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Flashcards in Hip OA Deck (20)
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What occurs to the cartilage in the early and progressive changes in oa

early- localized areas of softening presenting a pebbled texture at the surface

Progression-disruption along the collagen fibre planes


What occurs in the final stages of cartilaginous changes in oa- irregular cartilage

irregular cartilage- loss of PG content, there is subsequent focal proliferation on chondrocytes leading to irrecgular cartilage


What occurs in the final stages of cartilaginous changes in oa- osteophytes, subarticular cytes

osteophytes- new bone will occur in subchndral bone and jt margins- make it look larger

Subarticular cytes- predominate where overlying cartilage is thin or absenct


What occurs in the final stages of cartilaginous changes in oa- infiltration, calcification

infiltration- synovium will thicken and hypertrophy and the capsul conracts with infiltration of lymphoid follicles

Calcification- may occur as crystals deposit in cartilgae


What scale is the gold standard for grading severity of oa

kellgren and lawrence grading scale


What % of canadian adults have arthritis



mc pop with prevalence of oa

african americans


What jts are mc for oa

Hip and knee


modifiable risk factors of oa

-previous jt damage
-heavy work
-weaknes of leg mm


what gives you a 4x higher risk of oa

Obese women with BMI of 30-35
(obesity also a risk for bilateral oa) * most important risk factor


how does obesity contribute to oa

Adipose tissue itself is a source of inflammaory factors that are associated with alterations of cartilage homeostasis and degradation


1kg loss in weight lead to what % decrease in oa

10% decrease


What is the ratio of weight and compressive force

4:1 ratio in relation to weight and knee force


How well in knee injury associated with knee oa

knee injury was associated w a 6fold increased risk of knee oa (cruciate lig mc)


characteristics of primary oa

localized +/or generalized
small (peripheral jt) and or large (central its)
mixed and spine
erosive oa


Characteristics of secondary oa- cause

Congenital and developmental disorders, bone dysplasia
post surgery (meniscectomy)


90% of oa patient also have this

1 additional chronic condition
(knee oa associated with cv events and all cause mortality)


Eular diagnosis guidlines for oa : imaging

1. don’t need imaging to make an oa dx in pts w typical presentation

2. routine imaging not nessesary

3. pain film still best


what 3 signs and 3 symptoms should be used to dx oa

signs- crepitus, restricted mvmt, bony enlargemnt

symptoms- persistent knee pain, limited morning stiffness, reduced function

99% accurate w all 6


recommendations for non pharmacological therapy of oa

Strong for exercise + weight loss
-less strong for manual therapy and only in conjunction w exercise