Final Exam Flashcards

(49 cards)

1
Q

Overall - 4 Mechanisms of General Knee Injuries

A
  • Contact or Non-contact (more common)
  • Deceleration
  • Plant & Pivot (axial + rotation)
  • Multi-planar stress (rotation & valgus/varus stress)
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2
Q

History of Knee

T or F: patients will always sense/hear a “pop” with knee injuries

T or F: it is obvious if there’s ever a tear of ACL/PCL

Why is there variable magnitudes and locations of pain with knee injuries?

Why is WB status variable?

Describe the Locking Knee. Is it common? Why is it a good and bad thing?

A
  • not all injuries are 3rd degree….injury can happen with or without pop
  • large x-sectional - would be obvious
  • due to the varying # of nociceptors in the different structures of the knee

Status can change throughout the healing process

Locking knee: knee gets caught somewhere in the ROM; bad - there is something jamming the joint (loose ligament, meniscus); good - gets you high priority, straight into surgery

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3
Q

Recognition - 4 signs of a knee injury?

A

Swelling - where and when? (immediate=bleeding/ligament; slow - 24 hrs=syn fluid from syn membrane/meniscus)

Discoloration - if external (no discoloration with ACL/PCL)

Local muscular responses (muscle spasm, atrophy, limited ROM)

Change in WB status

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4
Q

MCL

Position of vulnerability

Pain location

Painful movements

Other damage?

Dynamic stabilitizers

A

multi-planar - valgus + knee FL

medial femoral condyle, tibial plateau (medial, anterior aspect)

EX, EX ROT (tibia out or femur in)

medial meniscus tear (connected), ACL (if force goes deep)

Pes anserine = 3 saviours of MCL - gracilis, sartorius, semitendinosus

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5
Q

LCL

Position of vulnerability

Rarely damaged because…

3 other potential structures that could be damaged

What might happen to the lateral chain?

A

varus stress (force running med-lat)

unnatural/uncommon force

PCL, biceps femoris (dynamic stabilizers), popliteus (avulsion)

tight lateral chain - as a response effect

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6
Q

ACL

2 mechanisms or situations where you’d get an ACL tear

3 non-contact positions/motions that could tear ACL?

A

quick change of direction, one leg landings

near full EX/slightly FL
hyper EX
IN AND EX ROT

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7
Q

T or F: in hyper-EX position, ACL and PCL can be taken out without rotational forces

A

T

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8
Q

ACL

Special tests?

Pain location

T or F: ACL rarely tears in isolation

A

lachman’s + others

deep knee pain, anterior tibia, medial joint line (medial to patellar tendon)

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9
Q

PCL

Common MOI(s)

Special test

A
  • dashboard knee (tibia translated posterior); tripping over another player/branch
  • HYPER EX (with ACL)

-posterior drawer test

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10
Q

Menisci

Mechanism of injury

ROM that would tear medial/lateral meniscus

Mobility

Joint line pain (M or L) - in what position is it palpable? what are the two theories for meniscus pain, even though they have few nociceptors?

A

FL + axial load + shear = plant & pivot

medial = EX ROT
lateral = IN ROT

palpable = knee FL, sitting on the table

small coronary LIGAMENT (anchors circumbference), articular cartilage (BONE)

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11
Q

Menisci (cont.)

1 good and 2 bad things about the meniscus

Special test

Size and location of the tear influences (ie. bucket-handle tear)

A

bad - if they don’t feel pain…can cause unnoticed damage,low blood supply - slow metabolic rate; good - meniscus senses little pain, so if it does sense pain it can keep patient off their knee and let it heal

special test - mcmurray

tears can happen anywhere but size/location of the tear can affect its effects or lack of effects on the joint; bucket-handle tear -anterior horn, body of the meniscus (depending on the size/location) it can flap back and forth with knee movement

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12
Q

2 other DDx with knee injury

A

bone bruising and articular cartilage damage

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13
Q

Potential for surgery

Priority (3 - high to low)

How about MCL?

Policy for surgery - do you always need surgery?

What happens over time if problem is not fixed?

A

ACL, PCL, unstable meniscus

MCL - if in isolation - heals by itself

athlete vs. normal person

arthritic change

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14
Q

Care for the menisci and ligaments

Acute Care (2) - WB status?

Surgical interventions

Exercise goals (4 focus areas)

Long term challenges

A

PWB - crutches, brace

remove loose meniscal fragments; full ACL = reconstruction

strengthen and increase control for hip, core, knee, ankle (mostly hip cause it dictates the rest)

long term: post injury = increase shear forces = degradation of cartilage

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15
Q

PFS

Pain locations

4 signs?

Inflammation?

Mechanical or acute damage problem?

4 factors (or rather Side FXs)

A

LOCALIZED behind patella (at its articulation area with femur)

axial load of BW (running, jumping)
PROLONGED low level fatigue = BUCKLING
theater sign = stiffness of knee after prolonged sitting, knee does not want to flex
/- crepitus (usually normal) - sense of something creaking/grinding

low inflammation

MECHANICAL - weight distribution, imbalance @ knee

hip control - impaired ABDs and EX ROTs (glutes) causing more VALGUS
arch control - OVER-PRO = medial shift of BW = VALGUS
quadriceps weakness - tight/weak RF, pulls on patella and quadriceps tendon to add pressure of it onto femur
restricted lateral chain - weak glutes med; tight ITB (blends into patella) = further pull on patella

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16
Q

Patellar Tendinopathy - “Jumper’s Knee”

Pain location

What action would cause ECC loading of the patellar tendon?

Recall: at what point does tedinopathy occur? What other feature of tendons furthers the problem?

Testing ROM + Resisted testing (pain)

Special test

Direction of patella movement/shift

Care/Supports (4) - identify it’s for PTS or PT

A

apex of the patella

SHOCK ABSORPTION/LANDING - passive knee FL + ECC load

tedinopathy = rate of damage > rate of repair; tendons usually have a lower blood supply and are often constantly used = overall SLOW HEAL

  • = active/passive knee FL, resisted knee EX

thomas, ober’s, step-down*

laterally

PTS: taping to pull patella medially IF QUADS ARE “LOOSE” (if tight, then you’d be further increasing grinding)
PTS: arch support - limit OVER-PRO/Valgus - orthotics, change of shoes, taping,
PTS: knee brace - C-shaped plastic to hold patella medial (can be used if they’re allergic to tape)
PT: patella tendon strap - counter pressure - compression of tendon for distribution of tensile load

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17
Q

Patellar Tendinopathy - “Jumper’s Knee” (cont.)

Mobility for? (3)

Resisted exercises for? (3)

What about exercises for the patellar tendon?

A

PTS: hip, knee, and tight lateral chain

PTS: ABDs, EX ROTs, knee EXs; more so PFS

PT: patellar tendon responds favourably to ECC training - but be careful because with tedinopathy…this is the MOI in the first place = single/double leg squat on a decline

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18
Q

Why do 1-2 leg squats on a decline? Which intensity level should this be at?

A

shift of work from ankles to knees as you go ECC down; mildly painful/uncomfortable

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19
Q

T or F: AC joint deals with a lot of rotation forces

A

F = mostly planar, little movement

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20
Q

Differentiate a shoulder dislocation and a separation

A

dislocation - completely out - GH joint

separation = structure holding AC joint together is SPRAINED (+/- supporting ligaments, syn. capsule)

21
Q

AC joint sprain is equiv to?

A

separated shoulder

22
Q

T or F: a lot of the ligaments of the AC joint do not cross its joint

A

T - ie. coracoid process to clavicle

23
Q

AC joint sprain/”Separated shoulder”

MOI

Patient presentation (name 4)

Aggravating motions (8) - high to low - any problems with rotations?

Other conditions to consider (2)

Rockwood classifications: Type I, Type II or III
-how is classified?

A

DIRECT LOAD onto ACROMION process due to falls

step deformity
LOW GRADE shoulder dislocation
bruising
discolouration

X-FL, X-EX, FL, ABD, EX, ADD, EX ROT, IN ROT

clavicle fx, impaired traps because the muscle blends into AC

grade or severity
OVERALL: KEEP AC JOINT IMMOBILIZE/CLOSED TO ALLOW HEALING vs. reducing pain
Type I: sling for days - little damage - support dropping of arm from gravity
Type II or II: sling up to 6 weeks - a bit more damage - longer time because more fibres are damaged

24
Q

Anterior GH Dislocation

Would you grade this?

Common MOI

Patient presentation (3)

Symptoms (3)

A

Nope, it’s 100% out

APPREHENSION POSITION (ABD, EX ROT, a little X-EX + posterior force) = pops out anterior

humeral head @ arm pit
gap @ acromion process
cradling arm

bruising
swelling
damage at anterior aspect of capsule

25
Anterior GH Dislocation (cont.) Other damages - labral - (2) As well as damage/impairments of 6 other muscles...
Bankart lesions = inferior, sightly anterior aspect of labrum; SLAP lesions = superior labrum anterior to posterior - less common damage to SITS (rotator cuff) TENDONS(4) Axillary nerve disruption = impaired deltoids and teres minor (2)
26
Anterior GH Dislocation (cont.) Reducing the dislocation.... Supports for immobilization (2)... - optimal time? - immobilization allows for? - optimum position for labral contact
manual relocation by PROFESSIONAL (high risk) slings and commercial braces - allow for the capsule to heal "tight", preventing further dislocation position: elbow FL, IN ROT (20-30 deg) = best labral contact
27
When it comes to Anterior GH Dislocation... Role/Effects of AGE (diff. between young/old) Surgical repair may be recommended because? -2 methods?
young (
28
Long term with Anterior GH Dislocation... Exercises to target which muscles? (2 groups) Why NM function training important?
scapular, RC stabilizers = increase their control/strength increase NM + proprioception = better coordination of a very mobile joint
29
Subacromial impingement (SAI) SAI does not tell us the pathology but rather... Decreased SA space: - 3 common origin points - bursitis?
it is a result of something.... subacromial bursa - bursitis = inflammation/fluid build up followed by improper maturation and thickening of the outer layer due degradation/irritation supraspinatus tendon LH of biceps tendon
30
SAI (cont.) Pain locations (3) & painful arc? Aggravating ROMs In sports, where is SAI commonly found?
pain location @ ACROMION, ANT aspect of head (LH biceps), LAT aspect of head (supraspinatus) ROM: elevation of arm = FL + ABD = most complex painful arc = MIDDLE ROM (60-120 deg) = most complex swimmers - triathletes, over head sports
31
Mechanical themes with shoulder injuries Glenohumeral control vs. _________ control
scapular note: combo of both - therefore ex.prescribe accordingly
32
Mechanical themes with shoulder injuries (cont.) GH control - ______ migration of the humerus - limited ROM? (1) - why?
superior limited EX ROT - SITS have downward line of pull - impairments would lead to superior migration and limit EX ROT b/c humeral head gets "caught"
33
Mechanical themes with shoulder injuries (cont.) Scapular control - ______ rotation is limited - Position of scapula (2)
superior rotation anteriorly tilted (acromion down), protracted (humerus anteriorly)
34
Valgus Extension Overload (VEO) Syndrome Acute or Chronic? Pain location Which movements/sports exert this overloading force? Effect of carrying angle?
chronic medial elbow pain that may come and go depending on use of elbow snapping elbow movements (pitching, javelin, tennis) carrying angle - more/naturally valgus - more traction on medial side
35
VEOs Diagram - what's happening at.... valgus stress: (distraction) Medial elbow @ MCL? Medial elbow @ olecranon valgus force: (compression) Lateral elbow @ at condyle/radial head
MCL - chronic stretch/lengthen olecranon "hook" - osteophytes formation and loose bodies radial head/lat condyle - avascular necrosis and loose bodies = COMPRESSION
36
Valgus Extension Overload (VEO) Syndrome (cont.) 2 muscles to target to reduce "snapping" mechanics? Modifications to training volume is ESSENTIAL Removal of bony fragments?
strength elbow flexors (biceps_ and GH EX ROTs (INFRASPINATUS, TERES MINOR) causes to increase ECC control (or deceleration) to decrease stress removal of fragments = prevent deterioration to cartilage - may require surgery
37
Epicondylalgia ECC overloading how? Local tendon/periosteum of epicondyle. Locations of pain?
PAIN AT EPICONDYLES pain @ med epicondyle= wrist FLs pain @ lat med epicondyle= wrist EXs ECC overloading due to local tendon/periosteum pull
38
Epicondylalgia (cont.) Lateral (TENNIS elbow) - 2 muscles involved and are ECC loaded? - which motion by player?
ECRL, ECRB - backhand swing (ball pushes wrist into FLs, EXs resist)
39
Epicondylalgia (cont.) Medial (GOLFERS elbow) - 2 muscle involved and are ECC loaded? - which motion by player?
FCR, pronator teres - golf swing - trailing arm (ball pushes wrist into EX and valgus deviation, FLs resist)
40
Epicondylalgia (cont.) Lat & Med - Aggravated by ________ tasks-why?
gripping - activate EXs and FLs
41
Epicondylalgia (cont.) 4 options for care/support?
reduce training volume correct form counterforce bracing train target muscle + its opposition: wrist drops - ECC training (quick ECC load of the same muscle)
42
Hyper-EX of elbow damages which two structures? Also, a risk of what? Another MOI of elbow injury?
anterior capsule and elbow flexors dislocation - olecranon hook pivots off and dislocates traction injury - arm gets caught/pulled = axial load (apart) - radial head slips out of annular ligament (dislocation)
43
What is FOOSH? MOIs?
FALLING ON OUT STRETCHED HAND hyper-ex by falling directly on hand olympic lifter - hyper-ex when weight falls BEHIND head
44
FOOSH (cont.) ROM Muscle guarding? 3 potential interrupted neural structures (and which movements)? Hinge bracing - limits what? Surgical - MUCL - important with which athletes?
EX - aggravating PRO-->SUP if there's a traction injury muscle guarding: increased activity in elbow FLs, decreased in EXs median - hyper-EX ulnar - valgus elbow radial - traction (radial head dislocation) elbow EX and naturally valgus as well - in order for healing process throwers
45
Neural compression if you're sleeping on your forearm/hand - loss in sensation AND muscle weakness is due to... in the case of athletes and clinically, what is usually causing neuropathology and loss of sensation?
restricted arterial supply to the nerve (whose blood supply is very superficial) muscle hypertrophy
46
Neural pathway & job of the median nerve. What happens if it's restricted proximally, distally?
``` 2 heads of pronator teres - symptoms in forearm carpal tunnel (median nerver + 4 FDS tendons + 4 deep FL tendons, + 1 thumb FL) - symptoms in hand ``` feed wrist FLs (1-3 digits)
47
Neural pathway & job of the ulnar nerve. What kind impairments? Impairment of this nerve is common in which athletes?
cubital tunnel tunnel of guyon (smaller than carpal tunnel) feed into 4-5 digits = critical to grip ulnar deviators + gripping cyclists who DIRECTLY put pressure on the tunnel of guyon causing tingling, reduced sensation and numbness
48
TFCC injures are either _____ or _____ TFCC is composed of what 2 structures? 2 functions of the TFCC
acute, chronic ligaments + meniscus shock absorption the ligaments hold together the inferior radioulnar joint
49
TFCC injury Which athletes would injury themselves via axial loading + slight EX? Which athletes would injury themselves via shear/twisting forces? Aggravating ROM How is the healing process here?
gymnasts, boxers = shock absorption swinging sports - tennis, baseball, clubs = repetitive PRO-->SUP ulnar deviation - which compresses the complex PRO-->SUP - shearing Wrist EX slow but relatively quicker than meniscus due to higher blood supply of ligaments+fibrocartilage (complex)