Lec 5 Flashcards

1
Q

force - velo relationship

A

as we increase force we decrease speed.
eccentric contractions provide the most force

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2
Q

motor unit

A

the nerve and all the muscles its connected to are recruited to exert force

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3
Q

musclr fibre ratio

coordinationo movement compared to size of muscle

A

the ratio of fibres innervated by the motoro neuron is determined by the precision accuracy and coordination of the moveemn, not the size of the muscle group

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4
Q

size principle

A

the body only recruits muscle units that it needs ot complete the job

in order from 1 - 2A-2X

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5
Q

muscle and neural adaptaiton to resistance training

A

we see that most traiing studies stop before they reach peak strenght and hypertrophy in the person at 8-12 weeks but we see that at this time is when neural adaptation beigins to peak and plateua.

most studies end at 8-12 weeks, serious strenght trainers peak after this

exogenous anabolic hormone use can break the hypertrophy plateau

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6
Q

majority. of population is what percent slow twitch

A

60%

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7
Q

fibre typing

A

using a biopsy provides moderate results

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8
Q

what determines contractile speed

A

myosin atpase

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9
Q

which fibres produce more lactate quicker

A

fast twtich fibres

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10
Q

swithvc from 2x to 2a

A

need 20 week 6-8 reps till failure

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11
Q

switch from 2a to 2x

A

21 week detraining

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12
Q

bone remodeling requires the right

A

stimulus and nutrients

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13
Q

horizontal both growth

A

shin kicks provide a stimulus horizontally allowing the bone to grow horizontally

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14
Q

BMD decreases with

A

age and if in space - no stimulus of tension

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15
Q

after one day of bed rest

A

we lose immense BMD

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16
Q

tibia BMD loss

A

38% in 60 days

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17
Q

BMD peaks and is created when

A

from 10 -20 is created peaks at 20

18
Q

osteoporosis

A

2 standard deviations from normal BMD

19
Q

suboptimal lifestyle factors include and what do they do

A

include lack of nutrients, vit d,, reduced physical activity

these decrease the peak of of BMD

20
Q

muscular hypertropy

A

muscular enlargment due to increase in muscle cross sectional area

the net increase in contractile proteins within the myofibril and the increase and myofibrils in the muscle

21
Q

sarcoplasmic hypetrophy

A

due to increase in volume in the uscle, from fluid swelling and glycogen

which can becoem a stimulus for growh

22
Q

3 ways to stimuluate mTOR

A

leucine
Resistance Training
Insulin

23
Q

what is the onjly direct stim of mTOR

A

eatong EAAs

24
Q

insulin

relaxation

A

insulin causes the relaxation of smooth muscle , which cause release of NO3 which vasodialates - which increases blood flow, which is driectly proportional to MPS

25
sarcoplasmic expansion hypothesis
this jsut says that the sarcoplasmic volume. increases allowing mroe roomn for ribosmomes and the constant swelling triggers fusion with sasatellite cells whihc birngs mre contractile proteins
26
myofribril expansion hypothesis
says that ribosomes increase tranascription during resistance traiing iand create more contractile proteins
27
tapering of myofibril hypertrophy but muscles still look bigger
we see that overtime myofibril hypertrophy tapers off and sarcoplasmic hypertrophy continues swelling - this saccounts for big muscles adn not always big strenght
28
is there an anabolic window
no 24 hours and 3 hours and 1 hour after exercise didnt chanfe the amount of MPS
29
the limit for mTOR
mTOR can only be stimmed for a an hour 60minutes until it taps out, then the refracotry period is about 4 hours which is why we should space our diet to be 3-4horus inbetween meals
30
how long is MPS stimulated after exercises?
48 hours so eat properly in the next 48 hours but to optimize hit same muscle 3 times a week
31
carbs effect on insulin
carbs stim inuslin response- 3 hr post eating shows a non-catabolic effect rather than a anabolic effect
32
carbs and piss with aa
when you eat carbs and aa after workout you excrete less aa acid in piss
33
why can't you train both endurance and RT and see gains
AMPK inhbiits mTOR but is turned on when enery is low (AMPK monitors our energy status), no anabolism, only catabolism to get us enrgy from carbs fat and some protein
34
AMPK-Akt master switch
during RT - akt stims mTOR to turn on and MPS - inhibiting AMPK during endurance training, AMPK senses low energy levels (glycogen) and stims TSC2 to inhbit mTOR for
35
interference model
the interference model says ig we want neural adaptation we need high load and if we want cardiovascular adaptation we need high intesity once we start doing both we are in the zone of interference 75% of HRmax = HIhg intesity 5RM hihg load dont train weight room shit on the field adn vice versa
36
hormone release after RT
the relase of hormones after RT are usually acute and trnasient not significantly effecting muscle growth
37
rest intervals inbetween sets
larger rest intervals past 120secons increase the amount of testosterone released and if you wiat 5 minutes you will get more atp then you had before the set even started (Supercompensation)
38
natural hormone fluctioation form RT
doesnt lead to increaesed mnuscle growth
39
frequency and volume of workouts
if we keep the volume constant, the amount of time we workout does not significantly change the amount of muscular hypertrophy
40
training till failure
it does not matter the amount of weight you are lifting, when training till failure we see the saem muscle growth
41
1rm boost if
you train heavy at low reps, physiological factor working at those heavy weights