decrease Vasoconstriction decrease shivering Core temperature allowed to decrease more
117
metabolic changes
Occurs after repeated cold exposures with heat loss | increase nonshivering and shivering thermogenesis
118
insulate changes
When increase metabolism cannot prevent heat loss | Enhanced skin VC ( increase peripheral tissue insulation)`
119
increase in insulation thickness:
increase white adipose
120
increase non shivering thermogenesis:
increase brown adipose
121
cold air is dry air:
- 37° C air is fully saturated at 47 mmHg - Air in lungs - 0° C air is minimally saturated at 5 mmHg
122
nasal breathing in the cold:
Good for: - increase temp of air before enters core - increase Water content before enters lungs
123
wet: water increases heat loss significantly
- Being in cold water will suck heat right out | - Standing vs Flowing water
124
wet: water on clothes
- Clothes getting wet will decrease insulation value by 30% - If clothing is breathable....you will have increase cooling via convection and evaporation - Layers are important to control temp during cold weather training
125
Altitude research is relatively young
First work major work in the 1960's • 1968 Mexico Summer Olympics - Low altitude residents performed poorly in long distance events • Dominated by Kenyans and Ethiopians
126
Partial pressures
-each gas has its own pressure -daltons law The total pressure (barometric pressure) is the sum of the individual par#al pressures
127
Barometric pressure at sea level
760 mmHg
128
Partial Pressures - Oxygen
Oxygen - 0.2093 x 760 = 159 (PO2)
129
Partial Pressure - Nitrogen
0.7903 x 760 = 600.4
130
Partial Pressure - CO2
0.03 x 760 = 0.223
131
Weight of a column of air in atmosphere
Pressure- exerting a force on a surface
132
high elevation- low pressure:
- Short column of air | - Lower force exerted
133
lower elevation- higher pressure:
- Taller column of air - Increased force exerted Hypobaria
134
colder air
Air tempdecreases 1°C every 150m(~500^)
135
dry air
- Cold air holds less water - Promotes evaporation • Skin • Respiration
136
increased solar radiation
- lower moisture content | - less cloud cover
137
ACUTE physio responses to altitude: Respiratory responses
-pulmonary ventilations -increases instantly -chemoreceptors -low PO2 -increased tidal volume -increase CO2 expelled -respiratory alkalosis(pH) compensation- bicarbonate excretion
138
physio response to altitude: oxygen transport
- Lower PO2(ambient) - gradient with alveoli - Lower Po2(alveoli) - gradient with blood - RBC transit time increases - Saturation of hemoglobin - Sea level-98% - 4300 m - 80%
139
ACUTE physio response to altitude: gas exchange at muscle
decrease at altitude - gradient - sea level(60mmHg gradient) - 4300. (15mmHg gradient) - 75% reduction in diffusion gradient - driving force for O2 - tissues not receiving adequate O2
140
ACUTE physio response to altitude: cardiovascular response
blood volume - increased respiration/urination - Plasma volume decreases rapidly - Increases hematocrit (relative) - Continued exposure (absolute) - EPO (kidneys) - Stimulates RBC production - *Compensation for lower PO2
141
cardiovascular response: submaximal exercise
``` Cardiac Output • Submaximal exercise - SV decreased (loss of plasma) - HR increases to compensate - Q elevated at a given workload » to compensate for lower PO2 ```
142
cardiovascular response: maximal exercise
``` cardiac output Maximal exercise - Both SV and HR decreased » Therefore Q decreased » HR decreases because the heart uses O2 as well ```
143
vo2 max drop with altitude:
30% decrease at 4300 m
144
vo2 max drop with altitude causes:
- lower oxygen gradients | - lower cardiac output
145
cardiovascular responses
``` - Blood pressure • Increase in BP - Increased blood viscosity - Increase sympathetic tone • Older individuals at risk of CVD may need to take precautions even will mild tasks above ADL's ```
146
Need to Increase caloric consump#on
- Thyroxine - SNS/Catacholamines - ~500 calories • CHO • Increased fluid intake - ~1 extra liter a day • Iron & Ferri#n
147
altitude optimizing performance: | Two strategies for sea-level athletes who must sometimes compete at altitude
1. Compete ASAP after arriving at altitude | 2. Be at altitude for 2-3 weeks before competing
148
1. Compete ASAP after arriving at altitude
* Does not confer benefits of acclimation | * Too soon for adverse effects of altitude
149
2. Be at altitude for 2-3 weeks before competing
* Worst adverse effects of altitude over | * Significant Acclimation will have occurred
150
acclimatization
• Physiological adjustments to lower PO2 - Reducing physiological strain • Never can reach sea level values*
151
pulmonary adaptations: increased pulmonary ventilation
- 40% higher than sea level (4,000 m) | • Elevated at rest and exercise
152
blood adaptations
``` • First two weeks - RBCs in circulation increases • Lack of oxygen (kidneys) - EPO • 4000 m (6 months) - 10% increase in RBCs • High mountain residents - HCT ~ 60% - Normal levels (~45%) ```
153
blood adaptations
As total RBC count increases so doesHemoglobin • PV increases within 2-3 weeks - Increased SV and Q - Assuming drinking enough water
154
muscle adaptations
Increased capillary density | - oxygen delivery
155
best mechanism for altitude:
Live High/Train Low is generally accepted as the best protocol for altitude training - Get acclimatization benefits of altitude at rest - Can still workout very hard training at higher pressures
156
Anaerobic activities
- No negative influence • 100, 200, 400m (reliance on anaerobic metabolism) - May be improved (why?) • Thinner air, less aerodynamic resistance • Mexico City Olympics - World records (100, 200, 400, long jump)
157
carbs
* 4 calories/gram * Energy source (45-65% of daily calories) * Fiber * Anabolic (insulin) * Glycogen - limited storage ~300 grams * Diet, environment, physical condition and exercise intensity determine glycogen use
158
carb loading
* Appropriate and beneficial for intense prolonged aerobic activities longer than ~1 hr * Normal diet can maintain glycogen stores otherwise * Greater the amount of glycogen stored = ↑ endurance performance as fatigue (BONKING) is delayed
159
proper way to load carbs
Not your night before competition CHO binge!
160
• 7 days before event: proper way to load carbs
* Reduce training intensity | * Eat a mixed diet of 55% CHO
161
3 days before event: proper way to load carbs
• 10-15 minutes of low intensity activity/day w/ an even higher CHO diet • 10g/kg of body weight (BW)
162
How many potatoes/Gatorade's would one consume to hit 350g?
* 70% of 2,000 calorie diet = 1,400 calories of CHO | * 350 grams of carbohydrates/day
163
Protein
* 4 calories/gram (10-35% of daily calories) * Used to produce enzymes, messenger proteins (hormones), antibodies, transport/storage, act as buffer, controls plasma volume as well as structural components * Responsible for nearly every task of cellular life!
164
How much protein to eat?
* General population = ~0.80g protein/kg of BW * Endurance = 1.2-1.4 g/kg BW * Strength = 1.6-1.7 g/kg BW • Example 176# (80kg) male who performs resistance training = 128 - 136 grams of protein/day
165
What does 126 grams of protein look like?
* 1 ounce of meat ~6 grams of protein | * Whey protein scoop ~25 grams of protein
166
Well what about my kidneys?!
...
167
• In healthy individuals:
high protein consumption is not detrimental to kidney function
168
One study showed, protein intakes at 2.8g/kg (175% higher than strength athlete rx) did not impair renal function in well-trained athletes
...
169
What to do post-workout?
• 4:1 ratio of CHO to protein within 2 hours • 1/2 scoop of protein in milk with ~2 large bananas
170
Fat
* 9 calories/gram (20-35% of daily calories) * Makes up cell membranes/nerve fibers/brain * Insulation, protection, vitamin absorption/storage/transport, steroid hormone production, bile * Provides ~70% of our energy needs when resting
171
Ethanol
* 7 empty calories per gram * Not a sleep aid! * Anti-anabolic * Dehydration of electrolytes not just water * ↑ vascular function or any ergogenic qualities? - no research to support it being ergogenic at this time
172
Ketogenic Diet (KD)
* Fat-notdepletedlikeCHO,↑fatreserve,↑ lipolysis and ↓ RER = fat loss = potential performance improvement * Elite gymnasts ↑ body composition and maintained strength on a 30 day KD * Aerobic performance in well-trained cyclists was not compromised by 4 weeks on a KD * Host of studies showing negative performance effects as well * Safety:constipation,lethargy,vomiting,increased serum lipids
173
Paleo Diet (PD)
* Blood pH - muscle breaks down w/ ↑ acidity * Eating ↑ CHO all day displaces high BCAA and nutrient rich foods. PD rx PWO CHO * Research - Very minimal RCT: ↓ fat mass, ↑ insulin sensitivity, ↑ glycemic control and controlled leptin in RCT of DM subjects * Safety: Possible calcium deficiency
174
Vegetarian Diet (VD)
* VD is followed for health benefits, weight management, nutrient density, alkalinity and proper CHO intake * 2016 Meta-Analyses of 7 RCT found no differences between VD and an omnivorous one regarding physical performance * Safety: depends on what VD is followed * Vegan: B12 deficiency, Omega 3s, Zinc, Iron
175
• Everyone is unique and must find out what lifestyle works best for you!
• All emphasize: leafy greens, vegetables, nuts/seeds, olive/avocado oil, non-starchy CHO, low CHO fruit (berries)
176
Ergogenic Aids
Any nutritional, mechanical, psychological or pharmacologic procedure that increases athletic performance
177
• Dietary Supplement -
product taken to supplement the diet
178
Supplements are NOT regulated!
• Caveatemptor • Potency,contamination, standardization, efficacy all a gamble when buying supplements • Proprietaryblendissue
179
Sodium Bicarbonate
• ActsasabuffersopHcanbe maintained during HIT • Shown to ↑ the performance of all out exercise lasting 1-7 minutes • 300 mg/kg of BW • Safety: GI distress (spread out dosage)
180
Creatine
* Body produces * ↑anaerobic production of ATP * Buffer * ↑FFM,strength, maybe boost brain health and immunity? * Safety:nottoxic for healthy individuals
181
Branched Chain Amino Acids
* Leucine, Isoleucine, Valine * Reduction of central fatigue = ↑ endurance and power d/t tryptophan absorption competition * ↑ muscle protein synthesis, ↑ glucose uptake, anti-catabolic mechanisms * Safety has been shown using 30g/day * Mixed research, fasted workout benefits?
182
Beta-Alanine
* Supplementtoproduce Carnosine (antioxidant) * Buffer of H+ * ImprovedHIT performance, ~3% ↑ in muscular endurance, fatigue ↓, possibly hypertrophic * Sideeffect:Paresthesia
183
Beet Root Juice
• NitrateNitriteNitric Oxide (NO) • Arginine and Citrulline = ↑ NO • ↑ Blood flow = more oxygen & nutrient delivery • ↑mitochondrial function • Time to exhaustion ↑, anti-cancer properties (in vitro) • Safety - hypotension, heart disease meds
184
1,3,7 Trimethylxanthine
* Acts on adenosine receptors * Adenosine = sedative * Nootropic * ↓RPE and reaction time * ↑in power output as well as aerobic and anaerobic exercise * ↑ thermogenesis, BMR & fat oxidation = fat loss - possibly * Cons: Tolerance and anti-sleep * Toxicity seen at 20mg/kg of BW * 1,454 mg for a 160# person
185
* CHO - glycogen * Protein - vital for nearly all cellular processes * Fat - vitamin absorption, hormones, insulation * Ketogenic, Paleo, Vegetarian - All safe, see if one works for you! * Supplements - Look for 3rd Party Verification * Come talk with me at Office Hours!
...
186
What is Euhydratedd
Normal hydration
187
what is hyperhydrated
over hydration
188
what is hypohydrated
dehydration
189
decrease in fluid leads to
ADH release Aldosterone Release Thirst
190
Fluid loss of 1-2% is likely is
of no major concern, but losses above this should be avoided
191
Fluid loss of 3-5% of body weight
results in cardiovasular strain and imparied ability to dissipate head
192
Fluid loss > 7%% is
very dangeous
193
Drink an extra ____ for every pound of body weight lost
16 fl oz
194
Normal urine production in a day is
~1.5 L/day
195
Urine Specific Gravity - Hyperhydrated
Below 1.015
196
Urine Specific Gravity - Euhydrated
1.015
197
Urine Specific Gravity - Hypohydrated
Above 1.02
198
Physiological Variable - Ventilation: Acute
Increased ~40%
199
Physiological Variable - Blood pH: Acute
Increased (More Alkaline)
200
Physiological Variable - Plasma Volume: Acute
Decreased due to incresase in ventilation and urination
201
Physiological Variable - EPO Levels: Acute
Increases in first 2-3 days
202
Physiological Variable - Hemoglobin: Acute
No Change
203
Physiological Variable - Arterial O2 Content: Acute
Decreased (~80% Saturation)
204
Physiological Variable - Q at rest and submax exercise: Acute
Increase
205
Physiological Variable - Q At Max Exercise: Acute
Decreased
206
Physiological Variable - Stroke Volume: Acute
PV Decrease --> SV Decrease
207
Physiological Variable - HR at Rest and Submax Exercise: Acute
Increased
208
Physiological Variable - HR at MAx Exercise: ACute
Decreased
209
Physiological Variable - Muscle Fiber Size: Acute
No Change
210
Physiological Variable - VO2 MAx: Acute
Significant decrease ~10% per 1000 m (above 1500 m)
211
Physiological Variable - Ventilation: Chronic
Remains above sea level
212
Physiological Variable - Blood pH: Chronic
Returns to near sea level - renal compensation
213
Physiological Variable - Plasma Volume: Chronic
Increases but remains slightly below sea level
214
Physiological Variable - EPO Levels: Chronic
Move toward sea level as O2 neds are being met. ~10% increase in hematocrit
215
Physiological Variable - Hemoglobin Mass: Chronic
Increase near 10%
216
Physiological Variable - Arterial O2 Content: Chronic
Approaches near sea level
217
Physiological Variable - Q at rest and submax exercise: Chronic
Increased
218
Physiological Variable - Qat max exercise: Chronic
Increase but remains at sea level
219
Physiological Variable - Stroke Volume: Chronic
Increases but remains slightly below sea level - plasma volume
220
Physiological Variable - HR at rest and submax exercise: Chronic
Increases but remains slightly below sea level
221
Physiological Variable - HR at max exercise: Chronic
remains below sea level values
222
Physiological Variable - muscle fiber size: Chronic
decreases below sea level value
223
Physiological Variable - VO2max: Chronic
Increases but remains slightly below sea level