Haemodynamic Shock Flashcards

1
Q

What is haemodynamic shock?

A

Acute condition of inadequate blood flow throughout the body

*a catastrophic fall in arterial blood pressure leads to circulatory shock

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What types of shocks are there due to fall in cardiac output?

A

Cardiogenic shock (pump failure)-V dont empty properly

Mechanical shock (obstructive)- v cannot fill properly

Hypovolaemic shock -reduced blood volume leads to poir venous return

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What is cardiogenic shock?

A

Acute failure of the heart to maintain cardiac output - pump failure

Similar to heart failure but different in that is it an acute condition

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

What are the potential causes of cardiogenic shock?

A
  • MI causing damage to left ventricle, part of myocardial has died therefore cannot pump enough blood to maintain BP
  • serious arrhythmias-heartmight be pumping too slowly (bradycardia) or too fast (tachycardia) which might mean the ventricle isn’t able to fill
  • acute worsening of heart failure
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

What issues does cardiogenic shock produce?

A

Raised CVP
Dramatic drop in arterial BP
Poorly perfused tissues (if the coronary arteries are affected it can exacerbate the problem, if the kidneys are poorly perfused it may result in poor urine production- oliguria)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What is cardiac arrest?

A

Unresponsiveness associated with lack of pulse

Heart has stopped or ceased to pump effectively

Asystole, pulseness electircal activity (no pulse but there is still electively activity- appears normal on ECG), ventricular fibrillation (can cause asystole)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

How do you treat cardiac arrest?

A

Basic life support- chest compression and external ventilation

Advances life support- defibrillation- puts all cells in refractory period allowing the pacemaker cells to start setting the pace again

Adrenaline- enhances myocardial function,increase peripheral resistance (acts on a1 receptors)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

What is mechanical shock?

A

When the ventricle cannot fill properly

Ie Cardiac tamponade
-blood or fluid builds up in pericardial space, restricts filling of the heart- limits end diastolic volume

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What does mechanical shock cause?

A

High central venous pressure (blood returns to heart but cant get in )

Low arterial blood pressure (very little cardiac output due to low stroke volume)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

During mechanical shock the heart will continue to beat and will attempt to increase the cardiac output. If stroke volume is being limited how can the heart do this?

A

Increase heart rate

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

How can a massive pulmonary embolism cause mechanical shock?

A
  1. Embolus occludes large pulmonary artery
  2. Pulmonary artery pressure rises
  3. Relight ventricle cannot empty
  4. Central venous pressure rises
  5. Reduced return of blood to left heart
  6. Limits filling of the left side of the heart
  7. Left atrial pressure is low
  8. Arterial pressure is low
  9. Shock
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What are the other side effects of PE aside from mechanical shock?

A

Chest pain, dyspnoea

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What is hypovolaemic shock?

A

Haemodynamic shock due to Reduced blood volume

(Most commonly due to haemorrhage, but can also be caused by severe burns, severe diarrhoea or vomiting and loss of Na+)

Severity of shock is related to amount and speed of blood loss

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

How does the body attempt to counteract hypovolaemic shock caused by a haemorrhage. How it is detected in the first place?

A
Haemorrhage 
Venous pressure falls (fall in stroke volume)
Cardiac output falls
Arterial reassure falls
Detected by baroreceptors 

Increased sympathetic stimulation
Cause; tachycardia, increased inotropy, peripheral vasoconstriction (NA acting on A1 receptors), venoconstriction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

How does internal transfusion occur during hypovolaemic shock?

A

Increased peripheral resistance reduced the capillary hydrostatic pressure causing net movement of fluid into capillaries

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

How will a patient present with hypovolaemic shock?

A

Tachycardia
Weak pulse (due to poor perfusion)
Pale skin (due to poor perfusion)
Cold and clammy extremities (due to sympathetic stimulation)

17
Q

What is a danger of hypovolaemic shock? How does it occur?and what further complication does it cause?

A

Decompensation

Caused by Peripheral vasoconstriction which impairs tissue perfusion which can lead to:

  • tissue damage due to hypoxia
  • release of chemical mediators (vasodilator)
  • TPR falls
  • BP falling dramatically
  • vital organs no longer being perfused
  • multi-system failure

*give fluid and electrolytes in order to maintain blood plasma volume

18
Q

What is the type of shock that refers to the shock that occurs when there is low TPR?

A

Distributive shock (low resistance shock/ normovolaemic)

19
Q

What are the two types of distributive shock?

A

Toxic/septic

Anaphylactic

20
Q

How is septic/toxic shock caused?

A

Endotoxins released by circulation bacteria

Triggered inflammatory response

Causes profound vasodilation

Dramatic fall in TPR

Fall in arterial pressure

Impaired perfusion of vital organs

(Capillaries become leaky-reduced blood volume)

Increased coagulation and localised hypo-prefusion

21
Q

What is septic shock?

A

Persisting hypotension requiring treatment to maintain blood pressure despite fluid resuscitation

22
Q

How does the body deal with septic shock?

A

Decreased arterial pressure is detected by baroreceptors and this increases sympathetic output. However, Vasoconstrictor effect overridden by mediators of vasodilation (endotoxins)

Body will increase heart rate and stroke volume too

23
Q

How would a patient with septic shock present?

A

Tachycardia
Warm, red extremities intimately but in the later stages of sepsis there is vasoconstriction which causes localised hypo-perfusion

24
Q

What is anaphylactic shock?

A

Server allergic reaction (anaphylaxis)

25
Q

How does anaphylactic shock occur?

A
  1. Histamine released from mast cells (and other mediators )
  2. Causes vasodilation = fall in TPR
  3. Dramatic drop in arterial pressure
  4. Increase sympathetic response to increase CO but vasodilation cannot be overcome
  5. Impaired perfusion of vital organs
  6. Mediators also cause bronchoconstriction ad laryngeal oedema = difficulty breathing
26
Q

How would a patient with anaphylactic shock present?

A

Difficulty breathing
Collapsed
Rapid heart rate
Red, warm extremities

27
Q

How is anaphylactic shock treated?

A

Give adrenaline which causes vasoconstriction via action at A1 adrenoreceptors =increases HR