Facial Nerve Disorders Flashcards

(103 cards)

1
Q

innervation of the skeletal muscles

A

somatic motor

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2
Q

innervation of smooth muscles

A

visceral motor

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3
Q

sensation from viscera (includes taste and smell as they are associated w/ digestive tract

A

visceral sensory

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4
Q

Sensation from sensory organs, skin, skeletal muscles, and connective tissue

A

somatic sensory

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5
Q

Cranial nerves III (CNIII) (oculomotor), IV (trochlear), and VI (abducens) control the position of the ______ through various muscles.

A

eyeballs

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6
Q

helps to adjust and coordinate eye position during movement. Several movements assist with this process: saccades, smooth pursuit, fixation, accommodation, vestibulo-ocular reflex, and optokinetic reflex.

A

occulomotor

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7
Q

largest of the cranial nerves,

A

trigeminal nerve

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8
Q

what are the sensory nerves

A

olfactory
optic
vestibulocochlear

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9
Q

what are motor nerves

A

oculomotor
trochlear
abducens
accessory
hypoglossal

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10
Q

what are mixed nerves

A

trigeminal
fanical
glossopharyngeal
vagus

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11
Q

CN 1 2 3 are somatic or visceral and motor or sensory?

A

cn 1 is visceral sensory
cn 2 and 3 are somatic sensory

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12
Q

CN 3, 4, 6, 11, 12 are somatic or visceral and motor or sensory?

A

all are somatic motor

cn 3 is visceral motor

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13
Q

cn 5, 7, 9, 10 are somatic or visceral and motor or sensory?

A

7,9, 10 are all 4
5 is somatic motor and somatic sensory

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14
Q

muscles of facial expression
furrowing forehead, raising eyebrow, pursing lips, closing eyes

A

somatic motor innervaton

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15
Q

responsible for PAM (post auricular muscle reflex)

A

postauricular muscle

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16
Q

somatic motor innervation

A

muscles of facial expression
postauricular muscle
stapedius muscle

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17
Q

ME acoustic reflex

A

stapedius

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18
Q

visceral motor innervation

A

lacrimal and salivary glands

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19
Q

tear ducts

A

lacrimal

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20
Q

taste in anterior 2/3 of tongue (chorda tympani nerve)

A

visceral sensory innervation

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21
Q

posterior eac, concha, ear lobe, deep parts of face

A

somatic sensory innervation

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22
Q

mixed nerve derived from the second pharyngeal arch

A

facial nerve

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23
Q

runs a complex three-dimensional course

A

facial nerve

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24
Q

facial nerve course in cranium

A

Arises from the anterior part of the pons
Exists the brainstem at the pontomedullary junction
Passes through the cerebellopontine angle (CPA) to enter the IAC

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25
This canal narrowing is a common site for facial nerve entrapment and associated disorders
Internal auditory canal or meatus (meatal) segment
26
runs between the VII and VIII N and provides vascular supply to this segment of the VII nerve
AICA
27
branch of VII N) lies between the VII and VIII N in the IAC
chorda tympani
28
Common site of pathology; temporal bone fractures & Bell's palsy
Labyrinthine segment aka Intratemporal portion
29
what are intratemporal portions of facial nerve
labyrinthine segment tympanic segment mastoid segment
30
The facial nerve is readily injured here in pathologic processes and during ME surgery
tympanic segment
31
It shows variable branching patterns in the face
mastoid segment
32
exits the temporal bone via the stylomastoid foramen
mastoid segment
33
The course of the facial nerve makes it vulnerable to many
neoplastic, traumatic, and infectious conditions.
34
The incidence of newborn facial paralysis is _______%
~ 0.2%
35
Pediatric facial nerve paralysis can be
Congenital Prenatal acquired Postnatal acquired
36
what is congenital pediatric fn paralysis
Developmental errors during embryogenesis
37
what is prenatal acquired pediatric fn paralysis
Typically, due to intrauterine trauma, for example, Forceps compression during delivery or compression of the side of face against the sacrum during labor Fetal exposure to teratogens, for example, Maternal rubella
38
what is postnatal pediatric fn paralysis
Many of the same conditions that can affect adults Most commonly trauma and infections
39
also called Albers-SchÖnberg Disease
Osteopetrosis
40
what is osteopetrosis
an ad genetic conditionpresent at birth with varying severity and is a milder form than the AR condition, which is more severe It is a bony dysplasia; the bones harden and become denser
41
Causes multiple cranial neuropathies because of bony obliteration of neural foramina with entrapment and compression of cranial nerves
osteopetrosis
42
Progressive or fluctuating involvement of CN II, V, VII, VIII in osteopetrosis
Congenital facial paralysis Vision (even blindness) and hearing loss (even deafness) are common
43
treatment for osteopetrosis
Symptomatic Facial nerve decompression if nerve entrapment and associated facial dysfunction
44
bony dysplasia
abnormal growth-dense bone
45
Rare congenital disorder associated with hypoplasia of 6th (Abducens – moves the eye laterally) and 7th (Facial) cranial nerve nuclei
Mobius syndrome
46
somatic efferent nerve that, in humans, controls the movement of a single muscle, the lateral rectus muscle of the eye, which moves the eye sideways, away from the nose.
Abducens (CN VI)
47
A condition in which tissue or an organ of the body fails to grow to normal size.
Hypoplasia
48
what is etiology of Mobius syndrome
Genetic with multiple genes and modes of inheritance (multifactorial) Exposure in utero to teratogens such as Cocaine Ergotamine Misoprostol (synthetic prostaglandin E1
49
alkaloid used as a vasoconstrictor to treat migraine, induce childbirth, and prevent post-partum hemorrhage
ergotamine
50
Vasodilator used to prevent stomach ulcers & sometimes used as a treatment for tinnitus
Misoprostol (synthetic prostaglandin E1)
51
symtpoms of mobius syndrom
Congenital facial diplegia (bilateral facial paralysis) Associated CN VI unilateral or bilateral paralysis Other cranial nerve deficits Deformities of extremities Musculoskeletal deformities Intellectual disability
52
mobius treatment
Ophthalmologic consultation Nerve reconstructive surgery
53
most common cause of acute unilateral facial paralysis Accounting for approximately 60 to 75% of such cases Although bilateral facial paralysis can also occur, the occurrence rate is <1% when compared to unilateral
Bell's palsy
54
It can also be recurrent in ~ 4 to 14% of affected individuals Right side is generally affected more often, ~ 63% of the time
bells palsy
55
affects both sexes equally Although the condition is more frequent in younger women (aged 10-19) compared to the same age group of men
bells palsy
56
Pregnancy can increase the risk threefold, most commonly during the third trimester Preeclampsia can increase the risk of occurrence
bell's palsy
57
(disorder of pregnancy with high blood pressure and proteins in the urine)
preeclampsia
58
etiology of bells palsy
Idiopathic (most common - diagnosis of exclusion) Herpes simplex virus (main infectious agent) Rarely caused by a otitis media, cerebello-pontine angle (CPA), parotid gland or skull-based tumors, or metastatic lesions Begins with the sensory fibers and then involves motor fibers
59
Inflammatory response (especially swelling) leading to compression/ischemia of the nerve at the meatal foramen and labyrinthine segment Nerve degeneration can occur
entrapment from herpes simplex virus
60
a diagnosis of a medical condition reached by a process of elimination, which may be necessary if presence cannot be established with complete confidence from history, examination or testing
exclusionary diagnosis
61
how to diagnose bells palsy
Onset of partial/total unilateral facial paralysis during a 48-hour period Fever and neck stiffness at the onset No hearing loss or vertigo No other cranial neuropathy Normal head and neck examination Drying of eye due to decreased eye closure and lack of lacrimation Rare cases of recurrence Some spontaneous recovery should be noted within 3 to 6 months in all patients
62
what is audio findings of bells palsy
normal otoscopy HL is rare in pure tones normal tymps ABNORMAL ARTS or PRESENT ARTs
63
Abnormal ARTS due to a lesion
proximal to the stapedius nerve
64
Presence of ARTs indicates
lesion distal to the stapedius nerve
65
Facial nerve tests most commonly used today include
electroneurography (ENOG) - most useful, electromyography (EMG), the nerve excitability test (NET), and the maximum stimulation test (MST)
66
what is ENOG
stimulating electrode is placed proximally over the nerve trunk and a second electrode is placed distally over muscle. Current is applied in the first electrode and response is detected in the second electrode. The response is measured bilaterally and the healthy side is compared to the weak side. The magnitude of the response on the weak side is calculated as a percentage of the response on the healthy side.
67
what is the advantage of ENOG
objective measure of the amount of intact axons relative to the healthy side. Degeneration greater than 90% is correlated with a poor prognosis.
68
what is done if function doesn't return 6 mos post paralysis
ENOG to assess degeneration of nerve fibers CT & MRI - for differential from VII n tumors
69
treatment for bells palsy
Decompression of the nerve is indicated when > 90% degeneration occurs within 2 weeks of onset Steroids indicated early in the course of the disease Acyclovir (antiviral) with steroids (inconsistent data re: benefits) Eye care to prevent permanent damage due to dryness of eye
70
involves relieving pressure on the nerve possibly by removing part of the surrounding bone
Decompression
71
what are differential diagnoses for bells palsy
CPA or skull-based tumors Vestibular schwannoma Otitis media Parotid gland tumors
72
what is prognosis of bells palsy
Majority of patients recover function within 3 to 6 months with no medical or surgical intervention except eye care as needed
73
what factors lead to good prognosis with recovery of function
younger patients partial paralysis &recovery within 2 mos intact ART EMG evidence of voluntary activity ENOG shows <90% degeneration of electrically evoked muscle action potential after about two weeks of onset
74
poor prognosis of recovery of function for bells palsy
Patients older than 65 years Greater than 90% nerve degeneration within the first two weeks as diagnosed with ENoG Diabetic patients
75
why diabetic pt's for bells palsy poor prognosis
because of a peripheral circulatory disorder that develops in the feeding vessels to the facial nerves
76
injury during surgical procedures
iatrogenic injury
77
Incidence 0.5 to 4% The most common site of injury is the tympanic segment over the oval window; e.g., injury during otosclerosis and cholesteatoma surgery
iatrogenic injury
78
Most common site of injury is adjacent to the geniculate ganglion Hearing loss (conductive, mixed or SNHL) is common
temporal bone fractures
79
what are traumas causing fn disorders
temporla bone fractures iatrogenic injury lacerations gunshot wounds
80
Invasion of pseudomonas pneumoniae and other bacteria into the soft tissue, cartilage, and bone
etiology of malignant otitis externa
81
treatment of malignant otitis externa
Debridement of infected tissue Decompression of facial nerve when needed Antibiotics
82
Caused by gram-positive bacteria and Haemophilus influenzae Invasion into the facial canal through a dehiscence may evoke an inflammatory response with edema, compression, and ischemia resulting in facial weakness
acute suppurative otitis media
83
treatment of facial paralysis secndary to inflammatory ME disease
Myringotomy Appropriate antibiotics Trans-mastoid decompression if nerve degeneration is progressive
84
Facial nerve paralysis secondary to _____ is fairly common Paralysis can occur with or without cholesteatoma
chronic otitis media
85
treatment of otitis media
urgent indication for surgical intervention Tympano-mastoidectomy is appropriate for the chronic ear infection followed by decompression of involved facial nerve Removal of cholesteatoma, if present, adherent to the nerve
86
Caused by the varicella zoster virus (herpes family) - shingles
Herpes zoster oticus (Ramsay-Hunt Syndrome)
87
signs of Herpes zoster oticus (Ramsay-Hunt Syndrome)
Otalgia and severe pain Vesicular eruption on the concha and/or external canal and along the sensory distribution of 7th cranial nerve Facial paralysis that tends to be more severe Hearing loss Vertigo Higher frequency of complete degeneration of the facial nerve
88
site of pathology of herpes
Labyrinthine segment of the facial nerve Can involve CNs V, IX, X, XI Herpes zoster cephalicus, which is rare and involves cervical dermatomes (skin areas)
89
treatment for herpes
antiviral medication, Acyclovir
90
prognosis of herpes
Less chances of complete spontaneous recovery than Bell’s palsy Chances of recovery low even with steroid administration
91
are benign neoplasms of schwann cells They comprise < 1% of all intrapetrous mass lesions
Primary facial neuromas or schwannomas
92
symptoms of facial neuroma
Facial weakness (2/3 of all cases) Most facial weakness or paralysis is gradual but sudden onset of paralysis has been reported Hearing loss (~ 50% of all patients) Can be SNHL, conductive, or mixed depending on tumor location
93
Other symptoms, in 10 to 15% of cases
Tinnitus Otorrhea Ear canal mass Otalgia Vestibular symptoms
94
If the lesion is on the mastoid segment of CN VII and invading the ME cavity, ear canal, or in the tympanic portion, a _______ is possible
conductive hearing loss
95
what is tumor location of facial neuroma
variable 58% in the tympanic segment 48% in the vertical segment (lesion proximal to stapedius nerve) 42% in the labrynthine segment and geniculate body 30% in the IAC 19% in the CPA 14% in the stylomastoid foramen
96
If tumor is confined to IAC or CPA, ______ facial symptoms maybe present
no
97
audios for facial nerve neuromas
Pure-tone audiometry – generally SNHL due to cochlear nerve compression by the tumor Immittance audiometry Normal tympanogram Abnormal ARTS due to a lesion proximal to the stapedius nerve Presence of ARTs indicates lesion distal to the stapedius nerve Lesion likely in the mastoid or extratemporal segment of the facial nerve
98
determine if a tumor is an acoustic or facial neuroma
abr
99
ARTs are abnormal whenever measured on the affected side
true
100
Whenever an ART is measured in the right ear (right ipsilateral and left contralateral) ARTs are absent
true
101
management of facial nerve
Controversy over whether or not to surgically remove small facial neuromas Radiotherapy to decrease tumor size before surgery or if causing pressure on critical structures such as the brainstem Decompression of facial nerve if paralysis or other symptoms Tumor resection if warranted, with facial nerve grafting from a sensory nerve
102
sensory nerve in the calf region (sura) of the leg. It is made up of collateral branches of the tibial nerve and common fibular nerve.
sural nerve
103
differential diagnosis of facial neuroma
Otitis media with conductive hearing loss With or without cholsteatoma Cholesteatoma Requires prompt decompression of the VII N and resection of the cholestatoma Glomus tumor (Paragangliommas – ME & jugular vein bulb) Meningiomas Tumor of the meninges `Acoustic neuroma (vestibular schwannoma) Both VII and VIII nerve schwannomas have been reported in some cases of NF2