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Flashcards in HTN Group Discussion Deck (15):

1. What are pertinent physical exam components to assess a patient for an elevated blood pressure/new diagnosis of hypertension.

check BP in both arms
obtain orthostatic blood pressures
palpate and auscultate the heart
check vascular pulses
check for abdominal masses and aortic pulsation
lower extremity edema
palpate thyroid
examine optic fund (retinopathy and papillary edema- long term damage)
S4 gallop (due to having to contract against high pressure systemic pressure)
check lungs for heart failure
auscultate for renal artery stenosis or adrenal masses
BMI to help target therapy, and assess for hyperlipidemia or diabetic risk


2. What are the recommended initial lab tests and indications for the test to assess a patient for elevated blood pressure.

urinalysis- DM, kidney disease
blood glucose
serum K (Kohn syndrome- aldosterone due to tumor)
Creatine (kidney disease)
12 lead EKG
TSH levels
fasting lipids (HDL, LDL and triglycerides-- risk of atherosclerosis, quantify CV risk)


3. What lifestyle changes might be suggested to a patient with b.p.

weight reduction (5-20mmHg)
DASH (8-14 mmHg)
dietary sodium (2-8 mmHg)
physical activity (4-9 mmHg)
moderation of alcohol consumption (2-4 mmHg)
smoking cessation (3.5 mmHg)


4. Do you prescribe medication after first visit with HTN?

see if lifestyle modifications, could this be caused by white coat HTN, next steps could be multiple measurements or home monitoring, unless there is clear end organ damage


5. What is the goal of HTN pressure?

goal to get the patient under 140/90, important to reduce cerebral vessels, protect kidney, protect coronary vessels; education on the side effects and consequences of HTN


6. What are the pros and cons by single or multiple drug therapy.

more drugs, can effect more interacting systems; clinically managing, paying for or picking up drugs can also be a problem


7. List the classes of antihypertensive drugs for treatment of HTN.

ACE-I (side effects of cough but cheap)

B-blockers (for heart failure patients)


8. What are the special conditions under situation of african american or asthmatics?

AA: increased risk of angioedema or slow acetylation due to ACE-I; B-blocker (B2) cause blocking of dilation of airways, (B1) specific can be sued but not at high doses; Asthma: ACE-I can cause bronchoconstriction (chose ARB)


9. What is the preference of HTN meds for diabetics?

for diabetics: ACEi or ARB then CCB and diuretics;

use blockers of RAS and check for increased hyperkalema
ARB can dilate efferent vessels to limit glomerular pressure and proteinuria and prevent end organ damage (although will show increased creatinine)


10. How do you decide to up an dose or add another med.

depends on how close you are to the goal, what is the cost to the patient? what is most likely to be biologically effective? (don't usually use ACEi and ARB together)


11. What are some concerns with persistent hypertension?

what is Mr. Jones doing (or not doing) to lower his BP- lifestyle changes, taking meds


12. In hypertensive emergency what is the course of action

needs to be admitted for IV drugs including nitroprusside (most power vasodilator) and has short half-life which makes it easier to manipulate response; remember withdrawal of meds esp. B-blockers and central sympathalytics can cause rebound of the pressure

goal is to decrease mean arterial blood pressure by no more than 25% within 2 hours or to decrease the range of 160/100 mmHg


13. What are the potential consequences if BP is or isn't treated quickly.

end organ damage (pailledema, encephylopathy), stroke risk

too aggressive of treatment can cause low perfusion to organs, stroke or syncope


14. What can be done to encourage compliance?

address patient concerns including side effects and cost, as well as other reasons about discontinuing meds as well as the significance of silent damage of the disease or importance of taking meds


15. What are the recommendations for HTN with additional cardiac event like MI?

MI is compelling indication for B-blocker especially if there is reduced systolic performance and it can help to reduce cardiovascular events in the future (although it takes 2 mo to titrate the drug dosage)

ARB and ACEi can cause fibrosis in the ventricle