Lecture 10 (HEENT)-Exam 4 Flashcards

(114 cards)

1
Q

Fill in the blanks? Is this a left or right TM?

A
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2
Q

What are the outer, middle and inner ear structures?

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3
Q

Left or right?

A

left

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4
Q

What type of questions do you need to ask for history?

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5
Q

Approach to the Ear- Exam
* What do you need to palpate?
* What do you need to look at?
* What are the tests for hearing loss?
* What other exams can you do?

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6
Q

What are the external ear disorders?

A

o Cerumen impaction
o Otitis externa
o Trauma
o Foreign Bodies
o Neoplasms

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7
Q

Cerumen Impaction
* What does the cerumen do?
* often what?
* What are symtomes?
* How do you dx?

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8
Q

What are the different types of txts for cerumen impaction?

A

Treatment (if symptomatic)
* cerumenolytic agents (1st line)
* irrigation (avoid if perforation)
* manual removal

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9
Q

What is first, second, third and 4th line for Cerumen Impaction txt

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10
Q

Otitis Externa (swimmer’s ear)
* What is it?
* Common or not?
* What are the organisms?
* Can be what?
* What are the Risk factors?
* What are the types?

A
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11
Q

What is the clinical presentation of OE?

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Clinical Presentation (acute)
* Rapid onset
* Ear ear pain/fullness
* Itching
* Drainage
* Tenderness

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12
Q

How do you clinical dx OE?

A

Clinical Diagnosis
* Visualize erythema and swelling of the ear canal
* Tenderness with palpation of tragus/auricle (tug test)
* otorrhea with otoscopy

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13
Q

Otitis Externa-
* What is the Treatment?
* What do you need to consider?

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14
Q

Otitis Externa-Treatment
* What are the different types of antibiotic drops/steriods? For how long?

A

Topical antibiotic drops +/-steroids x 7-10 days
* ciprofloxacin/dexamethasone otic (Ciprodex- can be $$$, less side effects, high potency)
* ciprofloxacin/hydrocortisone otic—(low-potency steroid)
* ofloxacinotic (no steroid)
* neomycin/polymyxin B/hydrocortisoneotic (Cortisporin- inexpensive, avoid if TM perforated)

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15
Q

Otitis Externa-Treatment
* if mild cases under a week, what can we try?
* What do you for a severe/ immunocompromised patients?
* What do you control?
* What can you do for supportive?
* What should you do if no improvement?

A
  • Mild cases <1 week- can try acetic acid (acidifying)
  • Severe/ immunocompromised-Topical + Oral Abx, consider wick placement
  • Pain control
  • Warm Compresses
  • Culture if no improvement
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16
Q

Otitis Externa-
* When does it resolve with meds and without meds?

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Prognosis- Resolves in approximately 6 days with combo antibiotic/steroid drop.
* Typically resolves in 6 weeks without treatment

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17
Q

What should you tell people to prevent recurrence of OE?

A
  • Counsel on proper ear hygiene (no Q tips!)
  • Ear plugs/blow drying ears/shake head after water exposure
  • Alcohol/acetic acid drops (no clear evidence to support)
  • Remove hearing aids nightly and clean regularly
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18
Q

Malignant Otitis Externa
* Where does the infection spread to?
* Potentially what?
* What are the risk factors?
* What is it ususally caused by?

A
  • Infection spreads to bones of the skull
  • Potentially life-threatening
  • Risk factors= DM, immunocompromised, elderly
  • Usually caused byPseudomonas aeruginosa
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19
Q

Malignant Otitis Externa
* What are the sx?
* how do you dx?
* What is the txt?

A

Sx:
* Foul discharge, granulations, severe ear pain (can progress to cranial nerves palsies)

Diagnostics
* CT/MRI showing bone erosion

Treatment
* Long antipseudomonal IV abx course (ciprofloxacin) 4-6 wks
* ENT Consult
* Surgery if no improvement

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20
Q

Progression of malignant otitis externa can affect what cranial nerves?

A

cranial nerve VII, IX, XI, or XII

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21
Q

Fungal OE:
* When should you consider this?
* How do you dx?
* What is the txt?

A

ex of med: clotrimazole

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22
Q

External Ear Trauma:Auricular Hematoma
* What is it?
* What is separated?
* What is the clinical presentation?

A
  • Direct trauma to the auricle
  • Separation of perichondrium from underlying cartilage, blood vessels torn, blood collects, hematoma forms
  • Clinical presentation- Bleeding/swelling from ear with history of trauma, swelling of the pinna +/- fluctuance
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23
Q

External Ear Trauma:Auricular Hematoma
* What is the txt?

A

Treatment: I&D to avoid deformity and necrosis (Cauliflower ear/wrestlers ear)
* Compression dressing (prevent reaccumulating)
* Empiric antibiotics

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24
Q

External Auditory Canal Abrasion
* MC occurs with what?
* What is the presentation?
* What is the txt?
* What is the complications?

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25
Foreign Bodies of the Ear Canal * Common in who? * What is the MC ages? and groups of people? * Often what? * How do you diagnosis?
* Fairly common in a Pediatrics (toys, beads, insects, etc) * Most common ages 1-6 * Adults in sports and outdoor activities * Often asymptomatic * Diagnosis: Visualization with otoscope (check the nose too!)
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Foreign Body Management->Insects * What do you do before removal? * Apply what to kill the bug? What can be the result? * What is an alternative? * May need what?
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Foreign Body Management (excluding insects) * What are the different options? What do you not do? * There may be more than one foreign body so what do you do? * ENT consult for what?
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When do you refer and follow up with a foreign body?
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If there is an abnormal lesion that doesn’t improve, what do you need to do and why?
If there is an abnormal lesion that doesn’t improve, refer! * Basal cell carcinoma * Squamous cell carcinoma * Melanoma
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Neoplasm: basal cell carcinoma * MC what? * Slow or fast? * What do you need to do? * What is the txt?
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Neoplasm: squamous cell carcinoma * Can be what? * What do you need to do? * What is the txt?
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Neoplasm: melanoma * What is it? * Can be what? * What do you need to do?
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What are the middle ear disorders?
* Otitis Media * Tympanic Membrane Perforation * Cholesteatoma * Mastoiditis
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What is Acute Otitis Media (AOM)?
Acute, suppurative infectious process marked by the presence of infected middle ear fluid and inflammation of the mucosa lining the middle ear space
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Otitis Media- Epidemiology * MC what? * What age groups gets it more? * What are the different types?
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Otitis Media- Pathogenesis * What are the different causes?
* Eustachian tube dysfunction with subsequent tube obstruction * Increased negative pressure * Accumulation of fluid * Microbial grown * Suppuration
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What are the most common pathogens for AOM?
* Most common pathogens – **Streptococcus Pneumoniae**, Haemophilus Influenzae, Moraxella catarrhalis, Staph aureus * Can also be viral (~ 16%)
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What are the risk factors for AOM?
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Otitis Media- Clinical Presentation * What are the most common sxs?
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how do you dx AOM? What do you see?
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What is this?
Pneumatic otoscope * TM does not move: OM * TM moves: normal
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What is this showing?
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What is this?
AOM
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What is this?
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What is this?
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Acute Otitis Media-Treatment * What is the first choice for txt? * What do you need to consider? * Who gets abx therapy? * What do adults get? * Obs in who?
Antibiotic therapy –> #1 Amoxicillin * Consider severity, age, risk factors, caregiver * <6 months, severe sx, toxic appearing- Abx therapy * Uncomplicated children Abx therapy * Adults: amoxicillin or amoxicillin/clavulanate (Augmentin) * Observation before abx if parental preference and low risk (explain risks/benefits/f/u)
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# AOM What is the dose of amox and for how long? What do you use for Pain control of AOM?
Amoxicillin 90 mg/kg per day ( high dose) -divided in two doses, max 3g/day Duration * 10 days (<2 years, perf TM, or hx of recurrence) * 5-7 days (> 2 years with intact TM and no recurrent hx) Pain control- ibuprofen/acetaminophen
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Otitis Media- Antibiotics Alternatives * What is first line? * What do you give if recurrence? * What do you give for PCN allergy (mild) * What do you give for severe PCN allergy?
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Otitis Media- When do you need to refer
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Otitis Media-Tympanostomy Tubes * Who gets this? * May cause what? * Tubes permit what? * Who do you refer to?
* Recurrent AOM (4 or more episodes per year) * May cause TM sclerosis * Tubes permit drainage of the middle ear fluid, aeration, and return of mucosa to normal * Refer to ENT for placement
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What is this?
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Otitis Media-Follow- up * When do you need to see patients who are txt with observation? * What do you do if not beter? * Monitor for what? * Who are reliable reporters? * What do you educate parents on?
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Otitis Media-Prevention & Prognosis * What are the vaccines? * What about breast feeding vs bottle? * Without antibiotics, AOM often resolves within what?
* Vaccine – Pneumococcal Vaccine (PCV) offers modest prevention (~7% risk reduction) * Influenza vaccine under age 6 offers modest prevention (~4%) * Exclusive breastfeeding until 6 months reduces risk (~43%) * Without antibiotics, AOM often resolves within 24 hours in ~ 60% ## Footnote Pneumococcal 15-valent conjugate vaccine, also PCV 20 now available
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Otitis Media Effusion (Serous Otitis Media) * What is it? * What is the presentation? * What are the otoscopic findings? * When does it resolve? * Differentiate from what?
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What is this?
Otitis Media with Effusion * Otoscopic findings of OME include visible fluid (often yellowish, but sometimes clear) behind an intact tympanic membrane.
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Otitis Media Effusion- Treatment * What is the txt? * When do you refer? What do they do? * What has not been shown to be useful?
* Watchful waiting (often spontaneous resolution <6 weeks) * Consider early surgical referral in children at risk for speech/learning problems * Tympanostomy tube placement if >3 months * Antibiotics, antihistamines, and steroids have not been shown to be useful in the treatment of OME
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What are the complications of AOM?
* Cholesteatoma * Tympanic Membrane Perforation * Mastoiditis * Facial Nerve Palsy
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Tympanic Membrane Perforation * occurs when? * Due to what? * What are the sx of rupture? * How will it heal? * Often resolves what? * What do you need to ensure? * When do you refer out?
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Cholesteatoma * What is it? What happens? * Rare complication of what?
* Abnormal accumulations of epithelium in middle ear and mastoid * Skin cells become trapped in middle ear, proliferate, and erode bone and surrounding structures * Rare complication of AOM or OME, ET dysfunction (9/100,000)
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Cholesteatoma * When do you suspect this? * What are the sx? * What does the exam show? * How do you dx?
* Suspect in any patient with a chronically draining ear * Sx: Ear drainage and hearing loss * Exam: Focal retractions and white mass behind intact TM * Dx: Temporal Bone CT
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Cholesteatoma * What is the txt? * What is the prognosis? What are the complications?
* Txt: ENT referral for surgery * Prognosis: Frequent recurrence * Complications: labyrinthitis, facial palsies and paralysis, meningitis and hearing loss
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What is this?$
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What is this?
Cholesteatoma
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Mastoiditis * Usually happens when? * What is the presentation? * What does it show on exam?
* Usually after weeks of inadequately treated AOM * Presentation: Postauricular pain/erythema, fevers * Exam: Tenderness and erythema posterior over mastoid bone
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Mastoiditis * how do you dx it? * How do you tx? * What happens if it does not improve?
* Dx: CT -reveals coalescence of the mastoid air cells due to bony septa destruction. * TX – IV antibiotics (first line) * No improvement->surgical drainage or mastoidectomy
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Tinnitus: * What is it? * Symptom with what? * How many people deal with this? * Hx of what? * usually what? * What type of escalation? * Most common cause? * What is the txt? * Sometimes due to what?
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What are the inner ear disorders?
* Acoustic neuroma * Barotrauma * Dysfunction of eustachian tube * Labyrinthitis * Vertigo (BPPV) * Meniere’s disease
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Acoustic neuroma (vestibular schwannoma) * What is the MC? * One of the mose common what? * What ages does this occur in? * Unilateral or bilateral? * May have what?
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Acoustic neuroma: * What is the presentation? * How do you diagnosis?
Presentation: * Unilateral hearing loss (95%-sensorineural), +/- disequilibrium (continuous), tinnitus Diagnosis: Enhanced MRI or CT * Can confirm hearing loss with audiogram
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Acoustic Neuroma * What is the txt?
Treatment – Refer to ENT Surgery * May do observation with repeat imaging
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Always get a MRI with what patients and why?
with unilateral, asymptomatic hearing loss * Any individual with a unilateral or asymmetric sensorineural hearing loss should be evaluated for an intracranial mass lesion
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What is this?
acoustic neuroma
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Barotrauma * What happens? * What are common causes?
* Increased barometric stress exerted on the middle ear can cause trauma * Common causes: **air travel (#1)**, scuba diving, blast injuries ## Footnote The problem is generally most acute during airplane descent, since the negative middle ear pressure tends to collapse and block the eustachian tube, causing pain.
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Barotrauma * What is the presentation? * How do you dx?
* Presentation: ear pressure, hearing loss (conductive), pain, +/- tinnitus * Diagnosis: Clinical (suspect based on symptoms and history) * May visualize trauma (**hemotympanum**) on otoscope exam
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Barotrauma * What is the txt? * What do you do if no improvement in few days? * What are some concering sx?
Treatment: **Conservative**-Usually resolves spontaneously * Recommend bedrest, head elevation, avoid anything that increases pressure * NSAIDS No improvement in a few days->requires urgent referral to ENT * **SNL hearing loss& vertigo->concern for perilymph fistula**
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Barotrauma * What do you educate patients on? * How do you prevent?
Patient Education: * Avoid diving with TM perforation or URI Prevention: Oral decongestants, antihistamines, and nasal decongestant sprays used prior to flying or diving * Counsel to swallow, yawn, and autoinsufflatation ## Footnote autoinsufflate (pinching nostrils closed while gently exhaling through the nose) 
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What is this?
Tympanic membrane barotrauma (bleeding)
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Eustachian Tube Dysfunction * ET connects to what? * What does it provide? * May dysfunction with that? * Increases in who? * Can be what?
* ET connects middle ear to the nasopharynx * Provides ventilation and drainage for the middle ear * May dysfunction with URI, sinusitis, diving * Increased in kids, improves by age 6 * Can hypo or hyper function
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Eustachian Tube Dysfunction * What is the presentation? * How do you dx?
Presentation: * Fullness in ear, decreased hearing, popping of ears Clinical diagnosis * TM may be retracted or normal * Can confirm with nasal endoscopy or tympanogram
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Eustachian Tube Dysfunction * How do yuo tx it? * What can you educate on?
Txt: Treat the underlying cause (rhinosinusitis, allergic, mass?) * Rhinosinusitis -pseudoephedrine, antihistamines, nasal steroids, pain control * autoinsufflation (if no active nasal infection)- Modified Valsalva Education: Avoid travel, diving, rapid altitude change
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Labyrinthitis * What is it? * Not what?
* Inflammation of the inner ear (labyrinth) or the nerves that connect the inner ear to the brain * Not dangerous, but incapacitating
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Labyrinthitis: * What are causes? * What is the presentation?
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Labyrinthitis- Diagnosis and Workup * What do you need to consider? * How do you dx?
* Broad differential (consider vascular events in >60, HA, neuro deficits, risk factors) * Diagnosis is clinical (consider MRI to r/o other diagnoses)
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Labyrinthitis- * What is the txt? * What do you need to differentiate from?
Txt: * Viral (secondary to URI)- supportive treatment (antihistamines, antiemetics, vestibular suppressants <24hrs) * Consider oral steroids (Grade 2C) if no contraindications (short-term relief, long term outcome uncertain) * Vestibular rehab in ongoing * Bacterial- (with otitis media or meningitis)- Abx treatment Differentiate from vestibular neuritis (auditory function preserved, no cochlear involvement)
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Vertigo * What is it? * Occurs in what conditions? * Difficult to do what?
* Sensation of self motion when no motion occurs * Occurs in conditions affecting the vestibular sensory organs of inner ear, cerebellum and brainstem and the connections between them. * Difficult to diagnose and determine etiology
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Vertigo: * What are the two types? * What is the most common cause of vertigo?
Must differentiate peripheral from central causes * Peripheral: Sudden onset, +/- tinnitus and hearing loss; horizontal nystagmus * Central: Onset is gradual, no associated auditory symptoms, neuro symptoms Most common:Benign paroxysmal positional vertigo (BPPV)
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What are some of the peripheral and central causes of vertigo?
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* Walk test before discharging from an ED! * Peripheral- hearing loss/tinnitus * Central- usually no ear complaints * Good neuro exam required either way document your neuro exam
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Benign Paroxysmal Positional Vertigo --(BPPV) * Most common what? * What is the pathogenesis?
* Most common form of peripheral vertigo (about 1⁄2 of vertigo cases) * Pathogenesis: Occurs when calcium debris are displaced spontaneously into the semicircular canals
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Benign Paroxysmal Positional Vertigo --(BPPV) * What is the presentation?
Presentation: **Recurrent, short episodes of vertigo** (seconds to 1 min) * **Usually provoked by head movements** * +/- N/V (intermittent) * +/-imbalance * Dizziness lasting minutes to hours is not BPPV
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Benign Paroxysmal Positional Vertigo --(BPPV) * how do you dx and txt it?
* Dx: Dix-Hallpike maneuver (move to supine position at head 45 angle) and normal neuro exam * DHM: delayed nystagmus (2-40secs) which lasts less than a minute + moderate vertigo * Treatment: Epley maneuver (can refer to physical therapy)
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Meniere’s Disease * What type of condition? * Believed to be associated with what?
* Idiopathic condition (exact cause unknown) * Believed to be associated with excess endolymph fluid in the inner ear (endolymphatic hydrops)
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Meniere's disease: * What is the presentation?
* Presentation (classic triad): Episodic vertigo, tinnitus, hearing loss (sensorineural, low frequency) * Though hearing loss comes and goes, there is an expected progressive loss of hearing in the low frequencies
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Meniere’s Disease- Diagnosis (Clinical) * What do you need to document? * What hearing loss and how do you know? * What else is present? * What do you need to exclused?
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Meniere’s Disease- Treatment options * What do you do first? * What are the pharm therapies?
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Meniere’s Disease- Treatment options * What do you do if refractory sx? * What do you do for perisitent disequilibrium?
Refractory Symptoms: Refer to ENT * Steroids orally or intratympanic * Middle ear injection of Gentamycin * Surgical options: decompression Persistent disequilibrium: Vestibular rehabilitation ## Footnote * For patients with MD and persistent disequilibrium symptoms between attacks, we suggest referral for vestibular rehabilitation therapy (Grade 2C). Helps compensate * Hearing loss may accompany Meniere disease or posterior circulation ischemia (due to infarction of the labyrinth) and is not pathognomonic of Meniere disease)
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Hearing Loss- Epidemiology * How much of the global population live with hearing loss? * ~15% of American adults aged 18 and over report what? * 2 to 3 out of every 1000 neonates are born with what? * 30 million people in US are exposed to what? * Increased prevalence of hearing loss with what?
* Nearly 20% of the global population live with hearing loss * ~15% of American adults aged 18 and over report some trouble hearing * 2 to 3 out of every 1000 neonates are born with a detectable hearing loss * 30 million people in US are exposed to dangerous noise levels on a regular basis * Increased prevalence of hearing loss with age (43% ages 65-84)
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Hearing Loss- Pathophysiology * What is conductive hearing loss? * What is sensorinerual hearing loss? * What is mixed loss?
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Hearing Loss- Screening * Should be part of what? * Screening for hearing is done during what? * Any detected hearing loss in primary care should be referred for what? * Screen who reg? * What is an audiologist?
* Should be a part of every yearly H & P (physical diagnosis/Bates) * Screening for hearing is done during well child visits for peds * Any detected hearing loss in primary care should be referred for audiologist testing * Screen the elderly regularly * Audiologist-professional licensed medical professional who works in various settings treating patients with hearing loss and balance disorders
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Sensorineural Hearing Loss * Associated with that? * What are the etiologies?
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Presbycusis * What is this? * What is do patients present with? * Common but what? * Cause is what? * Vastly undertreated, has what? * how do you screen?
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Presbycusis * What are common complaints? * how do you dx?
Common complaints: * inability to hear or understand speech in a noisy environment * difficulty understanding consonants * inability to hear high-pitched voices or noises * tinnitus Diagnostics: Screening questions, Weber/Rinne, otoscope exam, refer to audiology
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Presbycusis * What is the txt?
Treatments: * Adaptative techniques and environmental modifications * Hearing aids beneficial but underutilized * Cochlear implantation an option those who have not benefited from hearing aids
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Conductive Hearing Loss * What is it? * What are some causes?
Impairment of the passage of sound vibrations to the inner ear: * Obstruction (eg, cerumen) * Mass loading (eg, middle ear effusion) * Stiffness (eg, otosclerosis) * Discontinuity (eg, ossicular disruption)
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* What are some etiologies od conductive hearing loss? * Often correctable with what?
Etiologies: * Cerumen impaction * Transient eustachian tube dysfunction due to upper respiratory tract infection * Chronic ear infection (OM, cholesteatoma) * Trauma (perforation) * Otosclerosis * Perforations of the tympanic membrane Often correctable with medical or surgical therapy, or both
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Weber * What is a normal test? * What about conductive and sensorinerual hearing loss?
Weber –Normal test- sound heard equally in both ears * Tuning fork placed on midline of head –tone is heard loudest in ear with hearing loss * Unilateral conductive hearing loss- tone is heard loudest in ear with hearing loss * Unilateral sensorineural hearing loss – the tone is heard louder in the normal ear
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Rinne * how do you do it? * What is normal? * What is conductive?
* Tuning fork held against mastoid bone, then, comparing its sound when held lateral to the patient’s ear (air conduction) * Normal is when tone can still be heard (AC>BC) * Conductive hearing loss- Bone conduction sounds louder than air conduction (BC>AC)
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* What are hearing Loss Red Flags? * What do you need to do?
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Hearing Loss- Treatment * What do you need to stop or decrease? * Treat what? * What do you do if TM is an issue? * What can help hearing?
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Hearing loss-Prevention * Avoid what? (3) * What should you wear?
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Patient presents c/o hearing loss. Weber test lateralizes to the left ear. Rinne test of left ear is bone conduction greater than air conduction (BC>AC). Rinne test of the right ear is air conduction greater than bone conduction (AC>BC). What hearing loss does this suggest? A Right-sided conductive hearing loss B Left-sided conductive hearing loss C Right-sided sensorineural hearing loss D Left-sided sensorineural hearing loss E Mixed conductive and sensorineural hearing loss
B Left-sided conductive hearing loss
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Patient presents c/o hearing loss. Weber test lateralizes to the left ear. Rinne test of left ear is air conduction greater than bone condition (AC>BC). Rinne test of the right ear is air conduction greater than bone conduction (AC>BC). What hearing loss does this suggest? A Right-sided conductive hearing loss B Left-sided conductive hearing loss C Right-sided sensorineural hearing loss D Left-sided sensorineural hearing loss E Mixed conductive and sensorineural hearing loss
C Right-sided sensorineural hearing loss